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class="js-profile-total-view-text">Public Views</span></p><p class="data"><span class="js-profile-view-count"></span></p></div></span></div><div class="ri-section"><div class="ri-section-header"><span>Interests</span></div><div class="ri-tags-container"><a data-click-track="profile-user-info-expand-research-interests" data-has-card-for-ri-list="33654583" href="https://www.academia.edu/Documents/in/Critical_Terrorism_Studies"><div id="js-react-on-rails-context" style="display:none" data-rails-context="{"inMailer":false,"i18nLocale":"en","i18nDefaultLocale":"en","href":"https://independent.academia.edu/HarveyPollard1","location":"/HarveyPollard1","scheme":"https","host":"independent.academia.edu","port":null,"pathname":"/HarveyPollard1","search":null,"httpAcceptLanguage":null,"serverSide":false}"></div> <div class="js-react-on-rails-component" style="display:none" data-component-name="Pill" data-props="{"color":"gray","children":["Critical Terrorism Studies"]}" data-trace="false" 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data-props="{"color":"gray","children":["Parkinson's Disease"]}" data-trace="false" data-dom-id="Pill-react-component-04cd1a30-3d2f-4839-889d-b62b5934b1f1"></div> <div id="Pill-react-component-04cd1a30-3d2f-4839-889d-b62b5934b1f1"></div> </a></div></div></div></div><div class="right-panel-container"><div class="user-content-wrapper"><div class="uploads-container" id="social-redesign-work-container"><div class="upload-header"><h2 class="ds2-5-heading-sans-serif-xs">Uploads</h2></div><div class="documents-container backbone-social-profile-documents" style="width: 100%;"><div class="u-taCenter"></div><div class="profile--tab_content_container js-tab-pane tab-pane active" id="all"><div class="profile--tab_heading_container js-section-heading" data-section="Papers" id="Papers"><h3 class="profile--tab_heading_container">Papers by Harvey Pollard</h3></div><div class="js-work-strip profile--work_container" data-work-id="125970483"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/125970483/Genomic_organization_and_chromosomal_localization_of_the_mouse_synexin_gene"><img alt="Research paper thumbnail of Genomic organization and chromosomal localization of the mouse synexin gene" class="work-thumbnail" src="https://attachments.academia-assets.com/119920353/thumbnails/1.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/125970483/Genomic_organization_and_chromosomal_localization_of_the_mouse_synexin_gene">Genomic organization and chromosomal localization of the mouse synexin gene</a></div><div class="wp-workCard_item"><span>Biochemical Journal</span><span>, Aug 1, 1994</span></div><div class="wp-workCard_item wp-workCard--actions"><span 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class="work-thumbnail" src="https://attachments.academia-assets.com/119920352/thumbnails/1.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/125970482/Novel_isoforms_of_synexin_in_Xenopus_laevis_multiple_tandem_PGQM_repeats_distinguish_mRNAs_in_specific_adult_tissues_and_embryonic_stages">Novel isoforms of synexin in Xenopus laevis: multiple tandem PGQM repeats distinguish mRNAs in specific adult tissues and embryonic stages</a></div><div class="wp-workCard_item"><span>Biochemical Journal</span><span>, Jun 15, 1996</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><a id="0e20620c6c10e773e358c9343199a1cc" class="wp-workCard--action" rel="nofollow" data-click-track="profile-work-strip-download" 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synexins" class="work-thumbnail" src="https://attachments.academia-assets.com/119920351/thumbnails/1.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/125970481/Mouse_synexin_annexin_VII_polymorphisms_and_a_phylogenetic_comparison_with_other_synexins">Mouse synexin (annexin VII) polymorphisms and a phylogenetic comparison with other synexins</a></div><div class="wp-workCard_item"><span>Biochemical Journal</span><span>, Feb 1, 1993</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><a id="845f43c28d4045522ede36fca297cf04" class="wp-workCard--action" rel="nofollow" data-click-track="profile-work-strip-download" data-download="{"attachment_id":119920351,"asset_id":125970481,"asset_type":"Work","button_location":"profile"}" 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data-click-track="profile-work-strip-title" href="https://www.academia.edu/125970472/Signalling_Pathways_Have_Different_Expression_Profiles_in_Human_Platelets_Isolated_from_Men_and_Women">Signalling Pathways Have Different Expression Profiles in Human Platelets Isolated from Men and Women</a></div><div class="wp-workCard_item"><span>Blood</span><span>, 2006</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">Background: Cardiovascular diseases (CVD) are the leading causes of death for men and women in th...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">Background: Cardiovascular diseases (CVD) are the leading causes of death for men and women in the U.S. Platelets play a central role in the inflammation and coagulation mechanisms responsible for these disorders, and anti-platelet drugs are therefore the current mainstays in prevention and therapy. Understanding the signaling pathways involved in normal and abnormal platelet function using proteomic techniques offers a novel strategic approach to the discovery of potential new therapeutic targets. Hypothesis: The incidence and outcomes of cardiovascular disease are profoundly dependent on gender, and it is possible that differences in platelet signaling mechanisms may contribute to these disparities. We have therefore hypothesized that the platelet signaling proteome is gender-specific. Methods: Platelets were isolated and purified from four normal adult male and four normal adult female donors under an IRB-approved protocol. Conventional 2D-gel electrophoresis (2DGE) and mass spec...</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="125970472"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="125970472"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 125970472; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=125970472]").text(description); $(".js-view-count[data-work-id=125970472]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 125970472; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='125970472']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 125970472, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=125970472]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":125970472,"title":"Signalling Pathways Have Different Expression Profiles in Human Platelets Isolated from Men and Women","translated_title":"","metadata":{"abstract":"Background: Cardiovascular diseases (CVD) are the leading causes of death for men and women in the U.S. Platelets play a central role in the inflammation and coagulation mechanisms responsible for these disorders, and anti-platelet drugs are therefore the current mainstays in prevention and therapy. Understanding the signaling pathways involved in normal and abnormal platelet function using proteomic techniques offers a novel strategic approach to the discovery of potential new therapeutic targets. Hypothesis: The incidence and outcomes of cardiovascular disease are profoundly dependent on gender, and it is possible that differences in platelet signaling mechanisms may contribute to these disparities. We have therefore hypothesized that the platelet signaling proteome is gender-specific. Methods: Platelets were isolated and purified from four normal adult male and four normal adult female donors under an IRB-approved protocol. 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$(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="125970470"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" rel="nofollow" href="https://www.academia.edu/125970470/Synexin_Annexin_VII_Hypothesis_for_Ca2_GTP_Regulated_Exocytosis"><img alt="Research paper thumbnail of Synexin (Annexin VII) Hypothesis for Ca2+/GTP-Regulated Exocytosis" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" rel="nofollow" href="https://www.academia.edu/125970470/Synexin_Annexin_VII_Hypothesis_for_Ca2_GTP_Regulated_Exocytosis">Synexin (Annexin VII) Hypothesis for Ca2+/GTP-Regulated Exocytosis</a></div><div class="wp-workCard_item"><span>Advances in Pharmacology</span><span>, 1997</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">... 1. B id . Chem. 270, 23667-23671. 86 Harvey B. Pollard et ol. 10. Morgan, A., and Burgoyne, R...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">... 1. B id . Chem. 270, 23667-23671. 86 Harvey B. Pollard et ol. 10. Morgan, A., and Burgoyne, RD (1995). ... Essays Biochem. 30, 77-95. 12. O&#39;Connor, V., Augustine, GJ, and Betz, H. (1994).Synaptic vesicle exocytosis: Molecules and models. 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Fusion" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/125970468/Synexin_Calcium_and_the_Hydrophobic_Bridge_Hypothesis_for_Membrane_Fusion">Synexin, Calcium and the Hydrophobic Bridge Hypothesis for Membrane Fusion</a></div><div class="wp-workCard_item"><span>Molecular Mechanisms of Membrane Fusion</span><span>, 1988</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">Synexin is a calcium binding protein of 47,000da which causes chromaffin granules (1–5) and chrom...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">Synexin is a calcium binding protein of 47,000da which causes chromaffin granules (1–5) and chromaffin granule ghosts (6), as well as acidic liposomes (7,8) to aggregate and fuse. In addition to binding to granule membranes and liposomes, synexin also binds selectively to the inner aspect of chromaffin cell plasma membranes (9). Furthermore, synexin dependent fusion structures of chromaffin granules appear quite similar to the large vacuolar, compound exocytotic structures seen in secreting chromaffin cells (3–5). These facts have lead us to anticipate that synexin might be involved in fusion processes occurring during exocytosis (5). We have therefore devoted substantial efforts to trying to understand how synexin indeed fuses these biological and artificial membranes.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="125970468"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="125970468"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 125970468; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=125970468]").text(description); $(".js-view-count[data-work-id=125970468]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 125970468; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='125970468']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 125970468, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=125970468]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":125970468,"title":"Synexin, Calcium and the Hydrophobic Bridge Hypothesis for Membrane Fusion","translated_title":"","metadata":{"abstract":"Synexin is a calcium binding protein of 47,000da which causes chromaffin granules (1–5) and chromaffin granule ghosts (6), as well as acidic liposomes (7,8) to aggregate and fuse. 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$(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="123819412"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/123819412/Abstract_S01_03_A_prediction_of_prostate_cancer_deaths_spiking_by_SARS_CoV_2_infection"><img alt="Research paper thumbnail of Abstract S01-03: A prediction of prostate cancer deaths spiking by SARS-CoV-2 infection" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/123819412/Abstract_S01_03_A_prediction_of_prostate_cancer_deaths_spiking_by_SARS_CoV_2_infection">Abstract S01-03: A prediction of prostate cancer deaths spiking by SARS-CoV-2 infection</a></div><div class="wp-workCard_item"><span>Clinical Cancer Research</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">COVID-19 is a global issue, with over 6.25 million cases in 213 countries and territories on June...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">COVID-19 is a global issue, with over 6.25 million cases in 213 countries and territories on June 1, 2020. Although this virus infects all groups, data indicate that the risk for severe disease and death is much higher in older men, which coincides with the same group of patients at risk for prostate cancer. A recent Italian study investigated the prevalence and severity of COVID-19 in men with prostate cancer. This study indicated that of a total of 4,532 men with COVID-19, from the Veneto region of Italy, 9.5% (n=430) had cancer and out of those around 30% (n=118) had prostate cancer. Data also indicated that male cancer patients had a 1.8-fold increased risk of COVID-19 infection and developed a more severe disease. Interestingly, they observed that the prostate cancer patients (n=4) treated with androgen-deprivation therapy (ADT) were less likely to develop COVID-19, and in those who were infected, the disease was less severe. 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In this current study, we focused on determining the...","internal_url":"https://www.academia.edu/123819412/Abstract_S01_03_A_prediction_of_prostate_cancer_deaths_spiking_by_SARS_CoV_2_infection","translated_internal_url":"","created_at":"2024-09-12T12:42:56.958-07:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":33654583,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[],"downloadable_attachments":[],"slug":"Abstract_S01_03_A_prediction_of_prostate_cancer_deaths_spiking_by_SARS_CoV_2_infection","translated_slug":"","page_count":null,"language":"en","content_type":"Work","owner":{"id":33654583,"first_name":"Harvey","middle_initials":null,"last_name":"Pollard","page_name":"HarveyPollard1","domain_name":"independent","created_at":"2015-08-05T19:02:51.542-07:00","display_name":"Harvey Pollard","url":"https://independent.academia.edu/HarveyPollard1"},"attachments":[],"research_interests":[{"id":6021,"name":"Cancer","url":"https://www.academia.edu/Documents/in/Cancer"},{"id":12980,"name":"Prostate Cancer","url":"https://www.academia.edu/Documents/in/Prostate_Cancer"},{"id":26327,"name":"Medicine","url":"https://www.academia.edu/Documents/in/Medicine"},{"id":65390,"name":"Internal Medicine","url":"https://www.academia.edu/Documents/in/Internal_Medicine"},{"id":99773,"name":"Disease","url":"https://www.academia.edu/Documents/in/Disease"},{"id":161553,"name":"Prostate","url":"https://www.academia.edu/Documents/in/Prostate"}],"urls":[{"id":44634165,"url":"https://aacrjournals.org/clincancerres/article/26/18_Supplement/S01-03/202366/Abstract-S01-03-A-prediction-of-prostate-cancer"}]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="123819410"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/123819410/Posttraumatic_Stress_Disorder"><img alt="Research paper thumbnail of Posttraumatic Stress Disorder" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/123819410/Posttraumatic_Stress_Disorder">Posttraumatic Stress Disorder</a></div><div class="wp-workCard_item"><span>Conn's Translational Neuroscience</span><span>, 2017</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">This chapter provides an overview of the most up-to-date understanding of the neuroscience of pos...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">This chapter provides an overview of the most up-to-date understanding of the neuroscience of posttraumatic stress disorder (PTSD). A variety of topics are covered throughout this chapter in a systematic fashion with the goal that each section can be read and understood as a stand-alone piece of a greater whole. The first section presents an overview of the neurobiology of traumatic stress from receptors to structural brain changes. After this general overview, a discussion of the various animal models of PTSD is provided. These models include fear conditioning, fear-potentiated startle, and models of unpredictable stress. This review of animal models is then followed by a discussion of biomarkers, PTSD and somatic health, neuroimaging, and treatment. 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class="work-thumbnail" src="https://attachments.academia-assets.com/119920352/thumbnails/1.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/125970482/Novel_isoforms_of_synexin_in_Xenopus_laevis_multiple_tandem_PGQM_repeats_distinguish_mRNAs_in_specific_adult_tissues_and_embryonic_stages">Novel isoforms of synexin in Xenopus laevis: multiple tandem PGQM repeats distinguish mRNAs in specific adult tissues and embryonic stages</a></div><div class="wp-workCard_item"><span>Biochemical Journal</span><span>, Jun 15, 1996</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><a id="0e20620c6c10e773e358c9343199a1cc" class="wp-workCard--action" rel="nofollow" data-click-track="profile-work-strip-download" 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synexins" class="work-thumbnail" src="https://attachments.academia-assets.com/119920351/thumbnails/1.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/125970481/Mouse_synexin_annexin_VII_polymorphisms_and_a_phylogenetic_comparison_with_other_synexins">Mouse synexin (annexin VII) polymorphisms and a phylogenetic comparison with other synexins</a></div><div class="wp-workCard_item"><span>Biochemical Journal</span><span>, Feb 1, 1993</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><a id="845f43c28d4045522ede36fca297cf04" class="wp-workCard--action" rel="nofollow" data-click-track="profile-work-strip-download" data-download="{"attachment_id":119920351,"asset_id":125970481,"asset_type":"Work","button_location":"profile"}" 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data-click-track="profile-work-strip-title" href="https://www.academia.edu/125970472/Signalling_Pathways_Have_Different_Expression_Profiles_in_Human_Platelets_Isolated_from_Men_and_Women">Signalling Pathways Have Different Expression Profiles in Human Platelets Isolated from Men and Women</a></div><div class="wp-workCard_item"><span>Blood</span><span>, 2006</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">Background: Cardiovascular diseases (CVD) are the leading causes of death for men and women in th...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">Background: Cardiovascular diseases (CVD) are the leading causes of death for men and women in the U.S. Platelets play a central role in the inflammation and coagulation mechanisms responsible for these disorders, and anti-platelet drugs are therefore the current mainstays in prevention and therapy. Understanding the signaling pathways involved in normal and abnormal platelet function using proteomic techniques offers a novel strategic approach to the discovery of potential new therapeutic targets. Hypothesis: The incidence and outcomes of cardiovascular disease are profoundly dependent on gender, and it is possible that differences in platelet signaling mechanisms may contribute to these disparities. We have therefore hypothesized that the platelet signaling proteome is gender-specific. Methods: Platelets were isolated and purified from four normal adult male and four normal adult female donors under an IRB-approved protocol. Conventional 2D-gel electrophoresis (2DGE) and mass spec...</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="125970472"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="125970472"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 125970472; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=125970472]").text(description); $(".js-view-count[data-work-id=125970472]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 125970472; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='125970472']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 125970472, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=125970472]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":125970472,"title":"Signalling Pathways Have Different Expression Profiles in Human Platelets Isolated from Men and Women","translated_title":"","metadata":{"abstract":"Background: Cardiovascular diseases (CVD) are the leading causes of death for men and women in the U.S. Platelets play a central role in the inflammation and coagulation mechanisms responsible for these disorders, and anti-platelet drugs are therefore the current mainstays in prevention and therapy. Understanding the signaling pathways involved in normal and abnormal platelet function using proteomic techniques offers a novel strategic approach to the discovery of potential new therapeutic targets. Hypothesis: The incidence and outcomes of cardiovascular disease are profoundly dependent on gender, and it is possible that differences in platelet signaling mechanisms may contribute to these disparities. We have therefore hypothesized that the platelet signaling proteome is gender-specific. Methods: Platelets were isolated and purified from four normal adult male and four normal adult female donors under an IRB-approved protocol. 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B id . Chem. 270, 23667-23671. 86 Harvey B. Pollard et ol. 10. Morgan, A., and Burgoyne, R...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">... 1. B id . Chem. 270, 23667-23671. 86 Harvey B. Pollard et ol. 10. Morgan, A., and Burgoyne, RD (1995). ... Essays Biochem. 30, 77-95. 12. O&#39;Connor, V., Augustine, GJ, and Betz, H. (1994).Synaptic vesicle exocytosis: Molecules and models. 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Fusion" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/125970468/Synexin_Calcium_and_the_Hydrophobic_Bridge_Hypothesis_for_Membrane_Fusion">Synexin, Calcium and the Hydrophobic Bridge Hypothesis for Membrane Fusion</a></div><div class="wp-workCard_item"><span>Molecular Mechanisms of Membrane Fusion</span><span>, 1988</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">Synexin is a calcium binding protein of 47,000da which causes chromaffin granules (1–5) and chrom...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">Synexin is a calcium binding protein of 47,000da which causes chromaffin granules (1–5) and chromaffin granule ghosts (6), as well as acidic liposomes (7,8) to aggregate and fuse. In addition to binding to granule membranes and liposomes, synexin also binds selectively to the inner aspect of chromaffin cell plasma membranes (9). Furthermore, synexin dependent fusion structures of chromaffin granules appear quite similar to the large vacuolar, compound exocytotic structures seen in secreting chromaffin cells (3–5). These facts have lead us to anticipate that synexin might be involved in fusion processes occurring during exocytosis (5). We have therefore devoted substantial efforts to trying to understand how synexin indeed fuses these biological and artificial membranes.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="125970468"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="125970468"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 125970468; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=125970468]").text(description); $(".js-view-count[data-work-id=125970468]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 125970468; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='125970468']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 125970468, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=125970468]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":125970468,"title":"Synexin, Calcium and the Hydrophobic Bridge Hypothesis for Membrane Fusion","translated_title":"","metadata":{"abstract":"Synexin is a calcium binding protein of 47,000da which causes chromaffin granules (1–5) and chromaffin granule ghosts (6), as well as acidic liposomes (7,8) to aggregate and fuse. 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