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class="mw-page-container"> <div class="mw-page-container-inner"> <div class="vector-sitenotice-container"> <div id="siteNotice"></div> </div> <div class="vector-column-start"> <div class="vector-main-menu-container"> <div id="mw-navigation"> <nav id="mw-panel" class="vector-main-menu-landmark" aria-label="Wiki"> <div id="vector-main-menu-pinned-container" class="vector-pinned-container"> </div> </nav> </div> </div> <div class="vector-sticky-pinned-container"> <nav id="mw-panel-toc" aria-label="Tartalomjegyzék" data-event-name="ui.sidebar-toc" class="mw-table-of-contents-container vector-toc-landmark"> <div id="vector-toc-pinned-container" class="vector-pinned-container"> <div id="vector-toc" class="vector-toc vector-pinnable-element"> <div class="vector-pinnable-header vector-toc-pinnable-header vector-pinnable-header-pinned" data-feature-name="toc-pinned" data-pinnable-element-id="vector-toc" > <h2 class="vector-pinnable-header-label">Tartalomjegyzék</h2> <button class="vector-pinnable-header-toggle-button vector-pinnable-header-pin-button" data-event-name="pinnable-header.vector-toc.pin">áthelyezés az oldalsávba</button> <button class="vector-pinnable-header-toggle-button vector-pinnable-header-unpin-button" data-event-name="pinnable-header.vector-toc.unpin">elrejtés</button> </div> <ul class="vector-toc-contents" id="mw-panel-toc-list"> <li id="toc-mw-content-text" class="vector-toc-list-item vector-toc-level-1"> <a href="#" class="vector-toc-link"> <div class="vector-toc-text">Bevezető</div> </a> </li> <li id="toc-Elnevezésük" class="vector-toc-list-item vector-toc-level-1 vector-toc-list-item-expanded"> <a class="vector-toc-link" href="#Elnevezésük"> <div class="vector-toc-text"> <span class="vector-toc-numb">1</span> <span>Elnevezésük</span> </div> </a> <ul id="toc-Elnevezésük-sublist" class="vector-toc-list"> </ul> </li> <li id="toc-Fontosabb_képviselőik" class="vector-toc-list-item vector-toc-level-1 vector-toc-list-item-expanded"> <a class="vector-toc-link" href="#Fontosabb_képviselőik"> <div class="vector-toc-text"> <span class="vector-toc-numb">2</span> <span>Fontosabb képviselőik</span> </div> </a> <button aria-controls="toc-Fontosabb_képviselőik-sublist" class="cdx-button cdx-button--weight-quiet cdx-button--icon-only vector-toc-toggle"> <span class="vector-icon mw-ui-icon-wikimedia-expand"></span> <span>A(z) Fontosabb képviselőik alszakasz kinyitása/becsukása</span> </button> <ul id="toc-Fontosabb_képviselőik-sublist" class="vector-toc-list"> <li id="toc-Interleukin-1" class="vector-toc-list-item vector-toc-level-2"> <a class="vector-toc-link" href="#Interleukin-1"> <div class="vector-toc-text"> <span class="vector-toc-numb">2.1</span> <span>Interleukin-1</span> </div> </a> <ul id="toc-Interleukin-1-sublist" class="vector-toc-list"> </ul> </li> <li id="toc-Interleukin-2" class="vector-toc-list-item vector-toc-level-2"> <a class="vector-toc-link" href="#Interleukin-2"> <div class="vector-toc-text"> <span class="vector-toc-numb">2.2</span> <span>Interleukin-2</span> </div> </a> <ul id="toc-Interleukin-2-sublist" class="vector-toc-list"> </ul> </li> <li id="toc-Interleukin-3" class="vector-toc-list-item vector-toc-level-2"> <a class="vector-toc-link" href="#Interleukin-3"> <div class="vector-toc-text"> <span class="vector-toc-numb">2.3</span> <span>Interleukin-3</span> </div> </a> <ul id="toc-Interleukin-3-sublist" class="vector-toc-list"> </ul> </li> <li id="toc-Interleukin-4" class="vector-toc-list-item vector-toc-level-2"> <a class="vector-toc-link" href="#Interleukin-4"> <div class="vector-toc-text"> <span class="vector-toc-numb">2.4</span> <span>Interleukin-4</span> </div> </a> <ul id="toc-Interleukin-4-sublist" class="vector-toc-list"> </ul> </li> <li id="toc-Interleukin_5" class="vector-toc-list-item vector-toc-level-2"> <a class="vector-toc-link" href="#Interleukin_5"> <div class="vector-toc-text"> <span class="vector-toc-numb">2.5</span> <span>Interleukin 5</span> </div> </a> <ul id="toc-Interleukin_5-sublist" class="vector-toc-list"> </ul> </li> <li id="toc-Interleukin-6" class="vector-toc-list-item vector-toc-level-2"> <a class="vector-toc-link" href="#Interleukin-6"> <div class="vector-toc-text"> <span class="vector-toc-numb">2.6</span> <span>Interleukin-6</span> </div> </a> <ul id="toc-Interleukin-6-sublist" class="vector-toc-list"> </ul> </li> <li id="toc-Interleukin-7" class="vector-toc-list-item vector-toc-level-2"> <a class="vector-toc-link" href="#Interleukin-7"> <div class="vector-toc-text"> <span class="vector-toc-numb">2.7</span> <span>Interleukin-7</span> </div> </a> <ul id="toc-Interleukin-7-sublist" class="vector-toc-list"> </ul> </li> <li id="toc-Interleukin-8" class="vector-toc-list-item vector-toc-level-2"> <a class="vector-toc-link" href="#Interleukin-8"> <div class="vector-toc-text"> <span class="vector-toc-numb">2.8</span> <span>Interleukin-8</span> </div> </a> <ul id="toc-Interleukin-8-sublist" class="vector-toc-list"> </ul> </li> <li id="toc-Interleukin-9" class="vector-toc-list-item vector-toc-level-2"> <a class="vector-toc-link" href="#Interleukin-9"> <div class="vector-toc-text"> <span class="vector-toc-numb">2.9</span> <span>Interleukin-9</span> </div> </a> <ul id="toc-Interleukin-9-sublist" class="vector-toc-list"> </ul> </li> <li id="toc-Interleukin-10" class="vector-toc-list-item vector-toc-level-2"> <a class="vector-toc-link" href="#Interleukin-10"> <div class="vector-toc-text"> <span class="vector-toc-numb">2.10</span> <span>Interleukin-10</span> </div> </a> <ul id="toc-Interleukin-10-sublist" class="vector-toc-list"> </ul> </li> <li id="toc-Interleukin-11" class="vector-toc-list-item vector-toc-level-2"> <a class="vector-toc-link" href="#Interleukin-11"> <div class="vector-toc-text"> <span class="vector-toc-numb">2.11</span> <span>Interleukin-11</span> </div> </a> <ul id="toc-Interleukin-11-sublist" class="vector-toc-list"> </ul> </li> <li id="toc-Interleukin-12" class="vector-toc-list-item vector-toc-level-2"> <a class="vector-toc-link" href="#Interleukin-12"> <div class="vector-toc-text"> <span class="vector-toc-numb">2.12</span> <span>Interleukin-12</span> </div> </a> <ul id="toc-Interleukin-12-sublist" class="vector-toc-list"> </ul> </li> <li id="toc-Interleukin-13" class="vector-toc-list-item vector-toc-level-2"> <a class="vector-toc-link" href="#Interleukin-13"> <div class="vector-toc-text"> <span class="vector-toc-numb">2.13</span> <span>Interleukin-13</span> </div> </a> <ul id="toc-Interleukin-13-sublist" class="vector-toc-list"> </ul> </li> <li id="toc-Interleukin-15" class="vector-toc-list-item vector-toc-level-2"> <a class="vector-toc-link" href="#Interleukin-15"> <div class="vector-toc-text"> <span class="vector-toc-numb">2.14</span> <span>Interleukin-15</span> </div> </a> <ul id="toc-Interleukin-15-sublist" class="vector-toc-list"> </ul> </li> <li id="toc-Interleukin-17" class="vector-toc-list-item vector-toc-level-2"> <a class="vector-toc-link" href="#Interleukin-17"> <div class="vector-toc-text"> <span class="vector-toc-numb">2.15</span> <span>Interleukin-17</span> </div> </a> <ul id="toc-Interleukin-17-sublist" class="vector-toc-list"> </ul> </li> </ul> </li> <li id="toc-Interleukinok_az_emberben" class="vector-toc-list-item vector-toc-level-1 vector-toc-list-item-expanded"> <a class="vector-toc-link" href="#Interleukinok_az_emberben"> <div class="vector-toc-text"> <span class="vector-toc-numb">3</span> <span>Interleukinok az emberben</span> </div> </a> <ul id="toc-Interleukinok_az_emberben-sublist" class="vector-toc-list"> </ul> </li> <li id="toc-Jegyzetek" class="vector-toc-list-item vector-toc-level-1 vector-toc-list-item-expanded"> <a class="vector-toc-link" href="#Jegyzetek"> <div class="vector-toc-text"> <span class="vector-toc-numb">4</span> <span>Jegyzetek</span> </div> </a> <ul id="toc-Jegyzetek-sublist" class="vector-toc-list"> </ul> </li> <li id="toc-Fordítás" class="vector-toc-list-item vector-toc-level-1 vector-toc-list-item-expanded"> <a class="vector-toc-link" href="#Fordítás"> <div class="vector-toc-text"> <span class="vector-toc-numb">5</span> <span>Fordítás</span> </div> </a> <ul id="toc-Fordítás-sublist" class="vector-toc-list"> </ul> </li> </ul> </div> </div> </nav> </div> </div> <div class="mw-content-container"> <main id="content" class="mw-body"> <header class="mw-body-header vector-page-titlebar"> <nav aria-label="Tartalomjegyzék" class="vector-toc-landmark"> <div id="vector-page-titlebar-toc" class="vector-dropdown vector-page-titlebar-toc vector-button-flush-left" > <input type="checkbox" id="vector-page-titlebar-toc-checkbox" role="button" aria-haspopup="true" data-event-name="ui.dropdown-vector-page-titlebar-toc" class="vector-dropdown-checkbox " aria-label="Tartalomjegyzék kinyitása/becsukása" > <label id="vector-page-titlebar-toc-label" for="vector-page-titlebar-toc-checkbox" class="vector-dropdown-label cdx-button cdx-button--fake-button cdx-button--fake-button--enabled cdx-button--weight-quiet cdx-button--icon-only " aria-hidden="true" ><span class="vector-icon mw-ui-icon-listBullet mw-ui-icon-wikimedia-listBullet"></span> <span class="vector-dropdown-label-text">Tartalomjegyzék kinyitása/becsukása</span> </label> <div class="vector-dropdown-content"> <div id="vector-page-titlebar-toc-unpinned-container" class="vector-unpinned-container"> </div> </div> </div> </nav> <h1 id="firstHeading" class="firstHeading mw-first-heading"><span class="mw-page-title-main">Interleukin</span></h1> <div id="p-lang-btn" class="vector-dropdown mw-portlet mw-portlet-lang" > <input type="checkbox" id="p-lang-btn-checkbox" role="button" aria-haspopup="true" data-event-name="ui.dropdown-p-lang-btn" class="vector-dropdown-checkbox mw-interlanguage-selector" aria-label="Ugrás egy más nyelvű szócikkre. Elérhető 31 nyelven" > <label id="p-lang-btn-label" for="p-lang-btn-checkbox" class="vector-dropdown-label cdx-button cdx-button--fake-button cdx-button--fake-button--enabled cdx-button--weight-quiet cdx-button--action-progressive mw-portlet-lang-heading-31" aria-hidden="true" ><span class="vector-icon mw-ui-icon-language-progressive mw-ui-icon-wikimedia-language-progressive"></span> <span class="vector-dropdown-label-text">31 nyelv</span> </label> <div class="vector-dropdown-content"> <div class="vector-menu-content"> <ul class="vector-menu-content-list"> <li class="interlanguage-link interwiki-en mw-list-item"><a href="https://en.wikipedia.org/wiki/Interleukin" title="Interleukin – angol" lang="en" hreflang="en" data-title="Interleukin" data-language-autonym="English" data-language-local-name="angol" class="interlanguage-link-target"><span>English</span></a></li><li class="interlanguage-link interwiki-ar mw-list-item"><a href="https://ar.wikipedia.org/wiki/%D8%A5%D9%86%D8%AA%D8%B1%D9%84%D9%88%D9%83%D9%8A%D9%86" title="إنترلوكين – arab" lang="ar" hreflang="ar" data-title="إنترلوكين" data-language-autonym="العربية" data-language-local-name="arab" class="interlanguage-link-target"><span>العربية</span></a></li><li class="interlanguage-link interwiki-az mw-list-item"><a href="https://az.wikipedia.org/wiki/%C4%B0nterleykinl%C9%99r" title="İnterleykinlər – azerbajdzsáni" lang="az" hreflang="az" data-title="İnterleykinlər" data-language-autonym="Azərbaycanca" data-language-local-name="azerbajdzsáni" class="interlanguage-link-target"><span>Azərbaycanca</span></a></li><li class="interlanguage-link interwiki-bg mw-list-item"><a href="https://bg.wikipedia.org/wiki/%D0%98%D0%BD%D1%82%D0%B5%D1%80%D0%BB%D0%B5%D0%B2%D0%BA%D0%B8%D0%BD" title="Интерлевкин – bolgár" lang="bg" hreflang="bg" data-title="Интерлевкин" data-language-autonym="Български" data-language-local-name="bolgár" class="interlanguage-link-target"><span>Български</span></a></li><li class="interlanguage-link interwiki-bs mw-list-item"><a href="https://bs.wikipedia.org/wiki/Interleukin" title="Interleukin – bosnyák" lang="bs" hreflang="bs" data-title="Interleukin" data-language-autonym="Bosanski" data-language-local-name="bosnyák" class="interlanguage-link-target"><span>Bosanski</span></a></li><li class="interlanguage-link interwiki-ca mw-list-item"><a href="https://ca.wikipedia.org/wiki/Interleucina" title="Interleucina – katalán" lang="ca" hreflang="ca" data-title="Interleucina" data-language-autonym="Català" data-language-local-name="katalán" class="interlanguage-link-target"><span>Català</span></a></li><li class="interlanguage-link interwiki-cs mw-list-item"><a href="https://cs.wikipedia.org/wiki/Interleukiny" title="Interleukiny – cseh" lang="cs" hreflang="cs" data-title="Interleukiny" data-language-autonym="Čeština" data-language-local-name="cseh" class="interlanguage-link-target"><span>Čeština</span></a></li><li class="interlanguage-link interwiki-de mw-list-item"><a href="https://de.wikipedia.org/wiki/Interleukine" title="Interleukine – német" lang="de" hreflang="de" data-title="Interleukine" data-language-autonym="Deutsch" data-language-local-name="német" class="interlanguage-link-target"><span>Deutsch</span></a></li><li class="interlanguage-link interwiki-es mw-list-item"><a href="https://es.wikipedia.org/wiki/Interleucina" title="Interleucina – spanyol" lang="es" hreflang="es" data-title="Interleucina" data-language-autonym="Español" data-language-local-name="spanyol" class="interlanguage-link-target"><span>Español</span></a></li><li class="interlanguage-link interwiki-eu mw-list-item"><a href="https://eu.wikipedia.org/wiki/Interleukina" title="Interleukina – baszk" lang="eu" hreflang="eu" data-title="Interleukina" data-language-autonym="Euskara" data-language-local-name="baszk" class="interlanguage-link-target"><span>Euskara</span></a></li><li class="interlanguage-link interwiki-fa mw-list-item"><a href="https://fa.wikipedia.org/wiki/%D8%A7%DB%8C%D9%86%D8%AA%D8%B1%D9%84%D9%88%DA%A9%DB%8C%D9%86" title="اینترلوکین – perzsa" lang="fa" hreflang="fa" data-title="اینترلوکین" data-language-autonym="فارسی" data-language-local-name="perzsa" class="interlanguage-link-target"><span>فارسی</span></a></li><li class="interlanguage-link interwiki-fi mw-list-item"><a href="https://fi.wikipedia.org/wiki/Interleukiini" title="Interleukiini – finn" lang="fi" hreflang="fi" data-title="Interleukiini" data-language-autonym="Suomi" data-language-local-name="finn" class="interlanguage-link-target"><span>Suomi</span></a></li><li class="interlanguage-link interwiki-fr mw-list-item"><a href="https://fr.wikipedia.org/wiki/Interleukine" title="Interleukine – francia" lang="fr" hreflang="fr" data-title="Interleukine" data-language-autonym="Français" data-language-local-name="francia" class="interlanguage-link-target"><span>Français</span></a></li><li class="interlanguage-link interwiki-gl mw-list-item"><a href="https://gl.wikipedia.org/wiki/Interleucina" title="Interleucina – gallego" lang="gl" hreflang="gl" data-title="Interleucina" data-language-autonym="Galego" data-language-local-name="gallego" class="interlanguage-link-target"><span>Galego</span></a></li><li class="interlanguage-link interwiki-he mw-list-item"><a href="https://he.wikipedia.org/wiki/%D7%90%D7%99%D7%A0%D7%98%D7%A8%D7%9C%D7%95%D7%A7%D7%99%D7%9F" title="אינטרלוקין – héber" lang="he" hreflang="he" data-title="אינטרלוקין" data-language-autonym="עברית" data-language-local-name="héber" class="interlanguage-link-target"><span>עברית</span></a></li><li class="interlanguage-link interwiki-id mw-list-item"><a href="https://id.wikipedia.org/wiki/Interleukin" title="Interleukin – indonéz" lang="id" hreflang="id" data-title="Interleukin" data-language-autonym="Bahasa Indonesia" data-language-local-name="indonéz" class="interlanguage-link-target"><span>Bahasa Indonesia</span></a></li><li class="interlanguage-link interwiki-it mw-list-item"><a href="https://it.wikipedia.org/wiki/Interleuchine" title="Interleuchine – olasz" lang="it" hreflang="it" data-title="Interleuchine" data-language-autonym="Italiano" data-language-local-name="olasz" class="interlanguage-link-target"><span>Italiano</span></a></li><li class="interlanguage-link interwiki-ja mw-list-item"><a href="https://ja.wikipedia.org/wiki/%E3%82%A4%E3%83%B3%E3%82%BF%E3%83%BC%E3%83%AD%E3%82%A4%E3%82%AD%E3%83%B3" title="インターロイキン – japán" lang="ja" hreflang="ja" data-title="インターロイキン" data-language-autonym="日本語" data-language-local-name="japán" class="interlanguage-link-target"><span>日本語</span></a></li><li class="interlanguage-link interwiki-ko mw-list-item"><a href="https://ko.wikipedia.org/wiki/%EC%9D%B8%ED%84%B0%EB%A3%A8%ED%82%A8" title="인터루킨 – koreai" lang="ko" hreflang="ko" data-title="인터루킨" data-language-autonym="한국어" data-language-local-name="koreai" class="interlanguage-link-target"><span>한국어</span></a></li><li class="interlanguage-link interwiki-la mw-list-item"><a href="https://la.wikipedia.org/wiki/Interleukinum" title="Interleukinum – latin" lang="la" hreflang="la" data-title="Interleukinum" data-language-autonym="Latina" data-language-local-name="latin" class="interlanguage-link-target"><span>Latina</span></a></li><li class="interlanguage-link interwiki-ms mw-list-item"><a href="https://ms.wikipedia.org/wiki/Interleukin" title="Interleukin – maláj" lang="ms" hreflang="ms" data-title="Interleukin" data-language-autonym="Bahasa Melayu" data-language-local-name="maláj" class="interlanguage-link-target"><span>Bahasa Melayu</span></a></li><li class="interlanguage-link interwiki-nl mw-list-item"><a href="https://nl.wikipedia.org/wiki/Interleukine" title="Interleukine – holland" lang="nl" hreflang="nl" data-title="Interleukine" data-language-autonym="Nederlands" data-language-local-name="holland" class="interlanguage-link-target"><span>Nederlands</span></a></li><li class="interlanguage-link interwiki-pl mw-list-item"><a href="https://pl.wikipedia.org/wiki/Interleukiny" title="Interleukiny – lengyel" lang="pl" hreflang="pl" data-title="Interleukiny" data-language-autonym="Polski" data-language-local-name="lengyel" class="interlanguage-link-target"><span>Polski</span></a></li><li class="interlanguage-link interwiki-pt mw-list-item"><a href="https://pt.wikipedia.org/wiki/Interleucina" title="Interleucina – portugál" lang="pt" hreflang="pt" data-title="Interleucina" data-language-autonym="Português" data-language-local-name="portugál" class="interlanguage-link-target"><span>Português</span></a></li><li class="interlanguage-link interwiki-ru mw-list-item"><a href="https://ru.wikipedia.org/wiki/%D0%98%D0%BD%D1%82%D0%B5%D1%80%D0%BB%D0%B5%D0%B9%D0%BA%D0%B8%D0%BD%D1%8B" title="Интерлейкины – orosz" lang="ru" hreflang="ru" data-title="Интерлейкины" data-language-autonym="Русский" data-language-local-name="orosz" class="interlanguage-link-target"><span>Русский</span></a></li><li class="interlanguage-link interwiki-sh mw-list-item"><a href="https://sh.wikipedia.org/wiki/Interleukin" title="Interleukin – szerbhorvát" lang="sh" hreflang="sh" data-title="Interleukin" data-language-autonym="Srpskohrvatski / српскохрватски" data-language-local-name="szerbhorvát" class="interlanguage-link-target"><span>Srpskohrvatski / српскохрватски</span></a></li><li class="interlanguage-link interwiki-sl mw-list-item"><a href="https://sl.wikipedia.org/wiki/Interlevkin" title="Interlevkin – szlovén" lang="sl" hreflang="sl" data-title="Interlevkin" data-language-autonym="Slovenščina" data-language-local-name="szlovén" class="interlanguage-link-target"><span>Slovenščina</span></a></li><li class="interlanguage-link interwiki-sr mw-list-item"><a href="https://sr.wikipedia.org/wiki/%D0%98%D0%BD%D1%82%D0%B5%D1%80%D0%BB%D0%B5%D1%83%D0%BA%D0%B8%D0%BD%D0%B8" title="Интерлеукини – szerb" lang="sr" hreflang="sr" data-title="Интерлеукини" data-language-autonym="Српски / srpski" data-language-local-name="szerb" class="interlanguage-link-target"><span>Српски / srpski</span></a></li><li class="interlanguage-link interwiki-th mw-list-item"><a href="https://th.wikipedia.org/wiki/%E0%B8%AD%E0%B8%B4%E0%B8%99%E0%B9%80%E0%B8%95%E0%B8%AD%E0%B8%A3%E0%B9%8C%E0%B8%A5%E0%B8%B4%E0%B8%A7%E0%B8%84%E0%B8%B4%E0%B8%99" title="อินเตอร์ลิวคิน – thai" lang="th" hreflang="th" data-title="อินเตอร์ลิวคิน" data-language-autonym="ไทย" data-language-local-name="thai" class="interlanguage-link-target"><span>ไทย</span></a></li><li class="interlanguage-link interwiki-tr mw-list-item"><a href="https://tr.wikipedia.org/wiki/%C4%B0nterl%C3%B6kin" title="İnterlökin – török" lang="tr" hreflang="tr" data-title="İnterlökin" data-language-autonym="Türkçe" data-language-local-name="török" class="interlanguage-link-target"><span>Türkçe</span></a></li><li class="interlanguage-link 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id="mw-fr-revision-details" class="mw-fr-revision-details-dialog" style="display:none;"><div tabindex="0"></div><div class="cdx-dialog cdx-dialog--horizontal-actions"><header class="cdx-dialog__header cdx-dialog__header--default"><div class="cdx-dialog__header__title-group"><h2 class="cdx-dialog__header__title">Változat állapota</h2><p class="cdx-dialog__header__subtitle">Ez a lap egy ellenőrzött változata</p></div><button class="cdx-button cdx-button--action-default cdx-button--weight-quiet
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Többségüket helper <a href="/wiki/T-limfocita" title="T-limfocita">T-limfociták</a>, <a href="/wiki/Monocita" title="Monocita">monociták</a>, <a href="/wiki/Makrof%C3%A1g" title="Makrofág">makrofágok</a> és <a href="/w/index.php?title=Endot%C3%A9lium&action=edit&redlink=1" class="new" title="Endotélium (a lap nem létezik)">endoteliális</a> sejtek szintetizálják. Az interleukinek elősegítik a T- és <a href="/wiki/B-limfocita" title="B-limfocita">B-limfociták</a>, valamint a <a href="/wiki/Hemopoetikus_%C5%91ssejtek" title="Hemopoetikus őssejtek">vérképző</a> sejtek fejlődését és differenciálódását. Megfelelő működésük alapvető az <a href="/wiki/Immunrendszer" title="Immunrendszer">immunrendszer</a> számára, hiányuk általában <a href="/wiki/Autoimmun_betegs%C3%A9g" title="Autoimmun betegség">autoimmun</a> vagy immunhiányos betegségekhez vezet. Az emberi genomban több mint 50 interleukin és hasonló fehérje génje található meg.<sup id="cite_ref-1" class="reference"><a href="#cite_note-1"><span class="cite-bracket">[</span>1<span class="cite-bracket">]</span></a></sup> </p> <meta property="mw:PageProp/toc" /> <div class="mw-heading mw-heading2"><h2 id="Elnevezésük"><span id="Elnevez.C3.A9s.C3.BCk"></span>Elnevezésük</h2><span class="mw-editsection"><span class="mw-editsection-bracket">[</span><a href="/w/index.php?title=Interleukin&action=edit&section=1" title="Szakasz szerkesztése: Elnevezésük"><span>szerkesztés</span></a><span class="mw-editsection-bracket">]</span></span></div> <p>Az "interleukin" kifejezést 1979-ben vezették be a Svájcban megrendezett Második Nemzetközi Limfokin Konferencián, hogy egységesítsék a különböző kutatócsoportokban az interleukin-1-re (limfocita aktiváló faktor, mitogén protein, T-sejt helyettesítő faktor III, B-sejt aktiváló faktor, B-sejt differenciációs faktor, heidikin) és interleukin-2-re (TSF, stb.) alkalmazott elnevezéseket.<sup id="cite_ref-pmid2405873_2-0" class="reference"><a href="#cite_note-pmid2405873-2"><span class="cite-bracket">[</span>2<span class="cite-bracket">]</span></a></sup><sup id="cite_ref-3" class="reference"><a href="#cite_note-3"><span class="cite-bracket">[</span>3<span class="cite-bracket">]</span></a></sup><sup id="cite_ref-pmid91646_4-0" class="reference"><a href="#cite_note-pmid91646-4"><span class="cite-bracket">[</span>4<span class="cite-bracket">]</span></a></sup> </p><p>A szó összetevői a latin eredetű <i>inter</i> (jelentése "között", "közti") és a <i>leukin</i>, amely arra utal, hogy a molekulák többséget leukociták (fehérvérsejtek) termelik és azok is a célpontjaik (bár azóta kiderült, hogy más sejttípusok is bocsátanak ki interleukinokat). Az interleukinok alcsoportja a limfokinek, amelyeket limfociták állítanak elő és az immunválaszt szabályozzák. </p> <div class="mw-heading mw-heading2"><h2 id="Fontosabb_képviselőik"><span id="Fontosabb_k.C3.A9pvisel.C5.91ik"></span>Fontosabb képviselőik</h2><span class="mw-editsection"><span class="mw-editsection-bracket">[</span><a href="/w/index.php?title=Interleukin&action=edit&section=2" title="Szakasz szerkesztése: Fontosabb képviselőik"><span>szerkesztés</span></a><span class="mw-editsection-bracket">]</span></span></div> <div class="mw-heading mw-heading3"><h3 id="Interleukin-1">Interleukin-1</h3><span class="mw-editsection"><span class="mw-editsection-bracket">[</span><a href="/w/index.php?title=Interleukin&action=edit&section=3" title="Szakasz szerkesztése: Interleukin-1"><span>szerkesztés</span></a><span class="mw-editsection-bracket">]</span></span></div> <p>Az <a href="/w/index.php?title=Interleukin-1&action=edit&redlink=1" class="new" title="Interleukin-1 (a lap nem létezik)">interleukin-1</a> alfa és interleukin-1 béta (IL-1α és IL-1β) olyan citokinek, amelyek az immunválaszt, a <a href="/wiki/Gyullad%C3%A1s" title="Gyulladás">gyulladási</a> folyamatot és vérképzést szabályozzák.<sup id="cite_ref-pmid2969618_5-0" class="reference"><a href="#cite_note-pmid2969618-5"><span class="cite-bracket">[</span>5<span class="cite-bracket">]</span></a></sup> Emberből és egérből egyaránt kétféle IL-1 receptort (I és II típusú) sikerült klónozni; mindkettőnek három sejten kívüli <a href="/wiki/Ellenanyag" title="Ellenanyag">immunglobulin</a>-szerű doménja van, de szekvenciájuk csak 28%-ban egyezik.<sup id="cite_ref-pmid8702856_6-0" class="reference"><a href="#cite_note-pmid8702856-6"><span class="cite-bracket">[</span>6<span class="cite-bracket">]</span></a></sup> A receptorok a <a href="/wiki/Sejtmembr%C3%A1n" title="Sejtmembrán">sejtmembránba</a> ágyazott és szabadon úszó (szolubilis) formában egyaránt előfordulnak: feltételezik, hogy a szolubilis forma a transzmembrán alak <a href="/wiki/Transzl%C3%A1ci%C3%B3" title="Transzláció">transzláció</a> utáni módosításának maradéka. </p><p>Mindkét IL-1 receptor szekvenciája konzervatív, az evolúció során keveset változott és ugyanazon a kromoszómahelyen (ember esetében a <a href="/wiki/Hum%C3%A1n_2-es_kromosz%C3%B3ma" title="Humán 2-es kromoszóma">2. kromoszóma</a> q12 régiójában) találhatók.<sup id="cite_ref-pmid1833184_7-0" class="reference"><a href="#cite_note-pmid1833184-7"><span class="cite-bracket">[</span>7<span class="cite-bracket">]</span></a></sup> Mindkét receptor képes kötni az IL-1 mindhárom formáját (IL-1α, IL-1β és IL-1 receptor antagonista). </p><p>Interleukin-1-nek a központi idegrendszer működésében is szerepe lehet, legalábbis azoknál az egereknél, amelyekből hiányzik az IL-1 receptor génje romlott a <a href="/wiki/Hippokampusz" title="Hippokampusz">hippokampuszhoz</a> kapcsolódó memória.<sup id="cite_ref-pmid21093580_8-0" class="reference"><a href="#cite_note-pmid21093580-8"><span class="cite-bracket">[</span>8<span class="cite-bracket">]</span></a></sup><sup id="cite_ref-pmid14620878_9-0" class="reference"><a href="#cite_note-pmid14620878-9"><span class="cite-bracket">[</span>9<span class="cite-bracket">]</span></a></sup> Ez a hatás visszafordítható, ha a receptorgént tartalmazó prekurzor idegsejtet juttatnak az egér hippokampuszába és hagyják azokat <a href="/wiki/Asztrocita" title="Asztrocita">asztrocitákká</a> differenciálódni.<sup id="cite_ref-pmid21093580_8-1" class="reference"><a href="#cite_note-pmid21093580-8"><span class="cite-bracket">[</span>8<span class="cite-bracket">]</span></a></sup> </p> <div class="mw-heading mw-heading3"><h3 id="Interleukin-2">Interleukin-2</h3><span class="mw-editsection"><span class="mw-editsection-bracket">[</span><a href="/w/index.php?title=Interleukin&action=edit&section=4" title="Szakasz szerkesztése: Interleukin-2"><span>szerkesztés</span></a><span class="mw-editsection-bracket">]</span></span></div> <p>Az <a href="/w/index.php?title=Interleukin-2&action=edit&redlink=1" class="new" title="Interleukin-2 (a lap nem létezik)">interleukin-2</a>-t <a href="/wiki/Lektin" class="mw-redirect" title="Lektin">lektinnel</a> vagy antigénnel stimulált T-limfociták termelik és különféle élettani hatásokat indukál. Megindítja a receptorával rendelkező T-sejtek proliferációját, egyes B-sejtek esetében pedig növekedési faktorként, valamint az ellenanyagtermelés beindítójaként viselkedik.<sup id="cite_ref-PUB00004618_10-0" class="reference"><a href="#cite_note-PUB00004618-10"><span class="cite-bracket">[</span>10<span class="cite-bracket">]</span></a></sup><sup id="cite_ref-PUB00000368_11-0" class="reference"><a href="#cite_note-PUB00000368-11"><span class="cite-bracket">[</span>11<span class="cite-bracket">]</span></a></sup> Szerkezete alapján hasonlít az IL-4-hez és a <a href="/w/index.php?title=Granulocita-makrof%C3%A1g_kol%C3%B3niastimul%C3%A1l%C3%B3_faktor&action=edit&redlink=1" class="new" title="Granulocita-makrofág kolóniastimuláló faktor (a lap nem létezik)">granulocita-makrofág kolóniastimuláló faktorhoz</a> (GMCSF).<sup id="cite_ref-PUB00000368_11-1" class="reference"><a href="#cite_note-PUB00000368-11"><span class="cite-bracket">[</span>11<span class="cite-bracket">]</span></a></sup> </p> <div class="mw-heading mw-heading3"><h3 id="Interleukin-3">Interleukin-3</h3><span class="mw-editsection"><span class="mw-editsection-bracket">[</span><a href="/w/index.php?title=Interleukin&action=edit&section=5" title="Szakasz szerkesztése: Interleukin-3"><span>szerkesztés</span></a><span class="mw-editsection-bracket">]</span></span></div> <p>Az <a href="/w/index.php?title=Interleukin-3&action=edit&redlink=1" class="new" title="Interleukin-3 (a lap nem létezik)">IL-3</a> a hematopoiézist (a vérsejtek képződését) azon belül a <a href="/w/index.php?title=Granulocita&action=edit&redlink=1" class="new" title="Granulocita (a lap nem létezik)">granulociták</a> és <a href="/wiki/Makrof%C3%A1g" title="Makrofág">makrofágok</a> differenciálódását ás működését szabályozza.<sup id="cite_ref-PUB00001758_12-0" class="reference"><a href="#cite_note-PUB00001758-12"><span class="cite-bracket">[</span>12<span class="cite-bracket">]</span></a></sup><sup id="cite_ref-PUB00003991_13-0" class="reference"><a href="#cite_note-PUB00003991-13"><span class="cite-bracket">[</span>13<span class="cite-bracket">]</span></a></sup> A molekulát aktivált T-sejtek és <a href="/w/index.php?title=H%C3%ADz%C3%B3sejt&action=edit&redlink=1" class="new" title="Hízósejt (a lap nem létezik)">hízósejtek</a> állítják elő;<sup id="cite_ref-PUB00001758_12-1" class="reference"><a href="#cite_note-PUB00001758-12"><span class="cite-bracket">[</span>12<span class="cite-bracket">]</span></a></sup><sup id="cite_ref-PUB00003991_13-1" class="reference"><a href="#cite_note-PUB00003991-13"><span class="cite-bracket">[</span>13<span class="cite-bracket">]</span></a></sup> aktiváláshoz a transzláció után egy N-terminális szignálszakasz levágása szükséges.<sup id="cite_ref-PUB00003991_13-2" class="reference"><a href="#cite_note-PUB00003991-13"><span class="cite-bracket">[</span>13<span class="cite-bracket">]</span></a></sup> </p><p>Az egészséges T-limfociták (valamint a T-sejtes limfómák sejtjei) csak antigénekkel, mitogénekkel vagy kémiailag történő aktiválás után szintetizálnak IL-3-at. A mielomonocitikus leukémia sejtjei folyamatosan választják ki a molekulát, feltételezések szerint ez az állapot okozza a leukémia kialakulását.<sup id="cite_ref-PUB00003991_13-3" class="reference"><a href="#cite_note-PUB00003991-13"><span class="cite-bracket">[</span>13<span class="cite-bracket">]</span></a></sup> </p> <div class="mw-heading mw-heading3"><h3 id="Interleukin-4">Interleukin-4</h3><span class="mw-editsection"><span class="mw-editsection-bracket">[</span><a href="/w/index.php?title=Interleukin&action=edit&section=6" title="Szakasz szerkesztése: Interleukin-4"><span>szerkesztés</span></a><span class="mw-editsection-bracket">]</span></span></div> <p>Az <a href="/w/index.php?title=Interleukin-4&action=edit&redlink=1" class="new" title="Interleukin-4 (a lap nem létezik)">IL-4</a>-et a CD4<sup>+</sup> T-sejtek bocsátják ki, amelyek a B-sejtek osztódását és differenciálódását segítik. </p> <div class="mw-heading mw-heading3"><h3 id="Interleukin_5">Interleukin 5</h3><span class="mw-editsection"><span class="mw-editsection-bracket">[</span><a href="/w/index.php?title=Interleukin&action=edit&section=7" title="Szakasz szerkesztése: Interleukin 5"><span>szerkesztés</span></a><span class="mw-editsection-bracket">]</span></span></div> <p>Az <a href="/w/index.php?title=Interleukin-5&action=edit&redlink=1" class="new" title="Interleukin-5 (a lap nem létezik)">IL-5</a> (vagy eozinofil differenciációs faktor, EDF) az <a href="/wiki/Eozinofil_granulocita" title="Eozinofil granulocita">eozinofil granulociták</a> növekedését és aktivációját segíti,<sup id="cite_ref-PUB00004643_14-0" class="reference"><a href="#cite_note-PUB00004643-14"><span class="cite-bracket">[</span>14<span class="cite-bracket">]</span></a></sup><sup id="cite_ref-PUB00004643_14-1" class="reference"><a href="#cite_note-PUB00004643-14"><span class="cite-bracket">[</span>14<span class="cite-bracket">]</span></a></sup><sup id="cite_ref-PUB00004146_15-0" class="reference"><a href="#cite_note-PUB00004146-15"><span class="cite-bracket">[</span>15<span class="cite-bracket">]</span></a></sup> így fontos szerepet játszik azokban a betegségekben, ahol az eozinofilek száma emelkedett (pl. <a href="/wiki/Asztma" title="Asztma">asztma</a>).<sup id="cite_ref-PUB00004146_15-1" class="reference"><a href="#cite_note-PUB00004146-15"><span class="cite-bracket">[</span>15<span class="cite-bracket">]</span></a></sup> </p> <div class="mw-heading mw-heading3"><h3 id="Interleukin-6">Interleukin-6</h3><span class="mw-editsection"><span class="mw-editsection-bracket">[</span><a href="/w/index.php?title=Interleukin&action=edit&section=8" title="Szakasz szerkesztése: Interleukin-6"><span>szerkesztés</span></a><span class="mw-editsection-bracket">]</span></span></div> <p>Az <a href="/w/index.php?title=Interleukin-6&action=edit&redlink=1" class="new" title="Interleukin-6 (a lap nem létezik)">IL-6</a> (vagy B-sejt stimulációs faktor-2 és interferon béta-2) számos biológiai hatással rendelkezik.<sup id="cite_ref-PUB00003997_16-0" class="reference"><a href="#cite_note-PUB00003997-16"><span class="cite-bracket">[</span>16<span class="cite-bracket">]</span></a></sup> Alapvető szerepe van a B-sejtek végső differenciálódásban, hogy ellenanyagtermelő sejtekké váljanak; de fokozza a <a href="/wiki/Miel%C3%B3ma_multiplex" title="Mielóma multiplex">mielómák</a> és <a href="/w/index.php?title=Plazmacit%C3%B3ma&action=edit&redlink=1" class="new" title="Plazmacitóma (a lap nem létezik)">plazmacitómák</a> növekedését, az idegsejtek differenciálódását, <a href="/w/index.php?title=Hepatocita&action=edit&redlink=1" class="new" title="Hepatocita (a lap nem létezik)">hepatocitákban</a> pedig az akut fázisú reagensek (gyulladásjelző markerek) termelését.<sup id="cite_ref-PUB00003997_16-1" class="reference"><a href="#cite_note-PUB00003997-16"><span class="cite-bracket">[</span>16<span class="cite-bracket">]</span></a></sup><sup id="cite_ref-PUB00001608_17-0" class="reference"><a href="#cite_note-PUB00001608-17"><span class="cite-bracket">[</span>17<span class="cite-bracket">]</span></a></sup> </p><p>Proteinszekvenciája alapján több más citokin hasonlít az IL-6-ra;<sup id="cite_ref-PUB00003997_16-2" class="reference"><a href="#cite_note-PUB00003997-16"><span class="cite-bracket">[</span>16<span class="cite-bracket">]</span></a></sup><sup id="cite_ref-PUB00001608_17-1" class="reference"><a href="#cite_note-PUB00001608-17"><span class="cite-bracket">[</span>17<span class="cite-bracket">]</span></a></sup><sup id="cite_ref-PUB00000150_18-0" class="reference"><a href="#cite_note-PUB00000150-18"><span class="cite-bracket">[</span>18<span class="cite-bracket">]</span></a></sup> ide tartozik a <a href="/w/index.php?title=Granulocita_kol%C3%B3niastimul%C3%A1l%C3%B3_faktor&action=edit&redlink=1" class="new" title="Granulocita kolóniastimuláló faktor (a lap nem létezik)">granulocita kolóniastimuláló faktor</a> (GCSF) és a mielomonocitikus növekedési faktor (MGF). Mindkettő egyes vérsejtek képződését irányítja.<sup id="cite_ref-PUB00000150_18-1" class="reference"><a href="#cite_note-PUB00000150-18"><span class="cite-bracket">[</span>18<span class="cite-bracket">]</span></a></sup> Ennek a citokincsaládnak a tagjai 170-180 aminosavból álló glikoproteinek, amelyekben négy <a href="/wiki/Cisztein" title="Cisztein">cisztein</a> két diszulfid kötéssel stabilizálja a szerkezetet.<sup id="cite_ref-PUB00000150_18-2" class="reference"><a href="#cite_note-PUB00000150-18"><span class="cite-bracket">[</span>18<span class="cite-bracket">]</span></a></sup> Alakjuk a többi interleukinhez hasonlóan kompakt, globuláris.<sup id="cite_ref-PUB00002742_19-0" class="reference"><a href="#cite_note-PUB00002742-19"><span class="cite-bracket">[</span>19<span class="cite-bracket">]</span></a></sup> </p> <div class="mw-heading mw-heading3"><h3 id="Interleukin-7">Interleukin-7</h3><span class="mw-editsection"><span class="mw-editsection-bracket">[</span><a href="/w/index.php?title=Interleukin&action=edit&section=9" title="Szakasz szerkesztése: Interleukin-7"><span>szerkesztés</span></a><span class="mw-editsection-bracket">]</span></span></div> <p>Az <a href="/w/index.php?title=Interleukin-7&action=edit&redlink=1" class="new" title="Interleukin-7 (a lap nem létezik)">IL-7</a> a B- és T-sejtek korai fejlődési szakaszának növekedési faktora.<sup id="cite_ref-PUB00002018_20-0" class="reference"><a href="#cite_note-PUB00002018-20"><span class="cite-bracket">[</span>20<span class="cite-bracket">]</span></a></sup> </p> <div class="mw-heading mw-heading3"><h3 id="Interleukin-8">Interleukin-8</h3><span class="mw-editsection"><span class="mw-editsection-bracket">[</span><a href="/w/index.php?title=Interleukin&action=edit&section=10" title="Szakasz szerkesztése: Interleukin-8"><span>szerkesztés</span></a><span class="mw-editsection-bracket">]</span></span></div> <p>Az <a href="/w/index.php?title=Interleukin-8&action=edit&redlink=1" class="new" title="Interleukin-8 (a lap nem létezik)">IL-8</a>-at makrofágok, <a href="/wiki/H%C3%A1msz%C3%B6vet" title="Hámszövet">epiteliális</a> és endoteliális sejtek, valamint a légjáratok simaizomsejtjei termelik<sup id="cite_ref-pmid10873157_21-0" class="reference"><a href="#cite_note-pmid10873157-21"><span class="cite-bracket">[</span>21<span class="cite-bracket">]</span></a></sup> Az endoteliális sejtek vezikulumokban, az ún. Weibel-Palade testekben tárolják az IL-8-at.<sup id="cite_ref-pmid9802987_22-0" class="reference"><a href="#cite_note-pmid9802987-22"><span class="cite-bracket">[</span>22<span class="cite-bracket">]</span></a></sup><sup id="cite_ref-pmid9802986_23-0" class="reference"><a href="#cite_note-pmid9802986-23"><span class="cite-bracket">[</span>23<span class="cite-bracket">]</span></a></sup> Az ember genomjában az <i>CXCL8</i> gén kódolja.<sup id="cite_ref-pmid1967588_24-0" class="reference"><a href="#cite_note-pmid1967588-24"><span class="cite-bracket">[</span>24<span class="cite-bracket">]</span></a></sup> A génről egy 99 aminosavból álló prekurzor peptid íródik át, amelyből ezután utólagos módosulások által különféle aktív izoform változatok jönnek létre.<sup id="cite_ref-Brat_DJ_2005._pages_122-133_25-0" class="reference"><a href="#cite_note-Brat_DJ_2005._pages_122-133-25"><span class="cite-bracket">[</span>25<span class="cite-bracket">]</span></a></sup> A makrofágok sejtkultúrában alapvetően egy 72 aminosavból álló változatot választanak ki.<sup id="cite_ref-Brat_DJ_2005._pages_122-133_25-1" class="reference"><a href="#cite_note-Brat_DJ_2005._pages_122-133-25"><span class="cite-bracket">[</span>25<span class="cite-bracket">]</span></a></sup> </p><p>Elsődleges feladata a <a href="/wiki/Immunrendszer#Veleszületett_immunitás" title="Immunrendszer">veleszületett</a> immunválasz szabályozása. Több receptora is van, legismertebbek a CXCR1 és a CXCR2 (az előbbi gyakoribb és hatékonyabb is). </p> <div class="mw-heading mw-heading3"><h3 id="Interleukin-9">Interleukin-9</h3><span class="mw-editsection"><span class="mw-editsection-bracket">[</span><a href="/w/index.php?title=Interleukin&action=edit&section=11" title="Szakasz szerkesztése: Interleukin-9"><span>szerkesztés</span></a><span class="mw-editsection-bracket">]</span></span></div> <p>Az <a href="/w/index.php?title=Interleukin-9&action=edit&redlink=1" class="new" title="Interleukin-9 (a lap nem létezik)">IL-9</a> a helper T-sejtek IL-2 és IL-4-független fejlődését segíti elő.<sup id="cite_ref-PUB00003177_26-0" class="reference"><a href="#cite_note-PUB00003177-26"><span class="cite-bracket">[</span>26<span class="cite-bracket">]</span></a></sup> Közeli szerkezeti rokonai az IL-2 és az IL-15.<sup id="cite_ref-27" class="reference"><a href="#cite_note-27"><span class="cite-bracket">[</span>27<span class="cite-bracket">]</span></a></sup> </p> <div class="mw-heading mw-heading3"><h3 id="Interleukin-10">Interleukin-10</h3><span class="mw-editsection"><span class="mw-editsection-bracket">[</span><a href="/w/index.php?title=Interleukin&action=edit&section=12" title="Szakasz szerkesztése: Interleukin-10"><span>szerkesztés</span></a><span class="mw-editsection-bracket">]</span></span></div> <p>Az <a href="/w/index.php?title=Interleukin-10&action=edit&redlink=1" class="new" title="Interleukin-10 (a lap nem létezik)">IL-10</a> más, gyulladást elősegítő citokinek termelését gátolja. Célpontjai közé tartozik az aktivált makrofágok és helper T-sejtek által szekretált <a href="/w/index.php?title=Interferon-gamma&action=edit&redlink=1" class="new" title="Interferon-gamma (a lap nem létezik)">interferon-gamma</a>, az IL-2, az IL-3, a <a href="/wiki/Tumornekr%C3%B3zis-faktor_%CE%B1" title="Tumornekrózis-faktor α">tumornekrózis-faktor α</a> és a GMCSF. Szerkezetét tekintve egy 160 aminosavból álló peptid, melynek szerkezetét négy konzervatív helyen lévő cisztein és diszulfid kötéseik stabilizálják.<sup id="cite_ref-PUB00005262_28-0" class="reference"><a href="#cite_note-PUB00005262-28"><span class="cite-bracket">[</span>28<span class="cite-bracket">]</span></a></sup> Nagyon hasonlít az IL-10-hez az <a href="/wiki/Epstein%E2%80%93Barr-v%C3%ADrus" title="Epstein–Barr-vírus">Epstein-Barr vírus</a> BCRF1 proteinje és a ló herpeszvírus 2 E7 proteinje, amelyek szintén az antivirális immunválaszt indukáló interferon-gammát semlegesítik.<sup id="cite_ref-PUB00004497_29-0" class="reference"><a href="#cite_note-PUB00004497-29"><span class="cite-bracket">[</span>29<span class="cite-bracket">]</span></a></sup> </p> <div class="mw-heading mw-heading3"><h3 id="Interleukin-11">Interleukin-11</h3><span class="mw-editsection"><span class="mw-editsection-bracket">[</span><a href="/w/index.php?title=Interleukin&action=edit&section=13" title="Szakasz szerkesztése: Interleukin-11"><span>szerkesztés</span></a><span class="mw-editsection-bracket">]</span></span></div> <p>Az <a href="/w/index.php?title=Interleukin-11&action=edit&redlink=1" class="new" title="Interleukin-11 (a lap nem létezik)">IL-11</a> elősegíti a <a href="/w/index.php?title=Megakariocita&action=edit&redlink=1" class="new" title="Megakariocita (a lap nem létezik)">megakariociták</a> fejlődését, amelyekből aztán <a href="/wiki/V%C3%A9rlemezke" class="mw-redirect" title="Vérlemezke">vérlemezkék</a> lesznek, aktiválja a csontot lebontó <a href="/wiki/Oszteoklaszt" title="Oszteoklaszt">oszteoklasztokat</a> és gátolja a nyálkahártyák epitéliumában a sejtek növekedését.<sup id="cite_ref-PUB00013131_30-0" class="reference"><a href="#cite_note-PUB00013131-30"><span class="cite-bracket">[</span>30<span class="cite-bracket">]</span></a></sup> </p> <div class="mw-heading mw-heading3"><h3 id="Interleukin-12">Interleukin-12</h3><span class="mw-editsection"><span class="mw-editsection-bracket">[</span><a href="/w/index.php?title=Interleukin&action=edit&section=14" title="Szakasz szerkesztése: Interleukin-12"><span>szerkesztés</span></a><span class="mw-editsection-bracket">]</span></span></div> <p>Az <a href="/w/index.php?title=Interleukin-12&action=edit&redlink=1" class="new" title="Interleukin-12 (a lap nem létezik)">IL-12</a> diszulfidkötésekkel összetartott heterodimer, vagyis egy 35 kilodaltonos alfa és egy 40 kilodaltonos béta alegységből tevődik össze. Feladata a Th1 sejtes immunválasz stimulációja, vagyis az olyan kórokozók elleni védekezés, mint a <a href="/w/index.php?title=Leishmania&action=edit&redlink=1" class="new" title="Leishmania (a lap nem létezik)">Leishmania</a>, <a href="/wiki/Toxoplasma_gondii" title="Toxoplasma gondii">Toxoplasma</a>, <a href="/wiki/Kanyar%C3%B3" title="Kanyaró">kanyaróvírus</a> vagy <a href="/wiki/HIV" title="HIV">HIV</a>. Ezenkívül fokozza a <a href="/w/index.php?title=Term%C3%A9szetes_%C3%B6l%C5%91sejtek&action=edit&redlink=1" class="new" title="Természetes ölősejtek (a lap nem létezik)">természetes ölősejtek</a> hatékonyságát;<sup id="cite_ref-Abbas_2012_31-0" class="reference"><a href="#cite_note-Abbas_2012-31"><span class="cite-bracket">[</span>31<span class="cite-bracket">]</span></a></sup><sup id="cite_ref-pmid18619507_32-0" class="reference"><a href="#cite_note-pmid18619507-32"><span class="cite-bracket">[</span>32<span class="cite-bracket">]</span></a></sup> sajnos az autoimmun folyamatokban is (mint a <a href="/wiki/Sclerosis_multiplex" title="Sclerosis multiplex">sclerosis multiplex</a> vagy autoimmun bélgyulladás). Utóbbi esetben az IL-12 gátlásával enyhíthetőek a tünetek. A rendellenes Th2 immunválasszal járó kórképekben viszont segíthet a rekombináns IL-12 beadása.<sup id="cite_ref-PUB00007461_33-0" class="reference"><a href="#cite_note-PUB00007461-33"><span class="cite-bracket">[</span>33<span class="cite-bracket">]</span></a></sup><sup id="cite_ref-PUB00007462_34-0" class="reference"><a href="#cite_note-PUB00007462-34"><span class="cite-bracket">[</span>34<span class="cite-bracket">]</span></a></sup> </p> <div class="mw-heading mw-heading3"><h3 id="Interleukin-13">Interleukin-13</h3><span class="mw-editsection"><span class="mw-editsection-bracket">[</span><a href="/w/index.php?title=Interleukin&action=edit&section=15" title="Szakasz szerkesztése: Interleukin-13"><span>szerkesztés</span></a><span class="mw-editsection-bracket">]</span></span></div> <p>Az <a href="/w/index.php?title=Interleukin-13&action=edit&redlink=1" class="new" title="Interleukin-13 (a lap nem létezik)">IL-13</a> többféle hatással rendelkező citokin, amely az immunválasz és a gyulladás szabályozásában játszik szerepet.<sup id="cite_ref-PUB00004143_35-0" class="reference"><a href="#cite_note-PUB00004143-35"><span class="cite-bracket">[</span>35<span class="cite-bracket">]</span></a></sup> Gátolja a gyulladást elősegítő citokineket és az IL-2-vel együtt szabályozza az interferon-gamma szintézist. Szerkezete kissé hasonlít az IL-4-ére.<sup id="cite_ref-pmid26689873_36-0" class="reference"><a href="#cite_note-pmid26689873-36"><span class="cite-bracket">[</span>36<span class="cite-bracket">]</span></a></sup> </p> <div class="mw-heading mw-heading3"><h3 id="Interleukin-15">Interleukin-15</h3><span class="mw-editsection"><span class="mw-editsection-bracket">[</span><a href="/w/index.php?title=Interleukin&action=edit&section=16" title="Szakasz szerkesztése: Interleukin-15"><span>szerkesztés</span></a><span class="mw-editsection-bracket">]</span></span></div> <p>Az <a href="/w/index.php?title=Interleukin-15&action=edit&redlink=1" class="new" title="Interleukin-15 (a lap nem létezik)">IL-15</a>-nek is több funkciója van: stimulálja a sejtes immunválaszt<sup id="cite_ref-PUB00009488_37-0" class="reference"><a href="#cite_note-PUB00009488-37"><span class="cite-bracket">[</span>37<span class="cite-bracket">]</span></a></sup> és a T-limfociták proliferációját. </p> <div class="mw-heading mw-heading3"><h3 id="Interleukin-17">Interleukin-17</h3><span class="mw-editsection"><span class="mw-editsection-bracket">[</span><a href="/w/index.php?title=Interleukin&action=edit&section=17" title="Szakasz szerkesztése: Interleukin-17"><span>szerkesztés</span></a><span class="mw-editsection-bracket">]</span></span></div> <p>Az <a href="/w/index.php?title=Interleukin-17&action=edit&redlink=1" class="new" title="Interleukin-17 (a lap nem létezik)">IL-17</a>-et az aktivált memória T-sejtek bocsátják ki és a <a href="/wiki/Neutrofil_granulocita" title="Neutrofil granulocita">neutrofil granulociták</a> stimulálásával gyulladást indukál.<sup id="cite_ref-PUB00012254_38-0" class="reference"><a href="#cite_note-PUB00012254-38"><span class="cite-bracket">[</span>38<span class="cite-bracket">]</span></a></sup> Számos autoimmun betegség lefolyásában is szerepet játszik (allergiák, asztma, <a href="/wiki/Pikkelys%C3%B6m%C3%B6r" title="Pikkelysömör">pszoriázis</a>, stb.) Egyes kutatások szerint a tumorok kialakulásában és az átültett szervek kilökődésében is részt vesz.<sup id="cite_ref-39" class="reference"><a href="#cite_note-39"><span class="cite-bracket">[</span>39<span class="cite-bracket">]</span></a></sup> </p> <div class="mw-heading mw-heading2"><h2 id="Interleukinok_az_emberben">Interleukinok az emberben</h2><span class="mw-editsection"><span class="mw-editsection-bracket">[</span><a href="/w/index.php?title=Interleukin&action=edit&section=18" title="Szakasz szerkesztése: Interleukinok az emberben"><span>szerkesztés</span></a><span class="mw-editsection-bracket">]</span></span></div> <table class="wikitable"> <tbody><tr> <th rowspan="2">Név </th> <th rowspan="2">Termelő<sup id="cite_ref-Immunology68Unless_40-0" class="reference"><a href="#cite_note-Immunology68Unless-40"><span class="cite-bracket">[</span>40<span class="cite-bracket">]</span></a></sup> </th> <th colspan="2">Célpont </th> <th rowspan="2">Funkció<sup id="cite_ref-Immunology68Unless_40-1" class="reference"><a href="#cite_note-Immunology68Unless-40"><span class="cite-bracket">[</span>40<span class="cite-bracket">]</span></a></sup> </th></tr> <tr> <th>Receptor<sup id="cite_ref-Immunology68Unless_40-2" class="reference"><a href="#cite_note-Immunology68Unless-40"><span class="cite-bracket">[</span>40<span class="cite-bracket">]</span></a></sup><sup id="cite_ref-title_41-0" class="reference"><a href="#cite_note-title-41"><span class="cite-bracket">[</span>41<span class="cite-bracket">]</span></a></sup> </th> <th>Sejt<sup id="cite_ref-Immunology68Unless_40-3" class="reference"><a href="#cite_note-Immunology68Unless-40"><span class="cite-bracket">[</span>40<span class="cite-bracket">]</span></a></sup> </th></tr> <tr> <td rowspan="4"><a href="/w/index.php?title=Interleukin-1&action=edit&redlink=1" class="new" title="Interleukin-1 (a lap nem létezik)">IL-1</a> </td> <td rowspan="4"><a href="/wiki/Makrof%C3%A1g" title="Makrofág">makrofágok</a>, <a href="/wiki/B-limfocita" title="B-limfocita">B-sejtek</a>, <a href="/wiki/Monocita" title="Monocita">monociták</a>,<sup id="cite_ref-arizona_42-0" class="reference"><a href="#cite_note-arizona-42"><span class="cite-bracket">[</span>42<span class="cite-bracket">]</span></a></sup> <a href="/wiki/Dendritikus_sejt" title="Dendritikus sejt">dendritikus sejtek</a><sup id="cite_ref-arizona_42-1" class="reference"><a href="#cite_note-arizona-42"><span class="cite-bracket">[</span>42<span class="cite-bracket">]</span></a></sup> </td> <td rowspan="4">CD121a/IL1R1, CD121b/IL1R2 </td> <td><a href="/wiki/T-limfocita#Helper_T-sejtek" title="T-limfocita">Helper T-sejtek</a></td> <td>stimuláció <sup id="cite_ref-arizona_42-2" class="reference"><a href="#cite_note-arizona-42"><span class="cite-bracket">[</span>42<span class="cite-bracket">]</span></a></sup> </td></tr> <tr> <td><a href="/wiki/B-limfocita" title="B-limfocita">B-sejtek</a></td> <td>érés és proliferáció <sup id="cite_ref-arizona_42-3" class="reference"><a href="#cite_note-arizona-42"><span class="cite-bracket">[</span>42<span class="cite-bracket">]</span></a></sup> </td></tr> <tr> <td><a href="/w/index.php?title=Term%C3%A9szetes_%C3%B6l%C5%91sejtek&action=edit&redlink=1" class="new" title="Természetes ölősejtek (a lap nem létezik)">Természetes ölősejtek</a></td> <td>aktiváció<sup id="cite_ref-arizona_42-4" class="reference"><a href="#cite_note-arizona-42"><span class="cite-bracket">[</span>42<span class="cite-bracket">]</span></a></sup> </td></tr> <tr> <td>makrofágok, endotélium, egyéb</td> <td><a href="/wiki/Gyullad%C3%A1s" title="Gyulladás">gyulladás</a>,<sup id="cite_ref-arizona_42-5" class="reference"><a href="#cite_note-arizona-42"><span class="cite-bracket">[</span>42<span class="cite-bracket">]</span></a></sup> kis mennyiségbewn akut fázisú reakciót, nagyban lázat okoz </td></tr> <tr> <td><a href="/w/index.php?title=Interleukin-2&action=edit&redlink=1" class="new" title="Interleukin-2 (a lap nem létezik)">IL-2</a></td> <td>Th1-sejtek</td> <td>CD25/IL2RA, CD122/IL2RB, CD132/IL2RG</td> <td>aktivált<sup id="cite_ref-arizona_42-6" class="reference"><a href="#cite_note-arizona-42"><span class="cite-bracket">[</span>42<span class="cite-bracket">]</span></a></sup> T-sejtek és B-sejtek, természetes ölősejtek, makrofágok, <a href="/w/index.php?title=Oligodendrocita&action=edit&redlink=1" class="new" title="Oligodendrocita (a lap nem létezik)">oligodendrociták</a></td> <td>stimulálja a T-sejtek fejlődését és differenciálódását. Immunterápiában alkalmazzák rákos vagy szervátültetés miatt szupresszált immunrendszerű betegeknél. HIV-pozitív betegek esetében a CD4+ T-sejtek számának növelésére használhatják. </td></tr> <tr> <td rowspan="2"><a href="/w/index.php?title=Interleukin-3&action=edit&redlink=1" class="new" title="Interleukin-3 (a lap nem létezik)">IL-3</a></td> <td rowspan="2">aktivált T-sejtek,<sup id="cite_ref-arizona_42-7" class="reference"><a href="#cite_note-arizona-42"><span class="cite-bracket">[</span>42<span class="cite-bracket">]</span></a></sup> <a href="/w/index.php?title=H%C3%ADz%C3%B3sejt&action=edit&redlink=1" class="new" title="Hízósejt (a lap nem létezik)">hízósejtek</a>, természetes ölősejtek, endoteliális sejtek, <a href="/wiki/Eozinofil_granulocita" title="Eozinofil granulocita">eozinofilok</a></td> <td rowspan="2">IL3RA|CD123/IL3RA, CSF2RB|CD131/IL3RB</td> <td>hematopoietikus őssejtek</td> <td>a mieloid őssejtek differenciálódása és proliferációja<sup id="cite_ref-arizona_42-8" class="reference"><a href="#cite_note-arizona-42"><span class="cite-bracket">[</span>42<span class="cite-bracket">]</span></a></sup> pl. eritrocitává vagy granulocitává </td></tr> <tr> <td>hízósejtek</td> <td>növekedés, <a href="/wiki/Hisztamin" title="Hisztamin">hisztaminkibocsátás</a><sup id="cite_ref-arizona_42-9" class="reference"><a href="#cite_note-arizona-42"><span class="cite-bracket">[</span>42<span class="cite-bracket">]</span></a></sup> </td></tr> <tr> <td rowspan="3"><a href="/w/index.php?title=Interleukin-4&action=edit&redlink=1" class="new" title="Interleukin-4 (a lap nem létezik)">IL-4</a></td> <td rowspan="3">Th2-sejtek, frissen aktivált naív CD4+ sejtek, memória CD4+ sejtek, hízósejtek, makrofágok</td> <td rowspan="3">CD124/IL4R, CD132/IL2RG</td> <td>aktivált B-sejtek</td> <td>proliferáció és differenciáció, IgG1 és IgE szintézis.<sup id="cite_ref-arizona_42-10" class="reference"><a href="#cite_note-arizona-42"><span class="cite-bracket">[</span>42<span class="cite-bracket">]</span></a></sup> Fontos szerepet játszik az allergiás immunválaszban (IgE) </td></tr> <tr> <td>T-sejtek</td> <td>proliferáció<sup id="cite_ref-arizona_42-11" class="reference"><a href="#cite_note-arizona-42"><span class="cite-bracket">[</span>42<span class="cite-bracket">]</span></a></sup> </td></tr> <tr> <td><a href="/w/index.php?title=Endot%C3%A9lium&action=edit&redlink=1" class="new" title="Endotélium (a lap nem létezik)">endotélium</a></td> <td>A vaszkuláris sejt adhéziós molekula (VCAM-1) termelés fokozásával a limfociták tapadásának elősegítése.<sup id="cite_ref-43" class="reference"><a href="#cite_note-43"><span class="cite-bracket">[</span>43<span class="cite-bracket">]</span></a></sup> </td></tr> <tr> <td rowspan="2"><a href="/w/index.php?title=Interleukin-5&action=edit&redlink=1" class="new" title="Interleukin-5 (a lap nem létezik)">IL-5</a></td> <td rowspan="2">Th2-sejtek, hízósejtek, eozinofilok</td> <td rowspan="2">CD125/IL5RA, CD131/IL3RB</td> <td>eozinofilok</td> <td>termelés </td></tr> <tr> <td>B-sejtek</td> <td>differenciáció, IgA-termelés </td></tr> <tr> <td rowspan="4"><a href="/w/index.php?title=Interleukin-6&action=edit&redlink=1" class="new" title="Interleukin-6 (a lap nem létezik)">IL-6</a></td> <td rowspan="4">makrofágok, Th2 sejtek, B-sejtek, <a href="/wiki/Asztrocita" title="Asztrocita">asztrociták</a>, endotélium</td> <td rowspan="4">CD126/IL6RA, CD130/IR6RB</td> <td>aktivált B-sejtek</td> <td><a href="/w/index.php?title=Plazmasejt&action=edit&redlink=1" class="new" title="Plazmasejt (a lap nem létezik)">plazmasejtekké</a> való fejlődés </td></tr> <tr> <td>plazmasejtek</td> <td>ellenanyagkiválasztás </td></tr> <tr> <td>hematopoietikus őssejtek</td> <td>differenciáció </td></tr> <tr> <td>T-sejtek</td> <td>az akut fázis reakciót indukálja, vérképzés, sejtdifferenciáció, gyulladás </td></tr> <tr> <td><a href="/w/index.php?title=Interleukin-7&action=edit&redlink=1" class="new" title="Interleukin-7 (a lap nem létezik)">IL-7</a></td> <td>csontvelői sztrómasejtek és tímusz sztrómasejtek</td> <td>CD127/IL7RA, CD132/IL2RG</td> <td>pre-B-sejtek, pre-T-sejtek, természetes ölősejtek (NK-sejtek)</td> <td>a fehérvérsejtek differenciációja és proliferációja, B-, T- és NK-sejtek, fejlődése, fennmaradása </td></tr> <tr> <td><a href="/w/index.php?title=Interleukin-8&action=edit&redlink=1" class="new" title="Interleukin-8 (a lap nem létezik)">IL-8</a> vagy CXCL8</td> <td>makrofágok, limfociták, epiteliális sejtek, endoteliális sejtek</td> <td>CXCR1/IL8RA, CXCR2/IL8RB/CD128</td> <td><a href="/wiki/Neutrofil_granulocita" title="Neutrofil granulocita">neutrofilok</a>, <a href="/wiki/Bazofil_granulocita" title="Bazofil granulocita">bazofilok</a>, limfociták</td> <td>Neutrofil <a href="/wiki/Kemotaxis" title="Kemotaxis">kemotaxis</a> </td></tr> <tr> <td><a href="/w/index.php?title=Interleukin-9&action=edit&redlink=1" class="new" title="Interleukin-9 (a lap nem létezik)">IL-9</a></td> <td>Th2 sejtek, CD4+ helper sejtek</td> <td>CD129/IL9R</td> <td>T-sejtek, B-sejtek</td> <td>IgM, IgG, IgE termelés, stimulálja a hízósejteket </td></tr> <tr> <td rowspan="5"><a href="/w/index.php?title=Interleukin-10&action=edit&redlink=1" class="new" title="Interleukin-10 (a lap nem létezik)">IL-10</a></td> <td rowspan="5">monociták, Th2 sejtek, CD8+ T-sejtek, hízósejtek, makrofágok, egyes B-sejtek</td> <td rowspan="5">CD210/IL10RA, CDW210B/IL10RB</td> <td>makrofágok</td> <td>citokintermelés<sup id="cite_ref-arizona_42-12" class="reference"><a href="#cite_note-arizona-42"><span class="cite-bracket">[</span>42<span class="cite-bracket">]</span></a></sup> </td></tr> <tr> <td>B-sejtek</td> <td>aktiváció <sup id="cite_ref-arizona_42-13" class="reference"><a href="#cite_note-arizona-42"><span class="cite-bracket">[</span>42<span class="cite-bracket">]</span></a></sup> </td></tr> <tr> <td>hízósejtek</td> <td> </td></tr> <tr> <td>Th1 sejtek</td> <td>gátolja a Th1 sejtek citokintermelését (IFN-γ, TNF-β, IL-2) </td></tr> <tr> <td>Th2 sejtek</td> <td>stimuláció </td></tr> <tr> <td><a href="/w/index.php?title=Interleukin-11&action=edit&redlink=1" class="new" title="Interleukin-11 (a lap nem létezik)">IL-11</a></td> <td>csontvelő-sztróma</td> <td>IL11RA</td> <td>csontvelő-sztróma</td> <td>akut fázis proteinek termelése, <a href="/wiki/Oszteoklaszt" title="Oszteoklaszt">oszteoklasztok</a> képződése </td></tr> <tr> <td rowspan="2"><a href="/w/index.php?title=Interleukin-12&action=edit&redlink=1" class="new" title="Interleukin-12 (a lap nem létezik)">IL-12</a></td> <td rowspan="2"><a href="/wiki/Dendritikus_sejt" title="Dendritikus sejt">dendritikus sejtek</a>, B-sejtek, T-sejtek, makrofágok</td> <td rowspan="2">CD212/IL12RB1, IR12RB2</td> <td>aktivált <sup id="cite_ref-arizona_42-14" class="reference"><a href="#cite_note-arizona-42"><span class="cite-bracket">[</span>42<span class="cite-bracket">]</span></a></sup> T-sejtek</td> <td>citotixikus T-sejtté való differenciálódás (IL-2-vel),<sup id="cite_ref-arizona_42-15" class="reference"><a href="#cite_note-arizona-42"><span class="cite-bracket">[</span>42<span class="cite-bracket">]</span></a></sup> ↑ IFN-γ, TNF-α, ↓ IL-10 </td></tr> <tr> <td>természetes ölősejtek</td> <td>↑ IFN-γ, TNF-α </td></tr> <tr> <td><a href="/w/index.php?title=Interleukin-13&action=edit&redlink=1" class="new" title="Interleukin-13 (a lap nem létezik)">IL-13</a></td> <td>aktivált Th2 sejtek, hízósejtek, természetes ölősejtek</td> <td>IL13R</td> <td>Th2 sejtek, B-sejtek, makrfágok</td> <td>Stimulálja a B-sejtek (IgE) növekedését, gátolja a Th1 sejteket és a makrofágok gyulladásos citokinjeit ( IL-1, IL-6), ↓ IL-8, IL-10, IL-12 </td></tr> <tr> <td><a href="/w/index.php?title=Interleukin-14&action=edit&redlink=1" class="new" title="Interleukin-14 (a lap nem létezik)">IL-14</a></td> <td>T-sejtek és bizonyos rosszindulatú B-sejtek</td> <td></td> <td>aktivált B-sejtek</td> <td>szabályozza a B-sejtek növekedését és proliferációját, gátolja az immuglobulinkiválasztást </td></tr> <tr> <td><a href="/w/index.php?title=Interleukin-15&action=edit&redlink=1" class="new" title="Interleukin-15 (a lap nem létezik)">IL-15</a></td> <td>mononukleáris fagociták (és egyes egyéb sejtek), különösen makrofágok vírusfertőzés során</td> <td>IL15RA</td> <td>T-sejtek, aktivált B-sejtek</td> <td>megindítja a természetes ölősejtek képződését </td></tr> <tr> <td><a href="/w/index.php?title=Interleukin-16&action=edit&redlink=1" class="new" title="Interleukin-16 (a lap nem létezik)">IL-16</a></td> <td>limfociták, epiteliális sejtek, eozinofilok, CD8+ T-sejtek</td> <td><a href="/w/index.php?title=CD4&action=edit&redlink=1" class="new" title="CD4 (a lap nem létezik)">CD4</a></td> <td>CD4+ T-sejtek (Th-sejtek)</td> <td>CD4+ kemoattraktáns </td></tr> <tr> <td><a href="/w/index.php?title=Interleukin-17&action=edit&redlink=1" class="new" title="Interleukin-17 (a lap nem létezik)">IL-17</a></td> <td>T helper 17 sejtek (Th17)</td> <td>CDw217/IL17RA, IL17RB</td> <td>epitélium, endotélium, egyéb</td> <td>oszteoklasztok fejlődése, vérerek képződése, ↑ gyulladásos citokinek </td></tr> <tr> <td><a href="/w/index.php?title=Interleukin-18&action=edit&redlink=1" class="new" title="Interleukin-18 (a lap nem létezik)">IL-18</a></td> <td>makrofágok</td> <td>CDw218a/IL18R1</td> <td>Th1 sejtek, NK sejtek</td> <td>növeli az IFNγ termelést és az NK sejtek aktivitását </td></tr> <tr> <td><a href="/w/index.php?title=Interleukin-19&action=edit&redlink=1" class="new" title="Interleukin-19 (a lap nem létezik)">IL-19</a></td> <td>-</td> <td>IL20R</td> <td></td> <td>- </td></tr> <tr> <td><a href="/w/index.php?title=Interleukin-20&action=edit&redlink=1" class="new" title="Interleukin-20 (a lap nem létezik)">IL-20</a></td> <td>Aktivált <a href="/wiki/Keratinocita" title="Keratinocita">keratinociták</a> és monociták</td> <td>IL20R</td> <td></td> <td>A keratinociták proliferációját és differenciációját szabályozza </td></tr> <tr> <td><a href="/w/index.php?title=Interleukin-21&action=edit&redlink=1" class="new" title="Interleukin-21 (a lap nem létezik)">IL-21</a></td> <td>aktivált T helper sejtek, NK T sejtek</td> <td>IL21R</td> <td>Minden limfocita, dendritikus sejtek</td> <td>segít stimulálni a CD8+ T-sejtek aktivációját ás proliferációját, fokozza az NK sejtek citotoxicitását, fokozza a CD40 általi B-sejt proliferációt és differenciaciót, elősegíti a Th17 sejtek proliferációját </td></tr> <tr> <td><a href="/w/index.php?title=Interleukin-22&action=edit&redlink=1" class="new" title="Interleukin-22 (a lap nem létezik)">IL-22</a></td> <td>T helper 17 sejtek</td> <td>IL22R</td> <td></td> <td>Fokozza az epiteliűlis sejtek <a href="/w/index.php?title=Defenzin&action=edit&redlink=1" class="new" title="Defenzin (a lap nem létezik)">defenzintermelését</a>.<sup id="cite_ref-Abbas_2012_31-1" class="reference"><a href="#cite_note-Abbas_2012-31"><span class="cite-bracket">[</span>31<span class="cite-bracket">]</span></a></sup> Aktiválja a STAT1 és STAT3 transzkripciós faktorokat, növeli az akut fázis proteinek (szárum amiloid A, alfa 1-antikimotripszin, haptaglobin) termelését </td></tr> <tr> <td><a href="/w/index.php?title=Interleukin-23&action=edit&redlink=1" class="new" title="Interleukin-23 (a lap nem létezik)">IL-23</a></td> <td>makrofágok, dendritikus sejtek</td> <td>IL23R</td> <td></td> <td>Az IL-17-t termelő sejtek támogatása,<sup id="cite_ref-Abbas_2012_31-2" class="reference"><a href="#cite_note-Abbas_2012-31"><span class="cite-bracket">[</span>31<span class="cite-bracket">]</span></a></sup> fokozza az angiogenezist, de csökkenti a CD8+ T-sejtek infiltrációját </td></tr> <tr> <td><a href="/w/index.php?title=Interleukin-24&action=edit&redlink=1" class="new" title="Interleukin-24 (a lap nem létezik)">IL-24</a></td> <td><a href="/wiki/Melanocita" title="Melanocita">melanociták</a>, keratinociták, monocciták, T-sejtek</td> <td>IL20R</td> <td></td> <td>A tumorszupresszióban, sebgyógyulásban, pszoriázisban játszik fontos szerepet. </td></tr> <tr> <td><a href="/w/index.php?title=Interleukin-25&action=edit&redlink=1" class="new" title="Interleukin-25 (a lap nem létezik)">IL-25</a></td> <td>T-sejtek, hízósejtek, eozinofilok, makrofágok, nyálkahártya-sejtek</td> <td>LY6E</td> <td></td> <td>Fokozza az IL-4, IL-5 és IL-13 termelést </td></tr> <tr> <td><a href="/w/index.php?title=Interleukin-26&action=edit&redlink=1" class="new" title="Interleukin-26 (a lap nem létezik)">IL-26</a></td> <td>T-sejtek, monociták</td> <td>IL20R1</td> <td></td> <td>Fokozza az IL-10 és az IL-8 termelést, valamint az epiteliális sejtek felszínén a CD54 expresszióját </td></tr> <tr> <td><a href="/w/index.php?title=Interleukin-27&action=edit&redlink=1" class="new" title="Interleukin-27 (a lap nem létezik)">IL-27</a></td> <td>makrofágok, dendritikus sejtek</td> <td>IL27RA</td> <td></td> <td>A B- és T-sejtek aktivitását szabályozza </td></tr> <tr> <td><a href="/w/index.php?title=Interleukin-28&action=edit&redlink=1" class="new" title="Interleukin-28 (a lap nem létezik)">IL-28</a></td> <td>-</td> <td>IL28R</td> <td></td> <td>A vírusok elleni védekezésben játszik szerepet </td></tr> <tr> <td><a href="/w/index.php?title=Interleukin-29&action=edit&redlink=1" class="new" title="Interleukin-29 (a lap nem létezik)">IL-29</a></td> <td>-</td> <td></td> <td></td> <td>A baktériumok elleni védekezésben játszik szerepet </td></tr> <tr> <td><a href="/w/index.php?title=Interleukin-30&action=edit&redlink=1" class="new" title="Interleukin-30 (a lap nem létezik)">IL-30</a></td> <td>-</td> <td></td> <td></td> <td>A heterodimer IL-27 egyik lánca </td></tr> <tr> <td><a href="/w/index.php?title=Interleukin-31&action=edit&redlink=1" class="new" title="Interleukin-31 (a lap nem létezik)">IL-31</a></td> <td>Th2 sejtek</td> <td>IL31RA</td> <td></td> <td>A bőr gyulladásában játszhat szerepet </td></tr> <tr> <td><a href="/w/index.php?title=Interleukin-32&action=edit&redlink=1" class="new" title="Interleukin-32 (a lap nem létezik)">IL-32</a></td> <td>-</td> <td></td> <td></td> <td>Elősegíti a monociták és makrofágok TNF-α, IL-8 CXCL2 termelését </td></tr> <tr> <td><a href="/w/index.php?title=Interleukin-33&action=edit&redlink=1" class="new" title="Interleukin-33 (a lap nem létezik)">IL-33</a></td> <td>epiteliális sejtek</td> <td></td> <td></td> <td>Elősegíti a T helper sejtek 2. típusú citokintermelését </td></tr> <tr> <td><a href="/w/index.php?title=Interleukin-35&action=edit&redlink=1" class="new" title="Interleukin-35 (a lap nem létezik)">IL-35</a></td> <td>szabályozó T-sejtek</td> <td></td> <td></td> <td>Elnyomja a T helper sejtek aktivációját </td></tr> <tr> <td><a href="/w/index.php?title=Interleukin-36&action=edit&redlink=1" class="new" title="Interleukin-36 (a lap nem létezik)">IL-36</a></td> <td>-</td> <td></td> <td></td> <td>Szabályozza a dendtrikus sejtek és T-sejtek reakcióját </td></tr> </tbody></table> <div class="mw-heading mw-heading2"><h2 id="Jegyzetek">Jegyzetek</h2><span class="mw-editsection"><span class="mw-editsection-bracket">[</span><a href="/w/index.php?title=Interleukin&action=edit&section=19" title="Szakasz szerkesztése: Jegyzetek"><span>szerkesztés</span></a><span class="mw-editsection-bracket">]</span></span></div> <div class="ref-1col"> <div style="-moz-column-count:2; -webkit-column-count:2; column-count:2; -webkit-column-gap: 3em; -moz-column-gap: 3em; column-gap: 3em;"> <ol class="references"> <li id="cite_note-1"><span class="mw-cite-backlink"><a href="#cite_ref-1">↑</a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (2010. október 1.) „Evolutionary divergence and functions of the human interleukin (IL) gene family”. <i>Human Genomics</i> <b>5</b> (1), 30–55. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1186/1479-7364-5-1-30">10.1186/1479-7364-5-1-30</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/21106488">21106488</a>. <a href="/wiki/PubMed_Central" title="PubMed Central">PMC</a> <a rel="nofollow" class="external text" href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3390169/">3390169</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Evolutionary+divergence+and+functions+of+the+human+interleukin+%28IL%29+gene+family&rft.jtitle=Human+Genomics&rft.date=Oct+2010&rft.volume=5&rft.issue=1&rft.pages=30%E2%80%9355&rft_id=info:pmid/21106488&rft_id=info:doi/10.1186%2F1479-7364-5-1-30"><span style="display:none"> </span></span></span> </li> <li id="cite_note-pmid2405873-2"><span class="mw-cite-backlink"><a href="#cite_ref-pmid2405873_2-0">↑</a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (1990. január 1.) „Interleukin 1: the first interleukin”. <i>Immunology Today</i> <b>11</b> (1), 13–20. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1016/0167-5699(90)90005-t">10.1016/0167-5699(90)90005-t</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/2405873">2405873</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Interleukin+1%3A+the+first+interleukin&rft.jtitle=Immunology+Today&rft.date=Jan+1990&rft.volume=11&rft.issue=1&rft.pages=13%E2%80%9320&rft_id=info:pmid/2405873&rft_id=info:doi/10.1016%2F0167-5699%2890%2990005-t"><span style="display:none"> </span></span></span> </li> <li id="cite_note-3"><span class="mw-cite-backlink"><a href="#cite_ref-3">↑</a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal">szerk.: Habenicht A: Interleukin 1, <i>Growth Factors, Differentiation Factors, and Cytokines</i>. Berlin, Heidelberg: Springer, 85–102. o.. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>: <a rel="nofollow" class="external text" href="https://dx.doi.org/10.1007/978-3-642-74856-1_7">10.1007/978-3-642-74856-1_7</a> (1990). <a href="/wiki/Speci%C3%A1lis:K%C3%B6nyvforr%C3%A1sok/978-3-642-74856-1" title="Speciális:Könyvforrások/978-3-642-74856-1">ISBN 978-3-642-74856-1</a></cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Abook&rft.genre=book&rft.btitle=Growth+Factors%2C+Differentiation+Factors%2C+and+Cytokines&rft.atitle=Interleukin+1&rft.date=1990&rft.pub=Springer&rft.place=Berlin%2C+Heidelberg&rft.pages=85%E2%80%93102&rft.isbn=978-3-642-74856-1&rft_id=info:doi/10.1007%2F978-3-642-74856-1_7"><span style="display: none;"> </span></span></span> </li> <li id="cite_note-pmid91646-4"><span class="mw-cite-backlink"><a href="#cite_ref-pmid91646_4-0">↑</a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (1979. december 1.) „<a rel="nofollow" class="external text" href="http://www.jimmunol.org/content/123/6/2928.short">Revised nomenclature for antigen-nonspecific T cell proliferation and helper factors</a>”. <i>Journal of Immunology</i> <b>123</b> (6), 2928–9. o. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/91646">91646</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Revised+nomenclature+for+antigen-nonspecific+T+cell+proliferation+and+helper+factors&rft.jtitle=Journal+of+Immunology&rft.date=Dec+1979&rft.volume=123&rft.issue=6&rft.pages=2928%E2%80%939&rft_id=info:pmid/91646&rft_id=http%3A%2F%2Fwww.jimmunol.org%2Fcontent%2F123%2F6%2F2928.short"><span style="display:none"> </span></span></span> </li> <li id="cite_note-pmid2969618-5"><span class="mw-cite-backlink"><a href="#cite_ref-pmid2969618_5-0">↑</a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (1988. július 1.) „cDNA expression cloning of the IL-1 receptor, a member of the immunoglobulin superfamily”. <i>Science</i> <b>241</b> (4865), 585–9. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1126/science.2969618">10.1126/science.2969618</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/2969618">2969618</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=cDNA+expression+cloning+of+the+IL-1+receptor%2C+a+member+of+the+immunoglobulin+superfamily&rft.jtitle=Science&rft.date=Jul+1988&rft.volume=241&rft.issue=4865&rft.pages=585%E2%80%939&rft_id=info:pmid/2969618&rft_id=info:doi/10.1126%2Fscience.2969618"><span style="display:none"> </span></span></span> </li> <li id="cite_note-pmid8702856-6"><span class="mw-cite-backlink"><a href="#cite_ref-pmid8702856_6-0">↑</a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (1996. augusztus 1.) „Cloning and characterization of an alternatively processed human type II interleukin-1 receptor mRNA”. <i>The Journal of Biological Chemistry</i> <b>271</b> (34), 20965–72. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1074/jbc.271.34.20965">10.1074/jbc.271.34.20965</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/8702856">8702856</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Cloning+and+characterization+of+an+alternatively+processed+human+type+II+interleukin-1+receptor+mRNA&rft.jtitle=The+Journal+of+Biological+Chemistry&rft.date=Aug+1996&rft.volume=271&rft.issue=34&rft.pages=20965%E2%80%9372&rft_id=info:pmid/8702856&rft_id=info:doi/10.1074%2Fjbc.271.34.20965"><span style="display:none"> </span></span></span> </li> <li id="cite_note-pmid1833184-7"><span class="mw-cite-backlink"><a href="#cite_ref-pmid1833184_7-0">↑</a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (1991. október 1.) „<a rel="nofollow" class="external text" href="https://archive.org/details/sim_embo-journal_1991-10_10_10/page/2821">A novel IL-1 receptor, cloned from B cells by mammalian expression, is expressed in many cell types</a>”. <i>The EMBO Journal</i> <b>10</b> (10), 2821–32. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1002/j.1460-2075.1991.tb07831.x">10.1002/j.1460-2075.1991.tb07831.x</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/1833184">1833184</a>. <a href="/wiki/PubMed_Central" title="PubMed Central">PMC</a> <a rel="nofollow" class="external text" href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC452992/">452992</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=A+novel+IL-1+receptor%2C+cloned+from+B+cells+by+mammalian+expression%2C+is+expressed+in+many+cell+types&rft.jtitle=The+EMBO+Journal&rft.date=Oct+1991&rft.volume=10&rft.issue=10&rft.pages=2821%E2%80%9332&rft_id=info:pmid/1833184&rft_id=info:doi/10.1002%2Fj.1460-2075.1991.tb07831.x&rft_id=https%3A%2F%2Farchive.org%2Fdetails%2Fsim_embo-journal_1991-10_10_10%2Fpage%2F2821"><span style="display:none"> </span></span></span> </li> <li id="cite_note-pmid21093580-8"><span class="mw-cite-backlink">↑ <a href="#cite_ref-pmid21093580_8-0"><sup><i><b>a</b></i></sup></a> <a href="#cite_ref-pmid21093580_8-1"><sup><i><b>b</b></i></sup></a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (2011. július 1.) „Astrocytes support hippocampal-dependent memory and long-term potentiation via interleukin-1 signaling”. <i>Brain, Behavior, and Immunity</i> <b>25</b> (5), 1008–16. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1016/j.bbi.2010.11.007">10.1016/j.bbi.2010.11.007</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/21093580">21093580</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Astrocytes+support+hippocampal-dependent+memory+and+long-term+potentiation+via+interleukin-1+signaling&rft.jtitle=Brain%2C+Behavior%2C+and+Immunity&rft.date=Jul+2011&rft.volume=25&rft.issue=5&rft.pages=1008%E2%80%9316&rft_id=info:pmid/21093580&rft_id=info:doi/10.1016%2Fj.bbi.2010.11.007"><span style="display:none"> </span></span></span> </li> <li id="cite_note-pmid14620878-9"><span class="mw-cite-backlink"><a href="#cite_ref-pmid14620878_9-0">↑</a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (2003) „<a rel="nofollow" class="external text" href="https://archive.org/details/sim_hippocampus_2003_13_7/page/826">Impaired interleukin-1 signaling is associated with deficits in hippocampal memory processes and neural plasticity</a>”. <i>Hippocampus</i> <b>13</b> (7), 826–34. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1002/hipo.10135">10.1002/hipo.10135</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/14620878">14620878</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Impaired+interleukin-1+signaling+is+associated+with+deficits+in+hippocampal+memory+processes+and+neural+plasticity&rft.jtitle=Hippocampus&rft.date=2003&rft.volume=13&rft.issue=7&rft.pages=826%E2%80%9334&rft_id=info:pmid/14620878&rft_id=info:doi/10.1002%2Fhipo.10135&rft_id=https%3A%2F%2Farchive.org%2Fdetails%2Fsim_hippocampus_2003_13_7%2Fpage%2F826"><span style="display:none"> </span></span></span> </li> <li id="cite_note-PUB00004618-10"><span class="mw-cite-backlink"><a href="#cite_ref-PUB00004618_10-0">↑</a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (1986. május 1.) „Cloning, sequence, and expression of bovine interleukin 2”. <i>Proceedings of the National Academy of Sciences of the United States of America</i> <b>83</b> (10), 3223–7. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1073/pnas.83.10.3223">10.1073/pnas.83.10.3223</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/3517854">3517854</a>. <a href="/wiki/PubMed_Central" title="PubMed Central">PMC</a> <a rel="nofollow" class="external text" href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC323485/">323485</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Cloning%2C+sequence%2C+and+expression+of+bovine+interleukin+2&rft.jtitle=Proceedings+of+the+National+Academy+of+Sciences+of+the+United+States+of+America&rft.date=May+1986&rft.volume=83&rft.issue=10&rft.pages=3223%E2%80%937&rft_id=info:pmid/3517854&rft_id=info:doi/10.1073%2Fpnas.83.10.3223"><span style="display:none"> </span></span></span> </li> <li id="cite_note-PUB00000368-11"><span class="mw-cite-backlink">↑ <a href="#cite_ref-PUB00000368_11-0"><sup><i><b>a</b></i></sup></a> <a href="#cite_ref-PUB00000368_11-1"><sup><i><b>b</b></i></sup></a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (1992. augusztus 1.) „Secondary structure of human interleukin 2 from 3D heteronuclear NMR experiments”. <i>Biochemistry</i> <b>31</b> (33), 7741–4. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1021/bi00148a040">10.1021/bi00148a040</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/1510960">1510960</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Secondary+structure+of+human+interleukin+2+from+3D+heteronuclear+NMR+experiments&rft.jtitle=Biochemistry&rft.date=Aug+1992&rft.volume=31&rft.issue=33&rft.pages=7741%E2%80%934&rft_id=info:pmid/1510960&rft_id=info:doi/10.1021%2Fbi00148a040"><span style="display:none"> </span></span></span> </li> <li id="cite_note-PUB00001758-12"><span class="mw-cite-backlink">↑ <a href="#cite_ref-PUB00001758_12-0"><sup><i><b>a</b></i></sup></a> <a href="#cite_ref-PUB00001758_12-1"><sup><i><b>b</b></i></sup></a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (1987) „<a rel="nofollow" class="external text" href="https://archive.org/details/sim_gene_1987_55_1/page/115">Characterization of a human multilineage-colony-stimulating factor cDNA clone identified by a conserved noncoding sequence in mouse interleukin-3</a>”. <i>Gene</i> <b>55</b> (1), 115–24. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1016/0378-1119(87)90254-X">10.1016/0378-1119(87)90254-X</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/3497843">3497843</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Characterization+of+a+human+multilineage-colony-stimulating+factor+cDNA+clone+identified+by+a+conserved+noncoding+sequence+in+mouse+interleukin-3&rft.jtitle=Gene&rft.date=1987&rft.volume=55&rft.issue=1&rft.pages=115%E2%80%9324&rft_id=info:pmid/3497843&rft_id=info:doi/10.1016%2F0378-1119%2887%2990254-X&rft_id=https%3A%2F%2Farchive.org%2Fdetails%2Fsim_gene_1987_55_1%2Fpage%2F115"><span style="display:none"> </span></span></span> </li> <li id="cite_note-PUB00003991-13"><span class="mw-cite-backlink">↑ <a href="#cite_ref-PUB00003991_13-0"><sup><i><b>a</b></i></sup></a> <a href="#cite_ref-PUB00003991_13-1"><sup><i><b>b</b></i></sup></a> <a href="#cite_ref-PUB00003991_13-2"><sup><i><b>c</b></i></sup></a> <a href="#cite_ref-PUB00003991_13-3"><sup><i><b>d</b></i></sup></a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (1985) „Constitutive synthesis of interleukin-3 by leukaemia cell line WEHI-3B is due to retroviral insertion near the gene”. <i>Nature</i> <b>317</b> (6034), 255–8. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1038/317255a0">10.1038/317255a0</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/2413359">2413359</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Constitutive+synthesis+of+interleukin-3+by+leukaemia+cell+line+WEHI-3B+is+due+to+retroviral+insertion+near+the+gene&rft.jtitle=Nature&rft.date=1985&rft.volume=317&rft.issue=6034&rft.pages=255%E2%80%938&rft_id=info:pmid/2413359&rft_id=info:doi/10.1038%2F317255a0"><span style="display:none"> </span></span></span> </li> <li id="cite_note-PUB00004643-14"><span class="mw-cite-backlink">↑ <a href="#cite_ref-PUB00004643_14-0"><sup><i><b>a</b></i></sup></a> <a href="#cite_ref-PUB00004643_14-1"><sup><i><b>b</b></i></sup></a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (1987. október 1.) „Molecular cloning, nucleotide sequence, and expression of the gene encoding human eosinophil differentiation factor (interleukin 5)”. <i>Proceedings of the National Academy of Sciences of the United States of America</i> <b>84</b> (19), 6629–33. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1073/pnas.84.19.6629">10.1073/pnas.84.19.6629</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/3498940">3498940</a>. <a href="/wiki/PubMed_Central" title="PubMed Central">PMC</a> <a rel="nofollow" class="external text" href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC299136/">299136</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Molecular+cloning%2C+nucleotide+sequence%2C+and+expression+of+the+gene+encoding+human+eosinophil+differentiation+factor+%28interleukin+5%29&rft.jtitle=Proceedings+of+the+National+Academy+of+Sciences+of+the+United+States+of+America&rft.date=Oct+1987&rft.volume=84&rft.issue=19&rft.pages=6629%E2%80%9333&rft_id=info:pmid/3498940&rft_id=info:doi/10.1073%2Fpnas.84.19.6629"><span style="display:none"> </span></span></span> </li> <li id="cite_note-PUB00004146-15"><span class="mw-cite-backlink">↑ <a href="#cite_ref-PUB00004146_15-0"><sup><i><b>a</b></i></sup></a> <a href="#cite_ref-PUB00004146_15-1"><sup><i><b>b</b></i></sup></a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (1993. május 1.) „A novel dimer configuration revealed by the crystal structure at 2.4 A resolution of human interleukin-5”. <i>Nature</i> <b>363</b> (6425), 172–6. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1038/363172a0">10.1038/363172a0</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/8483502">8483502</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=A+novel+dimer+configuration+revealed+by+the+crystal+structure+at+2.4+A+resolution+of+human+interleukin-5&rft.jtitle=Nature&rft.date=May+1993&rft.volume=363&rft.issue=6425&rft.pages=172%E2%80%936&rft_id=info:pmid/8483502&rft_id=info:doi/10.1038%2F363172a0"><span style="display:none"> </span></span></span> </li> <li id="cite_note-PUB00003997-16"><span class="mw-cite-backlink">↑ <a href="#cite_ref-PUB00003997_16-0"><sup><i><b>a</b></i></sup></a> <a href="#cite_ref-PUB00003997_16-1"><sup><i><b>b</b></i></sup></a> <a href="#cite_ref-PUB00003997_16-2"><sup><i><b>c</b></i></sup></a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (1986) „Complementary DNA for a novel human interleukin (BSF-2) that induces B lymphocytes to produce immunoglobulin”. <i>Nature</i> <b>324</b> (6092), 73–6. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1038/324073a0">10.1038/324073a0</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/3491322">3491322</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Complementary+DNA+for+a+novel+human+interleukin+%28BSF-2%29+that+induces+B+lymphocytes+to+produce+immunoglobulin&rft.jtitle=Nature&rft.date=1986&rft.volume=324&rft.issue=6092&rft.pages=73%E2%80%936&rft_id=info:pmid/3491322&rft_id=info:doi/10.1038%2F324073a0"><span style="display:none"> </span></span></span> </li> <li id="cite_note-PUB00001608-17"><span class="mw-cite-backlink">↑ <a href="#cite_ref-PUB00001608_17-0"><sup><i><b>a</b></i></sup></a> <a href="#cite_ref-PUB00001608_17-1"><sup><i><b>b</b></i></sup></a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (1991. május 1.) „Evidence for the importance of a positive charge and an alpha-helical structure of the C-terminus for biological activity of human IL-6”. <i>FEBS Letters</i> <b>282</b> (2), 265–7. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1016/0014-5793(91)80491-K">10.1016/0014-5793(91)80491-K</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/2037043">2037043</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Evidence+for+the+importance+of+a+positive+charge+and+an+alpha-helical+structure+of+the+C-terminus+for+biological+activity+of+human+IL-6&rft.jtitle=FEBS+Letters&rft.date=May+1991&rft.volume=282&rft.issue=2&rft.pages=265%E2%80%937&rft_id=info:pmid/2037043&rft_id=info:doi/10.1016%2F0014-5793%2891%2980491-K"><span style="display:none"> </span></span></span> </li> <li id="cite_note-PUB00000150-18"><span class="mw-cite-backlink">↑ <a href="#cite_ref-PUB00000150_18-0"><sup><i><b>a</b></i></sup></a> <a href="#cite_ref-PUB00000150_18-1"><sup><i><b>b</b></i></sup></a> <a href="#cite_ref-PUB00000150_18-2"><sup><i><b>c</b></i></sup></a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (1989. július 1.) „Disulfide structures of human interleukin-6 are similar to those of human granulocyte colony stimulating factor”. <i>Archives of Biochemistry and Biophysics</i> <b>272</b> (1), 144–51. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1016/0003-9861(89)90205-1">10.1016/0003-9861(89)90205-1</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/2472117">2472117</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Disulfide+structures+of+human+interleukin-6+are+similar+to+those+of+human+granulocyte+colony+stimulating+factor&rft.jtitle=Archives+of+Biochemistry+and+Biophysics&rft.date=Jul+1989&rft.volume=272&rft.issue=1&rft.pages=144%E2%80%9351&rft_id=info:pmid/2472117&rft_id=info:doi/10.1016%2F0003-9861%2889%2990205-1"><span style="display:none"> </span></span></span> </li> <li id="cite_note-PUB00002742-19"><span class="mw-cite-backlink"><a href="#cite_ref-PUB00002742_19-0">↑</a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (1992. október 1.) „Crystal structure of recombinant human interleukin-4”. <i>The Journal of Biological Chemistry</i> <b>267</b> (28), 20371–6. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.2210/pdb2int/pdb">10.2210/pdb2int/pdb</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/1400355">1400355</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Crystal+structure+of+recombinant+human+interleukin-4&rft.jtitle=The+Journal+of+Biological+Chemistry&rft.date=Oct+1992&rft.volume=267&rft.issue=28&rft.pages=20371%E2%80%936&rft_id=info:pmid/1400355&rft_id=info:doi/10.2210%2Fpdb2int%2Fpdb"><span style="display:none"> </span></span></span> </li> <li id="cite_note-PUB00002018-20"><span class="mw-cite-backlink"><a href="#cite_ref-PUB00002018_20-0">↑</a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (1989. május 1.) „Interleukin 7: effects on early events in lymphopoiesis”. <i>Immunology Today</i> <b>10</b> (5), 170–3. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1016/0167-5699(89)90175-8">10.1016/0167-5699(89)90175-8</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/2663018">2663018</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Interleukin+7%3A+effects+on+early+events+in+lymphopoiesis&rft.jtitle=Immunology+Today&rft.date=May+1989&rft.volume=10&rft.issue=5&rft.pages=170%E2%80%933&rft_id=info:pmid/2663018&rft_id=info:doi/10.1016%2F0167-5699%2889%2990175-8"><span style="display:none"> </span></span></span> </li> <li id="cite_note-pmid10873157-21"><span class="mw-cite-backlink"><a href="#cite_ref-pmid10873157_21-0">↑</a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (2000) „Mitogen-activated protein kinases regulate cytokine gene expression in human airway myocytes”. <i>Am. J. Respir. Cell Mol. Biol.</i> <b>23</b> (1), 86–94. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1165/ajrcmb.23.1.4014">10.1165/ajrcmb.23.1.4014</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/10873157">10873157</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Mitogen-activated+protein+kinases+regulate+cytokine+gene+expression+in+human+airway+myocytes&rft.jtitle=Am.+J.+Respir.+Cell+Mol.+Biol.&rft.date=2000&rft.volume=23&rft.issue=1&rft.pages=86%E2%80%9394&rft_id=info:pmid/10873157&rft_id=info:doi/10.1165%2Fajrcmb.23.1.4014"><span style="display:none"> </span></span></span> </li> <li id="cite_note-pmid9802987-22"><span class="mw-cite-backlink"><a href="#cite_ref-pmid9802987_22-0">↑</a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (1998) „Endothelial cell "memory" of inflammatory stimulation: human venular endothelial cells store interleukin 8 in Weibel-Palade bodies”. <i>J. Exp. Med.</i> <b>188</b> (9), 1757–62. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1084/jem.188.9.1757">10.1084/jem.188.9.1757</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/9802987">9802987</a>. <a href="/wiki/PubMed_Central" title="PubMed Central">PMC</a> <a rel="nofollow" class="external text" href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2212526/">2212526</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Endothelial+cell+%22memory%22+of+inflammatory+stimulation%3A+human+venular+endothelial+cells+store+interleukin+8+in+Weibel-Palade+bodies&rft.jtitle=J.+Exp.+Med.&rft.date=1998&rft.volume=188&rft.issue=9&rft.pages=1757%E2%80%9362&rft_id=info:pmid/9802987&rft_id=info:doi/10.1084%2Fjem.188.9.1757"><span style="display:none"> </span></span></span> </li> <li id="cite_note-pmid9802986-23"><span class="mw-cite-backlink"><a href="#cite_ref-pmid9802986_23-0">↑</a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (1998) „Rapid secretion of prestored interleukin 8 from Weibel-Palade bodies of microvascular endothelial cells”. <i>J. Exp. Med.</i> <b>188</b> (9), 1751–6. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1084/jem.188.9.1751">10.1084/jem.188.9.1751</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/9802986">9802986</a>. <a href="/wiki/PubMed_Central" title="PubMed Central">PMC</a> <a rel="nofollow" class="external text" href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2212514/">2212514</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Rapid+secretion+of+prestored+interleukin+8+from+Weibel-Palade+bodies+of+microvascular+endothelial+cells&rft.jtitle=J.+Exp.+Med.&rft.date=1998&rft.volume=188&rft.issue=9&rft.pages=1751%E2%80%936&rft_id=info:pmid/9802986&rft_id=info:doi/10.1084%2Fjem.188.9.1751"><span style="display:none"> </span></span></span> </li> <li id="cite_note-pmid1967588-24"><span class="mw-cite-backlink"><a href="#cite_ref-pmid1967588_24-0">↑</a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (1990) „<a rel="nofollow" class="external text" href="https://archive.org/details/sim_human-genetics_1990-01_84_2/page/185">Monocyte-derived neutrophil chemotactic factor (MDNCF/IL-8) resides in a gene cluster along with several other members of the platelet factor 4 gene superfamily</a>”. <i>Hum. Genet.</i> <b>84</b> (2), 185–7. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1007/BF00208938">10.1007/BF00208938</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/1967588">1967588</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Monocyte-derived+neutrophil+chemotactic+factor+%28MDNCF%2FIL-8%29+resides+in+a+gene+cluster+along+with+several+other+members+of+the+platelet+factor+4+gene+superfamily&rft.jtitle=Hum.+Genet.&rft.date=1990&rft.volume=84&rft.issue=2&rft.pages=185%E2%80%937&rft_id=info:pmid/1967588&rft_id=info:doi/10.1007%2FBF00208938&rft_id=https%3A%2F%2Farchive.org%2Fdetails%2Fsim_human-genetics_1990-01_84_2%2Fpage%2F185"><span style="display:none"> </span></span></span> </li> <li id="cite_note-Brat_DJ_2005._pages_122-133-25"><span class="mw-cite-backlink">↑ <a href="#cite_ref-Brat_DJ_2005._pages_122-133_25-0"><sup><i><b>a</b></i></sup></a> <a href="#cite_ref-Brat_DJ_2005._pages_122-133_25-1"><sup><i><b>b</b></i></sup></a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (2005) „The role of interleukin-8 and its receptors in gliomagenesis and tumoral angiogenesis”. <i>Neuro-Oncology</i> <b>7</b> (2), 122–133. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1215/s1152851704001061">10.1215/s1152851704001061</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/15831231">15831231</a>. <a href="/wiki/PubMed_Central" title="PubMed Central">PMC</a> <a rel="nofollow" class="external text" href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1871893/">1871893</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=The+role+of+interleukin-8+and+its+receptors+in+gliomagenesis+and+tumoral+angiogenesis&rft.jtitle=Neuro-Oncology&rft.date=2005&rft.volume=7&rft.issue=2&rft.pages=122%E2%80%93133&rft_id=info:pmid/15831231&rft_id=info:doi/10.1215%2Fs1152851704001061"><span style="display:none"> </span></span></span> </li> <li id="cite_note-PUB00003177-26"><span class="mw-cite-backlink"><a href="#cite_ref-PUB00003177_26-0">↑</a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (1990. június 1.) „Human P40/IL-9. Expression in activated CD4+ T cells, genomic organization, and comparison with the mouse gene”. <i>Journal of Immunology</i> <b>144</b> (11), 4235–41. o. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/1971295">1971295</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Human+P40%2FIL-9.+Expression+in+activated+CD4%2B+T+cells%2C+genomic+organization%2C+and+comparison+with+the+mouse+gene&rft.jtitle=Journal+of+Immunology&rft.date=Jun+1990&rft.volume=144&rft.issue=11&rft.pages=4235%E2%80%9341&rft_id=info:pmid/1971295"><span style="display:none"> </span></span></span> </li> <li id="cite_note-27"><span class="mw-cite-backlink"><a href="#cite_ref-27">↑</a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (2019. február 1.) „The tertiary structure of γc cytokines dictates receptor sharing”. <i>Cytokine</i> <b>116</b>, 161–168. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1016/j.cyto.2019.01.007">10.1016/j.cyto.2019.01.007</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/30716660">30716660</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=The+tertiary+structure+of+%CE%B3c+cytokines+dictates+receptor+sharing&rft.jtitle=Cytokine&rft.date=February+2019&rft.volume=116&rft.pages=161%E2%80%93168&rft_id=info:pmid/30716660&rft_id=info:doi/10.1016%2Fj.cyto.2019.01.007"><span style="display:none"> </span></span></span> </li> <li id="cite_note-PUB00005262-28"><span class="mw-cite-backlink"><a href="#cite_ref-PUB00005262_28-0">↑</a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal">Zdanov A, Schalk-Hihi C, Gustchina A, Tsang M, Weatherbee J, Wlodawer A (1995. június 1.). „Crystal structure of interleukin-10 reveals the functional dimer with an unexpected topological similarity to interferon gamma”. <i>Structure</i> <b>3</b> (6), 591–601. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1016/S0969-2126(01)00193-9">10.1016/S0969-2126(01)00193-9</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/8590020">8590020</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Crystal+structure+of+interleukin-10+reveals+the+functional+dimer+with+an+unexpected+topological+similarity+to+interferon+gamma&rft.jtitle=Structure&rft.date=Jun+1995&rft.volume=3&rft.issue=6&rft.au=Zdanov+A%2C+Schalk-Hihi+C%2C+Gustchina+A%2C+Tsang+M%2C+Weatherbee+J%2C+Wlodawer+A&rft.pages=591%E2%80%93601&rft_id=info:pmid/8590020&rft_id=info:doi/10.1016%2FS0969-2126%2801%2900193-9"><span style="display:none"> </span></span></span> </li> <li id="cite_note-PUB00004497-29"><span class="mw-cite-backlink"><a href="#cite_ref-PUB00004497_29-0">↑</a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (1995. december 1.) „Subtraction hybridization identifies a novel melanoma differentiation associated gene, mda-7, modulated during human melanoma differentiation, growth and progression”. <i>Oncogene</i> <b>11</b> (12), 2477–86. o. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/8545104">8545104</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Subtraction+hybridization+identifies+a+novel+melanoma+differentiation+associated+gene%2C+mda-7%2C+modulated+during+human+melanoma+differentiation%2C+growth+and+progression&rft.jtitle=Oncogene&rft.date=Dec+1995&rft.volume=11&rft.issue=12&rft.pages=2477%E2%80%9386&rft_id=info:pmid/8545104"><span style="display:none"> </span></span></span> </li> <li id="cite_note-PUB00013131-30"><span class="mw-cite-backlink"><a href="#cite_ref-PUB00013131_30-0">↑</a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (1997) „Interleukin-11”. <i>The International Journal of Biochemistry & Cell Biology</i> <b>29</b> (8–9), 1059–62. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1016/S1357-2725(97)00017-4">10.1016/S1357-2725(97)00017-4</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/9416001">9416001</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Interleukin-11&rft.jtitle=The+International+Journal+of+Biochemistry+%26+Cell+Biology&rft.date=1997&rft.volume=29&rft.issue=8%E2%80%939&rft.pages=1059%E2%80%9362&rft_id=info:pmid/9416001&rft_id=info:doi/10.1016%2FS1357-2725%2897%2900017-4"><span style="display:none"> </span></span></span> </li> <li id="cite_note-Abbas_2012-31"><span class="mw-cite-backlink">↑ <a href="#cite_ref-Abbas_2012_31-0"><sup><i><b>a</b></i></sup></a> <a href="#cite_ref-Abbas_2012_31-1"><sup><i><b>b</b></i></sup></a> <a href="#cite_ref-Abbas_2012_31-2"><sup><i><b>c</b></i></sup></a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> <i><a rel="nofollow" class="external text" href="https://archive.org/details/cellularmolecula0000abba">Cellular and molecular immunology</a></i>, 7th, Philadelphia: Elsevier/Saunders (2012). <a href="/wiki/Speci%C3%A1lis:K%C3%B6nyvforr%C3%A1sok/978-1437715286" title="Speciális:Könyvforrások/978-1437715286">ISBN 978-1437715286</a></cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Abook&rft.genre=book&rft.btitle=Cellular+and+molecular+immunology&rft.date=2012&rft.edition=7th&rft.pub=Elsevier%2FSaunders&rft.place=Philadelphia&rft.isbn=978-1437715286&rft_id=https%3A%2F%2Farchive.org%2Fdetails%2Fcellularmolecula0000abba"><span style="display: none;"> </span></span></span> </li> <li id="cite_note-pmid18619507-32"><span class="mw-cite-backlink"><a href="#cite_ref-pmid18619507_32-0">↑</a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (2008. augusztus 1.) „Interleukin-12 improves cytotoxicity of natural killer cells via upregulated expression of NKG2D”. <i>Human Immunology</i> <b>69</b> (8), 490–500. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1016/j.humimm.2008.06.004">10.1016/j.humimm.2008.06.004</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/18619507">18619507</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Interleukin-12+improves+cytotoxicity+of+natural+killer+cells+via+upregulated+expression+of+NKG2D&rft.jtitle=Human+Immunology&rft.date=Aug+2008&rft.volume=69&rft.issue=8&rft.pages=490%E2%80%93500&rft_id=info:pmid/18619507&rft_id=info:doi/10.1016%2Fj.humimm.2008.06.004"><span style="display:none"> </span></span></span> </li> <li id="cite_note-PUB00007461-33"><span class="mw-cite-backlink"><a href="#cite_ref-PUB00007461_33-0">↑</a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (2001. június 1.) „Il-12: keeping cell-mediated immunity alive”. <i>Scandinavian Journal of Immunology</i> <b>53</b> (6), 529–32. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1046/j.1365-3083.2001.00917.x">10.1046/j.1365-3083.2001.00917.x</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/11422900">11422900</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Il-12%3A+keeping+cell-mediated+immunity+alive&rft.jtitle=Scandinavian+Journal+of+Immunology&rft.date=Jun+2001&rft.volume=53&rft.issue=6&rft.pages=529%E2%80%9332&rft_id=info:pmid/11422900&rft_id=info:doi/10.1046%2Fj.1365-3083.2001.00917.x"><span style="display:none"> </span></span></span> </li> <li id="cite_note-PUB00007462-34"><span class="mw-cite-backlink"><a href="#cite_ref-PUB00007462_34-0">↑</a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (1998) „<a rel="nofollow" class="external text" href="https://archive.org/details/sim_annual-review-of-immunology_1998_16/page/495">The interleukin-12/interleukin-12-receptor system: role in normal and pathologic immune responses</a>”. <i>Annual Review of Immunology</i> <b>16</b>, 495–521. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1146/annurev.immunol.16.1.495">10.1146/annurev.immunol.16.1.495</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/9597139">9597139</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=The+interleukin-12%2Finterleukin-12-receptor+system%3A+role+in+normal+and+pathologic+immune+responses&rft.jtitle=Annual+Review+of+Immunology&rft.date=1998&rft.volume=16&rft.pages=495%E2%80%93521&rft_id=info:pmid/9597139&rft_id=info:doi/10.1146%2Fannurev.immunol.16.1.495&rft_id=https%3A%2F%2Farchive.org%2Fdetails%2Fsim_annual-review-of-immunology_1998_16%2Fpage%2F495"><span style="display:none"> </span></span></span> </li> <li id="cite_note-PUB00004143-35"><span class="mw-cite-backlink"><a href="#cite_ref-PUB00004143_35-0">↑</a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (1993. március 1.) „Interleukin-13 is a new human lymphokine regulating inflammatory and immune responses”. <i>Nature</i> <b>362</b> (6417), 248–50. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1038/362248a0">10.1038/362248a0</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/8096327">8096327</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Interleukin-13+is+a+new+human+lymphokine+regulating+inflammatory+and+immune+responses&rft.jtitle=Nature&rft.date=Mar+1993&rft.volume=362&rft.issue=6417&rft.pages=248%E2%80%9350&rft_id=info:pmid/8096327&rft_id=info:doi/10.1038%2F362248a0"><span style="display:none"> </span></span></span> </li> <li id="cite_note-pmid26689873-36"><span class="mw-cite-backlink"><a href="#cite_ref-pmid26689873_36-0">↑</a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (2015. december 1.) „<a rel="nofollow" class="external text" href="http://real.mtak.hu/36921/1/030.62.2015.4.2.pdf">Interleukin-13 as an important cytokine: A review on its roles in some human diseases</a>”. <i>Acta Microbiologica et Immunologica Hungarica</i> <b>62</b> (4), 341–78. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1556/030.62.2015.4.2">10.1556/030.62.2015.4.2</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/26689873">26689873</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Interleukin-13+as+an+important+cytokine%3A+A+review+on+its+roles+in+some+human+diseases&rft.jtitle=Acta+Microbiologica+et+Immunologica+Hungarica&rft.date=December+2015&rft.volume=62&rft.issue=4&rft.pages=341%E2%80%9378&rft_id=info:pmid/26689873&rft_id=info:doi/10.1556%2F030.62.2015.4.2&rft_id=http%3A%2F%2Freal.mtak.hu%2F36921%2F1%2F030.62.2015.4.2.pdf"><span style="display:none"> </span></span></span> </li> <li id="cite_note-PUB00009488-37"><span class="mw-cite-backlink"><a href="#cite_ref-PUB00009488_37-0">↑</a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (2000. április 1.) „Role of IL-15 on monocytic resistance to human herpesvirus 6 infection”. <i>The New Microbiologica</i> <b>23</b> (2), 105–12. o. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/10872679">10872679</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Role+of+IL-15+on+monocytic+resistance+to+human+herpesvirus+6+infection&rft.jtitle=The+New+Microbiologica&rft.date=Apr+2000&rft.volume=23&rft.issue=2&rft.pages=105%E2%80%9312&rft_id=info:pmid/10872679"><span style="display:none"> </span></span></span> </li> <li id="cite_note-PUB00012254-38"><span class="mw-cite-backlink"><a href="#cite_ref-PUB00012254_38-0">↑</a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (2002. január 1.) „IL-17: prototype member of an emerging cytokine family”. <i>Journal of Leukocyte iology</i> <b>71</b> (1), 1–8. o. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/11781375">11781375</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=IL-17%3A+prototype+member+of+an+emerging+cytokine+family&rft.jtitle=Journal+of+Leukocyte+iology&rft.date=January+2002&rft.volume=71&rft.issue=1&rft.pages=1%E2%80%938&rft_id=info:pmid/11781375"><span style="display:none"> </span></span></span> </li> <li id="cite_note-39"><span class="mw-cite-backlink"><a href="#cite_ref-39">↑</a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (2008. június 1.) „Th17 cells in human disease”. <i>Immunological Reviews</i> <b>223</b>, 87–113. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1111/j.1600-065X.2008.00628.x">10.1111/j.1600-065X.2008.00628.x</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/18613831">18613831</a>. <a href="/wiki/PubMed_Central" title="PubMed Central">PMC</a> <a rel="nofollow" class="external text" href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3299089/">3299089</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Th17+cells+in+human+disease&rft.jtitle=Immunological+Reviews&rft.date=June+2008&rft.volume=223&rft.pages=87%E2%80%93113&rft_id=info:pmid/18613831&rft_id=info:doi/10.1111%2Fj.1600-065X.2008.00628.x"><span style="display:none"> </span></span></span> </li> <li id="cite_note-Immunology68Unless-40"><span class="mw-cite-backlink">↑ <a href="#cite_ref-Immunology68Unless_40-0"><sup><i><b>a</b></i></sup></a> <a href="#cite_ref-Immunology68Unless_40-1"><sup><i><b>b</b></i></sup></a> <a href="#cite_ref-Immunology68Unless_40-2"><sup><i><b>c</b></i></sup></a> <a href="#cite_ref-Immunology68Unless_40-3"><sup><i><b>d</b></i></sup></a></span> <span class="reference-text">Lippincott's Illustrated Reviews: Immunology. Paperback: 384 pages. Publisher: Lippincott Williams & Wilkins; (July 1, 2007). Language: English. <a href="/wiki/Speci%C3%A1lis:K%C3%B6nyvforr%C3%A1sok/0781795435" title="Speciális:Könyvforrások/0781795435">ISBN 0-7817-9543-5</a>. <a href="/wiki/Speci%C3%A1lis:K%C3%B6nyvforr%C3%A1sok/9780781795432" title="Speciális:Könyvforrások/9780781795432">ISBN 978-0-7817-9543-2</a>. Page 68</span> </li> <li id="cite_note-title-41"><span class="mw-cite-backlink"><a href="#cite_ref-title_41-0">↑</a></span> <span class="reference-text"><span class="citation"><a rel="nofollow" class="external text" href="https://web.archive.org/web/20060315211023/http://www.ebioscience.com/ebioscience/whatsnew/pdf/Cytokines.pdf"><i>Cytokines - Master Regulators of the Immune System</i></a>. eBioscience, 2007. május 1. [2006. március 15-i dátummal az <a rel="nofollow" class="external text" href="http://www.ebioscience.com/ebioscience/whatsnew/pdf/Cytokines.pdf">eredetiből</a> archiválva]. (Hozzáférés: 2008. február 28.)</span></span> </li> <li id="cite_note-arizona-42"><span class="mw-cite-backlink">↑ <a href="#cite_ref-arizona_42-0"><sup><i><b>a</b></i></sup></a> <a href="#cite_ref-arizona_42-1"><sup><i><b>b</b></i></sup></a> <a href="#cite_ref-arizona_42-2"><sup><i><b>c</b></i></sup></a> <a href="#cite_ref-arizona_42-3"><sup><i><b>d</b></i></sup></a> <a href="#cite_ref-arizona_42-4"><sup><i><b>e</b></i></sup></a> <a href="#cite_ref-arizona_42-5"><sup><i><b>f</b></i></sup></a> <a href="#cite_ref-arizona_42-6"><sup><i><b>g</b></i></sup></a> <a href="#cite_ref-arizona_42-7"><sup><i><b>h</b></i></sup></a> <a href="#cite_ref-arizona_42-8"><sup><i><b>i</b></i></sup></a> <a href="#cite_ref-arizona_42-9"><sup><i><b>j</b></i></sup></a> <a href="#cite_ref-arizona_42-10"><sup><i><b>k</b></i></sup></a> <a href="#cite_ref-arizona_42-11"><sup><i><b>l</b></i></sup></a> <a href="#cite_ref-arizona_42-12"><sup><i><b>m</b></i></sup></a> <a href="#cite_ref-arizona_42-13"><sup><i><b>n</b></i></sup></a> <a href="#cite_ref-arizona_42-14"><sup><i><b>o</b></i></sup></a> <a href="#cite_ref-arizona_42-15"><sup><i><b>p</b></i></sup></a></span> <span class="reference-text"><a rel="nofollow" class="external text" href="http://microvet.arizona.edu/Courses/MIC419/Tutorials/cytokines.html">Cytokine tutorial, The University of Arizona</a> <a rel="nofollow" class="external text" href="https://web.archive.org/web/20080202122645/http://microvet.arizona.edu/Courses/MIC419/Tutorials/cytokines.html">Archiválva</a> 2008. február 2-i dátummal a <a href="/wiki/Wayback_Machine" title="Wayback Machine">Wayback Machine</a>-ben.</span> </li> <li id="cite_note-43"><span class="mw-cite-backlink"><a href="#cite_ref-43">↑</a></span> <span class="reference-text"><cite class="book citation" style="font-style:normal"> (1996. december 1.) „Biological activity of IL-4 and IL-13 on human endothelial cells: functional evidence that both cytokines act through the same receptor”. <i>Int Immunol</i> <b>8</b> (12), 1915–25. o. <a href="/wiki/Digital_object_identifier" title="Digital object identifier">DOI</a>:<a rel="nofollow" class="external text" href="https://dx.doi.org/10.1093/intimm/8.12.1915">10.1093/intimm/8.12.1915</a>. <a href="/wiki/PubMed#PubMed-azonosító" title="PubMed">PMID</a> <a rel="nofollow" class="external text" href="https://pubmed.ncbi.nlm.nih.gov/8982776">8982776</a>.</cite><span class="Z3988" title="ctx_ver=Z39.88-2004&rft_val_fmt=info%3Aofi%2Ffmt%3Akev%3Amtx%3Ajournal&rft.genre=article&rft.atitle=Biological+activity+of+IL-4+and+IL-13+on+human+endothelial+cells%3A+functional+evidence+that+both+cytokines+act+through+the+same+receptor&rft.jtitle=Int+Immunol&rft.date=Dec+1996&rft.volume=8&rft.issue=12&rft.pages=1915%E2%80%9325&rft_id=info:pmid/8982776&rft_id=info:doi/10.1093%2Fintimm%2F8.12.1915"><span style="display:none"> </span></span></span> </li> </ol> </div> </div> <div class="mw-heading mw-heading2"><h2 id="Fordítás"><span id="Ford.C3.ADt.C3.A1s"></span>Fordítás</h2><span class="mw-editsection"><span class="mw-editsection-bracket">[</span><a href="/w/index.php?title=Interleukin&action=edit&section=20" title="Szakasz szerkesztése: Fordítás"><span>szerkesztés</span></a><span class="mw-editsection-bracket">]</span></span></div> <p>Ez a szócikk részben vagy egészben az <i><a href="https://en.wikipedia.org/wiki/Interleukin" class="extiw" title="en:Interleukin">Interleukin</a></i> című angol Wikipédia-szócikk <span class="plainlinks"><a class="external text" href="https://en.wikipedia.org/wiki/Interleukin?oldid=1089407020">ezen változatának</a> fordításán alapul.</span> Az eredeti cikk szerkesztőit annak laptörténete sorolja fel. Ez a jelzés csupán a megfogalmazás eredetét és a szerzői jogokat jelzi, nem szolgál a cikkben szereplő információk forrásmegjelöléseként. </p> <div class="navbox-styles"><style data-mw-deduplicate="TemplateStyles:r26593303">.mw-parser-output .hlist dl,.mw-parser-output .hlist ol,.mw-parser-output .hlist ul{margin:0;padding:0}.mw-parser-output .hlist dd,.mw-parser-output .hlist dt,.mw-parser-output .hlist li{margin:0;display:inline}.mw-parser-output .hlist.inline,.mw-parser-output .hlist.inline dl,.mw-parser-output .hlist.inline ol,.mw-parser-output .hlist.inline ul,.mw-parser-output .hlist dl dl,.mw-parser-output .hlist dl ol,.mw-parser-output .hlist dl ul,.mw-parser-output .hlist ol dl,.mw-parser-output .hlist ol ol,.mw-parser-output .hlist ol ul,.mw-parser-output .hlist ul dl,.mw-parser-output .hlist ul ol,.mw-parser-output .hlist ul ul{display:inline}.mw-parser-output .hlist .mw-empty-li{display:none}.mw-parser-output .hlist dt::after{content:": "}.mw-parser-output .hlist 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