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If you believe you were blocked by mistake, you can find additional information and instructions in the <a href="https://meta.wikimedia.org/wiki/Special:MyLanguage/No_open_proxies" class="extiw" title="m:Special:MyLanguage/No open proxies">No open proxies</a> global policy. </p> Otherwise, to discuss the block please <a href="https://meta.wikimedia.org/wiki/Steward_requests/Global" class="extiw" title="m:Steward requests/Global">post a request for review on Meta-Wiki</a>. You could also send an email to the <a href="https://meta.wikimedia.org/wiki/Special:MyLanguage/Stewards" class="extiw" title="m:Special:MyLanguage/Stewards">stewards</a> <a href="https://meta.wikimedia.org/wiki/Special:MyLanguage/VRT" class="extiw" title="m:Special:MyLanguage/VRT">VRT</a> queue at <kbd>stewards@wikimedia.org</kbd> including all above details.</div></div><hr /> <p>Jo kinne de boarnetekst fan dizze side besjen en kopiearje. </p><textarea readonly="" accesskey="," id="wpTextbox1" cols="80" rows="25" style="" class="mw-editfont-monospace" lang="fy" dir="ltr" name="wpTextbox1">== Oarsaken == De sykte wurdt neffens de meast akseptearre hypoteze feroarsake troch bepaalde aaiwiten (amyloïdprecursorproteïne:APP) abnormaal ôfbrutsen wurde. Hjirtroch ûntsteane abnormale ôfbraakprodukten (bèta amyloïd) dy't aggregaten (klûnten) foarmje bûten de harsenssellen. Boppedat is der in yntrasellulêre foarm fan amyloïd dat bestiet út Tau. Dizze foarmen fan amyloïd binne grif skealik foar harsenssellen dy't dêrnei ôfstjerre. By [[Post mortem|post-mortemûndersyk]] wurdt in sterk krompen harsensskors (atrofy) fûn, mei bûten de sel lizzende amyloïddelslaggen (senile ''plaques''), yn de sel lizzende amyloïddelslaggen (neurofibrillêre kleauwen) en delslaggen om bloedfetten. Dizze delslaggen binne net lykmjittich oer de harsenskors ferdield. Benammen gedielten fan de harsenskors dêr't ûnthâldfunksjes sitte, binne yn it begjin oandien. Ynearsten is de sykte fan Alzheimer in sykte oan de grize stof fan it searebrum. Iere deteksje of fêststelling fan de sykte fan Alzheimer foarmet op dit momint in wichtich ûnderwerp fan stúdzje. In soadbelovende technyk is it yn byld bringen fan it amyloïd yn de harsens mei in [[Positronemissietomografy|PET]]-scan. Dêrby wurdt trich in arts in radioaktive stof yn de bloedbaan fan de earm spuite. De stof (''Pittsburg compound B'' of PiB genaamd<ref>Klunk, W.E., et al., Imaging brain amyloid in Alzheimer's disease with Pittsburgh Compound-B.[see comment]. Annals of Neurology, 2004. 55(3): p. 306-19</ref>) giet dêrnei in gemyske ferbining oan mei it yn de harsens oanwêzige amyloïd.<ref>http://www.mirm.pitt.edu/news/article.asp?qEmpID=286</ref> De ''plaques'' of ôfbraakprodukten wurde dan sichtber yn it byld fan de scanner. Omdat ''plaques'' soms ek yn de harsens fan normale âlderen foarkomme, is it fan belang om te witten by hokker hoemannichte der in oanwizing bestiet foar de sykte fan Alzheimer. In oare technyk is de [[lumbale punksje]], wêrby't de rêgemurchfloeistof ûndersocht wurdt op amyloïd ôfbraakprodukten. [[Ofbyld:PiB PET Images AD.jpg|thumb|''In PET-scan byld fan de harsens fan in alzheimerpasjint (AD) en in sûne proefpersoan (Control)]] Der blykt in genetyske predisposysje te bestean foar Alzheimer. De erflikheid bedraacht nei skatting 76%. De let begjinnende alzheimer komt grif troch in yn de mande gean fan genetyske én omjouwingsynfloeden. De iere alzheimer liket dêr foaroer benammen troch genetyske faktoaren bepaald. It ApoE4 [[allel]] by minsken jout in ferhege kâns op de sykte, benammen foar de ier begjinnende ferzje. Minsken dy't oer it ApoE3 en ApoE4 beskikke hawwe in trijeris ferhege kâns op de sykte fan Alzheimer, en minsken dy't oer twa ApoE4 allelen besikke hawwe in 14 kear ferhege kâns op de sykte. Mar, 50% fan de minsken mei ApoE4 krijt gjin alzheimer, en in soad alzheimerpasjinten hawwe gjin ApoE4-allel(en). Resint hawwe ûndersikers nije risikogenen foar alzheimer fûn yn in ûndersyk fan in tige grutte populaasje.<ref>D. Harold et al. Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer’s disease. Nature Genetics. Advance online publication. 6 sept. 2009.</ref> Dizze genen kodearje aaiwiten dy't in rol sp[ylje by de cholesterolhúshâlding en ûnstekingsmeganismen yn de harsens. De rol fan [[aluminium]] en [[kwik]] (yn [[amalgaam]]-fullingen) as feroarsakers fan alzheimer is noch net opheldere. It grutte rol liket tige ûnwierskynlik. It tal ûndersiken is lyts en sprekt elkoar diels tsjin. Oare ko-faktoaren dy't mooglik in rol spylje, binne oare metalen (bygelyks [[Koper (elemint)|koper]]), [[ûntstekking (genêskunde)|ûntstekkingsreaksjes]] yn de harsens, in tekoart oan [[fitamine]]s, [[oksidative stress]] en [[metabolisme|metabole fersteuringen]].<ref>Forbes WF, Hill GB. Is exposure to aluminum a risk factor for the development of Alzheimer disease?--Yes. Arch Neurol. 1998;55:740-1.</ref><ref>Munoz DG. Is exposure to aluminum a risk factor for the development of Alzheimer disease?--No. Arch Neurol. 1998;55:737-9.</ref><ref>Schuurs AH, de Wolff FA.[Relation between mercury and Alzheimer's disease?] Ned Tijdschr Tandheelkd. 1997;104:219-22.</ref><ref>Mutter J, Naumann J, Schneider R, Walach H. [Mercury and Alzheimer's disease] Fortschr Neurol Psychiatr. 2007;75:528-38.</ref><ref>[http://www.alz.org/alzheimers_disease_myths_about_alzheimers.asp Alzheimers Association: Alzheimer Myths]</ref> </textarea><div class="templatesUsed"></div><p id="mw-returnto">Werom nei <a href="/wiki/Sykte_fan_Alzheimer" title="Sykte fan Alzheimer">Sykte fan Alzheimer</a>.</p> <noscript><img src="https://login.wikimedia.org/wiki/Special:CentralAutoLogin/start?type=1x1&useformat=desktop" alt="" width="1" height="1" style="border: none; position: absolute;"></noscript> <div class="printfooter" data-nosnippet="">Opfrege fan "<a dir="ltr" href="https://fy.wikipedia.org/wiki/Sykte_fan_Alzheimer">https://fy.wikipedia.org/wiki/Sykte_fan_Alzheimer</a>"</div></div> <div id="catlinks" class="catlinks catlinks-allhidden" data-mw="interface"></div> </div> </main> </div> <div class="mw-footer-container"> <footer id="footer" class="mw-footer" > <ul id="footer-info"> </ul> <ul id="footer-places"> <li id="footer-places-privacy"><a href="https://foundation.wikimedia.org/wiki/Special:MyLanguage/Policy:Privacy_policy">Privacybelied</a></li> <li id="footer-places-about"><a href="/wiki/Wikipedy:Ynfo">Oer Wikipedy</a></li> <li id="footer-places-disclaimers"><a href="/wiki/Wikipedy:Foarbeh%C3%A2ld">Foarbehâld</a></li> <li id="footer-places-wm-codeofconduct"><a href="https://foundation.wikimedia.org/wiki/Special:MyLanguage/Policy:Universal_Code_of_Conduct">Gedrachskoade</a></li> <li id="footer-places-developers"><a href="https://developer.wikimedia.org">Untwikkelders</a></li> <li id="footer-places-statslink"><a href="https://stats.wikimedia.org/#/fy.wikipedia.org">Statistiken</a></li> <li id="footer-places-cookiestatement"><a href="https://foundation.wikimedia.org/wiki/Special:MyLanguage/Policy:Cookie_statement">Taljochting koekjes</a></li> <li id="footer-places-mobileview"><a href="//fy.m.wikipedia.org/w/index.php?title=Sykte_fan_Alzheimer&action=edit&section=2&mobileaction=toggle_view_mobile" class="noprint stopMobileRedirectToggle">Mobile ferzje</a></li> </ul> <ul id="footer-icons" class="noprint"> <li id="footer-copyrightico"><a href="https://wikimediafoundation.org/" class="cdx-button cdx-button--fake-button cdx-button--size-large cdx-button--fake-button--enabled"><img src="/static/images/footer/wikimedia-button.svg" width="84" height="29" alt="Wikimedia Foundation" loading="lazy"></a></li> <li id="footer-poweredbyico"><a href="https://www.mediawiki.org/" class="cdx-button cdx-button--fake-button cdx-button--size-large cdx-button--fake-button--enabled"><img src="/w/resources/assets/poweredby_mediawiki.svg" alt="Powered by MediaWiki" width="88" height="31" loading="lazy"></a></li> </ul> </footer> </div> </div> </div> <div class="vector-settings" id="p-dock-bottom"> <ul></ul> </div><script>(RLQ=window.RLQ||[]).push(function(){mw.config.set({"wgHostname":"mw-web.codfw.main-5c59558b9d-d5pwq","wgBackendResponseTime":180,"wgPageParseReport":{"limitreport":{"cputime":"0.005","walltime":"0.006","ppvisitednodes":{"value":17,"limit":1000000},"postexpandincludesize":{"value":690,"limit":2097152},"templateargumentsize":{"value":0,"limit":2097152},"expansiondepth":{"value":2,"limit":100},"expensivefunctioncount":{"value":0,"limit":500},"unstrip-depth":{"value":0,"limit":20},"unstrip-size":{"value":0,"limit":5000000},"entityaccesscount":{"value":0,"limit":400},"timingprofile":["100.00% 0.000 1 -total"]},"cachereport":{"origin":"mw-web.codfw.main-5c59558b9d-d5pwq","timestamp":"20241130112318","ttl":2592000,"transientcontent":false}}});});</script> </body> </html>