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特异性抗体缺乏
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bg-color-teal-light"> <div class="container"> <div class="row"> <div class="col-sm-12"> <h1 class="font-size-30 fweight-400 color-white">原发性和获得性免疫缺陷研究杂志 </h1> </div> </div> </div> </div> <nav class="navbar navbar-expand-lg bg-navbar navbar-light mega-menu journal-menu topbar-shadow" id="mainNavbar"> <div class="container"> <button class="navbar-toggler" type="button" data-bs-toggle="collapse" data-bs-target="#navbarSupportedContent" aria-controls="navbarSupportedContent" aria-expanded="false" aria-label="Toggle navigation"> <span class="navbar-toggler-icon"></span> </button> <div class="collapse navbar-collapse justify-content-center" id="navbarSupportedContent"> <ul class="navbar-nav mb-auto mb-2 mb-lg-0"> <li class="nav-item"><a class="nav-link" href="https://chinese.scitechnol.com/journals/primary-acquired-immunodeficiency-research.html" title="点击这里"><i class="fa fa-home" aria-hidden="true"></i></a></li> <li class="nav-item dropdown"> <a class="nav-link dropdown-toggle" href="#" 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在预防感染方面发挥着主导作用。有些患者的免疫球蛋白和所有形式的 IgG 水平正常,但无法产生足够的特异性 IgG 抗体来保护我们免受某些病毒和细菌的侵害。那些产生正常免疫球蛋白水平但缺乏产生针对引起上呼吸道和下呼吸道感染的微生物类型的保护性 IgG 分子的能力的患者被认为患有特异性抗体缺乏症 (SAD)。 SAD 有时被称为部分抗体缺陷或多糖反应性受损。特异性 IgG 抗体对于抵抗感染很重要;然而,我们免疫系统的其他组成部分也可以消灭细菌和病毒。 T 细胞补体蛋白和 IgA 抗体(仅举几例)是我们免疫系统的一部分,在完整的免疫反应过程中协同工作。如果这些其他成分发挥良好作用,一些特异性抗体水平较低的患者可能很少生病。某些 IgG 亚类的抗体很容易与补体系统相互作用,而其他抗体与补体蛋白的相互作用则很差(如果有的话)。因此,无法产生特定亚类的抗体或免疫系统其他部分的轻微缺陷可能使个体容易受到某些类型的感染,但不易感染其他类型的感染。</font></font></p> </div> </div> <!-- sidebar --> <div class="col-xs-12 col-md-3"> <div class="divider-v4 divider-v4-left-double bg-color-white padding-10 margin-b-10"> <h2 class="divider-v4-title">探索科技</h2> <div> <ul class="list-unstyled angle-right"> <li class="color-base"><a href="https://chinese.scitechnol.com/author-guidelines.html" title="点击这里"> 作者指南</a></li> <li class="color-base"><a href="https://chinese.scitechnol.com/reviewer-guidelines.html" title="点击这里"> 审稿人指南</a></li> <li class="color-base"><a 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