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class="right-panel-container"><div class="user-content-wrapper"><div class="uploads-container" id="social-redesign-work-container"><div class="upload-header"><h2 class="ds2-5-heading-sans-serif-xs">Uploads</h2></div><div class="documents-container backbone-social-profile-documents" style="width: 100%;"><div class="u-taCenter"></div><div class="profile--tab_content_container js-tab-pane tab-pane active" id="all"><div class="profile--tab_heading_container js-section-heading" data-section="Papers" id="Papers"><h3 class="profile--tab_heading_container">Papers by Mikhail MM</h3></div><div class="js-work-strip profile--work_container" data-work-id="71556174"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/71556174/Periodicity_in_the_transient_regime_of_exhaustive_polling_systems"><img alt="Research paper thumbnail of Periodicity in the transient regime of exhaustive polling systems" class="work-thumbnail" src="https://attachments.academia-assets.com/80848681/thumbnails/1.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/71556174/Periodicity_in_the_transient_regime_of_exhaustive_polling_systems">Periodicity in the transient regime of exhaustive polling systems</a></div><div class="wp-workCard_item"><span>Annals of Applied Probability 2006 Vol 16 Pp 1816 1850 Peer Reviewed Journal</span><span>, Nov 1, 2006</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">We consider an exhaustive polling system with three nodes in its transient regime under a switchi...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">We consider an exhaustive polling system with three nodes in its transient regime under a switching rule of generalized greedy type. We show that, for the system with Poisson arrivals and service times with finite second moment, the sequence of nodes visited by the server is eventually periodic almost surely. To do this, we construct a dynamical system, the triangle process, which we show has eventually periodic trajectories for almost all sets of parameters and in this case we show that the stochastic trajectories follow the deterministic ones a.s. We also show there are infinitely many sets of parameters where the triangle process has aperiodic trajectories and in such cases trajectories of the stochastic model are aperiodic with positive probability.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><a id="75022166f97bc7ab19837d2d80657d94" class="wp-workCard--action" rel="nofollow" data-click-track="profile-work-strip-download" data-download="{"attachment_id":80848681,"asset_id":71556174,"asset_type":"Work","button_location":"profile"}" href="https://www.academia.edu/attachments/80848681/download_file?st=MTczMjc0NjQwNiw4LjIyMi4yMDguMTQ2&s=profile"><span><i class="fa fa-arrow-down"></i></span><span>Download</span></a><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="71556174"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="71556174"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 71556174; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=71556174]").text(description); $(".js-view-count[data-work-id=71556174]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 71556174; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='71556174']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 71556174, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (true){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "75022166f97bc7ab19837d2d80657d94" } } $('.js-work-strip[data-work-id=71556174]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":71556174,"title":"Periodicity in the transient regime of exhaustive polling systems","translated_title":"","metadata":{"abstract":"We consider an exhaustive polling system with three nodes in its transient regime under a switching rule of generalized greedy type. 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$(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="71556173"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/71556173/A_Mixture_of_the_Exclusion_Process_and_the_Voter_Model"><img alt="Research paper thumbnail of A Mixture of the Exclusion Process and the Voter Model" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/71556173/A_Mixture_of_the_Exclusion_Process_and_the_Voter_Model">A Mixture of the Exclusion Process and the Voter Model</a></div><div class="wp-workCard_item"><span>Eprint Arxiv Math 0002051</span><span>, Feb 1, 2000</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="71556173"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="71556173"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 71556173; 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The term multiplexed means that the model can be viewed as a nearest neighbours random walk on a tree whose vertices carry an internal degree of freedom from the finite set {1,. .. , d}, for some integer d. The consequence of the internal degree of freedom is an enhancement of the tree graph structure induced by the replacement of ordinary edges by multi-edges, indexed by the set {1,. .. , d} × {1,. .. , d}. This indexing conveys the information on the internal degree of freedom of the vertices contiguous to each edge. The term random environment means that the jumping rates for the random walk are a family of edge-indexed random variables, independent of the natural filtration generated by the random variables entering in the definition of the random walk; their joint distribution depends on the index of each component of the multi-edges. We study the large time asymptotic behaviour of this random walk and classify it with respect to positive recurrence or transience in terms of a specific parameter of the probability distribution of the jump rates. This classifying parameter is shown to coincide with the critical value of a matrix-valued multiplicative cascade on the ordinary tree (i.e. the one without internal degrees of freedom attached to the vertices) having the same vertex set as the state space of the random walk. 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By using a result about majorizing dependent random fields by Bernoulli fields, we prove that if the selection parameter is less than certain critical value, then, by choosing the scaling parameter large enough, we can assure that there is no percolation in the complementary set.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="71556164"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="71556164"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 71556164; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=71556164]").text(description); $(".js-view-count[data-work-id=71556164]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 71556164; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='71556164']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 71556164, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=71556164]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":71556164,"title":"On the connectivity properties of the complementary set in fractal percolation models","translated_title":"","metadata":{"abstract":"ABSTRACT We study the connectivity properties of the complementary set in Poisson multiscale percolation model and in Mandelbrot\u0026amp;#39;s percolation model in arbitrary dimension. 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By using a result about majorizing dependent random fields by Bernoulli fields, we prove that if the selection parameter is less than certain critical value, then, by choosing the scaling parameter large enough, we can assure that there is no percolation in the complementary set.","internal_url":"https://www.academia.edu/71556164/On_the_connectivity_properties_of_the_complementary_set_in_fractal_percolation_models","translated_internal_url":"","created_at":"2022-02-14T20:59:26.293-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":214780338,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[{"id":37690792,"work_id":71556164,"tagging_user_id":214780338,"tagged_user_id":185233567,"co_author_invite_id":null,"email":"s***v@gmail.com","display_order":0,"name":"Serguei Popov","title":"On the connectivity properties of the complementary set in fractal percolation models"}],"downloadable_attachments":[],"slug":"On_the_connectivity_properties_of_the_complementary_set_in_fractal_percolation_models","translated_slug":"","page_count":null,"language":"en","content_type":"Work","owner":{"id":214780338,"first_name":"Mikhail","middle_initials":null,"last_name":"MM","page_name":"MikhailMM","domain_name":"independent","created_at":"2022-02-14T20:58:13.540-08:00","display_name":"Mikhail MM","url":"https://independent.academia.edu/MikhailMM"},"attachments":[],"research_interests":[{"id":892,"name":"Statistics","url":"https://www.academia.edu/Documents/in/Statistics"},{"id":2177640,"name":"Random Field","url":"https://www.academia.edu/Documents/in/Random_Field"},{"id":3735670,"name":"Critical value","url":"https://www.academia.edu/Documents/in/Critical_value"}],"urls":[]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="71556163"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/71556163/Participation_of_the_urokinase_type_plasminogen_activator_receptor_uPAR_in_neutrophil_transendothelial_migration"><img alt="Research paper thumbnail of Participation of the urokinase-type plasminogen activator receptor (uPAR) in neutrophil transendothelial migration" class="work-thumbnail" src="https://attachments.academia-assets.com/80848703/thumbnails/1.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/71556163/Participation_of_the_urokinase_type_plasminogen_activator_receptor_uPAR_in_neutrophil_transendothelial_migration">Participation of the urokinase-type plasminogen activator receptor (uPAR) in neutrophil transendothelial migration</a></div><div class="wp-workCard_item"><span>Molecular Immunology</span><span>, 2011</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><a id="5d126fd0336624367913000e8867f7fb" class="wp-workCard--action" rel="nofollow" data-click-track="profile-work-strip-download" data-download="{"attachment_id":80848703,"asset_id":71556163,"asset_type":"Work","button_location":"profile"}" href="https://www.academia.edu/attachments/80848703/download_file?st=MTczMjc0NjQwNiw4LjIyMi4yMDguMTQ2&s=profile"><span><i class="fa fa-arrow-down"></i></span><span>Download</span></a><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="71556163"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="71556163"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 71556163; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=71556163]").text(description); $(".js-view-count[data-work-id=71556163]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 71556163; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='71556163']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 71556163, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (true){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "5d126fd0336624367913000e8867f7fb" } } $('.js-work-strip[data-work-id=71556163]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":71556163,"title":"Participation of the urokinase-type plasminogen activator receptor (uPAR) in neutrophil transendothelial migration","translated_title":"","metadata":{"publisher":"Elsevier BV","grobid_abstract":"The mechanisms underlying migration of neutrophils across endothelium are not completely understood. The urokinase-type plasminogen activator receptor (uPAR) plays a key role in neutrophil adhesion and migration. In the present study, we addressed whether uPAR regulates neutrophil transendothelial migration. We first showed that siRNA-mediated knockdown of uPAR in human umbilical vein endothelial cells (HUVECs) did not affect neutrophil migration across HUVEC monolayers indicating that endothelial uPAR does not regulate neutrophil transmigration. In contrast, the transmigration was significantly inhibited by Fab fragment of anti-uPAR monoclonal antibody and proteolytically inactive urokinase (uPA), whereas inhibition of proteolytical activity of endogenous uPA (with amiloride or plasminogen activator inhibitor-1) did not affect the transmigration. Both the anti-uPAR Fab fragment and proteolytically inactive uPA did not exert significant effects upon the transmigration conducted in the presence of F(ab) 2 fragment of blocking antibody to integrin Mac-1 indicating that uPAR regulates Mac-1-dependent transmigration. Mac-1-dependent, but not Mac-1-independent, transmigration was significantly reduced in the presence of N-acetyl-d-glucosamine and d-mannose, the saccharides that disrupt uPAR/Mac-1 association, but was unaffected in the presence of control saccharides (d-sorbitol and sucrose). We conclude that physical association of uPAR with Mac-1 mediates the regulatory effect of uPAR over the transmigration. Finally, we provide evidence that the functional cooperation between uPAR and Mac-1 is essential at both adhesion and diapedesis steps of neutrophil migration across endothelium. Thus, uPAR expressed on neutrophil plasma membrane regulates transendothelial migration independently of uPA proteolytical activity and acting as a cofactor for integrin Mac-1.","publication_date":{"day":null,"month":null,"year":2011,"errors":{}},"publication_name":"Molecular Immunology","grobid_abstract_attachment_id":80848703},"translated_abstract":null,"internal_url":"https://www.academia.edu/71556163/Participation_of_the_urokinase_type_plasminogen_activator_receptor_uPAR_in_neutrophil_transendothelial_migration","translated_internal_url":"","created_at":"2022-02-14T20:59:26.161-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":214780338,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[{"id":37690803,"work_id":71556163,"tagging_user_id":214780338,"tagged_user_id":50901029,"co_author_invite_id":null,"email":"a***a@compmechlab.com","display_order":0,"name":"Olga Antonova","title":"Participation of the urokinase-type 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Adhesion","url":"https://www.academia.edu/Documents/in/Cell_Adhesion"},{"id":63332,"name":"Molecular Immunology","url":"https://www.academia.edu/Documents/in/Molecular_Immunology"},{"id":71510,"name":"Endothelial Cells","url":"https://www.academia.edu/Documents/in/Endothelial_Cells"},{"id":147196,"name":"Monoclonal Antibodies","url":"https://www.academia.edu/Documents/in/Monoclonal_Antibodies"},{"id":273338,"name":"IL","url":"https://www.academia.edu/Documents/in/IL"},{"id":295260,"name":"Neutrophils","url":"https://www.academia.edu/Documents/in/Neutrophils"},{"id":754378,"name":"siRNA","url":"https://www.academia.edu/Documents/in/siRNA"},{"id":766014,"name":"Monoclonal Antibody","url":"https://www.academia.edu/Documents/in/Monoclonal_Antibody"},{"id":788667,"name":"ENDOTHELIUM","url":"https://www.academia.edu/Documents/in/ENDOTHELIUM"},{"id":1242344,"name":"Plasma Membrane","url":"https://www.academia.edu/Documents/in/Plasma_Membrane"},{"id":1661072,"name":"Plasminogen Activator Inhibitor","url":"https://www.academia.edu/Documents/in/Plasminogen_Activator_Inhibitor"},{"id":2412024,"name":"Proteolytic activity","url":"https://www.academia.edu/Documents/in/Proteolytic_activity"},{"id":2507489,"name":"Monosaccharides","url":"https://www.academia.edu/Documents/in/Monosaccharides"}],"urls":[]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="71556162"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/71556162/On_Range_and_Local_Time_of_Many_dimensional_Submartingales"><img alt="Research paper thumbnail of On Range and Local Time of Many-dimensional Submartingales" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard 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health systems and services research","url":"https://www.academia.edu/Documents/in/Public_health_systems_and_services_research-1"},{"id":546356,"name":"Clinical Study","url":"https://www.academia.edu/Documents/in/Clinical_Study"},{"id":3789879,"name":"Cardiovascular medicine and haematology","url":"https://www.academia.edu/Documents/in/Cardiovascular_medicine_and_haematology"}],"urls":[]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="71556160"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/71556160/Combination_Ethacizin_and_Ethmozin_Treatment_of_Resistant_Ventricular_Ectopy"><img alt="Research paper thumbnail of Combination Ethacizin and Ethmozin Treatment of Resistant Ventricular Ectopy" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/71556160/Combination_Ethacizin_and_Ethmozin_Treatment_of_Resistant_Ventricular_Ectopy">Combination Ethacizin and Ethmozin Treatment of Resistant Ventricular Ectopy</a></div><div class="wp-workCard_item"><span>Journal of Cardiovascular Pharmacology</span><span>, 1994</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">Ethmozin (Moricizine HCl) and ethacizin are two class I antiarrhythmic drugs with different rate ...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">Ethmozin (Moricizine HCl) and ethacizin are two class I antiarrhythmic drugs with different rate constants of interaction with the sodium channel. Computer simulation using the &amp;quot;guarded-receptor&amp;quot; model predicted that the combination of ethacizin and ethmozin should exert a greater decrease in excitability and conduction at short coupling intervals, but little effect at normal heart rates (HR). To test this prediction, we measured intraventricular conduction delay in canine hearts in vivo. In agreement with the model, the combination more potently prolonged the delay only at intervals &amp;lt; 600 ms as compared with ethacizin alone. Combination therapy was tested in 6 patients with idiopathic ventricular ectopic depolarizations (VEDs). Three patients were resistant to either ethmozin or ethacizin monotherapy, and three could not tolerate effective doses because of side effects. Quantitative continuous ECG monitoring showed that total VEDs in the resistant group decreased 0 and 17 +/- 13% for 400 and 800 mg/day ethmozin and 18 +/- 12 and 55 +/- 12% for 100 and 200 mg/day ethacizin, respectively. Combined therapy with ethmozin (400 mg/day) and ethacizin (100 mg/day) reduced the number of VEDs by 78 +/- 2% in these patients without side effects. In the &amp;quot;nonresistant&amp;quot; but intolerant group of patients, use of the combination allowed relief of symptomatic ectopy without side effects. A theoretical model correctly predicted an effective combination of class I antiarrhythmic drugs, one with &amp;quot;fast-off&amp;quot; and one with &amp;quot;slow-off&amp;quot; kinetics, which may provide a general rationale for choosing drug combinations.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="71556160"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="71556160"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 71556160; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=71556160]").text(description); $(".js-view-count[data-work-id=71556160]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 71556160; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='71556160']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 71556160, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=71556160]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":71556160,"title":"Combination Ethacizin and Ethmozin Treatment of Resistant Ventricular Ectopy","translated_title":"","metadata":{"abstract":"Ethmozin (Moricizine HCl) and ethacizin are two class I antiarrhythmic drugs with different rate constants of interaction with the sodium channel. Computer simulation using the \u0026amp;quot;guarded-receptor\u0026amp;quot; model predicted that the combination of ethacizin and ethmozin should exert a greater decrease in excitability and conduction at short coupling intervals, but little effect at normal heart rates (HR). To test this prediction, we measured intraventricular conduction delay in canine hearts in vivo. In agreement with the model, the combination more potently prolonged the delay only at intervals \u0026amp;lt; 600 ms as compared with ethacizin alone. Combination therapy was tested in 6 patients with idiopathic ventricular ectopic depolarizations (VEDs). Three patients were resistant to either ethmozin or ethacizin monotherapy, and three could not tolerate effective doses because of side effects. Quantitative continuous ECG monitoring showed that total VEDs in the resistant group decreased 0 and 17 +/- 13% for 400 and 800 mg/day ethmozin and 18 +/- 12 and 55 +/- 12% for 100 and 200 mg/day ethacizin, respectively. Combined therapy with ethmozin (400 mg/day) and ethacizin (100 mg/day) reduced the number of VEDs by 78 +/- 2% in these patients without side effects. In the \u0026amp;quot;nonresistant\u0026amp;quot; but intolerant group of patients, use of the combination allowed relief of symptomatic ectopy without side effects. A theoretical model correctly predicted an effective combination of class I antiarrhythmic drugs, one with \u0026amp;quot;fast-off\u0026amp;quot; and one with \u0026amp;quot;slow-off\u0026amp;quot; kinetics, which may provide a general rationale for choosing drug combinations.","publisher":"Ovid Technologies (Wolters Kluwer Health)","publication_date":{"day":null,"month":null,"year":1994,"errors":{}},"publication_name":"Journal of Cardiovascular Pharmacology"},"translated_abstract":"Ethmozin (Moricizine HCl) and ethacizin are two class I antiarrhythmic drugs with different rate constants of interaction with the sodium channel. Computer simulation using the \u0026amp;quot;guarded-receptor\u0026amp;quot; model predicted that the combination of ethacizin and ethmozin should exert a greater decrease in excitability and conduction at short coupling intervals, but little effect at normal heart rates (HR). To test this prediction, we measured intraventricular conduction delay in canine hearts in vivo. In agreement with the model, the combination more potently prolonged the delay only at intervals \u0026amp;lt; 600 ms as compared with ethacizin alone. Combination therapy was tested in 6 patients with idiopathic ventricular ectopic depolarizations (VEDs). Three patients were resistant to either ethmozin or ethacizin monotherapy, and three could not tolerate effective doses because of side effects. Quantitative continuous ECG monitoring showed that total VEDs in the resistant group decreased 0 and 17 +/- 13% for 400 and 800 mg/day ethmozin and 18 +/- 12 and 55 +/- 12% for 100 and 200 mg/day ethacizin, respectively. Combined therapy with ethmozin (400 mg/day) and ethacizin (100 mg/day) reduced the number of VEDs by 78 +/- 2% in these patients without side effects. In the \u0026amp;quot;nonresistant\u0026amp;quot; but intolerant group of patients, use of the combination allowed relief of symptomatic ectopy without side effects. A theoretical model correctly predicted an effective combination of class I antiarrhythmic drugs, one with \u0026amp;quot;fast-off\u0026amp;quot; and one with \u0026amp;quot;slow-off\u0026amp;quot; kinetics, which may provide a general rationale for choosing drug combinations.","internal_url":"https://www.academia.edu/71556160/Combination_Ethacizin_and_Ethmozin_Treatment_of_Resistant_Ventricular_Ectopy","translated_internal_url":"","created_at":"2022-02-14T20:59:25.731-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":214780338,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[{"id":37690782,"work_id":71556160,"tagging_user_id":214780338,"tagged_user_id":484537,"co_author_invite_id":null,"email":"f***r@gmail.com","display_order":0,"name":"Frank Starmer","title":"Combination Ethacizin and Ethmozin Treatment of Resistant Ventricular Ectopy"}],"downloadable_attachments":[],"slug":"Combination_Ethacizin_and_Ethmozin_Treatment_of_Resistant_Ventricular_Ectopy","translated_slug":"","page_count":null,"language":"en","content_type":"Work","owner":{"id":214780338,"first_name":"Mikhail","middle_initials":null,"last_name":"MM","page_name":"MikhailMM","domain_name":"independent","created_at":"2022-02-14T20:58:13.540-08:00","display_name":"Mikhail MM","url":"https://independent.academia.edu/MikhailMM"},"attachments":[],"research_interests":[{"id":26327,"name":"Medicine","url":"https://www.academia.edu/Documents/in/Medicine"},{"id":39000,"name":"Electrocardiography","url":"https://www.academia.edu/Documents/in/Electrocardiography"},{"id":52438,"name":"Dogs","url":"https://www.academia.edu/Documents/in/Dogs"},{"id":69542,"name":"Computer Simulation","url":"https://www.academia.edu/Documents/in/Computer_Simulation"},{"id":89987,"name":"Cardiovascular Pharmacology","url":"https://www.academia.edu/Documents/in/Cardiovascular_Pharmacology"},{"id":131298,"name":"Heart rate","url":"https://www.academia.edu/Documents/in/Heart_rate"},{"id":323090,"name":"Cardiac Arrhythmias","url":"https://www.academia.edu/Documents/in/Cardiac_Arrhythmias"},{"id":546356,"name":"Clinical Study","url":"https://www.academia.edu/Documents/in/Clinical_Study"},{"id":572282,"name":"Combination drug therapy","url":"https://www.academia.edu/Documents/in/Combination_drug_therapy"},{"id":1154249,"name":"Heart Ventricles","url":"https://www.academia.edu/Documents/in/Heart_Ventricles"},{"id":2572082,"name":"sodium channels","url":"https://www.academia.edu/Documents/in/sodium_channels"},{"id":3789879,"name":"Cardiovascular medicine and haematology","url":"https://www.academia.edu/Documents/in/Cardiovascular_medicine_and_haematology"},{"id":3789884,"name":"Pharmacology and pharmaceutical sciences","url":"https://www.academia.edu/Documents/in/Pharmacology_and_pharmaceutical_sciences"},{"id":4053335,"name":"Electric stimulation","url":"https://www.academia.edu/Documents/in/Electric_stimulation"}],"urls":[]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="71556159"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/71556159/The_hypertensive_heart_Pathogenesis_variants_and_prognostic_value"><img alt="Research paper thumbnail of The hypertensive heart: Pathogenesis, variants, and prognostic value" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/71556159/The_hypertensive_heart_Pathogenesis_variants_and_prognostic_value">The hypertensive heart: Pathogenesis, variants, and prognostic value</a></div><div class="wp-workCard_item"><span>Health Psychology</span><span>, 1988</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">We examine the heart&amp;#39;s involvement in arterial hypertension, reporting on several studies...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">We examine the heart&amp;#39;s involvement in arterial hypertension, reporting on several studies of hypertensive patients showing that left ventricular myocardial mass is a significant prognostic indicator of essential hypertension and that left ventricular hypertrophy (LVH) correlates with an induced increase in Ca2+ concentration in platelets. We also consider the LVH variant of asymmetric hypertrophy and the role of the hypertensive heart in coronary insufficiency, and we speculate on the significance of the degree of LVH and asymmetric hypertrophy as risk factors for predicting cardiac complications of essential hypertension.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="71556159"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="71556159"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 71556159; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=71556159]").text(description); $(".js-view-count[data-work-id=71556159]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 71556159; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='71556159']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 71556159, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=71556159]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":71556159,"title":"The hypertensive heart: Pathogenesis, variants, and prognostic value","translated_title":"","metadata":{"abstract":"We examine the heart\u0026amp;#39;s involvement in arterial hypertension, reporting on several studies of hypertensive patients showing that left ventricular myocardial mass is a significant prognostic indicator of essential hypertension and that left ventricular hypertrophy (LVH) correlates with an induced increase in Ca2+ concentration in platelets. We also consider the LVH variant of asymmetric hypertrophy and the role of the hypertensive heart in coronary insufficiency, and we speculate on the significance of the degree of LVH and asymmetric hypertrophy as risk factors for predicting cardiac complications of essential hypertension.","publisher":"American Psychological Association (APA)","publication_date":{"day":null,"month":null,"year":1988,"errors":{}},"publication_name":"Health Psychology"},"translated_abstract":"We examine the heart\u0026amp;#39;s involvement in arterial hypertension, reporting on several studies of hypertensive patients showing that left ventricular myocardial mass is a significant prognostic indicator of essential hypertension and that left ventricular hypertrophy (LVH) correlates with an induced increase in Ca2+ concentration in platelets. We also consider the LVH variant of asymmetric hypertrophy and the role of the hypertensive heart in coronary insufficiency, and we speculate on the significance of the degree of LVH and asymmetric hypertrophy as risk factors for predicting cardiac complications of essential hypertension.","internal_url":"https://www.academia.edu/71556159/The_hypertensive_heart_Pathogenesis_variants_and_prognostic_value","translated_internal_url":"","created_at":"2022-02-14T20:59:25.574-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":214780338,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[{"id":37690780,"work_id":71556159,"tagging_user_id":214780338,"tagged_user_id":134172687,"co_author_invite_id":null,"email":"a***a@yandex.ru","display_order":0,"name":"anna nesterova","title":"The hypertensive heart: Pathogenesis, variants, and prognostic value"}],"downloadable_attachments":[],"slug":"The_hypertensive_heart_Pathogenesis_variants_and_prognostic_value","translated_slug":"","page_count":null,"language":"en","content_type":"Work","owner":{"id":214780338,"first_name":"Mikhail","middle_initials":null,"last_name":"MM","page_name":"MikhailMM","domain_name":"independent","created_at":"2022-02-14T20:58:13.540-08:00","display_name":"Mikhail MM","url":"https://independent.academia.edu/MikhailMM"},"attachments":[],"research_interests":[{"id":260,"name":"Health Psychology","url":"https://www.academia.edu/Documents/in/Health_Psychology"},{"id":922,"name":"Education","url":"https://www.academia.edu/Documents/in/Education"},{"id":9534,"name":"Calcium","url":"https://www.academia.edu/Documents/in/Calcium"},{"id":14406,"name":"Health","url":"https://www.academia.edu/Documents/in/Health"},{"id":22997,"name":"Arterial hypertension","url":"https://www.academia.edu/Documents/in/Arterial_hypertension"},{"id":71399,"name":"Hypertension","url":"https://www.academia.edu/Documents/in/Hypertension"},{"id":74347,"name":"Hemodynamics","url":"https://www.academia.edu/Documents/in/Hemodynamics"},{"id":192721,"name":"Risk factors","url":"https://www.academia.edu/Documents/in/Risk_factors"},{"id":489727,"name":"Prognosis","url":"https://www.academia.edu/Documents/in/Prognosis"},{"id":515631,"name":"Left ventricular hypertrophy","url":"https://www.academia.edu/Documents/in/Left_ventricular_hypertrophy"},{"id":618800,"name":"Coronary Circulation","url":"https://www.academia.edu/Documents/in/Coronary_Circulation"},{"id":620049,"name":"Risk Factors","url":"https://www.academia.edu/Documents/in/Risk_Factors-1"},{"id":2515205,"name":"Essential Hypertension","url":"https://www.academia.edu/Documents/in/Essential_Hypertension"},{"id":2788426,"name":"Cardiomegaly","url":"https://www.academia.edu/Documents/in/Cardiomegaly"},{"id":2922956,"name":"Psychology and Cognitive Sciences","url":"https://www.academia.edu/Documents/in/Psychology_and_Cognitive_Sciences"},{"id":3187114,"name":"Blood platelets","url":"https://www.academia.edu/Documents/in/Blood_platelets"},{"id":3501173,"name":"Left Ventricular","url":"https://www.academia.edu/Documents/in/Left_Ventricular"},{"id":3763225,"name":"Medical and Health Sciences","url":"https://www.academia.edu/Documents/in/Medical_and_Health_Sciences"}],"urls":[]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="31783603"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/31783603/Urokinase_stimulates_inflammatory_response_in_damaged_vascular_wall_during_in_vivo_arterial_remodeling"><img alt="Research paper thumbnail of Urokinase stimulates inflammatory response in damaged vascular wall during in vivo arterial remodeling" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/31783603/Urokinase_stimulates_inflammatory_response_in_damaged_vascular_wall_during_in_vivo_arterial_remodeling">Urokinase stimulates inflammatory response in damaged vascular wall during in vivo arterial remodeling</a></div><div class="wp-workCard_item wp-workCard--coauthors"><span>by </span><span><a class="" data-click-track="profile-work-strip-authors" href="https://independent.academia.edu/MikhailMM">Mikhail MM</a>, <a class="" data-click-track="profile-work-strip-authors" href="https://independent.academia.edu/YelenaParfyonova">Yelena Parfyonova</a>, <a class="" data-click-track="profile-work-strip-authors" href="https://independent.academia.edu/ElizavetaRatner">Elizaveta Ratner</a>, and <a class="" data-click-track="profile-work-strip-authors" href="https://independent.academia.edu/OlgaPlekhanova">Olga Plekhanova</a></span></div><div class="wp-workCard_item"><span>Bulletin of Experimental Biology and Medicine</span><span>, 2008</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">Perivascular application of urokinase to the ballooned artery promoted the growth of neointima an...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">Perivascular application of urokinase to the ballooned artery promoted the growth of neointima and constrictive remodeling of the vessel and stimulated the inflammatory response in the damaged vascular wall in vivo. Recombinant tissue plasminogen activator did not induce these changes. Our results indicate that urokinase is involved in the regulation of the inflammatory response during in vivo remodeling of the damaged vascular wall.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="31783603"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="31783603"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 31783603; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=31783603]").text(description); $(".js-view-count[data-work-id=31783603]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 31783603; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='31783603']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 31783603, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=31783603]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":31783603,"title":"Urokinase stimulates inflammatory response in damaged vascular wall during in vivo arterial remodeling","translated_title":"","metadata":{"abstract":"Perivascular application of urokinase to the ballooned artery promoted the growth of neointima and constrictive remodeling of the vessel and stimulated the inflammatory response in the damaged vascular wall in vivo. 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Our results indicate that urokinase is involved in the regulation of the inflammatory response during in vivo remodeling of the damaged vascular wall.","internal_url":"https://www.academia.edu/31783603/Urokinase_stimulates_inflammatory_response_in_damaged_vascular_wall_during_in_vivo_arterial_remodeling","translated_internal_url":"","created_at":"2017-03-09T01:59:08.549-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":61244807,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[{"id":27950754,"work_id":31783603,"tagging_user_id":61244807,"tagged_user_id":214780338,"co_author_invite_id":6109124,"email":"m***f@gmail.com","display_order":0,"name":"Mikhail MM","title":"Urokinase stimulates inflammatory response in damaged vascular wall during in vivo arterial remodeling"},{"id":27950821,"work_id":31783603,"tagging_user_id":61244807,"tagged_user_id":61313753,"co_author_invite_id":6109162,"email":"e***r@gmail.com","display_order":4194304,"name":"Elizaveta Ratner","title":"Urokinase stimulates inflammatory response in damaged vascular wall during in vivo arterial remodeling"},{"id":27950843,"work_id":31783603,"tagging_user_id":61244807,"tagged_user_id":61509972,"co_author_invite_id":6106932,"email":"p***a@mail.ru","display_order":6291456,"name":"Olga Plekhanova","title":"Urokinase stimulates inflammatory response in damaged vascular wall during in vivo arterial remodeling"},{"id":27982071,"work_id":31783603,"tagging_user_id":61244807,"tagged_user_id":36516936,"co_author_invite_id":null,"email":"t***k@fbm.msu.ru","display_order":7340032,"name":"Vsevolod Tkachuk","title":"Urokinase stimulates inflammatory response in damaged vascular wall during in vivo arterial remodeling"}],"downloadable_attachments":[],"slug":"Urokinase_stimulates_inflammatory_response_in_damaged_vascular_wall_during_in_vivo_arterial_remodeling","translated_slug":"","page_count":null,"language":"en","content_type":"Work","owner":{"id":61244807,"first_name":"Yelena","middle_initials":"","last_name":"Parfyonova","page_name":"YelenaParfyonova","domain_name":"independent","created_at":"2017-03-09T01:58:50.424-08:00","display_name":"Yelena Parfyonova","url":"https://independent.academia.edu/YelenaParfyonova"},"attachments":[],"research_interests":[{"id":9334,"name":"Inflammation","url":"https://www.academia.edu/Documents/in/Inflammation"},{"id":375054,"name":"Rats","url":"https://www.academia.edu/Documents/in/Rats"},{"id":564879,"name":"Wistar Rats","url":"https://www.academia.edu/Documents/in/Wistar_Rats"},{"id":2454817,"name":"Inflammatory response","url":"https://www.academia.edu/Documents/in/Inflammatory_response"},{"id":2562018,"name":"Arteries","url":"https://www.academia.edu/Documents/in/Arteries"}],"urls":[]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="71556158"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/71556158/On_a_multiscale_continuous_percolation_model_with_unbounded_defects"><img alt="Research paper thumbnail of On a multiscale continuous percolation model with unbounded defects" class="work-thumbnail" src="https://attachments.academia-assets.com/80848705/thumbnails/1.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/71556158/On_a_multiscale_continuous_percolation_model_with_unbounded_defects">On a multiscale continuous percolation model with unbounded defects</a></div><div class="wp-workCard_item"><span>Bulletin of the Brazilian Mathematical Society</span><span>, 2003</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><a id="bc2f4cbc86629fb78251c1b2c70b2051" class="wp-workCard--action" rel="nofollow" data-click-track="profile-work-strip-download" data-download="{"attachment_id":80848705,"asset_id":71556158,"asset_type":"Work","button_location":"profile"}" href="https://www.academia.edu/attachments/80848705/download_file?st=MTczMjc0NjQwNyw4LjIyMi4yMDguMTQ2&s=profile"><span><i class="fa fa-arrow-down"></i></span><span>Download</span></a><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="71556158"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="71556158"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 71556158; 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The term multiplexed means that the model can be viewed as a nearest neighbours random walk on a tree whose vertices carry an internal degree of freedom from the finite set {1,. .. , d}, for some integer d. The consequence of the internal degree of freedom is an enhancement of the tree graph structure induced by the replacement of ordinary edges by multi-edges, indexed by the set {1,. .. , d} × {1,. .. , d}. This indexing conveys the information on the internal degree of freedom of the vertices contiguous to each edge. The term random environment means that the jumping rates for the random walk are a family of edge-indexed random variables, independent of the natural filtration generated by the random variables entering in the definition of the random walk; their joint distribution depends on the index of each component of the multi-edges. We study the large time asymptotic behaviour of this random walk and classify it with respect to positive recurrence or transience in terms of a specific parameter of the probability distribution of the jump rates. This classifying parameter is shown to coincide with the critical value of a matrix-valued multiplicative cascade on the ordinary tree (i.e. the one without internal degrees of freedom attached to the vertices) having the same vertex set as the state space of the random walk. 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$(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> </div><div class="profile--tab_content_container js-tab-pane tab-pane" data-section-id="14024609" id="papers"><div class="js-work-strip profile--work_container" data-work-id="71556174"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/71556174/Periodicity_in_the_transient_regime_of_exhaustive_polling_systems"><img alt="Research paper thumbnail of Periodicity in the transient regime of exhaustive polling systems" class="work-thumbnail" src="https://attachments.academia-assets.com/80848681/thumbnails/1.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/71556174/Periodicity_in_the_transient_regime_of_exhaustive_polling_systems">Periodicity in the transient regime of exhaustive polling systems</a></div><div class="wp-workCard_item"><span>Annals of Applied Probability 2006 Vol 16 Pp 1816 1850 Peer Reviewed Journal</span><span>, Nov 1, 2006</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">We consider an exhaustive polling system with three nodes in its transient regime under a switchi...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">We consider an exhaustive polling system with three nodes in its transient regime under a switching rule of generalized greedy type. We show that, for the system with Poisson arrivals and service times with finite second moment, the sequence of nodes visited by the server is eventually periodic almost surely. To do this, we construct a dynamical system, the triangle process, which we show has eventually periodic trajectories for almost all sets of parameters and in this case we show that the stochastic trajectories follow the deterministic ones a.s. We also show there are infinitely many sets of parameters where the triangle process has aperiodic trajectories and in such cases trajectories of the stochastic model are aperiodic with positive probability.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><a id="75022166f97bc7ab19837d2d80657d94" class="wp-workCard--action" rel="nofollow" data-click-track="profile-work-strip-download" data-download="{"attachment_id":80848681,"asset_id":71556174,"asset_type":"Work","button_location":"profile"}" href="https://www.academia.edu/attachments/80848681/download_file?st=MTczMjc0NjQwNyw4LjIyMi4yMDguMTQ2&st=MTczMjc0NjQwNiw4LjIyMi4yMDguMTQ2&s=profile"><span><i class="fa fa-arrow-down"></i></span><span>Download</span></a><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="71556174"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="71556174"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 71556174; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=71556174]").text(description); $(".js-view-count[data-work-id=71556174]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 71556174; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='71556174']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 71556174, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (true){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "75022166f97bc7ab19837d2d80657d94" } } $('.js-work-strip[data-work-id=71556174]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":71556174,"title":"Periodicity in the transient regime of exhaustive polling systems","translated_title":"","metadata":{"abstract":"We consider an exhaustive polling system with three nodes in its transient regime under a switching rule of generalized greedy type. 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By using a result about majorizing dependent random fields by Bernoulli fields, we prove that if the selection parameter is less than certain critical value, then, by choosing the scaling parameter large enough, we can assure that there is no percolation in the complementary set.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="71556164"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="71556164"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 71556164; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=71556164]").text(description); $(".js-view-count[data-work-id=71556164]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 71556164; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='71556164']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 71556164, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=71556164]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":71556164,"title":"On the connectivity properties of the complementary set in fractal percolation models","translated_title":"","metadata":{"abstract":"ABSTRACT We study the connectivity properties of the complementary set in Poisson multiscale percolation model and in Mandelbrot\u0026amp;#39;s percolation model in arbitrary dimension. 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By using a result about majorizing dependent random fields by Bernoulli fields, we prove that if the selection parameter is less than certain critical value, then, by choosing the scaling parameter large enough, we can assure that there is no percolation in the complementary set.","internal_url":"https://www.academia.edu/71556164/On_the_connectivity_properties_of_the_complementary_set_in_fractal_percolation_models","translated_internal_url":"","created_at":"2022-02-14T20:59:26.293-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":214780338,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[{"id":37690792,"work_id":71556164,"tagging_user_id":214780338,"tagged_user_id":185233567,"co_author_invite_id":null,"email":"s***v@gmail.com","display_order":0,"name":"Serguei Popov","title":"On the connectivity properties of the complementary set in fractal percolation models"}],"downloadable_attachments":[],"slug":"On_the_connectivity_properties_of_the_complementary_set_in_fractal_percolation_models","translated_slug":"","page_count":null,"language":"en","content_type":"Work","owner":{"id":214780338,"first_name":"Mikhail","middle_initials":null,"last_name":"MM","page_name":"MikhailMM","domain_name":"independent","created_at":"2022-02-14T20:58:13.540-08:00","display_name":"Mikhail MM","url":"https://independent.academia.edu/MikhailMM"},"attachments":[],"research_interests":[{"id":892,"name":"Statistics","url":"https://www.academia.edu/Documents/in/Statistics"},{"id":2177640,"name":"Random Field","url":"https://www.academia.edu/Documents/in/Random_Field"},{"id":3735670,"name":"Critical value","url":"https://www.academia.edu/Documents/in/Critical_value"}],"urls":[]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="71556163"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/71556163/Participation_of_the_urokinase_type_plasminogen_activator_receptor_uPAR_in_neutrophil_transendothelial_migration"><img alt="Research paper thumbnail of Participation of the urokinase-type plasminogen activator receptor (uPAR) in neutrophil transendothelial migration" class="work-thumbnail" src="https://attachments.academia-assets.com/80848703/thumbnails/1.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/71556163/Participation_of_the_urokinase_type_plasminogen_activator_receptor_uPAR_in_neutrophil_transendothelial_migration">Participation of the urokinase-type plasminogen activator receptor (uPAR) in neutrophil transendothelial migration</a></div><div class="wp-workCard_item"><span>Molecular Immunology</span><span>, 2011</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><a id="5d126fd0336624367913000e8867f7fb" class="wp-workCard--action" rel="nofollow" data-click-track="profile-work-strip-download" data-download="{"attachment_id":80848703,"asset_id":71556163,"asset_type":"Work","button_location":"profile"}" href="https://www.academia.edu/attachments/80848703/download_file?st=MTczMjc0NjQwNyw4LjIyMi4yMDguMTQ2&st=MTczMjc0NjQwNiw4LjIyMi4yMDguMTQ2&s=profile"><span><i class="fa fa-arrow-down"></i></span><span>Download</span></a><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="71556163"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="71556163"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 71556163; 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The urokinase-type plasminogen activator receptor (uPAR) plays a key role in neutrophil adhesion and migration. In the present study, we addressed whether uPAR regulates neutrophil transendothelial migration. We first showed that siRNA-mediated knockdown of uPAR in human umbilical vein endothelial cells (HUVECs) did not affect neutrophil migration across HUVEC monolayers indicating that endothelial uPAR does not regulate neutrophil transmigration. In contrast, the transmigration was significantly inhibited by Fab fragment of anti-uPAR monoclonal antibody and proteolytically inactive urokinase (uPA), whereas inhibition of proteolytical activity of endogenous uPA (with amiloride or plasminogen activator inhibitor-1) did not affect the transmigration. Both the anti-uPAR Fab fragment and proteolytically inactive uPA did not exert significant effects upon the transmigration conducted in the presence of F(ab) 2 fragment of blocking antibody to integrin Mac-1 indicating that uPAR regulates Mac-1-dependent transmigration. Mac-1-dependent, but not Mac-1-independent, transmigration was significantly reduced in the presence of N-acetyl-d-glucosamine and d-mannose, the saccharides that disrupt uPAR/Mac-1 association, but was unaffected in the presence of control saccharides (d-sorbitol and sucrose). We conclude that physical association of uPAR with Mac-1 mediates the regulatory effect of uPAR over the transmigration. Finally, we provide evidence that the functional cooperation between uPAR and Mac-1 is essential at both adhesion and diapedesis steps of neutrophil migration across endothelium. Thus, uPAR expressed on neutrophil plasma membrane regulates transendothelial migration independently of uPA proteolytical activity and acting as a cofactor for integrin Mac-1.","publication_date":{"day":null,"month":null,"year":2011,"errors":{}},"publication_name":"Molecular Immunology","grobid_abstract_attachment_id":80848703},"translated_abstract":null,"internal_url":"https://www.academia.edu/71556163/Participation_of_the_urokinase_type_plasminogen_activator_receptor_uPAR_in_neutrophil_transendothelial_migration","translated_internal_url":"","created_at":"2022-02-14T20:59:26.161-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":214780338,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[{"id":37690803,"work_id":71556163,"tagging_user_id":214780338,"tagged_user_id":50901029,"co_author_invite_id":null,"email":"a***a@compmechlab.com","display_order":0,"name":"Olga Antonova","title":"Participation of the urokinase-type 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Inhibitor","url":"https://www.academia.edu/Documents/in/Plasminogen_Activator_Inhibitor"},{"id":2412024,"name":"Proteolytic activity","url":"https://www.academia.edu/Documents/in/Proteolytic_activity"},{"id":2507489,"name":"Monosaccharides","url":"https://www.academia.edu/Documents/in/Monosaccharides"}],"urls":[]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="71556162"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/71556162/On_Range_and_Local_Time_of_Many_dimensional_Submartingales"><img alt="Research paper thumbnail of On Range and Local Time of Many-dimensional Submartingales" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard 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health systems and services research","url":"https://www.academia.edu/Documents/in/Public_health_systems_and_services_research-1"},{"id":546356,"name":"Clinical Study","url":"https://www.academia.edu/Documents/in/Clinical_Study"},{"id":3789879,"name":"Cardiovascular medicine and haematology","url":"https://www.academia.edu/Documents/in/Cardiovascular_medicine_and_haematology"}],"urls":[]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="71556160"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/71556160/Combination_Ethacizin_and_Ethmozin_Treatment_of_Resistant_Ventricular_Ectopy"><img alt="Research paper thumbnail of Combination Ethacizin and Ethmozin Treatment of Resistant Ventricular Ectopy" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/71556160/Combination_Ethacizin_and_Ethmozin_Treatment_of_Resistant_Ventricular_Ectopy">Combination Ethacizin and Ethmozin Treatment of Resistant Ventricular Ectopy</a></div><div class="wp-workCard_item"><span>Journal of Cardiovascular Pharmacology</span><span>, 1994</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">Ethmozin (Moricizine HCl) and ethacizin are two class I antiarrhythmic drugs with different rate ...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">Ethmozin (Moricizine HCl) and ethacizin are two class I antiarrhythmic drugs with different rate constants of interaction with the sodium channel. Computer simulation using the &amp;quot;guarded-receptor&amp;quot; model predicted that the combination of ethacizin and ethmozin should exert a greater decrease in excitability and conduction at short coupling intervals, but little effect at normal heart rates (HR). To test this prediction, we measured intraventricular conduction delay in canine hearts in vivo. In agreement with the model, the combination more potently prolonged the delay only at intervals &amp;lt; 600 ms as compared with ethacizin alone. Combination therapy was tested in 6 patients with idiopathic ventricular ectopic depolarizations (VEDs). Three patients were resistant to either ethmozin or ethacizin monotherapy, and three could not tolerate effective doses because of side effects. Quantitative continuous ECG monitoring showed that total VEDs in the resistant group decreased 0 and 17 +/- 13% for 400 and 800 mg/day ethmozin and 18 +/- 12 and 55 +/- 12% for 100 and 200 mg/day ethacizin, respectively. Combined therapy with ethmozin (400 mg/day) and ethacizin (100 mg/day) reduced the number of VEDs by 78 +/- 2% in these patients without side effects. In the &amp;quot;nonresistant&amp;quot; but intolerant group of patients, use of the combination allowed relief of symptomatic ectopy without side effects. A theoretical model correctly predicted an effective combination of class I antiarrhythmic drugs, one with &amp;quot;fast-off&amp;quot; and one with &amp;quot;slow-off&amp;quot; kinetics, which may provide a general rationale for choosing drug combinations.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="71556160"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="71556160"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 71556160; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=71556160]").text(description); $(".js-view-count[data-work-id=71556160]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 71556160; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='71556160']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 71556160, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=71556160]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":71556160,"title":"Combination Ethacizin and Ethmozin Treatment of Resistant Ventricular Ectopy","translated_title":"","metadata":{"abstract":"Ethmozin (Moricizine HCl) and ethacizin are two class I antiarrhythmic drugs with different rate constants of interaction with the sodium channel. Computer simulation using the \u0026amp;quot;guarded-receptor\u0026amp;quot; model predicted that the combination of ethacizin and ethmozin should exert a greater decrease in excitability and conduction at short coupling intervals, but little effect at normal heart rates (HR). To test this prediction, we measured intraventricular conduction delay in canine hearts in vivo. In agreement with the model, the combination more potently prolonged the delay only at intervals \u0026amp;lt; 600 ms as compared with ethacizin alone. Combination therapy was tested in 6 patients with idiopathic ventricular ectopic depolarizations (VEDs). Three patients were resistant to either ethmozin or ethacizin monotherapy, and three could not tolerate effective doses because of side effects. Quantitative continuous ECG monitoring showed that total VEDs in the resistant group decreased 0 and 17 +/- 13% for 400 and 800 mg/day ethmozin and 18 +/- 12 and 55 +/- 12% for 100 and 200 mg/day ethacizin, respectively. Combined therapy with ethmozin (400 mg/day) and ethacizin (100 mg/day) reduced the number of VEDs by 78 +/- 2% in these patients without side effects. In the \u0026amp;quot;nonresistant\u0026amp;quot; but intolerant group of patients, use of the combination allowed relief of symptomatic ectopy without side effects. A theoretical model correctly predicted an effective combination of class I antiarrhythmic drugs, one with \u0026amp;quot;fast-off\u0026amp;quot; and one with \u0026amp;quot;slow-off\u0026amp;quot; kinetics, which may provide a general rationale for choosing drug combinations.","publisher":"Ovid Technologies (Wolters Kluwer Health)","publication_date":{"day":null,"month":null,"year":1994,"errors":{}},"publication_name":"Journal of Cardiovascular Pharmacology"},"translated_abstract":"Ethmozin (Moricizine HCl) and ethacizin are two class I antiarrhythmic drugs with different rate constants of interaction with the sodium channel. Computer simulation using the \u0026amp;quot;guarded-receptor\u0026amp;quot; model predicted that the combination of ethacizin and ethmozin should exert a greater decrease in excitability and conduction at short coupling intervals, but little effect at normal heart rates (HR). To test this prediction, we measured intraventricular conduction delay in canine hearts in vivo. In agreement with the model, the combination more potently prolonged the delay only at intervals \u0026amp;lt; 600 ms as compared with ethacizin alone. Combination therapy was tested in 6 patients with idiopathic ventricular ectopic depolarizations (VEDs). Three patients were resistant to either ethmozin or ethacizin monotherapy, and three could not tolerate effective doses because of side effects. Quantitative continuous ECG monitoring showed that total VEDs in the resistant group decreased 0 and 17 +/- 13% for 400 and 800 mg/day ethmozin and 18 +/- 12 and 55 +/- 12% for 100 and 200 mg/day ethacizin, respectively. Combined therapy with ethmozin (400 mg/day) and ethacizin (100 mg/day) reduced the number of VEDs by 78 +/- 2% in these patients without side effects. In the \u0026amp;quot;nonresistant\u0026amp;quot; but intolerant group of patients, use of the combination allowed relief of symptomatic ectopy without side effects. A theoretical model correctly predicted an effective combination of class I antiarrhythmic drugs, one with \u0026amp;quot;fast-off\u0026amp;quot; and one with \u0026amp;quot;slow-off\u0026amp;quot; kinetics, which may provide a general rationale for choosing drug combinations.","internal_url":"https://www.academia.edu/71556160/Combination_Ethacizin_and_Ethmozin_Treatment_of_Resistant_Ventricular_Ectopy","translated_internal_url":"","created_at":"2022-02-14T20:59:25.731-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":214780338,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[{"id":37690782,"work_id":71556160,"tagging_user_id":214780338,"tagged_user_id":484537,"co_author_invite_id":null,"email":"f***r@gmail.com","display_order":0,"name":"Frank Starmer","title":"Combination Ethacizin and Ethmozin Treatment of Resistant Ventricular Ectopy"}],"downloadable_attachments":[],"slug":"Combination_Ethacizin_and_Ethmozin_Treatment_of_Resistant_Ventricular_Ectopy","translated_slug":"","page_count":null,"language":"en","content_type":"Work","owner":{"id":214780338,"first_name":"Mikhail","middle_initials":null,"last_name":"MM","page_name":"MikhailMM","domain_name":"independent","created_at":"2022-02-14T20:58:13.540-08:00","display_name":"Mikhail MM","url":"https://independent.academia.edu/MikhailMM"},"attachments":[],"research_interests":[{"id":26327,"name":"Medicine","url":"https://www.academia.edu/Documents/in/Medicine"},{"id":39000,"name":"Electrocardiography","url":"https://www.academia.edu/Documents/in/Electrocardiography"},{"id":52438,"name":"Dogs","url":"https://www.academia.edu/Documents/in/Dogs"},{"id":69542,"name":"Computer Simulation","url":"https://www.academia.edu/Documents/in/Computer_Simulation"},{"id":89987,"name":"Cardiovascular Pharmacology","url":"https://www.academia.edu/Documents/in/Cardiovascular_Pharmacology"},{"id":131298,"name":"Heart rate","url":"https://www.academia.edu/Documents/in/Heart_rate"},{"id":323090,"name":"Cardiac Arrhythmias","url":"https://www.academia.edu/Documents/in/Cardiac_Arrhythmias"},{"id":546356,"name":"Clinical Study","url":"https://www.academia.edu/Documents/in/Clinical_Study"},{"id":572282,"name":"Combination drug therapy","url":"https://www.academia.edu/Documents/in/Combination_drug_therapy"},{"id":1154249,"name":"Heart Ventricles","url":"https://www.academia.edu/Documents/in/Heart_Ventricles"},{"id":2572082,"name":"sodium channels","url":"https://www.academia.edu/Documents/in/sodium_channels"},{"id":3789879,"name":"Cardiovascular medicine and haematology","url":"https://www.academia.edu/Documents/in/Cardiovascular_medicine_and_haematology"},{"id":3789884,"name":"Pharmacology and pharmaceutical sciences","url":"https://www.academia.edu/Documents/in/Pharmacology_and_pharmaceutical_sciences"},{"id":4053335,"name":"Electric stimulation","url":"https://www.academia.edu/Documents/in/Electric_stimulation"}],"urls":[]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="71556159"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/71556159/The_hypertensive_heart_Pathogenesis_variants_and_prognostic_value"><img alt="Research paper thumbnail of The hypertensive heart: Pathogenesis, variants, and prognostic value" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/71556159/The_hypertensive_heart_Pathogenesis_variants_and_prognostic_value">The hypertensive heart: Pathogenesis, variants, and prognostic value</a></div><div class="wp-workCard_item"><span>Health Psychology</span><span>, 1988</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">We examine the heart&amp;#39;s involvement in arterial hypertension, reporting on several studies...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">We examine the heart&amp;#39;s involvement in arterial hypertension, reporting on several studies of hypertensive patients showing that left ventricular myocardial mass is a significant prognostic indicator of essential hypertension and that left ventricular hypertrophy (LVH) correlates with an induced increase in Ca2+ concentration in platelets. We also consider the LVH variant of asymmetric hypertrophy and the role of the hypertensive heart in coronary insufficiency, and we speculate on the significance of the degree of LVH and asymmetric hypertrophy as risk factors for predicting cardiac complications of essential hypertension.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="71556159"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="71556159"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 71556159; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=71556159]").text(description); $(".js-view-count[data-work-id=71556159]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 71556159; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='71556159']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 71556159, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=71556159]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":71556159,"title":"The hypertensive heart: Pathogenesis, variants, and prognostic value","translated_title":"","metadata":{"abstract":"We examine the heart\u0026amp;#39;s involvement in arterial hypertension, reporting on several studies of hypertensive patients showing that left ventricular myocardial mass is a significant prognostic indicator of essential hypertension and that left ventricular hypertrophy (LVH) correlates with an induced increase in Ca2+ concentration in platelets. We also consider the LVH variant of asymmetric hypertrophy and the role of the hypertensive heart in coronary insufficiency, and we speculate on the significance of the degree of LVH and asymmetric hypertrophy as risk factors for predicting cardiac complications of essential hypertension.","publisher":"American Psychological Association (APA)","publication_date":{"day":null,"month":null,"year":1988,"errors":{}},"publication_name":"Health Psychology"},"translated_abstract":"We examine the heart\u0026amp;#39;s involvement in arterial hypertension, reporting on several studies of hypertensive patients showing that left ventricular myocardial mass is a significant prognostic indicator of essential hypertension and that left ventricular hypertrophy (LVH) correlates with an induced increase in Ca2+ concentration in platelets. We also consider the LVH variant of asymmetric hypertrophy and the role of the hypertensive heart in coronary insufficiency, and we speculate on the significance of the degree of LVH and asymmetric hypertrophy as risk factors for predicting cardiac complications of essential hypertension.","internal_url":"https://www.academia.edu/71556159/The_hypertensive_heart_Pathogenesis_variants_and_prognostic_value","translated_internal_url":"","created_at":"2022-02-14T20:59:25.574-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":214780338,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[{"id":37690780,"work_id":71556159,"tagging_user_id":214780338,"tagged_user_id":134172687,"co_author_invite_id":null,"email":"a***a@yandex.ru","display_order":0,"name":"anna nesterova","title":"The hypertensive heart: Pathogenesis, variants, and prognostic value"}],"downloadable_attachments":[],"slug":"The_hypertensive_heart_Pathogenesis_variants_and_prognostic_value","translated_slug":"","page_count":null,"language":"en","content_type":"Work","owner":{"id":214780338,"first_name":"Mikhail","middle_initials":null,"last_name":"MM","page_name":"MikhailMM","domain_name":"independent","created_at":"2022-02-14T20:58:13.540-08:00","display_name":"Mikhail MM","url":"https://independent.academia.edu/MikhailMM"},"attachments":[],"research_interests":[{"id":260,"name":"Health Psychology","url":"https://www.academia.edu/Documents/in/Health_Psychology"},{"id":922,"name":"Education","url":"https://www.academia.edu/Documents/in/Education"},{"id":9534,"name":"Calcium","url":"https://www.academia.edu/Documents/in/Calcium"},{"id":14406,"name":"Health","url":"https://www.academia.edu/Documents/in/Health"},{"id":22997,"name":"Arterial hypertension","url":"https://www.academia.edu/Documents/in/Arterial_hypertension"},{"id":71399,"name":"Hypertension","url":"https://www.academia.edu/Documents/in/Hypertension"},{"id":74347,"name":"Hemodynamics","url":"https://www.academia.edu/Documents/in/Hemodynamics"},{"id":192721,"name":"Risk factors","url":"https://www.academia.edu/Documents/in/Risk_factors"},{"id":489727,"name":"Prognosis","url":"https://www.academia.edu/Documents/in/Prognosis"},{"id":515631,"name":"Left ventricular hypertrophy","url":"https://www.academia.edu/Documents/in/Left_ventricular_hypertrophy"},{"id":618800,"name":"Coronary Circulation","url":"https://www.academia.edu/Documents/in/Coronary_Circulation"},{"id":620049,"name":"Risk Factors","url":"https://www.academia.edu/Documents/in/Risk_Factors-1"},{"id":2515205,"name":"Essential Hypertension","url":"https://www.academia.edu/Documents/in/Essential_Hypertension"},{"id":2788426,"name":"Cardiomegaly","url":"https://www.academia.edu/Documents/in/Cardiomegaly"},{"id":2922956,"name":"Psychology and Cognitive Sciences","url":"https://www.academia.edu/Documents/in/Psychology_and_Cognitive_Sciences"},{"id":3187114,"name":"Blood platelets","url":"https://www.academia.edu/Documents/in/Blood_platelets"},{"id":3501173,"name":"Left Ventricular","url":"https://www.academia.edu/Documents/in/Left_Ventricular"},{"id":3763225,"name":"Medical and Health Sciences","url":"https://www.academia.edu/Documents/in/Medical_and_Health_Sciences"}],"urls":[]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="31783603"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/31783603/Urokinase_stimulates_inflammatory_response_in_damaged_vascular_wall_during_in_vivo_arterial_remodeling"><img alt="Research paper thumbnail of Urokinase stimulates inflammatory response in damaged vascular wall during in vivo arterial remodeling" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/31783603/Urokinase_stimulates_inflammatory_response_in_damaged_vascular_wall_during_in_vivo_arterial_remodeling">Urokinase stimulates inflammatory response in damaged vascular wall during in vivo arterial remodeling</a></div><div class="wp-workCard_item wp-workCard--coauthors"><span>by </span><span><a class="" data-click-track="profile-work-strip-authors" href="https://independent.academia.edu/MikhailMM">Mikhail MM</a>, <a class="" data-click-track="profile-work-strip-authors" href="https://independent.academia.edu/YelenaParfyonova">Yelena Parfyonova</a>, <a class="" data-click-track="profile-work-strip-authors" href="https://independent.academia.edu/ElizavetaRatner">Elizaveta Ratner</a>, and <a class="" data-click-track="profile-work-strip-authors" href="https://independent.academia.edu/OlgaPlekhanova">Olga Plekhanova</a></span></div><div class="wp-workCard_item"><span>Bulletin of Experimental Biology and Medicine</span><span>, 2008</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">Perivascular application of urokinase to the ballooned artery promoted the growth of neointima an...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">Perivascular application of urokinase to the ballooned artery promoted the growth of neointima and constrictive remodeling of the vessel and stimulated the inflammatory response in the damaged vascular wall in vivo. Recombinant tissue plasminogen activator did not induce these changes. Our results indicate that urokinase is involved in the regulation of the inflammatory response during in vivo remodeling of the damaged vascular wall.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="31783603"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="31783603"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 31783603; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=31783603]").text(description); $(".js-view-count[data-work-id=31783603]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 31783603; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='31783603']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 31783603, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=31783603]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":31783603,"title":"Urokinase stimulates inflammatory response in damaged vascular wall during in vivo arterial remodeling","translated_title":"","metadata":{"abstract":"Perivascular application of urokinase to the ballooned artery promoted the growth of neointima and constrictive remodeling of the vessel and stimulated the inflammatory response in the damaged vascular wall in vivo. 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Our results indicate that urokinase is involved in the regulation of the inflammatory response during in vivo remodeling of the damaged vascular wall.","internal_url":"https://www.academia.edu/31783603/Urokinase_stimulates_inflammatory_response_in_damaged_vascular_wall_during_in_vivo_arterial_remodeling","translated_internal_url":"","created_at":"2017-03-09T01:59:08.549-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":61244807,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[{"id":27950754,"work_id":31783603,"tagging_user_id":61244807,"tagged_user_id":214780338,"co_author_invite_id":6109124,"email":"m***f@gmail.com","display_order":0,"name":"Mikhail MM","title":"Urokinase stimulates inflammatory response in damaged vascular wall during in vivo arterial remodeling"},{"id":27950821,"work_id":31783603,"tagging_user_id":61244807,"tagged_user_id":61313753,"co_author_invite_id":6109162,"email":"e***r@gmail.com","display_order":4194304,"name":"Elizaveta Ratner","title":"Urokinase stimulates inflammatory response in damaged vascular wall during in vivo arterial remodeling"},{"id":27950843,"work_id":31783603,"tagging_user_id":61244807,"tagged_user_id":61509972,"co_author_invite_id":6106932,"email":"p***a@mail.ru","display_order":6291456,"name":"Olga Plekhanova","title":"Urokinase stimulates inflammatory response in damaged vascular wall during in vivo arterial remodeling"},{"id":27982071,"work_id":31783603,"tagging_user_id":61244807,"tagged_user_id":36516936,"co_author_invite_id":null,"email":"t***k@fbm.msu.ru","display_order":7340032,"name":"Vsevolod Tkachuk","title":"Urokinase stimulates inflammatory response in damaged vascular wall during in vivo arterial remodeling"}],"downloadable_attachments":[],"slug":"Urokinase_stimulates_inflammatory_response_in_damaged_vascular_wall_during_in_vivo_arterial_remodeling","translated_slug":"","page_count":null,"language":"en","content_type":"Work","owner":{"id":61244807,"first_name":"Yelena","middle_initials":"","last_name":"Parfyonova","page_name":"YelenaParfyonova","domain_name":"independent","created_at":"2017-03-09T01:58:50.424-08:00","display_name":"Yelena Parfyonova","url":"https://independent.academia.edu/YelenaParfyonova"},"attachments":[],"research_interests":[{"id":9334,"name":"Inflammation","url":"https://www.academia.edu/Documents/in/Inflammation"},{"id":375054,"name":"Rats","url":"https://www.academia.edu/Documents/in/Rats"},{"id":564879,"name":"Wistar Rats","url":"https://www.academia.edu/Documents/in/Wistar_Rats"},{"id":2454817,"name":"Inflammatory response","url":"https://www.academia.edu/Documents/in/Inflammatory_response"},{"id":2562018,"name":"Arteries","url":"https://www.academia.edu/Documents/in/Arteries"}],"urls":[]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="71556158"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/71556158/On_a_multiscale_continuous_percolation_model_with_unbounded_defects"><img alt="Research paper thumbnail of On a multiscale continuous percolation model with unbounded defects" class="work-thumbnail" src="https://attachments.academia-assets.com/80848705/thumbnails/1.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/71556158/On_a_multiscale_continuous_percolation_model_with_unbounded_defects">On a multiscale continuous percolation model with unbounded defects</a></div><div class="wp-workCard_item"><span>Bulletin of the Brazilian Mathematical Society</span><span>, 2003</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><a id="bc2f4cbc86629fb78251c1b2c70b2051" class="wp-workCard--action" rel="nofollow" data-click-track="profile-work-strip-download" data-download="{"attachment_id":80848705,"asset_id":71556158,"asset_type":"Work","button_location":"profile"}" href="https://www.academia.edu/attachments/80848705/download_file?st=MTczMjc0NjQwNyw4LjIyMi4yMDguMTQ2&st=MTczMjc0NjQwNyw4LjIyMi4yMDguMTQ2&s=profile"><span><i class="fa fa-arrow-down"></i></span><span>Download</span></a><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="71556158"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="71556158"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 71556158; 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