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<!doctype html> <html lang="hi"> <head> <title>Genetic Disorder of Obesity and Intellectual Disability of S | 88422</title> <meta name="keywords" content="Pinge Hui* , "/> <meta name="description" content="Prader-Willi condition is described by serious juvenile hypotonia with helpless suck and inability to flourish; hypogonadism causing genital hypoplasia and..88422"/> <meta name="citation_publisher" content="लॉन्गडम पब्लिशिंग एसएल" /> <meta name="citation_journal_title" content="डाउन सिंड्रोम और क्रोमोसोम असामान्यताओं का जर्नल"> <meta name="citation_title" content="Genetic Disorder of Obesity and Intellectual Disability of Specific Genes in Prader-Willi Syndrome "> <meta name="citation_author" content="Pinge Hui" /> <meta name="citation_year" content=""> <meta name="citation_volume" content="7"> <meta name="citation_issue" content="6"> <meta name="citation_abstract" content="Prader-Willi condition is described by serious juvenile hypotonia with helpless suck and inability to flourish; hypogonadism causing genital hypoplasia and pubertal deficiency; trademark facial highlights; youth beginning heftiness and hyperphagia; formative deferral/gentle scholarly incapacity; short height; and a particular conduct aggregate. Rest irregularities and scoliosis are normal. Development chemical deficiency is continuous, and substitution treatment gives improvement in development, body organization, and actual traits. The board is generally to a great extent steady. Agreement clinical symptomatic measures exist, yet finding ought to be affirmed through hereditary testing. Prader-Willi disorder is because of nonattendance of in a fatherly way communicated engraved qualities at 15q11.2-q13 through fatherly cancellation of this area, maternal uniparental disomy 15 (2030%), or an engraving imperfection (13%). Parent-explicit DNA methylation investigation will distinguish >99% of people. Notwithstanding, extra hereditary investigations are important to distinguish the sub-atomic class. There are different engraved qualities around here, the deficiency of which add to the total aggregate of Prader-Willi condition. In any case, nonattendance of a little nucleolar arranging RNA quality, SNORD116, appears to repeat a considerable lot of the clinical highlights. Kin repeat hazard is regularly <1%, however higher dangers might relate in specific cases. Pre-birth finding is accessible. 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Rest irregularities and scoliosis are normal. Development chemical deficiency is continuous, and substitution treatment gives improvement in development, body organization, and actual traits. The board is generally to a great extent steady. Agreement clinical symptomatic measures exist, yet finding ought to be affirmed through hereditary testing. Prader-Willi disorder is because of nonattendance of in a fatherly way communicated engraved qualities at 15q11.2-q13 through fatherly cancellation of this area, maternal uniparental disomy 15 (2030%), or an engraving imperfection (13%). Parent-explicit DNA methylation investigation will distinguish >99% of people. Notwithstanding, extra hereditary investigations are important to distinguish the sub-atomic class. There are different engraved qualities around here, the deficiency of which add to the total aggregate of Prader-Willi condition. In any case, nonattendance of a little nucleolar arranging RNA quality, SNORD116, appears to repeat a considerable lot of the clinical highlights. Kin repeat hazard is regularly <1%, however higher dangers might relate in specific cases. Pre-birth finding is accessible.</p> <div class="alert alert-info text-left"><b>अस्वीकरण:</b> इस सार का अनुवाद कृत्रिम बुद्धिमत्ता उपकरणों का उपयोग करके किया गया था और अभी तक इसकी समीक्षा या सत्यापन नहीं किया गया है।</div> <div class="nav social-icons"> <a class="nav-link w-auto">इस लेख का हिस्सा</a> <a title="यहाँ क्लिक करें" target="_blank" href="https://www.facebook.com/sharer.php?u=https://hindi.longdom.org/abstract/genetic-disorder-of-obesity-and-intellectual-disability-of-specific-genes-in-praderwilli-syndrome-88422.html" rel="noopener"><i class="fab fa-facebook-f"></i></a> <a class="nav-link" title="यहाँ क्लिक करें" target="_blank" href="https://twitter.com/share?url=https://hindi.longdom.org/abstract/genetic-disorder-of-obesity-and-intellectual-disability-of-specific-genes-in-praderwilli-syndrome-88422.html" rel="noopener"><i class="fab fa-twitter"></i></a> <a class="nav-link" title="यहाँ क्लिक करें" target="_blank" href="https://www.linkedin.com/shareArticle?mini=true&url=https://hindi.longdom.org/abstract/genetic-disorder-of-obesity-and-intellectual-disability-of-specific-genes-in-praderwilli-syndrome-88422.html" rel="noopener"><i class="fab fa-linkedin-in"></i></a> <a class="nav-link" title="यहाँ क्लिक करें" target="_blank" href="https://plus.google.com/share?url=https://hindi.longdom.org/abstract/genetic-disorder-of-obesity-and-intellectual-disability-of-specific-genes-in-praderwilli-syndrome-88422.html" rel="noopener"><i class="fab fa-google-plus-g"></i></a> </div> </div> </div> </div> </section> <footer class="bg-blue-grey-900 py-3"> <div class="container"> <div class="row"> <div class="col-12 col-sm-4"> <h4 class="white font-size-4 fweight-400 border-bottom-1 pb-2">सामग्री लिंक</h4> <ul class="list-unstyled footer-links font-size-3"> <li><a class="" href="https://hindi.longdom.org/privacy-policy.html" title="यहाँ क्लिक करें">गोपनीयता नीति</a></li> <li><a class="" href="https://hindi.longdom.org/terms-conditions.html" title="यहाँ क्लिक करें">नियम एवं शर्तें</a></li> <li><a class="" href="https://hindi.longdom.org/authors-reviewers-editors.html" title="यहाँ क्लिक करें">लेखक, समीक्षक और संपादक</a></li> </ul> </div> <div class="col-12 col-sm-4"> <h4 class="white font-size-4 fweight-400 border-bottom-1 pb-2">लॉन्गडोम से संपर्क करें</h4> <p><strong>लॉन्गडम ग्रुप एसए</strong><br /> एवेन्यू रोजर वांडेन्ड्रिस्शे,<br /> 18, 1150 ब्रुसेल्स, बेल्जियम<br /> फ़ोन: +442038085340<br/> <strong>ईमेल:</strong> <a class='white' href='mailto:info@longdom.org' title='यहां क्लिक करें'>info@longdom.org</a></p> </div> <div class="col-12 col-sm-4"> <h4 class="white font-size-4 fweight-400 border-bottom-1 pb-2">जोड़ना</h4> <nav class="nav nav-pills social-icons-footer flex-column a-pl-0"> <a href="https://www.facebook.com/longdompublisher" title="यहाँ क्लिक करें" target="_blank" class="nav-link bg-facebook-hover"><i class="fab fa-facebook-f bg-facebook"></i></a> <a href="https://www.linkedin.com/company/longdom-publishing-sl/" title="यहाँ क्लिक करें" target="_blank" class="nav-link bg-linkedin-hover"><i class="fab fa-linkedin-in bg-linkedin"></i></a> <a href="https://twitter.com/LongdomP" title="यहाँ क्लिक करें" target="_blank" class="nav-link bg-twitter-hover"><i class="fab fa-twitter bg-twitter"></i></a> <a href="https://www.instagram.com/longdom_publisher/" title="यहाँ क्लिक करें" target="_blank" class="nav-link bg-instagram-hover"><i class="fab fa-instagram bg-instagram"></i></a> </nav> </div> </div> <div class="row text-center"> <div class="col"> <p>कॉपीराइट © 2025 <a href="https://hindi.longdom.org/" title="यहाँ क्लिक करें" class="white">लॉन्गडम पब्लिशिंग एसएल</a>.</p> </div> </div> </div> </footer>  <a href="#0" class="cd-top js-cd-top">Top</a>   <script src="https://code.jquery.com/jquery-3.3.1.min.js"></script> <script src="https://cdnjs.cloudflare.com/ajax/libs/popper.js/1.14.7/umd/popper.min.js"></script> <script src="https://stackpath.bootstrapcdn.com/bootstrap/4.3.1/js/bootstrap.min.js"></script>  <script> jQuery(function($) { $(window).scroll(function fix_element() { $('#target').css( $(window).scrollTop() > 100 ? 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