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id="frame-backgroundimage-21401" class="frame-backgroundimage"><div class="copyright">漏 James Steidl und Sebastian Kaulitzki / fotolia.com</div></div></div></div></div><div class="frame-type-intro"><div class="frame-container frame-container-default"><div class="frame-inner"><header class="frame-header"><h1 class="element-header mb-2"><span>Inflammation in demyelinisierenden und neurodegenerativen Erkrankungen</span></h1></header></div></div></div></div><nav class="breadcrumb-section" aria-label="Brotkr眉melnavigation"><div class="container"><p class="sr-only" id="breadcrumb">Sie sind hier:</p><ol class="breadcrumb"><li class="breadcrumb-item"><a class="breadcrumb-link" href="https://www.uk-erlangen.de/" title="Zur Startseite des Universit盲tsklinikums Erlangen"><svg height="37" xmlns="http://www.w3.org/2000/svg" viewBox="0,0,63,64"><g><path d="M20.937+21.0438L0+21.0438L0+42.3789L20.937+42.3789L20.937+21.0438Z" opacity="1" fill="#00a579"/><path 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class="breadcrumb-text">Grundlagenforschung</span></a></li><li class="breadcrumb-item icon-arrow_right active" aria-current="page"><span class="breadcrumb-text">Inflammation in demyelinisierenden und neurodegenerativen Erkrankungen</span></li></ol></div></nav><main id="page-content" class="bp-page-content main-section"><div class="section section-before-content"></div><div class="section section-default"><div id="c67280" class=" frame frame-default frame-type-text frame-layout-default frame-size-default frame-height-default frame-background-none frame-space-before-none frame-space-after-none frame-no-backgroundimage"><div class="frame-group-container"><div class="frame-group-inner"><div class="frame-container frame-container-default"><div class="frame-inner"><div class="frame-header"><h2 class="element-header "><span>Inflammation in demyelinisierenden und neurodegenerativen Erkrankungen</span></h2><h3 class="element-subheader "><span>Projektleitung/ Mitarbeiter: Prof. Dr. J眉rgen Winkler/ PD Dr. Franz Marxreiter/Dr. Alana Hoffmann/Dr. Patrick S眉脽</span></h3></div><p>Neuroinflammation spielt eine entscheidende, aber bis heute weitgehend unverstandene Rolle bei neurodegenerativen Erkrankungen. In grundlagenwissenschaftlichen Ans盲tzen wird untersucht, inwieweit chronische Entzu虉ndungsvorga虉nge im zentralen Nervensystem (ZNS), aber auch in der Peripherie, zu einer Fehlfunktion oder sogar dem Absterben von Nerven- und Gliazellen im ZNS f眉hren k枚nnen. Insbesondere liegt hier das Hauptaugenmerk auf der Erforschung und Modulation von spezifischen Immunzellen und Entzu虉ndungssignalwegen, welche den Verlauf von neurodegenerativen Erkrankungen beeinflussen und so neue Therapieansa虉tze erm枚glichen. Ein wichtiger Bestandteil neurodegenerativer und -entz眉ndlicher Erkrankungen sind pathologische Proteinablagerungen in Nerven- und Gliazellen. Wir konnten in bisherigen Studien zeigen, dass 伪-Synuklein ein wichtiger Bestandteil in Parkinson und Parkinson-盲hnlichen Erkrankungen ist, jedoch auch in entz眉ndlichen Erkrankungen wie der Multiplen Sklerose eine Rolle spielt und Entz眉ndungsreaktionen beeinflussen kann. Um das Zusammenspiel von Neuroinflammation, 伪-Synuklein und zellul盲rer Pathologie besser zu verstehen untersuchen wir verschiedene Modelle neurodegenerativer und neuroinflammatorischer Erkrankungen.</p><p><strong>Kooperationspartner:</strong><br> Prof. Dr. R. Linker (Klinik und Poliklinik f眉r Neurologie, Regensburg)<br> Prof. Dr. B. Winner (Stammzellbiologische Abteilung, UKER)<br> Prof. Dr. T. Kuhlmann (Institut f眉r Neuropathologie, M眉nster)<br> Prof. Dr. M. Riemenschneider (Abteilung f眉r Neuropathologie, Universit盲tsklinikum Regensburg)<br> Prof. Dr. E. 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