<rdf:RDF xmlns:rdf="http://www.w3.org/1999/02/22-rdf-syntax-ns#" xmlns:prism="http://prismstandard.org/namespaces/basic/2.0/" xmlns:dc="http://purl.org/dc/elements/1.1/" xmlns:content="http://purl.org/rss/1.0/modules/content/" xmlns="http://purl.org/rss/1.0/" xmlns:admin="http://webns.net/mvcb/"> <channel rdf:about="http://feeds.nature.com/onc/rss/current"> <title>Oncogene</title> <description>Oncogene aims to make substantial advances in our knowledge of processes that contribute to cancer by publishing outstanding research. Oncogene propagates work that challenges standard conjecture and builds on previous studies, in particular those that lead to establishing new paradigms in the etio-pathogenesis, diagnosis, treatment, or prevention of cancers, and in processes that drive metastatic spread, and provide important insights into cancer biology beyond what has been revealed thus far. Areas covered by Oncogene include, but are not limited to, the cellular and molecular biology of cancer including resistance to cancer therapies, and development of better approaches to improving survival. Across cancer biology, we have a broad remit encompassing the life sciences and biomedicine, from the most fundamental and theoretical work, through to translational, applied, and clinical research.</description> <link>http://feeds.nature.com/onc/rss/current</link> <admin:generatorAgent rdf:resource="https://www.nature.com/"/> <admin:errorReportsTo rdf:resource="mailto:feedback@nature.com"/> <dc:publisher>Nature Publishing Group</dc:publisher> <dc:language>en</dc:language> <dc:rights>漏 2025 Macmillan Publishers Limited, part of Springer Nature. All rights reserved.</dc:rights> <prism:publicationName>Oncogene</prism:publicationName> <prism:copyright>漏 2025 Macmillan Publishers Limited, part of Springer Nature. All rights reserved.</prism:copyright> <prism:rightsAgent>permissions@nature.com</prism:rightsAgent> <image rdf:resource="https://www.nature.com/uploads/product/onc/rss.gif"/> <items> <rdf:Seq> <rdf:li rdf:resource="https://www.nature.com/articles/s41388-025-03301-7"/> <rdf:li rdf:resource="https://www.nature.com/articles/s41388-025-03300-8"/> <rdf:li rdf:resource="https://www.nature.com/articles/s41388-025-03306-2"/> <rdf:li rdf:resource="https://www.nature.com/articles/s41388-025-03307-1"/> <rdf:li rdf:resource="https://www.nature.com/articles/s41388-025-03297-0"/> <rdf:li rdf:resource="https://www.nature.com/articles/s41388-025-03305-3"/> <rdf:li rdf:resource="https://www.nature.com/articles/s41388-025-03304-4"/> <rdf:li rdf:resource="https://www.nature.com/articles/s41388-024-03265-0"/> </rdf:Seq> </items> </channel> <image rdf:about="https://www.nature.com/uploads/product/onc/rss.gif"> <title>Oncogene</title> <url>https://www.nature.com/uploads/product/onc/rss.gif</url> <link>http://feeds.nature.com/onc/rss/current</link> </image> <item rdf:about="https://www.nature.com/articles/s41388-025-03301-7"> <title><![CDATA[Novel role of general transcript factor IIH subunit 2 (GTF2H2) in the development and sex disparity of hepatocellular carcinoma]]></title> <link>https://www.nature.com/articles/s41388-025-03301-7</link> <content:encoded> <![CDATA[<p>Oncogene, Published online: 19 February 2025; <a href="https://www.nature.com/articles/s41388-025-03301-7">doi:10.1038/s41388-025-03301-7</a></p>Novel role of general transcript factor IIH subunit 2 (GTF2H2) in the development and sex disparity of hepatocellular carcinoma]]></content:encoded> <dc:title><![CDATA[Novel role of general transcript factor IIH subunit 2 (GTF2H2) in the development and sex disparity of hepatocellular carcinoma]]></dc:title> <dc:creator>Yanmeng Li</dc:creator><dc:creator>Qin Ouyang</dc:creator><dc:creator>Zhibin Chen</dc:creator><dc:creator>Donghu Zhou</dc:creator><dc:creator>Zhenkun Li</dc:creator><dc:creator>Xiaoxi Yang</dc:creator><dc:creator>Jiang Long</dc:creator><dc:creator>Guangyong Chen</dc:creator><dc:creator>Xiaojin Li</dc:creator><dc:creator>Siyu Jia</dc:creator><dc:creator>Huaduan Zi</dc:creator><dc:creator>Saiping Qi</dc:creator><dc:creator>Hengcheng Tang</dc:creator><dc:creator>Bei Zhang</dc:creator><dc:creator>Yamei Niu</dc:creator><dc:creator>Anjian Xu</dc:creator><dc:creator>Weimin Tong</dc:creator><dc:creator>Jidong Jia</dc:creator><dc:creator>Jian Huang</dc:creator> <dc:identifier>doi:10.1038/s41388-025-03301-7</dc:identifier> <dc:source>Oncogene, Published online: 2025-02-19; | doi:10.1038/s41388-025-03301-7</dc:source> <dc:date>2025-02-19</dc:date> <prism:publicationName>Oncogene</prism:publicationName> <prism:doi>10.1038/s41388-025-03301-7</prism:doi> <prism:url>https://www.nature.com/articles/s41388-025-03301-7</prism:url> </item> <item rdf:about="https://www.nature.com/articles/s41388-025-03300-8"> <title><![CDATA[Deletion of 17p in cancers: Guilt by (p53) association]]></title> <link>https://www.nature.com/articles/s41388-025-03300-8</link> <content:encoded> <![CDATA[<p>Oncogene, Published online: 18 February 2025; <a href="https://www.nature.com/articles/s41388-025-03300-8">doi:10.1038/s41388-025-03300-8</a></p>Deletion of 17p in cancers: Guilt by (p53) association]]></content:encoded> <dc:title><![CDATA[Deletion of 17p in cancers: Guilt by (p53) association]]></dc:title> <dc:creator>Francisca van Kampen</dc:creator><dc:creator>Abigail Clark</dc:creator><dc:creator>Jamie Soul</dc:creator><dc:creator>Aditi Kanhere</dc:creator><dc:creator>Mark A. 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Rellinger</dc:creator> <dc:identifier>doi:10.1038/s41388-025-03297-0</dc:identifier> <dc:source>Oncogene, Published online: 2025-02-16; | doi:10.1038/s41388-025-03297-0</dc:source> <dc:date>2025-02-16</dc:date> <prism:publicationName>Oncogene</prism:publicationName> <prism:doi>10.1038/s41388-025-03297-0</prism:doi> <prism:url>https://www.nature.com/articles/s41388-025-03297-0</prism:url> </item> <item rdf:about="https://www.nature.com/articles/s41388-025-03305-3"> <title><![CDATA[SUMOylation inhibition potentiates the glucocorticoid receptor to program growth arrest of acute lymphoblastic leukemia cells]]></title> <link>https://www.nature.com/articles/s41388-025-03305-3</link> <content:encoded> <![CDATA[<p>Oncogene, Published online: 14 February 2025; <a href="https://www.nature.com/articles/s41388-025-03305-3">doi:10.1038/s41388-025-03305-3</a></p>SUMOylation inhibition potentiates the glucocorticoid receptor to program growth arrest of acute lymphoblastic leukemia cells]]></content:encoded> <dc:title><![CDATA[SUMOylation inhibition potentiates the glucocorticoid receptor to program growth arrest of acute lymphoblastic leukemia cells]]></dc:title> <dc:creator>Emma Valima</dc:creator><dc:creator>Vera Varis</dc:creator><dc:creator>Kseniia Bureiko</dc:creator><dc:creator>Joanna K. Lempi盲inen</dc:creator><dc:creator>Anna-Mari Schroderus</dc:creator><dc:creator>Laura Oksa</dc:creator><dc:creator>Olli Lohi</dc:creator><dc:creator>Tuure Kinnunen</dc:creator><dc:creator>Markku Varjosalo</dc:creator><dc:creator>Einari A. Niskanen</dc:creator><dc:creator>Ville Paakinaho</dc:creator><dc:creator>Jorma J. Palvimo</dc:creator> <dc:identifier>doi:10.1038/s41388-025-03305-3</dc:identifier> <dc:source>Oncogene, Published online: 2025-02-14; | doi:10.1038/s41388-025-03305-3</dc:source> <dc:date>2025-02-14</dc:date> <prism:publicationName>Oncogene</prism:publicationName> <prism:doi>10.1038/s41388-025-03305-3</prism:doi> <prism:url>https://www.nature.com/articles/s41388-025-03305-3</prism:url> </item> <item rdf:about="https://www.nature.com/articles/s41388-025-03304-4"> <title><![CDATA[Correction: A novel amplification gene PCI domain containing 2 (PCID2) promotes colorectal cancer through directly degrading a tumor suppressor promyelocytic leukemia (PML)]]></title> <link>https://www.nature.com/articles/s41388-025-03304-4</link> <content:encoded> <![CDATA[<p>Oncogene, Published online: 12 February 2025; <a href="https://www.nature.com/articles/s41388-025-03304-4">doi:10.1038/s41388-025-03304-4</a></p>Correction: A novel amplification gene PCI domain containing 2 (PCID2) promotes colorectal cancer through directly degrading a tumor suppressor promyelocytic leukemia (PML)]]></content:encoded> <dc:title><![CDATA[Correction: A novel amplification gene PCI domain containing 2 (PCID2) promotes colorectal cancer through directly degrading a tumor suppressor promyelocytic leukemia (PML)]]></dc:title> <dc:creator>Jingwan Zhang</dc:creator><dc:creator>Jianning Zhai</dc:creator><dc:creator>Chi Chun Wong</dc:creator><dc:creator>Huarong Chen</dc:creator><dc:creator>Xiaohong Wang</dc:creator><dc:creator>Jiafu Ji</dc:creator><dc:creator>Jun Yu</dc:creator> <dc:identifier>doi:10.1038/s41388-025-03304-4</dc:identifier> <dc:source>Oncogene, Published online: 2025-02-12; | doi:10.1038/s41388-025-03304-4</dc:source> <dc:date>2025-02-12</dc:date> <prism:publicationName>Oncogene</prism:publicationName> <prism:doi>10.1038/s41388-025-03304-4</prism:doi> <prism:url>https://www.nature.com/articles/s41388-025-03304-4</prism:url> </item> <item rdf:about="https://www.nature.com/articles/s41388-024-03265-0"> <title><![CDATA[PPP2R1A mutations cause ATR inhibitor sensitivity in ovarian clear cell carcinoma]]></title> <link>https://www.nature.com/articles/s41388-024-03265-0</link> <content:encoded> <![CDATA[<p>Oncogene, Published online: 12 February 2025; <a href="https://www.nature.com/articles/s41388-024-03265-0">doi:10.1038/s41388-024-03265-0</a></p>PPP2R1A mutations cause ATR inhibitor sensitivity in ovarian clear cell carcinoma]]></content:encoded> <dc:title><![CDATA[PPP2R1A mutations cause ATR inhibitor sensitivity in ovarian clear cell carcinoma]]></dc:title> <dc:creator>James Stewart</dc:creator><dc:creator>Dragomir B. Krastev</dc:creator><dc:creator>Rachel Brough</dc:creator><dc:creator>Diana Zatreanu</dc:creator><dc:creator>Feifei Song</dc:creator><dc:creator>Joseph S. Baxter</dc:creator><dc:creator>Sandhya Sridhar</dc:creator><dc:creator>Jessica Frankum</dc:creator><dc:creator>Asha Konde</dc:creator><dc:creator>William Yang</dc:creator><dc:creator>Syed Haider</dc:creator><dc:creator>John Alexander</dc:creator><dc:creator>Kai Betteridge</dc:creator><dc:creator>Aditi Gulati</dc:creator><dc:creator>Ayoma D. Attygalle</dc:creator><dc:creator>Katherine Vroobel</dc:creator><dc:creator>Rachael Natrajan</dc:creator><dc:creator>Saira Khalique</dc:creator><dc:creator>Theodoros I. Roumeliotis</dc:creator><dc:creator>Jyoti S. Choudhary</dc:creator><dc:creator>Jason Yeung</dc:creator><dc:creator>Andrew J. Wicks</dc:creator><dc:creator>Rebecca Marlow</dc:creator><dc:creator>Susana Banerjee</dc:creator><dc:creator>Stephen J. Pettitt</dc:creator><dc:creator>Andrew N. J. Tutt</dc:creator><dc:creator>Christopher J. Lord</dc:creator> <dc:identifier>doi:10.1038/s41388-024-03265-0</dc:identifier> <dc:source>Oncogene, Published online: 2025-02-12; | doi:10.1038/s41388-024-03265-0</dc:source> <dc:date>2025-02-12</dc:date> <prism:publicationName>Oncogene</prism:publicationName> <prism:doi>10.1038/s41388-024-03265-0</prism:doi> <prism:url>https://www.nature.com/articles/s41388-024-03265-0</prism:url> </item> </rdf:RDF>