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Aβ imaging with 18F-florbetaben in prodromal Alzheimer's disease: a prospective outcome study | Journal of Neurology, Neurosurgery & Psychiatry

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For permission to use (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions" /> <meta name="DC.AccessRights" content="restricted" /> <meta name="DC.Description" content="Background We assessed the clinical utility of β-amyloid (Aβ) imaging with 18F-florbetaben (FBB) in mild cognitive impairment (MCI) by evaluating its prognostic accuracy for progression to Alzheimer's disease (AD), comparing semiquantitative with visual scan assessment, and exploring the relationships among Aβ, hippocampal volume (HV) and memory over time. Methods 45 MCI underwent FBB positron emission tomography, MRI and neuropsychological assessment at baseline and 2 years and clinical follow-up at 4 years. Positive FBB (FBB+), defined by a cortical to cerebellar cortex standardised uptake value ratio (SUVR) ≥1.45, was compared with visual assessment by five readers. Amnestic MCI (aMCI) was defined by a composite episodic memory (EM) Z-score of &lt;−1.5. Results At baseline, 24 (53%) MCI were FBB+. Majority reads agreed with SUVR classification (κ 0.96). In 2 years, 18 (75%) FBB+ progressed to AD compared with 2 (9.5%) FBB−, yielding a predictive accuracy of 83% (95% CI 61% to 94%). Four FBB− developed non-AD dementia. Predictive accuracies of HV (58% (95% CI 42% to 73%)) and aMCI status (73% (95% CI 58% to 81%)) were lower. Combinations did not improve accuracy. By 4 years, 21 (87.5%) FBB+ had AD whereas 5 (24%) FBB− had non-AD dementia yielding a predictive accuracy of 94% (95% CI 74% to 99%). While the strong baseline association between FBB SUVR and EM declined over 2 years, the association between EM and HV became stronger. FBB SUVR increased 2.2%/year in FBB+ with no change in FBB−. Conclusions 18F-florbetaben Aβ imaging facilitates accurate detection of prodromal AD. As neurodegeneration progresses, and in contrast with the early stages of the disease, hippocampal atrophy and not Aβ, seems to drive memory decline. Trial registration number NCT01138111." /> <meta name="DC.Contributor" content="Kevin T Ong" /> <meta name="DC.Contributor" content="Victor L Villemagne" /> <meta name="DC.Contributor" content="Alex Bahar-Fuchs" /> <meta name="DC.Contributor" content="Fiona Lamb" /> <meta name="DC.Contributor" content="Narelle Langdon" /> <meta name="DC.Contributor" content="Ana M Catafau" /> <meta name="DC.Contributor" content="Andrew W Stephens" /> <meta name="DC.Contributor" content="John Seibyl" /> <meta name="DC.Contributor" content="Ludger M Dinkelborg" /> <meta name="DC.Contributor" content="Cornelia B Reininger" /> <meta name="DC.Contributor" content="Barbara Putz" /> <meta name="DC.Contributor" content="Beate Rohde" /> <meta name="DC.Contributor" content="Colin L Masters" /> <meta name="DC.Contributor" content="Christopher C Rowe" /> <meta name="article:published_time" content="2015-04-01" /> <meta name="article:section" content="Neurodegeneration" /> <meta name="citation_title" content="Aβ imaging with 18F-florbetaben in prodromal Alzheimer's disease: a prospective outcome study" /> <meta name="citation_abstract" lang="en" content="&lt;h3&gt;Background&lt;/h3&gt; &lt;p&gt;We assessed the clinical utility of β-amyloid (Aβ) imaging with &lt;sup&gt;18&lt;/sup&gt;F-florbetaben (FBB) in mild cognitive impairment (MCI) by evaluating its prognostic accuracy for progression to Alzheimer9s disease (AD), comparing semiquantitative with visual scan assessment, and exploring the relationships among Aβ, hippocampal volume (HV) and memory over time.&lt;/p&gt;&lt;h3&gt;Methods&lt;/h3&gt; &lt;p&gt;45 MCI underwent FBB positron emission tomography, MRI and neuropsychological assessment at baseline and 2 years and clinical follow-up at 4 years. Positive FBB (FBB+), defined by a cortical to cerebellar cortex standardised uptake value ratio (SUVR) ≥1.45, was compared with visual assessment by five readers. Amnestic MCI (aMCI) was defined by a composite episodic memory (EM) Z-score of &amp;lt;−1.5.&lt;/p&gt;&lt;h3&gt;Results&lt;/h3&gt; &lt;p&gt;At baseline, 24 (53%) MCI were FBB+. Majority reads agreed with SUVR classification (κ 0.96). In 2 years, 18 (75%) FBB+ progressed to AD compared with 2 (9.5%) FBB−, yielding a predictive accuracy of 83% (95% CI 61% to 94%). Four FBB− developed non-AD dementia. Predictive accuracies of HV (58% (95% CI 42% to 73%)) and aMCI status (73% (95% CI 58% to 81%)) were lower. Combinations did not improve accuracy. By 4 years, 21 (87.5%) FBB+ had AD whereas 5 (24%) FBB− had non-AD dementia yielding a predictive accuracy of 94% (95% CI 74% to 99%). While the strong baseline association between FBB SUVR and EM declined over 2 years, the association between EM and HV became stronger. FBB SUVR increased 2.2%/year in FBB+ with no change in FBB−.&lt;/p&gt;&lt;h3&gt;Conclusions&lt;/h3&gt; &lt;p&gt;&lt;sup&gt;18&lt;/sup&gt;F-florbetaben Aβ imaging facilitates accurate detection of prodromal AD. As neurodegeneration progresses, and in contrast with the early stages of the disease, hippocampal atrophy and not Aβ, seems to drive memory decline.&lt;/p&gt;&lt;h3&gt;Trial registration number&lt;/h3&gt; &lt;p&gt;NCT01138111.&lt;/p&gt;" /> <meta name="citation_journal_title" content="Journal of Neurology, Neurosurgery &amp; Psychiatry" /> <meta name="citation_publisher" content="BMJ Publishing Group Ltd" /> <meta name="citation_publication_date" content="2015/04/01" /> <meta name="citation_mjid" content="jnnp;86/4/431" /> <meta name="citation_id" content="86/4/431" /> <meta name="citation_public_url" content="https://jnnp.bmj.com/content/86/4/431" /> <meta name="citation_abstract_html_url" content="https://jnnp.bmj.com/content/86/4/431.abstract" /> <meta name="citation_full_html_url" content="https://jnnp.bmj.com/content/86/4/431.full" /> <meta name="citation_pdf_url" content="https://jnnp.bmj.com/content/jnnp/86/4/431.full.pdf" /> <meta name="citation_issn" content="0022-3050" /> <meta name="citation_issn" content="1468-330X" /> <meta name="citation_journal_abbrev" content="J Neurol Neurosurg Psychiatry" /> <meta name="citation_doi" content="10.1136/jnnp-2014-308094" /> <meta name="citation_pmid" content="24970906" /> <meta name="citation_volume" content="86" /> <meta name="citation_issue" content="4" /> <meta name="citation_article_type" content="Research Article" /> <meta name="citation_section" content="Neurodegeneration" /> <meta name="citation_firstpage" content="431" /> <meta name="citation_lastpage" content="436" /> <meta name="citation_author" content="Kevin T Ong" /> <meta name="citation_author_institution" content="Centre for PET, Austin Health" /> <meta name="citation_author" content="Victor L Villemagne" /> <meta name="citation_author_institution" content="Centre for PET, Austin Health" /> <meta name="citation_author_institution" content="The Florey Institute of Neuroscience and Mental Health, The University of Melbourne" /> <meta name="citation_author" content="Alex Bahar-Fuchs" /> <meta name="citation_author_institution" content="Centre for PET, Austin Health" /> <meta name="citation_author_institution" content="Centre for Research on Aging, Health, and Wellbeing, The Australian National University" /> <meta name="citation_author" content="Fiona Lamb" /> <meta name="citation_author_institution" content="Centre for PET, Austin Health" /> <meta name="citation_author" content="Narelle Langdon" /> <meta name="citation_author_institution" content="Centre for PET, Austin Health" /> <meta name="citation_author" content="Ana M Catafau" /> <meta name="citation_author_institution" content="Piramal Imaging GmbH" /> <meta name="citation_author" content="Andrew W Stephens" /> <meta name="citation_author_institution" content="Piramal Imaging GmbH" /> <meta name="citation_author" content="John Seibyl" /> <meta name="citation_author_institution" content="Molecular NeuroImaging, L.L.C." /> <meta name="citation_author" content="Ludger M Dinkelborg" /> <meta name="citation_author_institution" content="Piramal Imaging GmbH" /> <meta name="citation_author" content="Cornelia B Reininger" /> <meta name="citation_author_institution" content="Bayer Healthcare" /> <meta name="citation_author" content="Barbara Putz" /> <meta name="citation_author_institution" content="Bayer Healthcare" /> <meta name="citation_author" content="Beate Rohde" /> <meta name="citation_author_institution" content="Bayer Healthcare" /> <meta name="citation_author" content="Colin L Masters" /> <meta name="citation_author_institution" content="The Florey Institute of Neuroscience and Mental Health, The University of Melbourne" /> <meta name="citation_author" content="Christopher C Rowe" /> <meta name="citation_author_institution" content="Centre for PET, Austin Health" /> <meta name="citation_reference" content="citation_journal_title=Annals of neurology;citation_journal_abbrev=Ann Neurol;citation_author=WE. 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Parisi;citation_title=Antemortem MRI findings correlate with hippocampal neuropathology in typical aging and dementia;citation_pages=750-7;citation_volume=58;citation_year=2002;citation_pmid=11889239;citation_doi=10.1212/WNL.58.5.750" /> <meta name="citation_reference" content="citation_journal_title=Lancet. Neurology;citation_journal_abbrev=Lancet Neurol;citation_author=CR. Jack;citation_author=DS. Knopman;citation_author=WJ. Jagust;citation_title=Hypothetical model of dynamic biomarkers of the Alzheimer's pathological cascade.;citation_pages=119-128;citation_volume=9;citation_year=2010;citation_issue=1;citation_pmid=20083042;citation_doi=10.1016/S1474-4422(09)70299-6" /> <meta name="citation_reference" content="citation_journal_title=Neurology;citation_author=CR. Jack;citation_author=HJ. Wiste;citation_author=TG. Lesnick;citation_title=Brain beta-amyloid load approaches a plateau;citation_pages=890-6;citation_volume=80;citation_year=2013;citation_doi=10.1212/WNL.0b013e3182840bbe" /> <meta name="twitter:title" content="Aβ imaging with 18F-florbetaben in prodromal Alzheimer's disease: a prospective outcome study" /> <meta name="twitter:card" content="summary_large_image" /> <meta name="twitter:image" content="https://jnnp.bmj.com/sites/default/files/highwire/jnnp/86/4.cover-source.jpg" /> <meta name="twitter:description" content="Background We assessed the clinical utility of β-amyloid (Aβ) imaging with 18F-florbetaben (FBB) in mild cognitive impairment (MCI) by evaluating its prognostic accuracy for progression to Alzheimer's disease (AD), comparing semiquantitative with visual scan assessment, and exploring the relationships among Aβ, hippocampal volume (HV) and memory over time. Methods 45 MCI underwent FBB positron emission tomography, MRI and neuropsychological assessment at baseline and 2 years and clinical follow-up at 4 years. Positive FBB (FBB+), defined by a cortical to cerebellar cortex standardised uptake value ratio (SUVR) ≥1.45, was compared with visual assessment by five readers. Amnestic MCI (aMCI) was defined by a composite episodic memory (EM) Z-score of &lt;−1.5. Results At baseline, 24 (53%) MCI were FBB+. Majority reads agreed with SUVR classification (κ 0.96). In 2 years, 18 (75%) FBB+ progressed to AD compared with 2 (9.5%) FBB−, yielding a predictive accuracy of 83% (95% CI 61% to 94%). Four FBB− developed non-AD dementia. Predictive accuracies of HV (58% (95% CI 42% to 73%)) and aMCI status (73% (95% CI 58% to 81%)) were lower. Combinations did not improve accuracy. By 4 years, 21 (87.5%) FBB+ had AD whereas 5 (24%) FBB− had non-AD dementia yielding a predictive accuracy of 94% (95% CI 74% to 99%). While the strong baseline association between FBB SUVR and EM declined over 2 years, the association between EM and HV became stronger. FBB SUVR increased 2.2%/year in FBB+ with no change in FBB−. Conclusions 18F-florbetaben Aβ imaging facilitates accurate detection of prodromal AD. As neurodegeneration progresses, and in contrast with the early stages of the disease, hippocampal atrophy and not Aβ, seems to drive memory decline. Trial registration number NCT01138111." /> <meta name="og-title" property="og:title" content="Aβ imaging with 18F-florbetaben in prodromal Alzheimer's disease: a prospective outcome study" /> <meta name="og-url" property="og:url" content="https://jnnp.bmj.com/content/86/4/431" /> <meta name="og-site-name" property="og:site_name" content="Journal of Neurology, Neurosurgery &amp; Psychiatry" /> <meta name="og-description" property="og:description" content="Background We assessed the clinical utility of β-amyloid (Aβ) imaging with 18F-florbetaben (FBB) in mild cognitive impairment (MCI) by evaluating its prognostic accuracy for progression to Alzheimer's disease (AD), comparing semiquantitative with visual scan assessment, and exploring the relationships among Aβ, hippocampal volume (HV) and memory over time. Methods 45 MCI underwent FBB positron emission tomography, MRI and neuropsychological assessment at baseline and 2 years and clinical follow-up at 4 years. Positive FBB (FBB+), defined by a cortical to cerebellar cortex standardised uptake value ratio (SUVR) ≥1.45, was compared with visual assessment by five readers. Amnestic MCI (aMCI) was defined by a composite episodic memory (EM) Z-score of &lt;−1.5. Results At baseline, 24 (53%) MCI were FBB+. Majority reads agreed with SUVR classification (κ 0.96). In 2 years, 18 (75%) FBB+ progressed to AD compared with 2 (9.5%) FBB−, yielding a predictive accuracy of 83% (95% CI 61% to 94%). Four FBB− developed non-AD dementia. Predictive accuracies of HV (58% (95% CI 42% to 73%)) and aMCI status (73% (95% CI 58% to 81%)) were lower. Combinations did not improve accuracy. By 4 years, 21 (87.5%) FBB+ had AD whereas 5 (24%) FBB− had non-AD dementia yielding a predictive accuracy of 94% (95% CI 74% to 99%). While the strong baseline association between FBB SUVR and EM declined over 2 years, the association between EM and HV became stronger. FBB SUVR increased 2.2%/year in FBB+ with no change in FBB−. Conclusions 18F-florbetaben Aβ imaging facilitates accurate detection of prodromal AD. As neurodegeneration progresses, and in contrast with the early stages of the disease, hippocampal atrophy and not Aβ, seems to drive memory decline. Trial registration number NCT01138111." /> <meta name="og-type" property="og:type" content="article" /> <meta name="og-image" property="og:image" content="https://jnnp.bmj.com/sites/default/files/highwire/jnnp/86/4.cover-source.jpg" /> <link rel="alternate" type="application/vnd.ms-powerpoint" title="Powerpoint" href="/content/86/4/431.ppt" /> <title>Aβ imaging with 18F-florbetaben in prodromal Alzheimer's disease: a prospective outcome study | Journal of Neurology, Neurosurgery &amp; Psychiatry</title> <link type="text/css" rel="stylesheet" href="/sites/default/files/advagg_css/css__Uwlgxr2XCTH3JEn8X9M0Zmw2O61ONTKbcuCCFzbUoWc__GaVzYzSnTbY94ZNnLRDkzT_0FKDQBbP8csnKKJ9LSZA__qw1DJnk-5gO1smAYQ6Cv-FvON600W_QtaIfAGcQ9MYA.css" media="all" /> <!-- OneTrust Cookies Consent Notice start --> <script src="https://cookie-cdn.cookiepro.com/scripttemplates/otSDKStub.js" type="text/javascript" charset="UTF-8" data-domain-script="565e50dd-f6e9-46d6-91cd-5c40a270264f"></script> <script 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Seibyl</span><a id="xref-aff-5-1" class="xref-aff" href="#aff-5">5</a>, </li><li class="contributor" id="contrib-9"><span class="name">Ludger M Dinkelborg</span><a id="xref-aff-4-3" class="xref-aff" href="#aff-4">4</a>, </li><li class="contributor" id="contrib-10"><span class="name">Cornelia B Reininger</span><a id="xref-aff-6-1" class="xref-aff" href="#aff-6">6</a>, </li><li class="contributor" id="contrib-11"><span class="name">Barbara Putz</span><a id="xref-aff-6-2" class="xref-aff" href="#aff-6">6</a>, </li><li class="contributor" id="contrib-12"><span class="name">Beate Rohde</span><a id="xref-aff-6-3" class="xref-aff" href="#aff-6">6</a>, </li><li class="contributor" id="contrib-13"><span class="name">Colin L Masters</span><a id="xref-aff-2-2" class="xref-aff" href="#aff-2">2</a>, </li><li class="last" id="contrib-14"><span class="name">Christopher C Rowe</span><a id="xref-aff-1-6" class="xref-aff" href="#aff-1">1</a></li></ol><ol class="affiliation-list"><li class="aff"><a id="aff-1" name="aff-1"></a><address> <sup>1</sup><span class="addr-line">Department of Nuclear Medicine</span>, <span class="institution">Centre for PET, Austin Health</span>, <span class="addr-line">Heidelberg, Victoria</span>, Australia</address></li><li class="aff"><a id="aff-2" name="aff-2"></a><address> <sup>2</sup><span class="institution">The Florey Institute of Neuroscience and Mental Health, The University of Melbourne</span>, <span class="addr-line">Parkville, Victoria</span>, Australia</address></li><li class="aff"><a id="aff-3" name="aff-3"></a><address> <sup>3</sup><span class="institution">Centre for Research on Aging, Health, and Wellbeing, The Australian National University</span>, <span class="addr-line">Acton, Australian Capital Territory</span>, Australia</address></li><li class="aff"><a id="aff-4" name="aff-4"></a><address> <sup>4</sup><span class="institution">Piramal Imaging GmbH</span>, <span class="addr-line">Berlin</span>, Germany</address></li><li class="aff"><a id="aff-5" name="aff-5"></a><address> <sup>5</sup><span class="institution">Molecular NeuroImaging, L.L.C.</span>, <span class="addr-line">New Haven, Connecticut</span>, USA</address></li><li class="aff"><a id="aff-6" name="aff-6"></a><address> <sup>6</sup><span class="institution">Bayer Healthcare</span>, <span class="addr-line">Berlin</span>, Germany</address></li></ol><ol class="corresp-list"><li class="corresp" id="corresp-1"><span class="corresp-label">Correspondence to</span> Professor Christopher Rowe, Department of Nuclear Medicine and Centre for PET, Austin Health, 145 Studley Road, Heidelberg, VIC 3084, Australia; <span class="em-link"><span class="em-addr">Christopher.Rowe{at}austin.org.au</span></span> </li></ol></div></div></div> </div> </div> <div class="panel-separator"></div><div class="panel-pane pane-highwire-markup abstract-with-bc" > <div class="pane-content"> <div class="highwire-markup"><div xmlns="http://www.w3.org/1999/xhtml" id="content-block" xmlns:xhtml="http://www.w3.org/1999/xhtml"><div class="article abstract-view "><span class="highwire-journal-article-marker-start"></span><div class="section abstract" id="abstract-1"><h2>Abstract</h2><div id="sec-1" class="subsection"> <p id="p-1"><strong>Background</strong> We assessed the clinical utility of β-amyloid (Aβ) imaging with <sup>18</sup>F-florbetaben (FBB) in mild cognitive impairment (MCI) by evaluating its prognostic accuracy for progression to Alzheimer's disease (AD), comparing semiquantitative with visual scan assessment, and exploring the relationships among Aβ, hippocampal volume (HV) and memory over time.</p> </div><div id="sec-2" class="subsection"> <p id="p-2"><strong>Methods</strong> 45 MCI underwent FBB positron emission tomography, MRI and neuropsychological assessment at baseline and 2 years and clinical follow-up at 4 years. Positive FBB (FBB+), defined by a cortical to cerebellar cortex standardised uptake value ratio (SUVR) ≥1.45, was compared with visual assessment by five readers. Amnestic MCI (aMCI) was defined by a composite episodic memory (EM) Z-score of &lt;−1.5.</p> </div><div id="sec-3" class="subsection"> <p id="p-3"><strong>Results</strong> At baseline, 24 (53%) MCI were FBB+. Majority reads agreed with SUVR classification (κ 0.96). In 2 years, 18 (75%) FBB+ progressed to AD compared with 2 (9.5%) FBB−, yielding a predictive accuracy of 83% (95% CI 61% to 94%). Four FBB− developed non-AD dementia. Predictive accuracies of HV (58% (95% CI 42% to 73%)) and aMCI status (73% (95% CI 58% to 81%)) were lower. Combinations did not improve accuracy. By 4 years, 21 (87.5%) FBB+ had AD whereas 5 (24%) FBB− had non-AD dementia yielding a predictive accuracy of 94% (95% CI 74% to 99%). While the strong baseline association between FBB SUVR and EM declined over 2 years, the association between EM and HV became stronger. FBB SUVR increased 2.2%/year in FBB+ with no change in FBB−.</p> </div><div id="sec-4" class="subsection"> <p id="p-4"><strong>Conclusions</strong> <sup>18</sup>F-florbetaben Aβ imaging facilitates accurate detection of prodromal AD. As neurodegeneration progresses, and in contrast with the early stages of the disease, hippocampal atrophy and not Aβ, seems to drive memory decline.</p> </div><div id="sec-5" class="subsection"> <p id="p-5"><strong>Trial registration number</strong> NCT01138111.</p> </div> </div><ul class="kwd-group"><li class="kwd">ALZHEIMER'S DISEASE</li><li class="kwd">AMYLOID</li><li class="kwd">COGNITION</li><li class="kwd">DEMENTIA</li><li class="kwd">MRI</li></ul><span class="highwire-journal-article-marker-end"></span></div><span class="related-urls"></span></div></div> </div> </div> <div class="panel-separator"></div><div class="panel-pane pane-dfp-pane oas-ads oas-ads-mid pull-right" > <div class="pane-content"> <div id="dfp-ad-mpu-wrapper" class="dfp-tag-wrapper"> <div id="dfp-ad-mpu" class="dfp-tag-wrapper"> <script type="text/javascript"> googletag.cmd.push(function() { googletag.display("dfp-ad-mpu"); }); </script> </div> </div> </div> </div> <div class="panel-separator"></div><div class="panel-pane pane-custom pane-1" > <div class="pane-content"> <p><a href="https://doi.org/10.1136/jnnp-2014-308094" target="_new">https://doi.org/10.1136/jnnp-2014-308094</a></p> </div> </div> <div class="panel-separator"></div><div class="panel-pane pane-highwire-altmetrics" > <h2 class="pane-title">Statistics from Altmetric.com</h2> <div class="pane-content"> <div data-badge-details="right" data-badge-type="medium-donut" data-doi="10.1136/jnnp-2014-308094" data-hide-no-mentions="true" class="altmetric-embed"></div> </div> </div> <div class="panel-separator"></div><div class="panel-pane pane-bmjj-jumplinks" > <div class="pane-content"> <div class="highwire-list-wrapper"><div class="highwire-list"><ul></ul></div></div> </div> </div> <div class="panel-separator"></div><div class="panel-pane pane-custom pane-2 permissions-box" > <h2 class="pane-title">Request Permissions</h2> <div class="pane-content"> <p>If you wish to reuse any or all of this article please use the link below which will take you to the Copyright Clearance Center&rsquo;s RightsLink service. 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