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He underwent computed tomography of the abdomen and pelvis because of persistent abdominal pain. Computed tomography revealed hepatomegaly with liver span of 15 cm (Fig. 1). The patient had intermittent transaminitis over the past year with negative workup for autoimmune and infectious etiologies. He had been diagnosed with nonalcoholic steatohepatitis. He reported no alcohol or drug use; acetaminophen level was undetectable. Coagulation studies and total protein and albumin were normal. His hepatic enzyme concentrations had risen from before hospitalization: aspartate aminotransferase, from 44 IU/L to 262 IU/L; alanine aminotransferase, from 125 IU/L to 519 IU/L; alkaline phosphatase, from 113 IU/L to 161 IU/L. There were no elevations in bilirubin or creatinine kinase, nor was there a decrease in renal function. By the day of discharge, his hepatic transaminases had trended back to baseline. Ultrasonography-guided liver biopsy was performed, and diffusely pale-staining hepatocytes with excessive glycogen accumulation on periodic acid–Schiff stain were seen (Fig. 2). Glycogenated nuclei were scattered throughout the parenchyma, and rare macrovesicular fat droplets were present. What is the diagnosis?</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="114873059"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="114873059"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 114873059; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=114873059]").text(description); $(".js-view-count[data-work-id=114873059]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 114873059; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='114873059']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 114873059, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=114873059]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":114873059,"title":"Visual Vignette","translated_title":"","metadata":{"abstract":"Case Presentation: A 19-year-old white man with uncontrolled type 1 diabetes mellitus (hemoglobin A1c, 12.7%) was admitted to the intensive care unit and treated for diabetic ketoacidosis. He underwent computed tomography of the abdomen and pelvis because of persistent abdominal pain. Computed tomography revealed hepatomegaly with liver span of 15 cm (Fig. 1). The patient had intermittent transaminitis over the past year with negative workup for autoimmune and infectious etiologies. He had been diagnosed with nonalcoholic steatohepatitis. He reported no alcohol or drug use; acetaminophen level was undetectable. Coagulation studies and total protein and albumin were normal. His hepatic enzyme concentrations had risen from before hospitalization: aspartate aminotransferase, from 44 IU/L to 262 IU/L; alanine aminotransferase, from 125 IU/L to 519 IU/L; alkaline phosphatase, from 113 IU/L to 161 IU/L. There were no elevations in bilirubin or creatinine kinase, nor was there a decrease in renal function. By the day of discharge, his hepatic transaminases had trended back to baseline. Ultrasonography-guided liver biopsy was performed, and diffusely pale-staining hepatocytes with excessive glycogen accumulation on periodic acid–Schiff stain were seen (Fig. 2). Glycogenated nuclei were scattered throughout the parenchyma, and rare macrovesicular fat droplets were present. What is the diagnosis?","publisher":"AACE Corp (American Association of Clinical Endocrinologists)","publication_date":{"day":null,"month":null,"year":2010,"errors":{}},"publication_name":"Endocrine Practice"},"translated_abstract":"Case Presentation: A 19-year-old white man with uncontrolled type 1 diabetes mellitus (hemoglobin A1c, 12.7%) was admitted to the intensive care unit and treated for diabetic ketoacidosis. He underwent computed tomography of the abdomen and pelvis because of persistent abdominal pain. Computed tomography revealed hepatomegaly with liver span of 15 cm (Fig. 1). The patient had intermittent transaminitis over the past year with negative workup for autoimmune and infectious etiologies. He had been diagnosed with nonalcoholic steatohepatitis. He reported no alcohol or drug use; acetaminophen level was undetectable. Coagulation studies and total protein and albumin were normal. His hepatic enzyme concentrations had risen from before hospitalization: aspartate aminotransferase, from 44 IU/L to 262 IU/L; alanine aminotransferase, from 125 IU/L to 519 IU/L; alkaline phosphatase, from 113 IU/L to 161 IU/L. There were no elevations in bilirubin or creatinine kinase, nor was there a decrease in renal function. By the day of discharge, his hepatic transaminases had trended back to baseline. Ultrasonography-guided liver biopsy was performed, and diffusely pale-staining hepatocytes with excessive glycogen accumulation on periodic acid–Schiff stain were seen (Fig. 2). 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What is the diagnosis?","internal_url":"https://www.academia.edu/114873059/Visual_Vignette","translated_internal_url":"","created_at":"2024-02-13T18:13:18.405-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":5615739,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[],"downloadable_attachments":[],"slug":"Visual_Vignette","translated_slug":"","page_count":null,"language":"en","content_type":"Work","owner":{"id":5615739,"first_name":"ratan","middle_initials":null,"last_name":"jha","page_name":"ratanjha","domain_name":"independent","created_at":"2013-09-18T01:17:38.903-07:00","display_name":"ratan jha","url":"https://independent.academia.edu/ratanjha"},"attachments":[],"research_interests":[{"id":26327,"name":"Medicine","url":"https://www.academia.edu/Documents/in/Medicine"},{"id":64568,"name":"Humans","url":"https://www.academia.edu/Documents/in/Humans"},{"id":71437,"name":"Liver","url":"https://www.academia.edu/Documents/in/Liver"},{"id":111545,"name":"Male","url":"https://www.academia.edu/Documents/in/Male"},{"id":244814,"name":"Clinical Sciences","url":"https://www.academia.edu/Documents/in/Clinical_Sciences"},{"id":382075,"name":"Adult","url":"https://www.academia.edu/Documents/in/Adult"},{"id":915951,"name":"Type 2 Diabetes Mellitus","url":"https://www.academia.edu/Documents/in/Type_2_Diabetes_Mellitus"},{"id":3789883,"name":"Paediatrics and reproductive medicine","url":"https://www.academia.edu/Documents/in/Paediatrics_and_reproductive_medicine"},{"id":4178099,"name":"Vignette","url":"https://www.academia.edu/Documents/in/Vignette-1"}],"urls":[]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="109496826"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/109496826/Effect_of_lockdown_on_patient_care_during_COVID_19_pandemic"><img alt="Research paper thumbnail of Effect of lockdown on patient care during COVID-19 pandemic" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/109496826/Effect_of_lockdown_on_patient_care_during_COVID_19_pandemic">Effect of lockdown on patient care during COVID-19 pandemic</a></div><div class="wp-workCard_item"><span>Journal of Renal Nutrition and Metabolism</span><span>, 2020</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109496826"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109496826"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109496826; 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$(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="109496805"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" rel="nofollow" href="https://www.academia.edu/109496805/Glycemic_control_in_patients_of_chronic_kidney_disease"><img alt="Research paper thumbnail of Glycemic control in patients of chronic kidney disease" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" rel="nofollow" href="https://www.academia.edu/109496805/Glycemic_control_in_patients_of_chronic_kidney_disease">Glycemic control in patients of chronic kidney disease</a></div><div class="wp-workCard_item"><span>International Journal of Diabetes in Developing Countries</span><span>, 2007</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">ABSTRACT</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109496805"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109496805"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109496805; 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$(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="109496804"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" rel="nofollow" href="https://www.academia.edu/109496804/Pregnancy_predisposes_to_rhabdomyolysis_due_to_hypokalemia"><img alt="Research paper thumbnail of Pregnancy predisposes to rhabdomyolysis due to hypokalemia" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" rel="nofollow" href="https://www.academia.edu/109496804/Pregnancy_predisposes_to_rhabdomyolysis_due_to_hypokalemia">Pregnancy predisposes to rhabdomyolysis due to hypokalemia</a></div><div class="wp-workCard_item"><span>Saudi journal of kidney diseases and transplantation : an official publication of the Saudi Center for Organ Transplantation, Saudi Arabia</span><span>, 2010</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">Increased predilection for hypokalemia-induced rhabdomyolysis has been noted in pregnant women. W...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">Increased predilection for hypokalemia-induced rhabdomyolysis has been noted in pregnant women. We managed a woman with distal renal tubular acidosis (RTA) with persistent hypokalemia who presented with recurrent rhabdomyolysis in her consecutive pregnancies despite adequate potassium citrate therapy.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109496804"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109496804"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109496804; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=109496804]").text(description); $(".js-view-count[data-work-id=109496804]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 109496804; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='109496804']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 109496804, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=109496804]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":109496804,"title":"Pregnancy predisposes to rhabdomyolysis due to hypokalemia","translated_title":"","metadata":{"abstract":"Increased predilection for hypokalemia-induced rhabdomyolysis has been noted in pregnant women. 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Interpretation of such elevated enzymes thus warrants caution. It necessitates adequate awareness amongst clinicians, of conditions with such elevation in the absence of myocardial ischemia/infarction as well as those that harbinger false positives. We discuss one such case that posed a diagnostic dilemma and review the pertinent literature.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109496803"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109496803"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109496803; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=109496803]").text(description); $(".js-view-count[data-work-id=109496803]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 109496803; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='109496803']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 109496803, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=109496803]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":109496803,"title":"Raised cardiac enzymes in sepsis with renal failure: An encompassing umbrella or a masquerader?","translated_title":"","metadata":{"abstract":"Elevation of cardiac enzymes and troponins particularly in settings of sepsis and renal failure may cloud the diagnostic picture of acute coronary syndrome in many cases. Interpretation of such elevated enzymes thus warrants caution. It necessitates adequate awareness amongst clinicians, of conditions with such elevation in the absence of myocardial ischemia/infarction as well as those that harbinger false positives. We discuss one such case that posed a diagnostic dilemma and review the pertinent literature.","publisher":"Medknow","publication_date":{"day":null,"month":null,"year":2014,"errors":{}},"publication_name":"Journal of the Scientific Society"},"translated_abstract":"Elevation of cardiac enzymes and troponins particularly in settings of sepsis and renal failure may cloud the diagnostic picture of acute coronary syndrome in many cases. Interpretation of such elevated enzymes thus warrants caution. It necessitates adequate awareness amongst clinicians, of conditions with such elevation in the absence of myocardial ischemia/infarction as well as those that harbinger false positives. 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href="https://www.academia.edu/109496802/Distal_Renal_Tubular_Acidosis_Due_to_Primary_Hyperparathyroidism"><img alt="Research paper thumbnail of Distal Renal Tubular Acidosis Due to Primary Hyperparathyroidism" class="work-thumbnail" src="https://attachments.academia-assets.com/107605145/thumbnails/1.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/109496802/Distal_Renal_Tubular_Acidosis_Due_to_Primary_Hyperparathyroidism">Distal Renal Tubular Acidosis Due to Primary Hyperparathyroidism</a></div><div class="wp-workCard_item"><span>Endocrine Practice</span><span>, 2008</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><a id="69f3af28c1ba7c6842cf8c63901aa1bf" class="wp-workCard--action" rel="nofollow" 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retrospective analysis of fifty one cases from a single centre" class="work-thumbnail" src="https://attachments.academia-assets.com/108758525/thumbnails/1.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/109496784/Symptomatic_primary_hyperparathyroidism_a_retrospective_analysis_of_fifty_one_cases_from_a_single_centre">Symptomatic primary hyperparathyroidism: a retrospective analysis of fifty one cases from a single centre</a></div><div class="wp-workCard_item"><span>The Journal of the Association of Physicians of India</span><span>, 2008</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">Widely prevalent vitamin D deficiency and delayed diagnosis contributes to severe symptomatic pri...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">Widely prevalent vitamin D deficiency and delayed diagnosis contributes to severe symptomatic primary hyperparathyroidism in India. We analysed fifty one consecutive patients of primary hyperparathyroidism managed at our centre. All of them were symptomatic. Retrospective analysis. Fifty one consecutive cases of symptomatic primary hyperparathyroidism, presenting to our centre from January 1994 to May 2007 were retrospectively analyzed. Clinical presentation, biochemical, radiological and details of underlying parathyroid lesion were noted. A total of 51 cases of primary hyperparathyroidism were studied. Mean age was 39.5 +/- 11.5 yrs (Range 13 to 70 years, Female: Male 2:1). Mean duration of symptoms was 35.8 + 29.1 months. Bone pains and painful proximal myopathy were the commonest presentation (24/51), followed by pathological fractures in 12 cases. Distal Renal tubular acidosis was diagnosed in 4 cases, 3 of whom normalized after surgery. At initial evaluation, twenty one patien...</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><a id="dd40fcceab0853742140527c1cf63687" class="wp-workCard--action" rel="nofollow" data-click-track="profile-work-strip-download" data-download="{"attachment_id":108758525,"asset_id":109496784,"asset_type":"Work","button_location":"profile"}" href="https://www.academia.edu/attachments/108758525/download_file?st=MTczMjM4MDE2Nyw4LjIyMi4yMDguMTQ2&s=profile"><span><i class="fa fa-arrow-down"></i></span><span>Download</span></a><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109496784"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109496784"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109496784; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=109496784]").text(description); $(".js-view-count[data-work-id=109496784]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 109496784; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='109496784']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 109496784, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (true){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "dd40fcceab0853742140527c1cf63687" } } $('.js-work-strip[data-work-id=109496784]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":109496784,"title":"Symptomatic primary hyperparathyroidism: a retrospective analysis of fifty one cases from a single centre","translated_title":"","metadata":{"abstract":"Widely prevalent vitamin D deficiency and delayed diagnosis contributes to severe symptomatic primary hyperparathyroidism in India. 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The associations vary from absence of testis alone to high anorectal anomalies in other patients. We present two interesting patients with URA, encountered recently. Our first case was diagnosed with URA at the age of 11 years, which was detected on sonography, when he presented with pain abdomen. The presence of an epididymal cyst masked the absence of ipsilateral testes leading to delay in the diagnosis. Our second case was diagnosed with URA during the neonatal period when he presented with anorectal agenesis. He underwent abdomino-anal pull-through operation and later clinical course was complicated by recurrent cystitis, secondary vesicoureteral reflux and hydroureteronephrosis of solitary kidney, progressing to chronic kidney disease.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="70976560"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="70976560"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 70976560; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=70976560]").text(description); $(".js-view-count[data-work-id=70976560]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 70976560; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='70976560']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 70976560, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=70976560]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":70976560,"title":"The right sided syndrome, congenital absence of kidney and testis","translated_title":"","metadata":{"abstract":"Unilateral renal agenesis (URA) is a developmental defect associated with ano-malies of the genitourinary system. The associations vary from absence of testis alone to high anorectal anomalies in other patients. We present two interesting patients with URA, encountered recently. Our first case was diagnosed with URA at the age of 11 years, which was detected on sonography, when he presented with pain abdomen. The presence of an epididymal cyst masked the absence of ipsilateral testes leading to delay in the diagnosis. Our second case was diagnosed with URA during the neonatal period when he presented with anorectal agenesis. 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$(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="70976558"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" rel="nofollow" href="https://www.academia.edu/70976558/Uremic_toxins_oxidative_stress_and_inflammation_in_chronic_kidney_disease"><img alt="Research paper thumbnail of Uremic toxins, oxidative stress, and inflammation in chronic kidney disease" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" rel="nofollow" href="https://www.academia.edu/70976558/Uremic_toxins_oxidative_stress_and_inflammation_in_chronic_kidney_disease">Uremic toxins, oxidative stress, and inflammation in chronic kidney disease</a></div><div class="wp-workCard_item"><span>Journal of Renal Nutrition and Metabolism</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">Chronic kidney disease (CKD) is a state of exaggerated oxidative stress and chronic inflammation....</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">Chronic kidney disease (CKD) is a state of exaggerated oxidative stress and chronic inflammation. The syndrome of CKD culminates in three spheres of dysfunction namely: accumulation of uremic toxins, oxidative stress and progressive systemic inflammation. It is postulated that uremic as well as non-uremic solutes may play an important role in worsening oxidative stress and activating many other inimical pathways which results in endothelial dysfunction and cytokine driven inflammatory process. 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The syndrome of CKD culminates in three spheres of dysfunction namely: accumulation of uremic toxins, oxidative stress and progressive systemic inflammation. It is postulated that uremic as well as non-uremic solutes may play an important role in worsening oxidative stress and activating many other inimical pathways which results in endothelial dysfunction and cytokine driven inflammatory process. This article briefly touches upon biomarkers and treatment strategies targeting uremia, oxidative stress and inflammation.","publisher":"Medknow","publication_name":"Journal of Renal Nutrition and Metabolism"},"translated_abstract":"Chronic kidney disease (CKD) is a state of exaggerated oxidative stress and chronic inflammation. The syndrome of CKD culminates in three spheres of dysfunction namely: accumulation of uremic toxins, oxidative stress and progressive systemic inflammation. 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$(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="70976552"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/70976552/Transverse_myelitis_in_antiphospholipid_antibody_negative_systemic_lupus_erythematosus"><img alt="Research paper thumbnail of Transverse myelitis in antiphospholipid antibody negative systemic lupus erythematosus" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/70976552/Transverse_myelitis_in_antiphospholipid_antibody_negative_systemic_lupus_erythematosus">Transverse myelitis in antiphospholipid antibody negative systemic lupus erythematosus</a></div><div class="wp-workCard_item"><span>Journal of the Indian Medical Association</span><span>, 2013</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">Acute transverse myelitis is a well known neurological complication occurring in systemic lupus e...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">Acute transverse myelitis is a well known neurological complication occurring in systemic lupus erythematosus. Many prior studies have shown a link between transverse myelitis and the presence of antiphospholipid antibodies. Earlier theories have linked thrombotic tendency to be the culprit in such manifestations but currently there is evidence to support other causative mechanisms. A case of a young female diagnosed as systemic lupus erythematosus has been reported who presented with acute transverse myelitis and was found to be seronegative for antiphospholipid antibody. It is important to pay heed to and accordingly treat complications like acute transverse myelitis that occur regardless of antiphospholipid antibody positivity in a systemic lupus erythematosus setting.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="70976552"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="70976552"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 70976552; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=70976552]").text(description); $(".js-view-count[data-work-id=70976552]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 70976552; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='70976552']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 70976552, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=70976552]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":70976552,"title":"Transverse myelitis in antiphospholipid antibody negative systemic lupus erythematosus","translated_title":"","metadata":{"abstract":"Acute transverse myelitis is a well known neurological complication occurring in systemic lupus erythematosus. Many prior studies have shown a link between transverse myelitis and the presence of antiphospholipid antibodies. Earlier theories have linked thrombotic tendency to be the culprit in such manifestations but currently there is evidence to support other causative mechanisms. A case of a young female diagnosed as systemic lupus erythematosus has been reported who presented with acute transverse myelitis and was found to be seronegative for antiphospholipid antibody. It is important to pay heed to and accordingly treat complications like acute transverse myelitis that occur regardless of antiphospholipid antibody positivity in a systemic lupus erythematosus setting.","publication_date":{"day":null,"month":null,"year":2013,"errors":{}},"publication_name":"Journal of the Indian Medical Association"},"translated_abstract":"Acute transverse myelitis is a well known neurological complication occurring in systemic lupus erythematosus. Many prior studies have shown a link between transverse myelitis and the presence of antiphospholipid antibodies. Earlier theories have linked thrombotic tendency to be the culprit in such manifestations but currently there is evidence to support other causative mechanisms. A case of a young female diagnosed as systemic lupus erythematosus has been reported who presented with acute transverse myelitis and was found to be seronegative for antiphospholipid antibody. It is important to pay heed to and accordingly treat complications like acute transverse myelitis that occur regardless of antiphospholipid antibody positivity in a systemic lupus erythematosus setting.","internal_url":"https://www.academia.edu/70976552/Transverse_myelitis_in_antiphospholipid_antibody_negative_systemic_lupus_erythematosus","translated_internal_url":"","created_at":"2022-02-08T21:20:43.476-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":5615739,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[],"downloadable_attachments":[],"slug":"Transverse_myelitis_in_antiphospholipid_antibody_negative_systemic_lupus_erythematosus","translated_slug":"","page_count":null,"language":"en","content_type":"Work","owner":{"id":5615739,"first_name":"ratan","middle_initials":null,"last_name":"jha","page_name":"ratanjha","domain_name":"independent","created_at":"2013-09-18T01:17:38.903-07:00","display_name":"ratan jha","url":"https://independent.academia.edu/ratanjha"},"attachments":[],"research_interests":[{"id":26327,"name":"Medicine","url":"https://www.academia.edu/Documents/in/Medicine"},{"id":64568,"name":"Humans","url":"https://www.academia.edu/Documents/in/Humans"},{"id":66614,"name":"Systemic Lupus Erythematosus","url":"https://www.academia.edu/Documents/in/Systemic_Lupus_Erythematosus"},{"id":98925,"name":"Female","url":"https://www.academia.edu/Documents/in/Female"},{"id":227337,"name":"Antiphospholipid antibodies","url":"https://www.academia.edu/Documents/in/Antiphospholipid_antibodies"},{"id":382075,"name":"Adult","url":"https://www.academia.edu/Documents/in/Adult"}],"urls":[]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> </div><div class="profile--tab_content_container js-tab-pane tab-pane" data-section-id="746521" id="papers"><div class="js-work-strip profile--work_container" data-work-id="114873059"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/114873059/Visual_Vignette"><img alt="Research paper thumbnail of Visual Vignette" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/114873059/Visual_Vignette">Visual Vignette</a></div><div class="wp-workCard_item"><span>Endocrine Practice</span><span>, 2010</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">Case Presentation: A 19-year-old white man with uncontrolled type 1 diabetes mellitus (hemoglobin...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">Case Presentation: A 19-year-old white man with uncontrolled type 1 diabetes mellitus (hemoglobin A1c, 12.7%) was admitted to the intensive care unit and treated for diabetic ketoacidosis. He underwent computed tomography of the abdomen and pelvis because of persistent abdominal pain. Computed tomography revealed hepatomegaly with liver span of 15 cm (Fig. 1). The patient had intermittent transaminitis over the past year with negative workup for autoimmune and infectious etiologies. He had been diagnosed with nonalcoholic steatohepatitis. He reported no alcohol or drug use; acetaminophen level was undetectable. Coagulation studies and total protein and albumin were normal. His hepatic enzyme concentrations had risen from before hospitalization: aspartate aminotransferase, from 44 IU/L to 262 IU/L; alanine aminotransferase, from 125 IU/L to 519 IU/L; alkaline phosphatase, from 113 IU/L to 161 IU/L. There were no elevations in bilirubin or creatinine kinase, nor was there a decrease in renal function. By the day of discharge, his hepatic transaminases had trended back to baseline. Ultrasonography-guided liver biopsy was performed, and diffusely pale-staining hepatocytes with excessive glycogen accumulation on periodic acid–Schiff stain were seen (Fig. 2). Glycogenated nuclei were scattered throughout the parenchyma, and rare macrovesicular fat droplets were present. 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He underwent computed tomography of the abdomen and pelvis because of persistent abdominal pain. Computed tomography revealed hepatomegaly with liver span of 15 cm (Fig. 1). The patient had intermittent transaminitis over the past year with negative workup for autoimmune and infectious etiologies. He had been diagnosed with nonalcoholic steatohepatitis. He reported no alcohol or drug use; acetaminophen level was undetectable. Coagulation studies and total protein and albumin were normal. His hepatic enzyme concentrations had risen from before hospitalization: aspartate aminotransferase, from 44 IU/L to 262 IU/L; alanine aminotransferase, from 125 IU/L to 519 IU/L; alkaline phosphatase, from 113 IU/L to 161 IU/L. There were no elevations in bilirubin or creatinine kinase, nor was there a decrease in renal function. By the day of discharge, his hepatic transaminases had trended back to baseline. Ultrasonography-guided liver biopsy was performed, and diffusely pale-staining hepatocytes with excessive glycogen accumulation on periodic acid–Schiff stain were seen (Fig. 2). Glycogenated nuclei were scattered throughout the parenchyma, and rare macrovesicular fat droplets were present. What is the diagnosis?","publisher":"AACE Corp (American Association of Clinical Endocrinologists)","publication_date":{"day":null,"month":null,"year":2010,"errors":{}},"publication_name":"Endocrine Practice"},"translated_abstract":"Case Presentation: A 19-year-old white man with uncontrolled type 1 diabetes mellitus (hemoglobin A1c, 12.7%) was admitted to the intensive care unit and treated for diabetic ketoacidosis. He underwent computed tomography of the abdomen and pelvis because of persistent abdominal pain. Computed tomography revealed hepatomegaly with liver span of 15 cm (Fig. 1). The patient had intermittent transaminitis over the past year with negative workup for autoimmune and infectious etiologies. He had been diagnosed with nonalcoholic steatohepatitis. He reported no alcohol or drug use; acetaminophen level was undetectable. Coagulation studies and total protein and albumin were normal. His hepatic enzyme concentrations had risen from before hospitalization: aspartate aminotransferase, from 44 IU/L to 262 IU/L; alanine aminotransferase, from 125 IU/L to 519 IU/L; alkaline phosphatase, from 113 IU/L to 161 IU/L. There were no elevations in bilirubin or creatinine kinase, nor was there a decrease in renal function. By the day of discharge, his hepatic transaminases had trended back to baseline. Ultrasonography-guided liver biopsy was performed, and diffusely pale-staining hepatocytes with excessive glycogen accumulation on periodic acid–Schiff stain were seen (Fig. 2). 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$(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="109496805"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" rel="nofollow" href="https://www.academia.edu/109496805/Glycemic_control_in_patients_of_chronic_kidney_disease"><img alt="Research paper thumbnail of Glycemic control in patients of chronic kidney disease" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" rel="nofollow" href="https://www.academia.edu/109496805/Glycemic_control_in_patients_of_chronic_kidney_disease">Glycemic control in patients of chronic kidney disease</a></div><div class="wp-workCard_item"><span>International Journal of Diabetes in Developing Countries</span><span>, 2007</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">ABSTRACT</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109496805"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109496805"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109496805; 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$(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="109496804"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" rel="nofollow" href="https://www.academia.edu/109496804/Pregnancy_predisposes_to_rhabdomyolysis_due_to_hypokalemia"><img alt="Research paper thumbnail of Pregnancy predisposes to rhabdomyolysis due to hypokalemia" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" rel="nofollow" href="https://www.academia.edu/109496804/Pregnancy_predisposes_to_rhabdomyolysis_due_to_hypokalemia">Pregnancy predisposes to rhabdomyolysis due to hypokalemia</a></div><div class="wp-workCard_item"><span>Saudi journal of kidney diseases and transplantation : an official publication of the Saudi Center for Organ Transplantation, Saudi Arabia</span><span>, 2010</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">Increased predilection for hypokalemia-induced rhabdomyolysis has been noted in pregnant women. W...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">Increased predilection for hypokalemia-induced rhabdomyolysis has been noted in pregnant women. We managed a woman with distal renal tubular acidosis (RTA) with persistent hypokalemia who presented with recurrent rhabdomyolysis in her consecutive pregnancies despite adequate potassium citrate therapy.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109496804"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109496804"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109496804; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=109496804]").text(description); $(".js-view-count[data-work-id=109496804]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 109496804; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='109496804']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 109496804, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=109496804]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":109496804,"title":"Pregnancy predisposes to rhabdomyolysis due to hypokalemia","translated_title":"","metadata":{"abstract":"Increased predilection for hypokalemia-induced rhabdomyolysis has been noted in pregnant women. 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Interpretation of such elevated enzymes thus warrants caution. It necessitates adequate awareness amongst clinicians, of conditions with such elevation in the absence of myocardial ischemia/infarction as well as those that harbinger false positives. We discuss one such case that posed a diagnostic dilemma and review the pertinent literature.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109496803"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109496803"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109496803; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=109496803]").text(description); $(".js-view-count[data-work-id=109496803]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 109496803; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='109496803']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 109496803, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=109496803]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":109496803,"title":"Raised cardiac enzymes in sepsis with renal failure: An encompassing umbrella or a masquerader?","translated_title":"","metadata":{"abstract":"Elevation of cardiac enzymes and troponins particularly in settings of sepsis and renal failure may cloud the diagnostic picture of acute coronary syndrome in many cases. 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href="https://www.academia.edu/109496802/Distal_Renal_Tubular_Acidosis_Due_to_Primary_Hyperparathyroidism"><img alt="Research paper thumbnail of Distal Renal Tubular Acidosis Due to Primary Hyperparathyroidism" class="work-thumbnail" src="https://attachments.academia-assets.com/107605145/thumbnails/1.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/109496802/Distal_Renal_Tubular_Acidosis_Due_to_Primary_Hyperparathyroidism">Distal Renal Tubular Acidosis Due to Primary Hyperparathyroidism</a></div><div class="wp-workCard_item"><span>Endocrine Practice</span><span>, 2008</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><a id="69f3af28c1ba7c6842cf8c63901aa1bf" class="wp-workCard--action" rel="nofollow" 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retrospective analysis of fifty one cases from a single centre" class="work-thumbnail" src="https://attachments.academia-assets.com/108758525/thumbnails/1.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/109496784/Symptomatic_primary_hyperparathyroidism_a_retrospective_analysis_of_fifty_one_cases_from_a_single_centre">Symptomatic primary hyperparathyroidism: a retrospective analysis of fifty one cases from a single centre</a></div><div class="wp-workCard_item"><span>The Journal of the Association of Physicians of India</span><span>, 2008</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">Widely prevalent vitamin D deficiency and delayed diagnosis contributes to severe symptomatic pri...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">Widely prevalent vitamin D deficiency and delayed diagnosis contributes to severe symptomatic primary hyperparathyroidism in India. We analysed fifty one consecutive patients of primary hyperparathyroidism managed at our centre. All of them were symptomatic. Retrospective analysis. Fifty one consecutive cases of symptomatic primary hyperparathyroidism, presenting to our centre from January 1994 to May 2007 were retrospectively analyzed. Clinical presentation, biochemical, radiological and details of underlying parathyroid lesion were noted. A total of 51 cases of primary hyperparathyroidism were studied. Mean age was 39.5 +/- 11.5 yrs (Range 13 to 70 years, Female: Male 2:1). Mean duration of symptoms was 35.8 + 29.1 months. Bone pains and painful proximal myopathy were the commonest presentation (24/51), followed by pathological fractures in 12 cases. Distal Renal tubular acidosis was diagnosed in 4 cases, 3 of whom normalized after surgery. At initial evaluation, twenty one patien...</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><a id="dd40fcceab0853742140527c1cf63687" class="wp-workCard--action" rel="nofollow" data-click-track="profile-work-strip-download" data-download="{"attachment_id":108758525,"asset_id":109496784,"asset_type":"Work","button_location":"profile"}" href="https://www.academia.edu/attachments/108758525/download_file?st=MTczMjM4MDE2Nyw4LjIyMi4yMDguMTQ2&st=MTczMjM4MDE2Nyw4LjIyMi4yMDguMTQ2&s=profile"><span><i class="fa fa-arrow-down"></i></span><span>Download</span></a><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109496784"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109496784"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109496784; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=109496784]").text(description); $(".js-view-count[data-work-id=109496784]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 109496784; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='109496784']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 109496784, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (true){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "dd40fcceab0853742140527c1cf63687" } } $('.js-work-strip[data-work-id=109496784]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":109496784,"title":"Symptomatic primary hyperparathyroidism: a retrospective analysis of fifty one cases from a single centre","translated_title":"","metadata":{"abstract":"Widely prevalent vitamin D deficiency and delayed diagnosis contributes to severe symptomatic primary hyperparathyroidism in India. 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Mortality due to HBV in hemodialysis: Effect of vaccination. J Nephrol 1993; 6: 98-102. 3. Th...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">... Mortality due to HBV in hemodialysis: Effect of vaccination. J Nephrol 1993; 6: 98-102. 3. Thomas PP, Kirubhakaran MG, Jacob CK, Srinivas NS, Hariharan S, John TJ, Shastry JCM. Hepatitis B infection in dialysis unit in South India. 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$(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="70976560"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/70976560/The_right_sided_syndrome_congenital_absence_of_kidney_and_testis"><img alt="Research paper thumbnail of The right sided syndrome, congenital absence of kidney and testis" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/70976560/The_right_sided_syndrome_congenital_absence_of_kidney_and_testis">The right sided syndrome, congenital absence of kidney and testis</a></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">Unilateral renal agenesis (URA) is a developmental defect associated with ano-malies of the genit...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">Unilateral renal agenesis (URA) is a developmental defect associated with ano-malies of the genitourinary system. 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$(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="70976552"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/70976552/Transverse_myelitis_in_antiphospholipid_antibody_negative_systemic_lupus_erythematosus"><img alt="Research paper thumbnail of Transverse myelitis in antiphospholipid antibody negative systemic lupus erythematosus" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/70976552/Transverse_myelitis_in_antiphospholipid_antibody_negative_systemic_lupus_erythematosus">Transverse myelitis in antiphospholipid antibody negative systemic lupus erythematosus</a></div><div class="wp-workCard_item"><span>Journal of the Indian Medical Association</span><span>, 2013</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">Acute transverse myelitis is a well known neurological complication occurring in systemic lupus e...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">Acute transverse myelitis is a well known neurological complication occurring in systemic lupus erythematosus. Many prior studies have shown a link between transverse myelitis and the presence of antiphospholipid antibodies. Earlier theories have linked thrombotic tendency to be the culprit in such manifestations but currently there is evidence to support other causative mechanisms. A case of a young female diagnosed as systemic lupus erythematosus has been reported who presented with acute transverse myelitis and was found to be seronegative for antiphospholipid antibody. 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