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class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/109190305/Amoebic_Liver_Abscess_Communicating_with_Intrahepatic_Bile_Ducts"><img alt="Research paper thumbnail of Amoebic Liver Abscess Communicating with Intrahepatic Bile Ducts" class="work-thumbnail" src="https://attachments.academia-assets.com/107388546/thumbnails/1.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/109190305/Amoebic_Liver_Abscess_Communicating_with_Intrahepatic_Bile_Ducts">Amoebic Liver Abscess Communicating with Intrahepatic Bile Ducts</a></div><div class="wp-workCard_item"><span>Tropical Doctor</span><span>, 1997</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><a 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Liver Abscess","url":"https://www.academia.edu/Documents/in/Amoebic_Liver_Abscess"},{"id":1303572,"name":"Metronidazole","url":"https://www.academia.edu/Documents/in/Metronidazole"},{"id":1436065,"name":"Bile Duct Diseases","url":"https://www.academia.edu/Documents/in/Bile_Duct_Diseases"}],"urls":[{"id":35398409,"url":"http://journals.sagepub.com/doi/pdf/10.1177/004947559702700326"}]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="109190299"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/109190299/Effect_of_Esophageal_Variceal_Band_Ligation_on_Hemorrhoids_Anorectal_Varices_and_Portal_Hypertensive_Colopathy"><img alt="Research paper thumbnail of Effect of Esophageal Variceal Band Ligation on Hemorrhoids, Anorectal Varices, and Portal Hypertensive Colopathy" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/109190299/Effect_of_Esophageal_Variceal_Band_Ligation_on_Hemorrhoids_Anorectal_Varices_and_Portal_Hypertensive_Colopathy">Effect of Esophageal Variceal Band Ligation on Hemorrhoids, Anorectal Varices, and Portal Hypertensive Colopathy</a></div><div class="wp-workCard_item"><span>Endoscopy</span><span>, 2002</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">Although it is known that obliteration of esophageal varices following endoscopic variceal band l...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">Although it is known that obliteration of esophageal varices following endoscopic variceal band ligation results in an increase in the incidence of portal hypertensive gastropathy, the effect of variceal ligation on hemorrhoids, anorectal/colonic varices and portal hypertensive colopathy is not known. The aim of this study was to investigate the effect of endoscopic variceal band ligation on hemorrhoids, anorectal/colonic varices and portal hypertensive colopathy. A total of 60 consecutive patients with cirrhosis of the liver and portal hypertension were prospectively studied. Upper gastrointestinal endoscopy and full-length colonoscopy were carried out before the patients underwent endoscopic variceal band ligation for esophageal varices and after obliteration of the varices following band ligation. Obliteration of esophageal varices by endoscopic variceal band ligation did not affect the incidence of hemorrhoids (37 % before and after), anorectal varices (40 % before and after), and portal hypertensive colopathy (57 % before and after). It is concluded that esophageal variceal band ligation does not affect the incidence of hemorrhoids, anorectal varices or portal hypertensive colopathy in patients with cirrhosis of the liver.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109190299"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109190299"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109190299; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=109190299]").text(description); $(".js-view-count[data-work-id=109190299]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 109190299; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='109190299']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 109190299, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=109190299]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":109190299,"title":"Effect of Esophageal Variceal Band Ligation on Hemorrhoids, Anorectal Varices, and Portal Hypertensive Colopathy","translated_title":"","metadata":{"abstract":"Although it is known that obliteration of esophageal varices following endoscopic variceal band ligation results in an increase in the incidence of portal hypertensive gastropathy, the effect of variceal ligation on hemorrhoids, anorectal/colonic varices and portal hypertensive colopathy is not known. The aim of this study was to investigate the effect of endoscopic variceal band ligation on hemorrhoids, anorectal/colonic varices and portal hypertensive colopathy. A total of 60 consecutive patients with cirrhosis of the liver and portal hypertension were prospectively studied. Upper gastrointestinal endoscopy and full-length colonoscopy were carried out before the patients underwent endoscopic variceal band ligation for esophageal varices and after obliteration of the varices following band ligation. Obliteration of esophageal varices by endoscopic variceal band ligation did not affect the incidence of hemorrhoids (37 % before and after), anorectal varices (40 % before and after), and portal hypertensive colopathy (57 % before and after). It is concluded that esophageal variceal band ligation does not affect the incidence of hemorrhoids, anorectal varices or portal hypertensive colopathy in patients with cirrhosis of the liver.","publisher":"Georg Thieme Verlag KG","publication_date":{"day":null,"month":null,"year":2002,"errors":{}},"publication_name":"Endoscopy"},"translated_abstract":"Although it is known that obliteration of esophageal varices following endoscopic variceal band ligation results in an increase in the incidence of portal hypertensive gastropathy, the effect of variceal ligation on hemorrhoids, anorectal/colonic varices and portal hypertensive colopathy is not known. The aim of this study was to investigate the effect of endoscopic variceal band ligation on hemorrhoids, anorectal/colonic varices and portal hypertensive colopathy. A total of 60 consecutive patients with cirrhosis of the liver and portal hypertension were prospectively studied. Upper gastrointestinal endoscopy and full-length colonoscopy were carried out before the patients underwent endoscopic variceal band ligation for esophageal varices and after obliteration of the varices following band ligation. Obliteration of esophageal varices by endoscopic variceal band ligation did not affect the incidence of hemorrhoids (37 % before and after), anorectal varices (40 % before and after), and portal hypertensive colopathy (57 % before and after). It is concluded that esophageal variceal band ligation does not affect the incidence of hemorrhoids, anorectal varices or portal hypertensive colopathy in patients with cirrhosis of the liver.","internal_url":"https://www.academia.edu/109190299/Effect_of_Esophageal_Variceal_Band_Ligation_on_Hemorrhoids_Anorectal_Varices_and_Portal_Hypertensive_Colopathy","translated_internal_url":"","created_at":"2023-11-15T01:49:39.619-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":48000299,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[],"downloadable_attachments":[],"slug":"Effect_of_Esophageal_Variceal_Band_Ligation_on_Hemorrhoids_Anorectal_Varices_and_Portal_Hypertensive_Colopathy","translated_slug":"","page_count":null,"language":"en","content_type":"Work","owner":{"id":48000299,"first_name":"Vatsala","middle_initials":null,"last_name":"Misra","page_name":"VatsalaMisra","domain_name":"independent","created_at":"2016-04-30T07:36:38.954-07:00","display_name":"Vatsala Misra","url":"https://independent.academia.edu/VatsalaMisra"},"attachments":[],"research_interests":[{"id":3274,"name":"Gastroenterology","url":"https://www.academia.edu/Documents/in/Gastroenterology"},{"id":22506,"name":"Adolescent","url":"https://www.academia.edu/Documents/in/Adolescent"},{"id":26327,"name":"Medicine","url":"https://www.academia.edu/Documents/in/Medicine"},{"id":37805,"name":"Endoscopy","url":"https://www.academia.edu/Documents/in/Endoscopy"},{"id":63890,"name":"Complication","url":"https://www.academia.edu/Documents/in/Complication"},{"id":65390,"name":"Internal Medicine","url":"https://www.academia.edu/Documents/in/Internal_Medicine"},{"id":71342,"name":"Portal hypertension","url":"https://www.academia.edu/Documents/in/Portal_hypertension"},{"id":174502,"name":"Incidence","url":"https://www.academia.edu/Documents/in/Incidence"},{"id":227324,"name":"Colonoscopy","url":"https://www.academia.edu/Documents/in/Colonoscopy"},{"id":244814,"name":"Clinical Sciences","url":"https://www.academia.edu/Documents/in/Clinical_Sciences"},{"id":600778,"name":"Cirrhosis","url":"https://www.academia.edu/Documents/in/Cirrhosis"},{"id":1486844,"name":"Hemorrhoids","url":"https://www.academia.edu/Documents/in/Hemorrhoids"},{"id":2922786,"name":"Esophageal varices","url":"https://www.academia.edu/Documents/in/Esophageal_varices"},{"id":3530956,"name":"Esophageal and gastric varices","url":"https://www.academia.edu/Documents/in/Esophageal_and_gastric_varices"},{"id":3589281,"name":"Ligation","url":"https://www.academia.edu/Documents/in/Ligation"}],"urls":[{"id":35398406,"url":"http://www.thieme-connect.de/products/ejournals/pdf/10.1055/s-2002-20290.pdf"}]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="109190298"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/109190298/Ileocecal_masses_in_patients_with_amebic_liver_abscess_Etiology_and_management"><img alt="Research paper thumbnail of Ileocecal masses in patients with amebic liver abscess: Etiology and management" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/109190298/Ileocecal_masses_in_patients_with_amebic_liver_abscess_Etiology_and_management">Ileocecal masses in patients with amebic liver abscess: Etiology and management</a></div><div class="wp-workCard_item"><span>World Journal of Gastroenterology</span><span>, 2006</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">To assess the causes of ileocecal mass in patients with amebic liver abscess. Patients with amebi...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">To assess the causes of ileocecal mass in patients with amebic liver abscess. Patients with amebic liver abscess and ileocecal mass were carefully examined and investigated by contrast-enhanced CT scan followed by colonoscopy and histological examination of biopsy materials from lesions during colonoscopy. Ileocecal masses were found in seventeen patients with amebic liver abscess. The cause of the mass was ameboma in 14 patients, cecal tuberculosis in 2 patients and adenocarcinoma of the cecum in 1 patient. Colonic ulcers were noted in five of the six (83%) patients with active diarrhea at presentation. The ileocecal mass in all these patients was ameboma. Ulcers were seen in only one of the 11 (9%) patients without diarrhea. The difference was statistically significant from the group with diarrhea (P&amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;lt; 0.005). Ileocecal mass is not an uncommon finding in patients with amebic liver abscess. Although, the ileocecal mass is due to ameboma formation in most cases, it should not be assumed that this is the case in all patients. Colonoscopy and histological examination of the target biopsies are mandatory to avoid missing a more sinister lesion.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109190298"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109190298"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109190298; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=109190298]").text(description); $(".js-view-count[data-work-id=109190298]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 109190298; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='109190298']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 109190298, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=109190298]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":109190298,"title":"Ileocecal masses in patients with amebic liver abscess: Etiology and management","translated_title":"","metadata":{"abstract":"To assess the causes of ileocecal mass in patients with amebic liver abscess. Patients with amebic liver abscess and ileocecal mass were carefully examined and investigated by contrast-enhanced CT scan followed by colonoscopy and histological examination of biopsy materials from lesions during colonoscopy. Ileocecal masses were found in seventeen patients with amebic liver abscess. The cause of the mass was ameboma in 14 patients, cecal tuberculosis in 2 patients and adenocarcinoma of the cecum in 1 patient. Colonic ulcers were noted in five of the six (83%) patients with active diarrhea at presentation. The ileocecal mass in all these patients was ameboma. Ulcers were seen in only one of the 11 (9%) patients without diarrhea. The difference was statistically significant from the group with diarrhea (P\u0026amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;lt; 0.005). Ileocecal mass is not an uncommon finding in patients with amebic liver abscess. Although, the ileocecal mass is due to ameboma formation in most cases, it should not be assumed that this is the case in all patients. Colonoscopy and histological examination of the target biopsies are mandatory to avoid missing a more sinister lesion.","publisher":"Baishideng Publishing Group Inc.","publication_date":{"day":null,"month":null,"year":2006,"errors":{}},"publication_name":"World Journal of Gastroenterology"},"translated_abstract":"To assess the causes of ileocecal mass in patients with amebic liver abscess. Patients with amebic liver abscess and ileocecal mass were carefully examined and investigated by contrast-enhanced CT scan followed by colonoscopy and histological examination of biopsy materials from lesions during colonoscopy. Ileocecal masses were found in seventeen patients with amebic liver abscess. The cause of the mass was ameboma in 14 patients, cecal tuberculosis in 2 patients and adenocarcinoma of the cecum in 1 patient. Colonic ulcers were noted in five of the six (83%) patients with active diarrhea at presentation. The ileocecal mass in all these patients was ameboma. Ulcers were seen in only one of the 11 (9%) patients without diarrhea. The difference was statistically significant from the group with diarrhea (P\u0026amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;lt; 0.005). Ileocecal mass is not an uncommon finding in patients with amebic liver abscess. Although, the ileocecal mass is due to ameboma formation in most cases, it should not be assumed that this is the case in all patients. Colonoscopy and histological examination of the target biopsies are mandatory to avoid missing a more sinister lesion.","internal_url":"https://www.academia.edu/109190298/Ileocecal_masses_in_patients_with_amebic_liver_abscess_Etiology_and_management","translated_internal_url":"","created_at":"2023-11-15T01:49:33.626-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":48000299,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[],"downloadable_attachments":[],"slug":"Ileocecal_masses_in_patients_with_amebic_liver_abscess_Etiology_and_management","translated_slug":"","page_count":null,"language":"en","content_type":"Work","owner":{"id":48000299,"first_name":"Vatsala","middle_initials":null,"last_name":"Misra","page_name":"VatsalaMisra","domain_name":"independent","created_at":"2016-04-30T07:36:38.954-07:00","display_name":"Vatsala Misra","url":"https://independent.academia.edu/VatsalaMisra"},"attachments":[],"research_interests":[{"id":26327,"name":"Medicine","url":"https://www.academia.edu/Documents/in/Medicine"},{"id":37826,"name":"Biopsy","url":"https://www.academia.edu/Documents/in/Biopsy"},{"id":44259,"name":"Diarrhea","url":"https://www.academia.edu/Documents/in/Diarrhea"},{"id":227324,"name":"Colonoscopy","url":"https://www.academia.edu/Documents/in/Colonoscopy"},{"id":244814,"name":"Clinical Sciences","url":"https://www.academia.edu/Documents/in/Clinical_Sciences"},{"id":988280,"name":"Abscess","url":"https://www.academia.edu/Documents/in/Abscess"},{"id":4191298,"name":"Ileal diseases","url":"https://www.academia.edu/Documents/in/Ileal_diseases"}],"urls":[]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="109190297"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/109190297/Endoscopic_and_histological_changes_in_upper_gastrointestinal_tract_of_patients_with_chronic_renal_failure"><img alt="Research paper thumbnail of Endoscopic and histological changes in upper gastrointestinal tract of patients with chronic renal failure" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/109190297/Endoscopic_and_histological_changes_in_upper_gastrointestinal_tract_of_patients_with_chronic_renal_failure">Endoscopic and histological changes in upper gastrointestinal tract of patients with chronic renal failure</a></div><div class="wp-workCard_item"><span>Indian journal of pathology & microbiology</span><span>, 2004</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">To evaluate the endoscopic and histological changes in upper gastrointestinal tract of patients w...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">To evaluate the endoscopic and histological changes in upper gastrointestinal tract of patients with chronic renal failure 50 patients and 50 controls were studied. Upper gastrointestinal endoscopy was done and 2 biopsies each were taken from oesophagus, corpus and antrum of the stomach and duodenum. Sections were stained with haematoxylin & eosin, Alcian blue--Periodic acid Schiff&#39;s (pH 2.5), and Loeffler&#39;s methylene blue stains. Oesophagus was endoscopically normal in most of the patients. Predominant histological finding was chronic oesophagitis which was significantly higher in patients than controls (47.1% Vs 26%; p&lt;0.05). Significantly higher (p&lt;0.001) number of patients had gastritis, oedema and pale mucosa on endoscopic examination of stomach. Predominant histological changes were mucosal oedema (82.35%), gastritis (23.5%) and increase in number of bi- and multinucleated parietal cells with vacuolation and fragmentation of the cytoplasm (29%). Prevalence of H. ...</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109190297"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109190297"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109190297; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=109190297]").text(description); $(".js-view-count[data-work-id=109190297]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 109190297; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='109190297']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 109190297, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=109190297]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":109190297,"title":"Endoscopic and histological changes in upper gastrointestinal tract of patients with chronic renal failure","translated_title":"","metadata":{"abstract":"To evaluate the endoscopic and histological changes in upper gastrointestinal tract of patients with chronic renal failure 50 patients and 50 controls were studied. Upper gastrointestinal endoscopy was done and 2 biopsies each were taken from oesophagus, corpus and antrum of the stomach and duodenum. Sections were stained with haematoxylin \u0026 eosin, Alcian blue--Periodic acid Schiff\u0026#39;s (pH 2.5), and Loeffler\u0026#39;s methylene blue stains. Oesophagus was endoscopically normal in most of the patients. Predominant histological finding was chronic oesophagitis which was significantly higher in patients than controls (47.1% Vs 26%; p\u0026lt;0.05). Significantly higher (p\u0026lt;0.001) number of patients had gastritis, oedema and pale mucosa on endoscopic examination of stomach. Predominant histological changes were mucosal oedema (82.35%), gastritis (23.5%) and increase in number of bi- and multinucleated parietal cells with vacuolation and fragmentation of the cytoplasm (29%). Prevalence of H. ...","publication_date":{"day":null,"month":null,"year":2004,"errors":{}},"publication_name":"Indian journal of pathology \u0026 microbiology"},"translated_abstract":"To evaluate the endoscopic and histological changes in upper gastrointestinal tract of patients with chronic renal failure 50 patients and 50 controls were studied. Upper gastrointestinal endoscopy was done and 2 biopsies each were taken from oesophagus, corpus and antrum of the stomach and duodenum. Sections were stained with haematoxylin \u0026 eosin, Alcian blue--Periodic acid Schiff\u0026#39;s (pH 2.5), and Loeffler\u0026#39;s methylene blue stains. Oesophagus was endoscopically normal in most of the patients. Predominant histological finding was chronic oesophagitis which was significantly higher in patients than controls (47.1% Vs 26%; p\u0026lt;0.05). Significantly higher (p\u0026lt;0.001) number of patients had gastritis, oedema and pale mucosa on endoscopic examination of stomach. Predominant histological changes were mucosal oedema (82.35%), gastritis (23.5%) and increase in number of bi- and multinucleated parietal cells with vacuolation and fragmentation of the cytoplasm (29%). Prevalence of H. ...","internal_url":"https://www.academia.edu/109190297/Endoscopic_and_histological_changes_in_upper_gastrointestinal_tract_of_patients_with_chronic_renal_failure","translated_internal_url":"","created_at":"2023-11-15T01:49:33.407-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":48000299,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[],"downloadable_attachments":[],"slug":"Endoscopic_and_histological_changes_in_upper_gastrointestinal_tract_of_patients_with_chronic_renal_failure","translated_slug":"","page_count":null,"language":"en","content_type":"Work","owner":{"id":48000299,"first_name":"Vatsala","middle_initials":null,"last_name":"Misra","page_name":"VatsalaMisra","domain_name":"independent","created_at":"2016-04-30T07:36:38.954-07:00","display_name":"Vatsala Misra","url":"https://independent.academia.edu/VatsalaMisra"},"attachments":[],"research_interests":[{"id":3274,"name":"Gastroenterology","url":"https://www.academia.edu/Documents/in/Gastroenterology"},{"id":6947,"name":"Medical Microbiology","url":"https://www.academia.edu/Documents/in/Medical_Microbiology"},{"id":21460,"name":"Helicobacter pylori","url":"https://www.academia.edu/Documents/in/Helicobacter_pylori"},{"id":26327,"name":"Medicine","url":"https://www.academia.edu/Documents/in/Medicine"},{"id":65390,"name":"Internal Medicine","url":"https://www.academia.edu/Documents/in/Internal_Medicine"},{"id":98939,"name":"Pubmed","url":"https://www.academia.edu/Documents/in/Pubmed"},{"id":131233,"name":"Indian","url":"https://www.academia.edu/Documents/in/Indian"},{"id":405739,"name":"Esophagus","url":"https://www.academia.edu/Documents/in/Esophagus"},{"id":537759,"name":"Gastrointestinal Tract","url":"https://www.academia.edu/Documents/in/Gastrointestinal_Tract"},{"id":1030661,"name":"Stomach","url":"https://www.academia.edu/Documents/in/Stomach"},{"id":1106287,"name":"Chronic Kidney Failure","url":"https://www.academia.edu/Documents/in/Chronic_Kidney_Failure"},{"id":1819399,"name":"Case Control Studies","url":"https://www.academia.edu/Documents/in/Case_Control_Studies"},{"id":2090674,"name":"Duodenum","url":"https://www.academia.edu/Documents/in/Duodenum"}],"urls":[]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="109190296"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" rel="nofollow" href="https://www.academia.edu/109190296/Duodenal_metastases_from_squamous_cell_carcinoma_of_the_lung_endoscopic_management_of_bleeding_and_biliary_and_duodenal_obstruction"><img alt="Research paper thumbnail of Duodenal metastases from squamous cell carcinoma of the lung: endoscopic management of bleeding and biliary and duodenal obstruction" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" rel="nofollow" href="https://www.academia.edu/109190296/Duodenal_metastases_from_squamous_cell_carcinoma_of_the_lung_endoscopic_management_of_bleeding_and_biliary_and_duodenal_obstruction">Duodenal metastases from squamous cell carcinoma of the lung: endoscopic management of bleeding and biliary and duodenal obstruction</a></div><div class="wp-workCard_item"><span>Indian journal of gastroenterology : official journal of the Indian Society of Gastroenterology</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">Intestinal metastases from cancer of the lung are rare. We report a 65-year-old man with duodenal...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">Intestinal metastases from cancer of the lung are rare. We report a 65-year-old man with duodenal metastases from squamous cell cancer causing GI bleeding and biliary and duodenal obstruction; these were managed endoscopically.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109190296"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109190296"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109190296; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=109190296]").text(description); $(".js-view-count[data-work-id=109190296]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 109190296; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='109190296']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 109190296, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=109190296]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":109190296,"title":"Duodenal metastases from squamous cell carcinoma of the lung: endoscopic management of bleeding and biliary and duodenal obstruction","translated_title":"","metadata":{"abstract":"Intestinal metastases from cancer of the lung are rare. 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Sections were stained with hematoxylene and eosin, AB/PAS (Ph 2.5) and Loeffler&#39;s methylene blue stains. Infection by H. pylori, inflammation and atrophy were found to be significantly more in leprosy patients as compared to controls (p &lt; 0.01, &lt; 0.005 and &lt; 0.02 respectively). On comparing the histopathological changes in various subgroups of leprosy, H. pylori, inflammation and activity showed a statistically decreasing trend from tuberculoid to lepromatous subgroups (p &lt; 0.05, &lt; 0.001, &lt; 0.01 respectively). Atrophy showed a significant increasing trend from tuberculoid to lepromatous group (&lt; 0.001), it is concluded that despite a low prevalence of H. pylori and associated gastritis in patients with lepromatous leprosy, gastric epithelial damage is more marked due to altered immune response.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109190295"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109190295"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109190295; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=109190295]").text(description); $(".js-view-count[data-work-id=109190295]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 109190295; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='109190295']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 109190295, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=109190295]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":109190295,"title":"Helicobacter pylori and associated histopathological changes in gastric biopsies of patients with leprosy","translated_title":"","metadata":{"abstract":"Two antral biopsies each from 104 patients of leprosy and 100 controls were studied to find out the prevalence of H. pylori and associated histopathological changes. Sections were stained with hematoxylene and eosin, AB/PAS (Ph 2.5) and Loeffler\u0026#39;s methylene blue stains. Infection by H. pylori, inflammation and atrophy were found to be significantly more in leprosy patients as compared to controls (p \u0026lt; 0.01, \u0026lt; 0.005 and \u0026lt; 0.02 respectively). On comparing the histopathological changes in various subgroups of leprosy, H. pylori, inflammation and activity showed a statistically decreasing trend from tuberculoid to lepromatous subgroups (p \u0026lt; 0.05, \u0026lt; 0.001, \u0026lt; 0.01 respectively). Atrophy showed a significant increasing trend from tuberculoid to lepromatous group (\u0026lt; 0.001), it is concluded that despite a low prevalence of H. pylori and associated gastritis in patients with lepromatous leprosy, gastric epithelial damage is more marked due to altered immune response.","publication_date":{"day":null,"month":null,"year":2001,"errors":{}},"publication_name":"Indian journal of pathology \u0026 microbiology"},"translated_abstract":"Two antral biopsies each from 104 patients of leprosy and 100 controls were studied to find out the prevalence of H. pylori and associated histopathological changes. Sections were stained with hematoxylene and eosin, AB/PAS (Ph 2.5) and Loeffler\u0026#39;s methylene blue stains. Infection by H. pylori, inflammation and atrophy were found to be significantly more in leprosy patients as compared to controls (p \u0026lt; 0.01, \u0026lt; 0.005 and \u0026lt; 0.02 respectively). On comparing the histopathological changes in various subgroups of leprosy, H. pylori, inflammation and activity showed a statistically decreasing trend from tuberculoid to lepromatous subgroups (p \u0026lt; 0.05, \u0026lt; 0.001, \u0026lt; 0.01 respectively). Atrophy showed a significant increasing trend from tuberculoid to lepromatous group (\u0026lt; 0.001), it is concluded that despite a low prevalence of H. pylori and associated gastritis in patients with lepromatous leprosy, gastric epithelial damage is more marked due to altered immune response.","internal_url":"https://www.academia.edu/109190295/Helicobacter_pylori_and_associated_histopathological_changes_in_gastric_biopsies_of_patients_with_leprosy","translated_internal_url":"","created_at":"2023-11-15T01:49:32.696-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":48000299,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[],"downloadable_attachments":[],"slug":"Helicobacter_pylori_and_associated_histopathological_changes_in_gastric_biopsies_of_patients_with_leprosy","translated_slug":"","page_count":null,"language":"en","content_type":"Work","owner":{"id":48000299,"first_name":"Vatsala","middle_initials":null,"last_name":"Misra","page_name":"VatsalaMisra","domain_name":"independent","created_at":"2016-04-30T07:36:38.954-07:00","display_name":"Vatsala Misra","url":"https://independent.academia.edu/VatsalaMisra"},"attachments":[],"research_interests":[{"id":3274,"name":"Gastroenterology","url":"https://www.academia.edu/Documents/in/Gastroenterology"},{"id":6947,"name":"Medical Microbiology","url":"https://www.academia.edu/Documents/in/Medical_Microbiology"},{"id":21460,"name":"Helicobacter pylori","url":"https://www.academia.edu/Documents/in/Helicobacter_pylori"},{"id":22506,"name":"Adolescent","url":"https://www.academia.edu/Documents/in/Adolescent"},{"id":26327,"name":"Medicine","url":"https://www.academia.edu/Documents/in/Medicine"},{"id":73489,"name":"Leprosy","url":"https://www.academia.edu/Documents/in/Leprosy"},{"id":98939,"name":"Pubmed","url":"https://www.academia.edu/Documents/in/Pubmed"},{"id":131233,"name":"Indian","url":"https://www.academia.edu/Documents/in/Indian"},{"id":289271,"name":"Aged","url":"https://www.academia.edu/Documents/in/Aged"},{"id":357850,"name":"Atrophy","url":"https://www.academia.edu/Documents/in/Atrophy"},{"id":925054,"name":"Gastritis","url":"https://www.academia.edu/Documents/in/Gastritis"},{"id":1819399,"name":"Case Control Studies","url":"https://www.academia.edu/Documents/in/Case_Control_Studies"},{"id":2547982,"name":"Gastric mucosa","url":"https://www.academia.edu/Documents/in/Gastric_mucosa"}],"urls":[]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="109190294"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/109190294/AgNOR_counts_are_not_altered_in_alcoholic_cirrhosis"><img alt="Research paper thumbnail of AgNOR counts are not altered in alcoholic cirrhosis" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/109190294/AgNOR_counts_are_not_altered_in_alcoholic_cirrhosis">AgNOR counts are not altered in alcoholic cirrhosis</a></div><div class="wp-workCard_item"><span>Indian journal of pathology & microbiology</span><span>, 2003</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">One hundred and two liver biopsy specimens were stained for Argyrophilic nucleolar organizer regi...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">One hundred and two liver biopsy specimens were stained for Argyrophilic nucleolar organizer regions and associated proteins to assess its utility in differentiating normal, cirrhotic and neoplastic liver tissue. A statistically significant (p &lt; 0.001) difference was observed between mean AgNOR counts of normal (1.53 +/- 0.4), post-hepatitic cirrhosis (3.65 +/- 0.53) and hepatocellular carcinoma (7.94 +/- 1.18). In contrast the mean AgNOR count of biopsies with alcoholic cirrhosis (1.57 +/- 0.06) was significantly less (p &lt; 0.001) than post-hepatitic cirrhosis and was similar to that of normal liver tissue. It is concluded that AgNORs can act as a good adjuvant to histology in diagnosing liver diseases especially in differentiating post-hepatitic and alcoholic cirrhosis.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109190294"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109190294"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109190294; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=109190294]").text(description); $(".js-view-count[data-work-id=109190294]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 109190294; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='109190294']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 109190294, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=109190294]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":109190294,"title":"AgNOR counts are not altered in alcoholic cirrhosis","translated_title":"","metadata":{"abstract":"One hundred and two liver biopsy specimens were stained for Argyrophilic nucleolar organizer regions and associated proteins to assess its utility in differentiating normal, cirrhotic and neoplastic liver tissue. A statistically significant (p \u0026lt; 0.001) difference was observed between mean AgNOR counts of normal (1.53 +/- 0.4), post-hepatitic cirrhosis (3.65 +/- 0.53) and hepatocellular carcinoma (7.94 +/- 1.18). In contrast the mean AgNOR count of biopsies with alcoholic cirrhosis (1.57 +/- 0.06) was significantly less (p \u0026lt; 0.001) than post-hepatitic cirrhosis and was similar to that of normal liver tissue. It is concluded that AgNORs can act as a good adjuvant to histology in diagnosing liver diseases especially in differentiating post-hepatitic and alcoholic cirrhosis.","publication_date":{"day":null,"month":null,"year":2003,"errors":{}},"publication_name":"Indian journal of pathology \u0026 microbiology"},"translated_abstract":"One hundred and two liver biopsy specimens were stained for Argyrophilic nucleolar organizer regions and associated proteins to assess its utility in differentiating normal, cirrhotic and neoplastic liver tissue. A statistically significant (p \u0026lt; 0.001) difference was observed between mean AgNOR counts of normal (1.53 +/- 0.4), post-hepatitic cirrhosis (3.65 +/- 0.53) and hepatocellular carcinoma (7.94 +/- 1.18). In contrast the mean AgNOR count of biopsies with alcoholic cirrhosis (1.57 +/- 0.06) was significantly less (p \u0026lt; 0.001) than post-hepatitic cirrhosis and was similar to that of normal liver tissue. It is concluded that AgNORs can act as a good adjuvant to histology in diagnosing liver diseases especially in differentiating post-hepatitic and alcoholic cirrhosis.","internal_url":"https://www.academia.edu/109190294/AgNOR_counts_are_not_altered_in_alcoholic_cirrhosis","translated_internal_url":"","created_at":"2023-11-15T01:49:32.437-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":48000299,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[],"downloadable_attachments":[],"slug":"AgNOR_counts_are_not_altered_in_alcoholic_cirrhosis","translated_slug":"","page_count":null,"language":"en","content_type":"Work","owner":{"id":48000299,"first_name":"Vatsala","middle_initials":null,"last_name":"Misra","page_name":"VatsalaMisra","domain_name":"independent","created_at":"2016-04-30T07:36:38.954-07:00","display_name":"Vatsala Misra","url":"https://independent.academia.edu/VatsalaMisra"},"attachments":[],"research_interests":[{"id":165,"name":"Pathology","url":"https://www.academia.edu/Documents/in/Pathology"},{"id":6947,"name":"Medical Microbiology","url":"https://www.academia.edu/Documents/in/Medical_Microbiology"},{"id":12069,"name":"Histology","url":"https://www.academia.edu/Documents/in/Histology"},{"id":26327,"name":"Medicine","url":"https://www.academia.edu/Documents/in/Medicine"},{"id":37826,"name":"Biopsy","url":"https://www.academia.edu/Documents/in/Biopsy"},{"id":71343,"name":"Liver Cirrhosis","url":"https://www.academia.edu/Documents/in/Liver_Cirrhosis"},{"id":71437,"name":"Liver","url":"https://www.academia.edu/Documents/in/Liver"},{"id":80694,"name":"Silver","url":"https://www.academia.edu/Documents/in/Silver"},{"id":98939,"name":"Pubmed","url":"https://www.academia.edu/Documents/in/Pubmed"},{"id":111972,"name":"Hepatocellular Carcinoma","url":"https://www.academia.edu/Documents/in/Hepatocellular_Carcinoma"},{"id":144048,"name":"Hepatitis","url":"https://www.academia.edu/Documents/in/Hepatitis"},{"id":558694,"name":"Nucleolus Organizer Region","url":"https://www.academia.edu/Documents/in/Nucleolus_Organizer_Region"},{"id":600778,"name":"Cirrhosis","url":"https://www.academia.edu/Documents/in/Cirrhosis"},{"id":1819399,"name":"Case Control Studies","url":"https://www.academia.edu/Documents/in/Case_Control_Studies"},{"id":2815868,"name":"Alcoholic liver disease","url":"https://www.academia.edu/Documents/in/Alcoholic_liver_disease"},{"id":3562342,"name":"Liver neoplasms","url":"https://www.academia.edu/Documents/in/Liver_neoplasms"}],"urls":[]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="109190293"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/109190293/Diagnosing_ascites_Value_of_ascitic_fluid_total_protein_albumin_cholesterol_their_ratios_serum_ascites_albumin_and_cholesterol_gradient"><img alt="Research paper thumbnail of Diagnosing ascites: Value of ascitic fluid total protein, albumin, cholesterol, their ratios, serum-ascites albumin and cholesterol gradient" class="work-thumbnail" src="https://attachments.academia-assets.com/107388529/thumbnails/1.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/109190293/Diagnosing_ascites_Value_of_ascitic_fluid_total_protein_albumin_cholesterol_their_ratios_serum_ascites_albumin_and_cholesterol_gradient">Diagnosing ascites: Value of ascitic fluid total protein, albumin, cholesterol, their ratios, serum-ascites albumin and cholesterol gradient</a></div><div class="wp-workCard_item"><span>Journal of Gastroenterology and Hepatology</span><span>, 1995</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><a id="4200a97401e41ffdf22c3362a2482beb" class="wp-workCard--action" rel="nofollow" data-click-track="profile-work-strip-download" data-download="{"attachment_id":107388529,"asset_id":109190293,"asset_type":"Work","button_location":"profile"}" href="https://www.academia.edu/attachments/107388529/download_file?st=MTczMjc1MTU1Nyw4LjIyMi4yMDguMTQ2&s=profile"><span><i class="fa fa-arrow-down"></i></span><span>Download</span></a><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109190293"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109190293"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109190293; 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The mean f s.d. ascitic fluid total protein, albumin, cholesterol, their respective ascitic fluid/serum ratios in cirrhotic ascites were lower than malignant and tuberculous groups (P \u003c 0.001 for each). The difference between malignant and tuberculous groups was sigtllficant for ascitic fluid/serum total protein (P \u003c 0.05) and ascitic fluidherum albumin (P \u003c 0.01) only. Mean serum-ascites albumin gradient in cirrhotics was higher than in the malignant and tuberculous groups (P \u003c 0.001 for each). The difference between malignant and tuberculous groups was sigtllficant (P \u003c 0.01). Mean f s.d. serum-ascites cholesterol gradient in cirrhotics was higher than that in malignant and tuberculous groups (P\u003cO.001 for each). The difference between malignant and tuberculous groups was also si@icant (P \u003c 0.01). Both serudascitic fluid total protein less than 0.5 and ascitic fluid cholesterol less than 55 mg/dL had 94% diagnostic accuracy for differentiating cirrhotic from malignant and tuberculous ascites. Serum ascitic fluid albumin gradient greater than 1.1 g/ dL, ascitic fluid/serum albumin less than 0.65 and ascitic fluid albumin less than 2 g/dL had diagnostic accuracy of 92, 92 and 91%, respectively. Ascitic fluid total protein had diagnostic accuracy of 88%. None of the tests was able to differentiate between malignant and tuberculous ascites. Measurement of ascitic fluid cholesterol concentration is a simple method of differentiating cirrhotic from non-cirrhotic ascites.","publication_date":{"day":null,"month":null,"year":1995,"errors":{}},"publication_name":"Journal of Gastroenterology and Hepatology","grobid_abstract_attachment_id":107388529},"translated_abstract":null,"internal_url":"https://www.academia.edu/109190293/Diagnosing_ascites_Value_of_ascitic_fluid_total_protein_albumin_cholesterol_their_ratios_serum_ascites_albumin_and_cholesterol_gradient","translated_internal_url":"","created_at":"2023-11-15T01:49:31.708-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":48000299,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[],"downloadable_attachments":[{"id":107388529,"title":"","file_type":"pdf","scribd_thumbnail_url":"https://attachments.academia-assets.com/107388529/thumbnails/1.jpg","file_name":"j.1440-1746.1995.tb01096.x20231115-1-yeiz6s.pdf","download_url":"https://www.academia.edu/attachments/107388529/download_file?st=MTczMjc1MTU1Nyw4LjIyMi4yMDguMTQ2&","bulk_download_file_name":"Diagnosing_ascites_Value_of_ascitic_flui.pdf","bulk_download_url":"https://d1wqtxts1xzle7.cloudfront.net/107388529/j.1440-1746.1995.tb01096.x20231115-1-yeiz6s-libre.pdf?1700048619=\u0026response-content-disposition=attachment%3B+filename%3DDiagnosing_ascites_Value_of_ascitic_flui.pdf\u0026Expires=1732708236\u0026Signature=Wpb1AURv3o67ywEec2ZXbuoaRthkC-CjjocL58cw6m2QFOVY7nBwYpz5JoB~RipTvAVFjSpjjqpR~GHxT9700PETmCZjnpPRyU2WW9Uu1ZfYj45XDVSUVNVyKszggQ4knw~SVW5RVtvdVE0L3KfDqhcJYlvVN10evUxt9ygQgX1GdSUQaFPC6bDWlOTvM~IQ0ZAAY6zfXWF-X9ZasPTZBYOsc5H2AEdOytZ-RtiZZB-LTYx47OReaa9l82QTflEpBN0WVEAW8jvwmXjN4M5wuf3kLsxHso5gxtrvyC6Gw6uv~SJ7W6VEFEnJNcTSaMmf6mIPU2JD7XSdjW3RPykf9w__\u0026Key-Pair-Id=APKAJLOHF5GGSLRBV4ZA"}],"slug":"Diagnosing_ascites_Value_of_ascitic_fluid_total_protein_albumin_cholesterol_their_ratios_serum_ascites_albumin_and_cholesterol_gradient","translated_slug":"","page_count":5,"language":"en","content_type":"Work","owner":{"id":48000299,"first_name":"Vatsala","middle_initials":null,"last_name":"Misra","page_name":"VatsalaMisra","domain_name":"independent","created_at":"2016-04-30T07:36:38.954-07:00","display_name":"Vatsala Misra","url":"https://independent.academia.edu/VatsalaMisra"},"attachments":[{"id":107388529,"title":"","file_type":"pdf","scribd_thumbnail_url":"https://attachments.academia-assets.com/107388529/thumbnails/1.jpg","file_name":"j.1440-1746.1995.tb01096.x20231115-1-yeiz6s.pdf","download_url":"https://www.academia.edu/attachments/107388529/download_file?st=MTczMjc1MTU1Nyw4LjIyMi4yMDguMTQ2&","bulk_download_file_name":"Diagnosing_ascites_Value_of_ascitic_flui.pdf","bulk_download_url":"https://d1wqtxts1xzle7.cloudfront.net/107388529/j.1440-1746.1995.tb01096.x20231115-1-yeiz6s-libre.pdf?1700048619=\u0026response-content-disposition=attachment%3B+filename%3DDiagnosing_ascites_Value_of_ascitic_flui.pdf\u0026Expires=1732708236\u0026Signature=Wpb1AURv3o67ywEec2ZXbuoaRthkC-CjjocL58cw6m2QFOVY7nBwYpz5JoB~RipTvAVFjSpjjqpR~GHxT9700PETmCZjnpPRyU2WW9Uu1ZfYj45XDVSUVNVyKszggQ4knw~SVW5RVtvdVE0L3KfDqhcJYlvVN10evUxt9ygQgX1GdSUQaFPC6bDWlOTvM~IQ0ZAAY6zfXWF-X9ZasPTZBYOsc5H2AEdOytZ-RtiZZB-LTYx47OReaa9l82QTflEpBN0WVEAW8jvwmXjN4M5wuf3kLsxHso5gxtrvyC6Gw6uv~SJ7W6VEFEnJNcTSaMmf6mIPU2JD7XSdjW3RPykf9w__\u0026Key-Pair-Id=APKAJLOHF5GGSLRBV4ZA"}],"research_interests":[{"id":26327,"name":"Medicine","url":"https://www.academia.edu/Documents/in/Medicine"},{"id":62112,"name":"Prospective studies","url":"https://www.academia.edu/Documents/in/Prospective_studies"},{"id":65390,"name":"Internal Medicine","url":"https://www.academia.edu/Documents/in/Internal_Medicine"},{"id":71343,"name":"Liver Cirrhosis","url":"https://www.academia.edu/Documents/in/Liver_Cirrhosis"},{"id":76408,"name":"Albumin","url":"https://www.academia.edu/Documents/in/Albumin"},{"id":90514,"name":"Cholesterol","url":"https://www.academia.edu/Documents/in/Cholesterol"},{"id":111972,"name":"Hepatocellular Carcinoma","url":"https://www.academia.edu/Documents/in/Hepatocellular_Carcinoma"},{"id":181569,"name":"Proteins","url":"https://www.academia.edu/Documents/in/Proteins"},{"id":244814,"name":"Clinical Sciences","url":"https://www.academia.edu/Documents/in/Clinical_Sciences"},{"id":260020,"name":"GASTROENTEROLOGY AND HEPATOLOGY.","url":"https://www.academia.edu/Documents/in/GASTROENTEROLOGY_AND_HEPATOLOGY"},{"id":790533,"name":"Liver Failure","url":"https://www.academia.edu/Documents/in/Liver_Failure"},{"id":901876,"name":"Sensitivity and Specificity","url":"https://www.academia.edu/Documents/in/Sensitivity_and_Specificity"},{"id":1281483,"name":"Ascites","url":"https://www.academia.edu/Documents/in/Ascites"},{"id":1593060,"name":"Blood Proteins","url":"https://www.academia.edu/Documents/in/Blood_Proteins"},{"id":2349258,"name":"Serum albumin","url":"https://www.academia.edu/Documents/in/Serum_albumin"},{"id":2836761,"name":"Total protein","url":"https://www.academia.edu/Documents/in/Total_protein"},{"id":3562342,"name":"Liver neoplasms","url":"https://www.academia.edu/Documents/in/Liver_neoplasms"},{"id":3960847,"name":"Gastroenterology and Hepatology","url":"https://www.academia.edu/Documents/in/Gastroenterology_and_Hepatology-1"},{"id":4074011,"name":" Ascitic Fluid","url":"https://www.academia.edu/Documents/in/Ascitic_Fluid"}],"urls":[{"id":35398403,"url":"http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1440-1746.1995.tb01096.x/fullpdf"}]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="109190292"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" rel="nofollow" href="https://www.academia.edu/109190292/Nodular_gastritis_in_adults_Clinical_features_endoscopic_appearance_histopathological_features_and_response_to_therapy"><img alt="Research paper thumbnail of Nodular gastritis in adults: Clinical features, endoscopic appearance, histopathological features, and response to therapy" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" rel="nofollow" href="https://www.academia.edu/109190292/Nodular_gastritis_in_adults_Clinical_features_endoscopic_appearance_histopathological_features_and_response_to_therapy">Nodular gastritis in adults: Clinical features, endoscopic appearance, histopathological features, and response to therapy</a></div><div class="wp-workCard_item"><span>Journal of Gastroenterology and Hepatology</span><span>, 2008</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">Background and Aims: The present study aims to determine the prevalence of nodular gastritis (NG...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">Background and Aims: The present study aims to determine the prevalence of nodular gastritis (NG) and ascertain its clinical presentation and histopathological features in adults. It also assesses its association with Helicobacter pylori and the normalization of endoscopic features, symptoms, and histology after anti H. pylori therapy.Methods: A total of 7140 patients undergoing upper gastrointestinal endoscopy were studied. Patients showing nodularity of the gastric mucosa at endoscopy and an age‐ and sex‐matched control group with normal gastric mucosa underwent biopsies from the gastric antrum and fundus. The biopsies were assessed for the presence of mucosal inflammation, activity, eosinophils, atrophy, lymphoid follicles, H. pylori, and the presence of intestinal metaplasia. Patients with NG were given triple therapy. Endoscopy and biopsy was repeated after 4 weeks of stopping therapy. The symptoms of the patients and histology were assessed pre‐ and post‐therapy.Results: Th...</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109190292"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109190292"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109190292; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=109190292]").text(description); $(".js-view-count[data-work-id=109190292]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 109190292; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='109190292']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 109190292, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=109190292]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":109190292,"title":"Nodular gastritis in adults: Clinical features, endoscopic appearance, histopathological features, and response to therapy","translated_title":"","metadata":{"abstract":"Background and Aims: The present study aims to determine the prevalence of nodular gastritis (NG) and ascertain its clinical presentation and histopathological features in adults. It also assesses its association with Helicobacter pylori and the normalization of endoscopic features, symptoms, and histology after anti H. pylori therapy.Methods: A total of 7140 patients undergoing upper gastrointestinal endoscopy were studied. Patients showing nodularity of the gastric mucosa at endoscopy and an age‐ and sex‐matched control group with normal gastric mucosa underwent biopsies from the gastric antrum and fundus. The biopsies were assessed for the presence of mucosal inflammation, activity, eosinophils, atrophy, lymphoid follicles, H. pylori, and the presence of intestinal metaplasia. Patients with NG were given triple therapy. Endoscopy and biopsy was repeated after 4 weeks of stopping therapy. The symptoms of the patients and histology were assessed pre‐ and post‐therapy.Results: Th...","publisher":"Wiley","publication_date":{"day":null,"month":null,"year":2008,"errors":{}},"publication_name":"Journal of Gastroenterology and Hepatology"},"translated_abstract":"Background and Aims: The present study aims to determine the prevalence of nodular gastritis (NG) and ascertain its clinical presentation and histopathological features in adults. It also assesses its association with Helicobacter pylori and the normalization of endoscopic features, symptoms, and histology after anti H. pylori therapy.Methods: A total of 7140 patients undergoing upper gastrointestinal endoscopy were studied. Patients showing nodularity of the gastric mucosa at endoscopy and an age‐ and sex‐matched control group with normal gastric mucosa underwent biopsies from the gastric antrum and fundus. The biopsies were assessed for the presence of mucosal inflammation, activity, eosinophils, atrophy, lymphoid follicles, H. pylori, and the presence of intestinal metaplasia. Patients with NG were given triple therapy. Endoscopy and biopsy was repeated after 4 weeks of stopping therapy. The symptoms of the patients and histology were assessed pre‐ and post‐therapy.Results: Th...","internal_url":"https://www.academia.edu/109190292/Nodular_gastritis_in_adults_Clinical_features_endoscopic_appearance_histopathological_features_and_response_to_therapy","translated_internal_url":"","created_at":"2023-11-15T01:49:31.346-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":48000299,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[],"downloadable_attachments":[],"slug":"Nodular_gastritis_in_adults_Clinical_features_endoscopic_appearance_histopathological_features_and_response_to_therapy","translated_slug":"","page_count":null,"language":"en","content_type":"Work","owner":{"id":48000299,"first_name":"Vatsala","middle_initials":null,"last_name":"Misra","page_name":"VatsalaMisra","domain_name":"independent","created_at":"2016-04-30T07:36:38.954-07:00","display_name":"Vatsala Misra","url":"https://independent.academia.edu/VatsalaMisra"},"attachments":[],"research_interests":[{"id":3274,"name":"Gastroenterology","url":"https://www.academia.edu/Documents/in/Gastroenterology"},{"id":21460,"name":"Helicobacter pylori","url":"https://www.academia.edu/Documents/in/Helicobacter_pylori"},{"id":26327,"name":"Medicine","url":"https://www.academia.edu/Documents/in/Medicine"},{"id":37826,"name":"Biopsy","url":"https://www.academia.edu/Documents/in/Biopsy"},{"id":43838,"name":"India","url":"https://www.academia.edu/Documents/in/India"},{"id":65390,"name":"Internal Medicine","url":"https://www.academia.edu/Documents/in/Internal_Medicine"},{"id":244814,"name":"Clinical Sciences","url":"https://www.academia.edu/Documents/in/Clinical_Sciences"},{"id":289271,"name":"Aged","url":"https://www.academia.edu/Documents/in/Aged"},{"id":289330,"name":"Prevalence","url":"https://www.academia.edu/Documents/in/Prevalence"},{"id":335984,"name":"Anti-Bacterial Agents","url":"https://www.academia.edu/Documents/in/Anti-Bacterial_Agents"},{"id":925054,"name":"Gastritis","url":"https://www.academia.edu/Documents/in/Gastritis"},{"id":1295203,"name":"Gastroscopy","url":"https://www.academia.edu/Documents/in/Gastroscopy"},{"id":1819399,"name":"Case Control Studies","url":"https://www.academia.edu/Documents/in/Case_Control_Studies"},{"id":2452785,"name":"Proton Pump Inhibitors","url":"https://www.academia.edu/Documents/in/Proton_Pump_Inhibitors"},{"id":2547982,"name":"Gastric mucosa","url":"https://www.academia.edu/Documents/in/Gastric_mucosa"}],"urls":[{"id":35398402,"url":"https://onlinelibrary.wiley.com/doi/pdf/10.1111/j.1440-1746.2007.05044.x"}]}, dispatcherData: dispatcherData }); 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$(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="109190290"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" rel="nofollow" href="https://www.academia.edu/109190290/Preoperative_sonographic_diagnosis_of_acute_appendicitis_caused_byAscaris_lumbricoides"><img alt="Research paper thumbnail of Preoperative sonographic diagnosis of acute appendicitis caused byAscaris lumbricoides" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" rel="nofollow" href="https://www.academia.edu/109190290/Preoperative_sonographic_diagnosis_of_acute_appendicitis_caused_byAscaris_lumbricoides">Preoperative sonographic diagnosis of acute appendicitis caused byAscaris lumbricoides</a></div><div class="wp-workCard_item"><span>Journal of Clinical Ultrasound</span><span>, 1999</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">Acute appendicitis caused by Ascaris lumbricoides is an uncommon variant of a common disease. We ...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">Acute appendicitis caused by Ascaris lumbricoides is an uncommon variant of a common disease. We describe a case in which sonography was used for preoperative diagnosis of ascaris appendicitis.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109190290"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109190290"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109190290; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=109190290]").text(description); $(".js-view-count[data-work-id=109190290]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 109190290; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='109190290']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 109190290, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=109190290]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":109190290,"title":"Preoperative sonographic diagnosis of acute appendicitis caused byAscaris lumbricoides","translated_title":"","metadata":{"abstract":"Acute appendicitis caused by Ascaris lumbricoides is an uncommon variant of a common disease. 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The factors influencing colonic involvement also are unknown. Methods: Seventy-one patients with amebic liver abscess were studied. Colonoscopy was performed in all patients. Results: Abdominal pain and fever were the most common presenting manifestations. Ten patients (14%) had diarrhea at admission. Ten other patients had a history of diarrhea during the preceding 2 months. Thirty-nine (55%) patients had colonic ulcers. Colonic ulcers were present in 18 of the 20 (90%) patients with ongoing diarrhea or a history of recent diarrhea, and in 21 of 51 (41%) patients without diarrhea (p \u003c 0.001). Thirty (42%) patients had small, discrete ulcers in the cecum, the ascending colon, or the region of the hepatic flexure. Nine patients had large, multiple ulcers with surrounding inflammation. In these patients, the ulcers were present either in the left colon (n = 7) or throughout the colon (n = 2). The mean (standard deviation) age of patients with 5 or more ulcers was significantly greater than that of patients with fewer than 5 ulcers (49.8 [14.6] years vs. 37 [11.7] years; p \u003c 0.05). Multiple ulcers were noted in 7 of the 10 patients (70%) with diarrhea at admission and in two of the 61 patients (3%) in whom diarrhea was not a presenting symptom (p \u003c 0.001). No association was noted between the location of the abscess in the liver and the colonic lesions. Conclusions: Colonic ulcers are a common finding, occurring in more than half of patients with amebic liver abscess. They are more likely to be present if the patient has diarrhea as a presenting symptom or has had diarrhea in the recent past. Multiple, large, and left-sided ulcers are more common in elderly patients and in those in whom diarrhea is the presenting symptom. However, there is no association between the location of the abscess in the liver and colonic lesions.","publication_date":{"day":null,"month":null,"year":2004,"errors":{}},"publication_name":"Gastrointestinal 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$(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="109190288"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/109190288/Esophageal_stricture_as_a_late_sequel_of_Stevens_Johnson_syndrome_in_adults_incidental_detection_because_of_foreign_body_impaction"><img alt="Research paper thumbnail of Esophageal stricture as a late sequel of Stevens-Johnson syndrome in adults: incidental detection because of foreign body impaction" class="work-thumbnail" src="https://attachments.academia-assets.com/107388527/thumbnails/1.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/109190288/Esophageal_stricture_as_a_late_sequel_of_Stevens_Johnson_syndrome_in_adults_incidental_detection_because_of_foreign_body_impaction">Esophageal stricture as a late sequel of Stevens-Johnson syndrome in adults: incidental detection because of foreign body impaction</a></div><div class="wp-workCard_item"><span>Gastrointestinal Endoscopy</span><span>, 2004</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><a id="ba06929e4c752a949764eb9563cea16c" class="wp-workCard--action" rel="nofollow" data-click-track="profile-work-strip-download" data-download="{"attachment_id":107388527,"asset_id":109190288,"asset_type":"Work","button_location":"profile"}" href="https://www.academia.edu/attachments/107388527/download_file?st=MTczMjc1MTU1Nyw4LjIyMi4yMDguMTQ2&s=profile"><span><i class="fa fa-arrow-down"></i></span><span>Download</span></a><span class="wp-workCard--action 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Endoscopy","url":"https://www.academia.edu/Documents/in/Gastrointestinal_Endoscopy"},{"id":364282,"name":"Impaction","url":"https://www.academia.edu/Documents/in/Impaction"},{"id":405739,"name":"Esophagus","url":"https://www.academia.edu/Documents/in/Esophagus"},{"id":620049,"name":"Risk Factors","url":"https://www.academia.edu/Documents/in/Risk_Factors-1"},{"id":1295297,"name":"Foreign Bodies","url":"https://www.academia.edu/Documents/in/Foreign_Bodies"},{"id":1971419,"name":"Esophagoscopy","url":"https://www.academia.edu/Documents/in/Esophagoscopy"},{"id":2460250,"name":"Esophageal Stenosis","url":"https://www.academia.edu/Documents/in/Esophageal_Stenosis"},{"id":2560984,"name":"foreign body","url":"https://www.academia.edu/Documents/in/foreign_body"}],"urls":[{"id":35398400,"url":"https://api.elsevier.com/content/article/PII:S001651070302710X?httpAccept=text/xml"}]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="109190287"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/109190287/Endoscopic_Biopsies_from_Normal_Appearing_Terminal_Ileum_and_Cecum_in_Patients_with_Suspected_Colonic_Tuberculosis"><img alt="Research paper thumbnail of Endoscopic Biopsies from Normal-Appearing Terminal Ileum and Cecum in Patients with Suspected Colonic Tuberculosis" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/109190287/Endoscopic_Biopsies_from_Normal_Appearing_Terminal_Ileum_and_Cecum_in_Patients_with_Suspected_Colonic_Tuberculosis">Endoscopic Biopsies from Normal-Appearing Terminal Ileum and Cecum in Patients with Suspected Colonic Tuberculosis</a></div><div class="wp-workCard_item"><span>Endoscopy</span><span>, 2004</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">Colonic tuberculosis is generally diagnosed by colonoscopy and targeted biopsy of lesions. Howeve...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">Colonic tuberculosis is generally diagnosed by colonoscopy and targeted biopsy of lesions. However, the diagnostic yield of colonic biopsies is not very good. So far as we are aware, there have been no studies investigating the role of biopsies from endoscopically normal-appearing cecum and terminal ileum in diagnosing colonic or ileal tuberculosis, or both. Patients with a clinical suspicion of colonic tuberculosis, in whom no endoscopic abnormalities were found on colonoscopy or ileoscopy, were included in the study. Multiple biopsies were obtained from the cecum and ileum. Fifty patients were studied. Intubation of the terminal ileum was possible in 43 patients (86 %). Histological examination of biopsies obtained from the cecum and terminal ileum showed noncaseating granuloma in two patients. Both of these biopsies were from the terminal ileum. In two other patients, collections of loosely arranged epithelioid cells were observed. This established the diagnosis in these four patients (8 %). In the remaining 46 patients, histology showed nonspecific inflammation in 18 patients (in the cecum in 15 and in the terminal ileum in seven). The other biopsies did not show any abnormalities (33 from the cecum, 34 from the terminal ileum). Histological examination of biopsies from the normal-appearing cecum and terminal ileum is useful in a small but significant number of patients with colonic tuberculosis.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109190287"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109190287"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109190287; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=109190287]").text(description); $(".js-view-count[data-work-id=109190287]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 109190287; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='109190287']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 109190287, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=109190287]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":109190287,"title":"Endoscopic Biopsies from Normal-Appearing Terminal Ileum and Cecum in Patients with Suspected Colonic Tuberculosis","translated_title":"","metadata":{"abstract":"Colonic tuberculosis is generally diagnosed by colonoscopy and targeted biopsy of lesions. However, the diagnostic yield of colonic biopsies is not very good. So far as we are aware, there have been no studies investigating the role of biopsies from endoscopically normal-appearing cecum and terminal ileum in diagnosing colonic or ileal tuberculosis, or both. Patients with a clinical suspicion of colonic tuberculosis, in whom no endoscopic abnormalities were found on colonoscopy or ileoscopy, were included in the study. Multiple biopsies were obtained from the cecum and ileum. Fifty patients were studied. Intubation of the terminal ileum was possible in 43 patients (86 %). Histological examination of biopsies obtained from the cecum and terminal ileum showed noncaseating granuloma in two patients. Both of these biopsies were from the terminal ileum. In two other patients, collections of loosely arranged epithelioid cells were observed. This established the diagnosis in these four patients (8 %). In the remaining 46 patients, histology showed nonspecific inflammation in 18 patients (in the cecum in 15 and in the terminal ileum in seven). The other biopsies did not show any abnormalities (33 from the cecum, 34 from the terminal ileum). Histological examination of biopsies from the normal-appearing cecum and terminal ileum is useful in a small but significant number of patients with colonic tuberculosis.","publisher":"Georg Thieme Verlag KG","publication_date":{"day":null,"month":null,"year":2004,"errors":{}},"publication_name":"Endoscopy"},"translated_abstract":"Colonic tuberculosis is generally diagnosed by colonoscopy and targeted biopsy of lesions. However, the diagnostic yield of colonic biopsies is not very good. So far as we are aware, there have been no studies investigating the role of biopsies from endoscopically normal-appearing cecum and terminal ileum in diagnosing colonic or ileal tuberculosis, or both. Patients with a clinical suspicion of colonic tuberculosis, in whom no endoscopic abnormalities were found on colonoscopy or ileoscopy, were included in the study. Multiple biopsies were obtained from the cecum and ileum. Fifty patients were studied. Intubation of the terminal ileum was possible in 43 patients (86 %). Histological examination of biopsies obtained from the cecum and terminal ileum showed noncaseating granuloma in two patients. Both of these biopsies were from the terminal ileum. In two other patients, collections of loosely arranged epithelioid cells were observed. This established the diagnosis in these four patients (8 %). In the remaining 46 patients, histology showed nonspecific inflammation in 18 patients (in the cecum in 15 and in the terminal ileum in seven). The other biopsies did not show any abnormalities (33 from the cecum, 34 from the terminal ileum). Histological examination of biopsies from the normal-appearing cecum and terminal ileum is useful in a small but significant number of patients with colonic tuberculosis.","internal_url":"https://www.academia.edu/109190287/Endoscopic_Biopsies_from_Normal_Appearing_Terminal_Ileum_and_Cecum_in_Patients_with_Suspected_Colonic_Tuberculosis","translated_internal_url":"","created_at":"2023-11-15T01:49:30.307-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":48000299,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[],"downloadable_attachments":[],"slug":"Endoscopic_Biopsies_from_Normal_Appearing_Terminal_Ileum_and_Cecum_in_Patients_with_Suspected_Colonic_Tuberculosis","translated_slug":"","page_count":null,"language":"en","content_type":"Work","owner":{"id":48000299,"first_name":"Vatsala","middle_initials":null,"last_name":"Misra","page_name":"VatsalaMisra","domain_name":"independent","created_at":"2016-04-30T07:36:38.954-07:00","display_name":"Vatsala Misra","url":"https://independent.academia.edu/VatsalaMisra"},"attachments":[],"research_interests":[{"id":3274,"name":"Gastroenterology","url":"https://www.academia.edu/Documents/in/Gastroenterology"},{"id":26327,"name":"Medicine","url":"https://www.academia.edu/Documents/in/Medicine"},{"id":37805,"name":"Endoscopy","url":"https://www.academia.edu/Documents/in/Endoscopy"},{"id":37826,"name":"Biopsy","url":"https://www.academia.edu/Documents/in/Biopsy"},{"id":62235,"name":"Tuberculosis","url":"https://www.academia.edu/Documents/in/Tuberculosis"},{"id":65390,"name":"Internal Medicine","url":"https://www.academia.edu/Documents/in/Internal_Medicine"},{"id":162159,"name":"Differential Diagnosis","url":"https://www.academia.edu/Documents/in/Differential_Diagnosis"},{"id":227324,"name":"Colonoscopy","url":"https://www.academia.edu/Documents/in/Colonoscopy"},{"id":244814,"name":"Clinical Sciences","url":"https://www.academia.edu/Documents/in/Clinical_Sciences"},{"id":829416,"name":"Colon","url":"https://www.academia.edu/Documents/in/Colon"},{"id":1901112,"name":"Ileum","url":"https://www.academia.edu/Documents/in/Ileum"}],"urls":[{"id":35398399,"url":"http://www.thieme-connect.de/products/ejournals/pdf/10.1055/s-2004-814518.pdf"}]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="109190286"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/109190286/AgNORs_in_benign_borderline_and_transitional_cell_neoplasms_of_the_urinary_bladder"><img alt="Research paper thumbnail of AgNORs in benign, borderline and transitional cell neoplasms of the urinary bladder" class="work-thumbnail" src="https://attachments.academia-assets.com/107388525/thumbnails/1.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/109190286/AgNORs_in_benign_borderline_and_transitional_cell_neoplasms_of_the_urinary_bladder">AgNORs in benign, borderline and transitional cell neoplasms of the urinary bladder</a></div><div class="wp-workCard_item"><span>APMIS</span><span>, 1998</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">Argyrophilic nucleolar organizer regions (AgNORs) were studied in 106 tissue samples from the uri...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">Argyrophilic nucleolar organizer regions (AgNORs) were studied in 106 tissue samples from the urinary bladder (6 normal transitional epithelium, 5 cystitis, 12 hyperplastic, 14 dysplastic lesions, 12 carcinoma in situ, 4 transitional cell carcinoma grade 0, 12 grade 1, 15 grade II and 12 grade III) to evaluate their role in differentiating benign, borderline and malignant lesions. The NOR counts presented a rising scale from normal (2.21), inflammatory (3.93 for both cystitis and hyperplasia), dysplastic (4.16), carcinoma in situ (5.08) to malignant lesions (5.28 grade I, 6.59 grade II and 8.37 grade III). It was concluded that AgNORs do not have a diagnostic role in these lesions, but that they can act as a reliable adjunct to existing parameters in the early detection of tumour recurrence and grading of malignant neoplasms.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><a id="304b2443c7acf6d249cf0b0a4e1a6ae6" class="wp-workCard--action" rel="nofollow" data-click-track="profile-work-strip-download" data-download="{"attachment_id":107388525,"asset_id":109190286,"asset_type":"Work","button_location":"profile"}" href="https://www.academia.edu/attachments/107388525/download_file?st=MTczMjc1MTU1OCw4LjIyMi4yMDguMTQ2&s=profile"><span><i class="fa fa-arrow-down"></i></span><span>Download</span></a><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109190286"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109190286"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109190286; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=109190286]").text(description); $(".js-view-count[data-work-id=109190286]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 109190286; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='109190286']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 109190286, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (true){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "304b2443c7acf6d249cf0b0a4e1a6ae6" } } $('.js-work-strip[data-work-id=109190286]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":109190286,"title":"AgNORs in benign, borderline and transitional cell neoplasms of the urinary bladder","translated_title":"","metadata":{"abstract":"Argyrophilic nucleolar organizer regions (AgNORs) were studied in 106 tissue samples from the urinary bladder (6 normal transitional epithelium, 5 cystitis, 12 hyperplastic, 14 dysplastic lesions, 12 carcinoma in situ, 4 transitional cell carcinoma grade 0, 12 grade 1, 15 grade II and 12 grade III) to evaluate their role in differentiating benign, borderline and malignant lesions. The NOR counts presented a rising scale from normal (2.21), inflammatory (3.93 for both cystitis and hyperplasia), dysplastic (4.16), carcinoma in situ (5.08) to malignant lesions (5.28 grade I, 6.59 grade II and 8.37 grade III). It was concluded that AgNORs do not have a diagnostic role in these lesions, but that they can act as a reliable adjunct to existing parameters in the early detection of tumour recurrence and grading of malignant neoplasms.","publisher":"Wiley","publication_date":{"day":null,"month":null,"year":1998,"errors":{}},"publication_name":"APMIS"},"translated_abstract":"Argyrophilic nucleolar organizer regions (AgNORs) were studied in 106 tissue samples from the urinary bladder (6 normal transitional epithelium, 5 cystitis, 12 hyperplastic, 14 dysplastic lesions, 12 carcinoma in situ, 4 transitional cell carcinoma grade 0, 12 grade 1, 15 grade II and 12 grade III) to evaluate their role in differentiating benign, borderline and malignant lesions. The NOR counts presented a rising scale from normal (2.21), inflammatory (3.93 for both cystitis and hyperplasia), dysplastic (4.16), carcinoma in situ (5.08) to malignant lesions (5.28 grade I, 6.59 grade II and 8.37 grade III). It was concluded that AgNORs do not have a diagnostic role in these lesions, but that they can act as a reliable adjunct to existing parameters in the early detection of tumour recurrence and grading of malignant neoplasms.","internal_url":"https://www.academia.edu/109190286/AgNORs_in_benign_borderline_and_transitional_cell_neoplasms_of_the_urinary_bladder","translated_internal_url":"","created_at":"2023-11-15T01:49:30.090-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":48000299,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[],"downloadable_attachments":[{"id":107388525,"title":"","file_type":"pdf","scribd_thumbnail_url":"https://attachments.academia-assets.com/107388525/thumbnails/1.jpg","file_name":"j.1699-0463.1998.tb00250.x20231115-1-xkw8vv.pdf","download_url":"https://www.academia.edu/attachments/107388525/download_file?st=MTczMjc1MTU1OCw4LjIyMi4yMDguMTQ2&","bulk_download_file_name":"AgNORs_in_benign_borderline_and_transiti.pdf","bulk_download_url":"https://d1wqtxts1xzle7.cloudfront.net/107388525/j.1699-0463.1998.tb00250.x20231115-1-xkw8vv-libre.pdf?1700048619=\u0026response-content-disposition=attachment%3B+filename%3DAgNORs_in_benign_borderline_and_transiti.pdf\u0026Expires=1732755158\u0026Signature=EMJ0HspAZcp33YrlNbpU9Z8Z0ZEcKaCiZWhCQ3P-RS4g~ODq6QZzKqoIASWEPIiCSi5oLZq~sYxz7q1lJpliJbfDN-DS9aUaocHafwLT3Jna4M4hJKVXth6AnabyzVaWRbtzPUSYSyqAMMGef3MXMXJttx2WpzcUZyHxBecl1dU2i3gxibGYpQoMnWPmBhxYBA~OsKqoWwj-ZZuu3tKvunOV28DJd4UOI1eJfvhviPIDz4-irPuGzpHTqPyDZvSWo5T332h~88yLl-CqI5wwEfYAUeUNVvsRfoq4lnzWEPcksohvJRDgZ3UqPU5dXQLt8mV8VNxdmBsQObrJN0HatA__\u0026Key-Pair-Id=APKAJLOHF5GGSLRBV4ZA"}],"slug":"AgNORs_in_benign_borderline_and_transitional_cell_neoplasms_of_the_urinary_bladder","translated_slug":"","page_count":6,"language":"en","content_type":"Work","owner":{"id":48000299,"first_name":"Vatsala","middle_initials":null,"last_name":"Misra","page_name":"VatsalaMisra","domain_name":"independent","created_at":"2016-04-30T07:36:38.954-07:00","display_name":"Vatsala Misra","url":"https://independent.academia.edu/VatsalaMisra"},"attachments":[{"id":107388525,"title":"","file_type":"pdf","scribd_thumbnail_url":"https://attachments.academia-assets.com/107388525/thumbnails/1.jpg","file_name":"j.1699-0463.1998.tb00250.x20231115-1-xkw8vv.pdf","download_url":"https://www.academia.edu/attachments/107388525/download_file?st=MTczMjc1MTU1OCw4LjIyMi4yMDguMTQ2&","bulk_download_file_name":"AgNORs_in_benign_borderline_and_transiti.pdf","bulk_download_url":"https://d1wqtxts1xzle7.cloudfront.net/107388525/j.1699-0463.1998.tb00250.x20231115-1-xkw8vv-libre.pdf?1700048619=\u0026response-content-disposition=attachment%3B+filename%3DAgNORs_in_benign_borderline_and_transiti.pdf\u0026Expires=1732755158\u0026Signature=EMJ0HspAZcp33YrlNbpU9Z8Z0ZEcKaCiZWhCQ3P-RS4g~ODq6QZzKqoIASWEPIiCSi5oLZq~sYxz7q1lJpliJbfDN-DS9aUaocHafwLT3Jna4M4hJKVXth6AnabyzVaWRbtzPUSYSyqAMMGef3MXMXJttx2WpzcUZyHxBecl1dU2i3gxibGYpQoMnWPmBhxYBA~OsKqoWwj-ZZuu3tKvunOV28DJd4UOI1eJfvhviPIDz4-irPuGzpHTqPyDZvSWo5T332h~88yLl-CqI5wwEfYAUeUNVvsRfoq4lnzWEPcksohvJRDgZ3UqPU5dXQLt8mV8VNxdmBsQObrJN0HatA__\u0026Key-Pair-Id=APKAJLOHF5GGSLRBV4ZA"}],"research_interests":[{"id":165,"name":"Pathology","url":"https://www.academia.edu/Documents/in/Pathology"},{"id":1290,"name":"Immunology","url":"https://www.academia.edu/Documents/in/Immunology"},{"id":6947,"name":"Medical Microbiology","url":"https://www.academia.edu/Documents/in/Medical_Microbiology"},{"id":26327,"name":"Medicine","url":"https://www.academia.edu/Documents/in/Medicine"},{"id":176968,"name":"Hyperplasia","url":"https://www.academia.edu/Documents/in/Hyperplasia"},{"id":244814,"name":"Clinical Sciences","url":"https://www.academia.edu/Documents/in/Clinical_Sciences"},{"id":558694,"name":"Nucleolus Organizer Region","url":"https://www.academia.edu/Documents/in/Nucleolus_Organizer_Region"},{"id":1433630,"name":"Transitional Cell Carcinoma","url":"https://www.academia.edu/Documents/in/Transitional_Cell_Carcinoma"},{"id":1902592,"name":"Urinary Bladder","url":"https://www.academia.edu/Documents/in/Urinary_Bladder"},{"id":3287751,"name":"cystitis","url":"https://www.academia.edu/Documents/in/cystitis"},{"id":3364752,"name":"Carcinoma in situ","url":"https://www.academia.edu/Documents/in/Carcinoma_in_situ"},{"id":4102912,"name":"Urinary bladder neoplasms","url":"https://www.academia.edu/Documents/in/Urinary_bladder_neoplasms"}],"urls":[{"id":35398398,"url":"https://onlinelibrary.wiley.com/doi/pdf/10.1111/j.1699-0463.1998.tb00250.x"}]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="109190235"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" rel="nofollow" href="https://www.academia.edu/109190235/Helicobacter_pylori_and_gastric_cancer"><img alt="Research paper thumbnail of Helicobacter pylori and gastric cancer" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" rel="nofollow" href="https://www.academia.edu/109190235/Helicobacter_pylori_and_gastric_cancer">Helicobacter pylori and gastric cancer</a></div><div class="wp-workCard_item"><span>The American Journal of Medicine</span><span>, 1999</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">This review focuses on the similarities between the epidemiology of gastric cancer and the epidem...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">This review focuses on the similarities between the epidemiology of gastric cancer and the epidemiology of Helicobacter pylori. Their demographic patterns and the results of studies regarding familial and environmental risk factors are described. The association of gastric cancer and H. pylori infection with both gastric ulcer and chronic atrophic gastritis is also characterized and the possibility that a H. pylori infection could lead to gastric cancer is discussed.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109190235"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109190235"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109190235; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=109190235]").text(description); $(".js-view-count[data-work-id=109190235]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 109190235; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='109190235']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 109190235, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=109190235]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":109190235,"title":"Helicobacter pylori and gastric cancer","translated_title":"","metadata":{"abstract":"This review focuses on the similarities between the epidemiology of gastric cancer and the epidemiology of Helicobacter pylori. Their demographic patterns and the results of studies regarding familial and environmental risk factors are described. The association of gastric cancer and H. pylori infection with both gastric ulcer and chronic atrophic gastritis is also characterized and the possibility that a H. pylori infection could lead to gastric cancer is discussed.","publisher":"Elsevier BV","publication_date":{"day":null,"month":null,"year":1999,"errors":{}},"publication_name":"The American Journal of Medicine"},"translated_abstract":"This review focuses on the similarities between the epidemiology of gastric cancer and the epidemiology of Helicobacter pylori. Their demographic patterns and the results of studies regarding familial and environmental risk factors are described. 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$(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="104662124"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/104662124/Morphometric_Assessment_of_Chronic_Inflammatory_Cells_in_Colonic_Biopsies_of_Patients_with_Irritable_Bowel_Syndrome"><img alt="Research paper thumbnail of Morphometric Assessment of Chronic Inflammatory Cells in Colonic Biopsies of Patients with Irritable Bowel Syndrome" class="work-thumbnail" src="https://attachments.academia-assets.com/104330772/thumbnails/1.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/104662124/Morphometric_Assessment_of_Chronic_Inflammatory_Cells_in_Colonic_Biopsies_of_Patients_with_Irritable_Bowel_Syndrome">Morphometric Assessment of Chronic Inflammatory Cells in Colonic Biopsies of Patients with Irritable Bowel Syndrome</a></div><div class="wp-workCard_item"><span>JOURNAL OF CLINICAL AND DIAGNOSTIC RESEARCH</span><span>, 2019</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><a id="6a33a05de4b8adb2700230cf817b1129" class="wp-workCard--action" rel="nofollow" data-click-track="profile-work-strip-download" data-download="{"attachment_id":104330772,"asset_id":104662124,"asset_type":"Work","button_location":"profile"}" href="https://www.academia.edu/attachments/104330772/download_file?st=MTczMjc1MTU1OCw4LjIyMi4yMDguMTQ2&s=profile"><span><i class="fa fa-arrow-down"></i></span><span>Download</span></a><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="104662124"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="104662124"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 104662124; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=104662124]").text(description); $(".js-view-count[data-work-id=104662124]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 104662124; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='104662124']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 104662124, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (true){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); 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It is a functional bowel disorder characterised by abdominal pain, cramps, changes in bowel habit, gassiness, bloating, and nausea, but without any specific diagnostic markers. Thus, the diagnosis is based on clinical presentation for which several symptoms based diagnostic criteria have been suggested time to time over the last two decades [1]. These include Manning, Rome I, Rome II , Rome III and most recently Rome IV criteria. The Rome criteria is the most widely accepted worldwide because of its reasonable sensitivity and specificity [2]. IBS presents clinically as one of these three predominant subtypes-(1) IBS with constipation (IBS-C); (2) IBS with diarrhoea (IBS-D) and (3) Mixed IBS (IBS-M). According to onset of symptoms, IBS patients are further divided into Post Infectious IBS (PI-IBS) and Non Postinfectious IBS (NPI-IBS). PI-IBS is defined as Rome criteria positive IBS of acute onset developing after an infectious aetiology [3]. Lack of disease defining biomarkers makes the aetiology of this disease complicated with various factors like altered gastrointestinal motility, visceral hypersensitivity, post infectious reactivity, brain-gut interactions, alteration in faecal micro flora, bacterial overgrowth, barrier dysfunctions, food sensitivity, carbohydrate malabsorption, and intestinal inflammation being implicated in its pathogenesis. A genetic contribution also seems likely based upon the high degree of concordance seen among monozygotic twins as well as familial aggregations observed [4].","publication_date":{"day":null,"month":null,"year":2019,"errors":{}},"publication_name":"JOURNAL OF CLINICAL AND DIAGNOSTIC RESEARCH","grobid_abstract_attachment_id":104330772},"translated_abstract":null,"internal_url":"https://www.academia.edu/104662124/Morphometric_Assessment_of_Chronic_Inflammatory_Cells_in_Colonic_Biopsies_of_Patients_with_Irritable_Bowel_Syndrome","translated_internal_url":"","created_at":"2023-07-17T09:44:59.810-07:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":48000299,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[],"downloadable_attachments":[{"id":104330772,"title":"","file_type":"pdf","scribd_thumbnail_url":"https://attachments.academia-assets.com/104330772/thumbnails/1.jpg","file_name":"40569_PDKM_V-2PoG_KM_CE_5BRa1_5D_PF1AG_OM_PFASL_PBNC_AG_PF2TR_SL_PNSL.pdf","download_url":"https://www.academia.edu/attachments/104330772/download_file?st=MTczMjc1MTU1OCw4LjIyMi4yMDguMTQ2&","bulk_download_file_name":"Morphometric_Assessment_of_Chronic_Infla.pdf","bulk_download_url":"https://d1wqtxts1xzle7.cloudfront.net/104330772/40569_PDKM_V-2PoG_KM_CE_5BRa1_5D_PF1AG_OM_PFASL_PBNC_AG_PF2TR_SL_PNSL-libre.pdf?1689612376=\u0026response-content-disposition=attachment%3B+filename%3DMorphometric_Assessment_of_Chronic_Infla.pdf\u0026Expires=1732755158\u0026Signature=Dk0RLf2HgFjC2rSxnG58jlw7aZB4B2WfhwECyDVxgnu59sRS5MbQRslraZTEEWP0iaodpNB99BC-xsq-uVsuch-nxf6ckSlenEt5stxK5OaJtMLKYQOfb4Gckx8w6eD49QbTMzDNVUcsKKt8Sodl9Km8FDrR1k9jrHukYJAdIJrbxp24wOxjcMEqb~I3bqNdTYF5AF4ArFwGUSjMLtjnus8NMD-OGyB~amcXypR3SL2ANvkJwA58JjG~PKFSJCrtykqzqzHlYP0hm0Xjaqxvdh5T8472qYHFTqcF0TgO~pWdYX4vN1WP1S9xROZl-IpTHFOZgZIRJKU8OLkzxtjung__\u0026Key-Pair-Id=APKAJLOHF5GGSLRBV4ZA"}],"slug":"Morphometric_Assessment_of_Chronic_Inflammatory_Cells_in_Colonic_Biopsies_of_Patients_with_Irritable_Bowel_Syndrome","translated_slug":"","page_count":4,"language":"en","content_type":"Work","owner":{"id":48000299,"first_name":"Vatsala","middle_initials":null,"last_name":"Misra","page_name":"VatsalaMisra","domain_name":"independent","created_at":"2016-04-30T07:36:38.954-07:00","display_name":"Vatsala Misra","url":"https://independent.academia.edu/VatsalaMisra"},"attachments":[{"id":104330772,"title":"","file_type":"pdf","scribd_thumbnail_url":"https://attachments.academia-assets.com/104330772/thumbnails/1.jpg","file_name":"40569_PDKM_V-2PoG_KM_CE_5BRa1_5D_PF1AG_OM_PFASL_PBNC_AG_PF2TR_SL_PNSL.pdf","download_url":"https://www.academia.edu/attachments/104330772/download_file?st=MTczMjc1MTU1OCw4LjIyMi4yMDguMTQ2&","bulk_download_file_name":"Morphometric_Assessment_of_Chronic_Infla.pdf","bulk_download_url":"https://d1wqtxts1xzle7.cloudfront.net/104330772/40569_PDKM_V-2PoG_KM_CE_5BRa1_5D_PF1AG_OM_PFASL_PBNC_AG_PF2TR_SL_PNSL-libre.pdf?1689612376=\u0026response-content-disposition=attachment%3B+filename%3DMorphometric_Assessment_of_Chronic_Infla.pdf\u0026Expires=1732755158\u0026Signature=Dk0RLf2HgFjC2rSxnG58jlw7aZB4B2WfhwECyDVxgnu59sRS5MbQRslraZTEEWP0iaodpNB99BC-xsq-uVsuch-nxf6ckSlenEt5stxK5OaJtMLKYQOfb4Gckx8w6eD49QbTMzDNVUcsKKt8Sodl9Km8FDrR1k9jrHukYJAdIJrbxp24wOxjcMEqb~I3bqNdTYF5AF4ArFwGUSjMLtjnus8NMD-OGyB~amcXypR3SL2ANvkJwA58JjG~PKFSJCrtykqzqzHlYP0hm0Xjaqxvdh5T8472qYHFTqcF0TgO~pWdYX4vN1WP1S9xROZl-IpTHFOZgZIRJKU8OLkzxtjung__\u0026Key-Pair-Id=APKAJLOHF5GGSLRBV4ZA"}],"research_interests":[{"id":3274,"name":"Gastroenterology","url":"https://www.academia.edu/Documents/in/Gastroenterology"},{"id":26327,"name":"Medicine","url":"https://www.academia.edu/Documents/in/Medicine"},{"id":65390,"name":"Internal Medicine","url":"https://www.academia.edu/Documents/in/Internal_Medicine"},{"id":214598,"name":"Irritable Bowel Syndrome","url":"https://www.academia.edu/Documents/in/Irritable_Bowel_Syndrome"}],"urls":[{"id":32938474,"url":"https://jcdr.net/article_fulltext.asp?issn=0973-709x\u0026year=2019\u0026volume=13\u0026issue=5\u0026page=EC06\u0026issn=0973-709x\u0026id=12852"}]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="104662123"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/104662123/Ileoscopy_in_patients_with_ileocolonic_tuberculosis"><img alt="Research paper thumbnail of Ileoscopy in patients with ileocolonic tuberculosis" class="work-thumbnail" src="https://attachments.academia-assets.com/104330773/thumbnails/1.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/104662123/Ileoscopy_in_patients_with_ileocolonic_tuberculosis">Ileoscopy in patients with ileocolonic tuberculosis</a></div><div class="wp-workCard_item"><span>World Journal of Gastroenterology</span><span>, 2007</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><a id="80e54b12649c6ee5bee95ba446abad9a" class="wp-workCard--action" rel="nofollow" data-click-track="profile-work-strip-download" data-download="{"attachment_id":104330773,"asset_id":104662123,"asset_type":"Work","button_location":"profile"}" href="https://www.academia.edu/attachments/104330773/download_file?st=MTczMjc1MTU1OCw4LjIyMi4yMDguMTQ2&s=profile"><span><i class="fa fa-arrow-down"></i></span><span>Download</span></a><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="104662123"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="104662123"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 104662123; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=104662123]").text(description); $(".js-view-count[data-work-id=104662123]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 104662123; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='104662123']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 104662123, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (true){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "80e54b12649c6ee5bee95ba446abad9a" } } $('.js-work-strip[data-work-id=104662123]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":104662123,"title":"Ileoscopy in patients with ileocolonic tuberculosis","translated_title":"","metadata":{"publisher":"Baishideng Publishing Group Inc.","grobid_abstract":"AIM: To study the role of retrograde terminal ileoscopy in patients suspected to have ileocolonic tuberculosis. METHODS: A retrospective analysis was performed for patients undergoing colonoscopy for suspected ileocolonic tuberculosis between January 2000 and June 2004, in whom retrograde ileoscopy had been performed. Only patients diagnosed with tuberculosis on the basis of histological findings of either a caseating granuloma or those having non-caseating granuloma or a collection of epithelioid cells at a minimum of one endoscopic lesion (either in the colon or the terminal ileum) on histology with good responses to conventional anti-tuberculous drugs were enrolled for the study. RESULTS: Fifty-three patients were included. The terminal ileum was involved in only 11 patients. Eight of these patients had involvement of the cecum too. Two patients had ileal lesions without cecal involvement; however, lesions were noted in the ascending colon. In one patient the whole colon was normal and only the terminal ileum showed nodularity and ulceration. Histological examination of the ileal biopsies obtained from the ileal lesions showed noncaseating granulomas in five, collection of epithelioid cells in four and nonspecific histology in two patients. Mucosal biopsies obtained from the lesion in the terminal ileum, in the patient in whom mucosal abnormality was observed to be confined to the ileum, showed noncaseating granulomas. In two other patients the ileal biopsies alone showed histological evidence of tuberculosis with biopsies from the colonic lesions showing non-specific inflammatory changes only. CONCLUSION: Re t ro g ra d e i l e o s c o py s h o u l d b e performed in all patients undergoing colonoscopy for suspected ileocolonic tuberculosis. In some patients only the terminal ileum may be involved and histological examination may reveal evidence of tuberculosis only from the lesions in the terminal ileum. 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The appearance, behavior and management of the gastric mass was more like a bezoar than that of a foreign body, and therefore we strongly feel that it should be called a \"plastobezoar\". CASE REPORT An 11-year-old boy was admitted with complaints of vomiting and abdominal pain of six months duration and swelling over the face and feet for the last one month. On clinical examination the child was pale with puffiness over the face. Pedal edema was present. Abdominal examination found a fi rm lump palpable in the epigastrium, which moved with respiration. No crepitus was felt over the lump. There was mild tenderness present over the mass. The hemoglobin was 5.6 g/dL, total and differential leukocyte counts were normal. The serum urea was 36.4 mmol/L, creatinine 314 μmol/L, and serum albumin was 2 g/dL. Serum globulin, fasting and post-parandial blood glucose, serum amylase, bilirubin and transaminases were normal. Urine examination did not reveal any abnormality. Serum sodium was 135 mmol/L, potassium 2.8 mmol/L and calcium 1.8 mmol/L. Just after admission, the patient developed typical grandmal epileptic attack, which was managed by intravenous diazepam injection. Intravenous and oral supplements of potassium and calcium were given and the child did not have any further episodes of epilepsy. The patient was considered to have pre-renal failure due to severe vomiting and dehydration and was managed conservatively with intravenous fl uid. Two packs of whole blood were transfused and 100 mL of 20% albumin was given intravenously. Gradually, the serum creatinine and urea became normal. Clinically the puffiness over the face and pedal edema disappeared and the patient started feeling better. Dialysis was not required. A contrast enhanced CT scan revealed an irregular, large, non-attached mass of varying attenuation with pockets of air, occupying the stomach (Figure 1). A portion of the mass was also encroaching the duodenum. A few polypoidal masses were seen in the antrum of the stomach near the pylorus. There was no post-contrast enhancement of the mass lesion. UGI endoscopy revealed a large bezoar occupying nearly the whole stomach (Figure 2). 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$(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> </div><div class="profile--tab_content_container js-tab-pane tab-pane" data-section-id="5136580" id="papers"><div class="js-work-strip profile--work_container" data-work-id="109190305"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/109190305/Amoebic_Liver_Abscess_Communicating_with_Intrahepatic_Bile_Ducts"><img alt="Research paper thumbnail of Amoebic Liver Abscess Communicating with Intrahepatic Bile Ducts" class="work-thumbnail" src="https://attachments.academia-assets.com/107388546/thumbnails/1.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/109190305/Amoebic_Liver_Abscess_Communicating_with_Intrahepatic_Bile_Ducts">Amoebic Liver Abscess Communicating with Intrahepatic Bile Ducts</a></div><div class="wp-workCard_item"><span>Tropical Doctor</span><span>, 1997</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><a id="9f18124c743354c018cca78fe87bb1d1" class="wp-workCard--action" rel="nofollow" data-click-track="profile-work-strip-download" data-download="{"attachment_id":107388546,"asset_id":109190305,"asset_type":"Work","button_location":"profile"}" href="https://www.academia.edu/attachments/107388546/download_file?st=MTczMjc1MTU1OCw4LjIyMi4yMDguMTQ2&st=MTczMjc1MTU1Nyw4LjIyMi4yMDguMTQ2&s=profile"><span><i class="fa fa-arrow-down"></i></span><span>Download</span></a><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109190305"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109190305"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109190305; 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$(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="109190299"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/109190299/Effect_of_Esophageal_Variceal_Band_Ligation_on_Hemorrhoids_Anorectal_Varices_and_Portal_Hypertensive_Colopathy"><img alt="Research paper thumbnail of Effect of Esophageal Variceal Band Ligation on Hemorrhoids, Anorectal Varices, and Portal Hypertensive Colopathy" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/109190299/Effect_of_Esophageal_Variceal_Band_Ligation_on_Hemorrhoids_Anorectal_Varices_and_Portal_Hypertensive_Colopathy">Effect of Esophageal Variceal Band Ligation on Hemorrhoids, Anorectal Varices, and Portal Hypertensive Colopathy</a></div><div class="wp-workCard_item"><span>Endoscopy</span><span>, 2002</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">Although it is known that obliteration of esophageal varices following endoscopic variceal band l...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">Although it is known that obliteration of esophageal varices following endoscopic variceal band ligation results in an increase in the incidence of portal hypertensive gastropathy, the effect of variceal ligation on hemorrhoids, anorectal/colonic varices and portal hypertensive colopathy is not known. The aim of this study was to investigate the effect of endoscopic variceal band ligation on hemorrhoids, anorectal/colonic varices and portal hypertensive colopathy. A total of 60 consecutive patients with cirrhosis of the liver and portal hypertension were prospectively studied. Upper gastrointestinal endoscopy and full-length colonoscopy were carried out before the patients underwent endoscopic variceal band ligation for esophageal varices and after obliteration of the varices following band ligation. Obliteration of esophageal varices by endoscopic variceal band ligation did not affect the incidence of hemorrhoids (37 % before and after), anorectal varices (40 % before and after), and portal hypertensive colopathy (57 % before and after). It is concluded that esophageal variceal band ligation does not affect the incidence of hemorrhoids, anorectal varices or portal hypertensive colopathy in patients with cirrhosis of the liver.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109190299"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109190299"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109190299; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=109190299]").text(description); $(".js-view-count[data-work-id=109190299]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 109190299; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='109190299']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 109190299, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=109190299]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":109190299,"title":"Effect of Esophageal Variceal Band Ligation on Hemorrhoids, Anorectal Varices, and Portal Hypertensive Colopathy","translated_title":"","metadata":{"abstract":"Although it is known that obliteration of esophageal varices following endoscopic variceal band ligation results in an increase in the incidence of portal hypertensive gastropathy, the effect of variceal ligation on hemorrhoids, anorectal/colonic varices and portal hypertensive colopathy is not known. The aim of this study was to investigate the effect of endoscopic variceal band ligation on hemorrhoids, anorectal/colonic varices and portal hypertensive colopathy. A total of 60 consecutive patients with cirrhosis of the liver and portal hypertension were prospectively studied. Upper gastrointestinal endoscopy and full-length colonoscopy were carried out before the patients underwent endoscopic variceal band ligation for esophageal varices and after obliteration of the varices following band ligation. Obliteration of esophageal varices by endoscopic variceal band ligation did not affect the incidence of hemorrhoids (37 % before and after), anorectal varices (40 % before and after), and portal hypertensive colopathy (57 % before and after). It is concluded that esophageal variceal band ligation does not affect the incidence of hemorrhoids, anorectal varices or portal hypertensive colopathy in patients with cirrhosis of the liver.","publisher":"Georg Thieme Verlag KG","publication_date":{"day":null,"month":null,"year":2002,"errors":{}},"publication_name":"Endoscopy"},"translated_abstract":"Although it is known that obliteration of esophageal varices following endoscopic variceal band ligation results in an increase in the incidence of portal hypertensive gastropathy, the effect of variceal ligation on hemorrhoids, anorectal/colonic varices and portal hypertensive colopathy is not known. The aim of this study was to investigate the effect of endoscopic variceal band ligation on hemorrhoids, anorectal/colonic varices and portal hypertensive colopathy. A total of 60 consecutive patients with cirrhosis of the liver and portal hypertension were prospectively studied. Upper gastrointestinal endoscopy and full-length colonoscopy were carried out before the patients underwent endoscopic variceal band ligation for esophageal varices and after obliteration of the varices following band ligation. Obliteration of esophageal varices by endoscopic variceal band ligation did not affect the incidence of hemorrhoids (37 % before and after), anorectal varices (40 % before and after), and portal hypertensive colopathy (57 % before and after). It is concluded that esophageal variceal band ligation does not affect the incidence of hemorrhoids, anorectal varices or portal hypertensive colopathy in patients with cirrhosis of the liver.","internal_url":"https://www.academia.edu/109190299/Effect_of_Esophageal_Variceal_Band_Ligation_on_Hemorrhoids_Anorectal_Varices_and_Portal_Hypertensive_Colopathy","translated_internal_url":"","created_at":"2023-11-15T01:49:39.619-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":48000299,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[],"downloadable_attachments":[],"slug":"Effect_of_Esophageal_Variceal_Band_Ligation_on_Hemorrhoids_Anorectal_Varices_and_Portal_Hypertensive_Colopathy","translated_slug":"","page_count":null,"language":"en","content_type":"Work","owner":{"id":48000299,"first_name":"Vatsala","middle_initials":null,"last_name":"Misra","page_name":"VatsalaMisra","domain_name":"independent","created_at":"2016-04-30T07:36:38.954-07:00","display_name":"Vatsala Misra","url":"https://independent.academia.edu/VatsalaMisra"},"attachments":[],"research_interests":[{"id":3274,"name":"Gastroenterology","url":"https://www.academia.edu/Documents/in/Gastroenterology"},{"id":22506,"name":"Adolescent","url":"https://www.academia.edu/Documents/in/Adolescent"},{"id":26327,"name":"Medicine","url":"https://www.academia.edu/Documents/in/Medicine"},{"id":37805,"name":"Endoscopy","url":"https://www.academia.edu/Documents/in/Endoscopy"},{"id":63890,"name":"Complication","url":"https://www.academia.edu/Documents/in/Complication"},{"id":65390,"name":"Internal Medicine","url":"https://www.academia.edu/Documents/in/Internal_Medicine"},{"id":71342,"name":"Portal hypertension","url":"https://www.academia.edu/Documents/in/Portal_hypertension"},{"id":174502,"name":"Incidence","url":"https://www.academia.edu/Documents/in/Incidence"},{"id":227324,"name":"Colonoscopy","url":"https://www.academia.edu/Documents/in/Colonoscopy"},{"id":244814,"name":"Clinical Sciences","url":"https://www.academia.edu/Documents/in/Clinical_Sciences"},{"id":600778,"name":"Cirrhosis","url":"https://www.academia.edu/Documents/in/Cirrhosis"},{"id":1486844,"name":"Hemorrhoids","url":"https://www.academia.edu/Documents/in/Hemorrhoids"},{"id":2922786,"name":"Esophageal varices","url":"https://www.academia.edu/Documents/in/Esophageal_varices"},{"id":3530956,"name":"Esophageal and gastric varices","url":"https://www.academia.edu/Documents/in/Esophageal_and_gastric_varices"},{"id":3589281,"name":"Ligation","url":"https://www.academia.edu/Documents/in/Ligation"}],"urls":[{"id":35398406,"url":"http://www.thieme-connect.de/products/ejournals/pdf/10.1055/s-2002-20290.pdf"}]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="109190298"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/109190298/Ileocecal_masses_in_patients_with_amebic_liver_abscess_Etiology_and_management"><img alt="Research paper thumbnail of Ileocecal masses in patients with amebic liver abscess: Etiology and management" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/109190298/Ileocecal_masses_in_patients_with_amebic_liver_abscess_Etiology_and_management">Ileocecal masses in patients with amebic liver abscess: Etiology and management</a></div><div class="wp-workCard_item"><span>World Journal of Gastroenterology</span><span>, 2006</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">To assess the causes of ileocecal mass in patients with amebic liver abscess. Patients with amebi...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">To assess the causes of ileocecal mass in patients with amebic liver abscess. Patients with amebic liver abscess and ileocecal mass were carefully examined and investigated by contrast-enhanced CT scan followed by colonoscopy and histological examination of biopsy materials from lesions during colonoscopy. Ileocecal masses were found in seventeen patients with amebic liver abscess. The cause of the mass was ameboma in 14 patients, cecal tuberculosis in 2 patients and adenocarcinoma of the cecum in 1 patient. Colonic ulcers were noted in five of the six (83%) patients with active diarrhea at presentation. The ileocecal mass in all these patients was ameboma. Ulcers were seen in only one of the 11 (9%) patients without diarrhea. The difference was statistically significant from the group with diarrhea (P&amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;lt; 0.005). Ileocecal mass is not an uncommon finding in patients with amebic liver abscess. Although, the ileocecal mass is due to ameboma formation in most cases, it should not be assumed that this is the case in all patients. Colonoscopy and histological examination of the target biopsies are mandatory to avoid missing a more sinister lesion.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109190298"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109190298"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109190298; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=109190298]").text(description); $(".js-view-count[data-work-id=109190298]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 109190298; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='109190298']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 109190298, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=109190298]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":109190298,"title":"Ileocecal masses in patients with amebic liver abscess: Etiology and management","translated_title":"","metadata":{"abstract":"To assess the causes of ileocecal mass in patients with amebic liver abscess. Patients with amebic liver abscess and ileocecal mass were carefully examined and investigated by contrast-enhanced CT scan followed by colonoscopy and histological examination of biopsy materials from lesions during colonoscopy. Ileocecal masses were found in seventeen patients with amebic liver abscess. The cause of the mass was ameboma in 14 patients, cecal tuberculosis in 2 patients and adenocarcinoma of the cecum in 1 patient. Colonic ulcers were noted in five of the six (83%) patients with active diarrhea at presentation. The ileocecal mass in all these patients was ameboma. Ulcers were seen in only one of the 11 (9%) patients without diarrhea. The difference was statistically significant from the group with diarrhea (P\u0026amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;lt; 0.005). Ileocecal mass is not an uncommon finding in patients with amebic liver abscess. Although, the ileocecal mass is due to ameboma formation in most cases, it should not be assumed that this is the case in all patients. Colonoscopy and histological examination of the target biopsies are mandatory to avoid missing a more sinister lesion.","publisher":"Baishideng Publishing Group Inc.","publication_date":{"day":null,"month":null,"year":2006,"errors":{}},"publication_name":"World Journal of Gastroenterology"},"translated_abstract":"To assess the causes of ileocecal mass in patients with amebic liver abscess. Patients with amebic liver abscess and ileocecal mass were carefully examined and investigated by contrast-enhanced CT scan followed by colonoscopy and histological examination of biopsy materials from lesions during colonoscopy. Ileocecal masses were found in seventeen patients with amebic liver abscess. The cause of the mass was ameboma in 14 patients, cecal tuberculosis in 2 patients and adenocarcinoma of the cecum in 1 patient. Colonic ulcers were noted in five of the six (83%) patients with active diarrhea at presentation. The ileocecal mass in all these patients was ameboma. Ulcers were seen in only one of the 11 (9%) patients without diarrhea. The difference was statistically significant from the group with diarrhea (P\u0026amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;amp;lt; 0.005). Ileocecal mass is not an uncommon finding in patients with amebic liver abscess. Although, the ileocecal mass is due to ameboma formation in most cases, it should not be assumed that this is the case in all patients. Colonoscopy and histological examination of the target biopsies are mandatory to avoid missing a more sinister lesion.","internal_url":"https://www.academia.edu/109190298/Ileocecal_masses_in_patients_with_amebic_liver_abscess_Etiology_and_management","translated_internal_url":"","created_at":"2023-11-15T01:49:33.626-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":48000299,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[],"downloadable_attachments":[],"slug":"Ileocecal_masses_in_patients_with_amebic_liver_abscess_Etiology_and_management","translated_slug":"","page_count":null,"language":"en","content_type":"Work","owner":{"id":48000299,"first_name":"Vatsala","middle_initials":null,"last_name":"Misra","page_name":"VatsalaMisra","domain_name":"independent","created_at":"2016-04-30T07:36:38.954-07:00","display_name":"Vatsala Misra","url":"https://independent.academia.edu/VatsalaMisra"},"attachments":[],"research_interests":[{"id":26327,"name":"Medicine","url":"https://www.academia.edu/Documents/in/Medicine"},{"id":37826,"name":"Biopsy","url":"https://www.academia.edu/Documents/in/Biopsy"},{"id":44259,"name":"Diarrhea","url":"https://www.academia.edu/Documents/in/Diarrhea"},{"id":227324,"name":"Colonoscopy","url":"https://www.academia.edu/Documents/in/Colonoscopy"},{"id":244814,"name":"Clinical Sciences","url":"https://www.academia.edu/Documents/in/Clinical_Sciences"},{"id":988280,"name":"Abscess","url":"https://www.academia.edu/Documents/in/Abscess"},{"id":4191298,"name":"Ileal diseases","url":"https://www.academia.edu/Documents/in/Ileal_diseases"}],"urls":[]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="109190297"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/109190297/Endoscopic_and_histological_changes_in_upper_gastrointestinal_tract_of_patients_with_chronic_renal_failure"><img alt="Research paper thumbnail of Endoscopic and histological changes in upper gastrointestinal tract of patients with chronic renal failure" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/109190297/Endoscopic_and_histological_changes_in_upper_gastrointestinal_tract_of_patients_with_chronic_renal_failure">Endoscopic and histological changes in upper gastrointestinal tract of patients with chronic renal failure</a></div><div class="wp-workCard_item"><span>Indian journal of pathology & microbiology</span><span>, 2004</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">To evaluate the endoscopic and histological changes in upper gastrointestinal tract of patients w...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">To evaluate the endoscopic and histological changes in upper gastrointestinal tract of patients with chronic renal failure 50 patients and 50 controls were studied. Upper gastrointestinal endoscopy was done and 2 biopsies each were taken from oesophagus, corpus and antrum of the stomach and duodenum. Sections were stained with haematoxylin & eosin, Alcian blue--Periodic acid Schiff&#39;s (pH 2.5), and Loeffler&#39;s methylene blue stains. Oesophagus was endoscopically normal in most of the patients. Predominant histological finding was chronic oesophagitis which was significantly higher in patients than controls (47.1% Vs 26%; p&lt;0.05). Significantly higher (p&lt;0.001) number of patients had gastritis, oedema and pale mucosa on endoscopic examination of stomach. Predominant histological changes were mucosal oedema (82.35%), gastritis (23.5%) and increase in number of bi- and multinucleated parietal cells with vacuolation and fragmentation of the cytoplasm (29%). Prevalence of H. ...</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109190297"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109190297"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109190297; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=109190297]").text(description); $(".js-view-count[data-work-id=109190297]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 109190297; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='109190297']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 109190297, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=109190297]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":109190297,"title":"Endoscopic and histological changes in upper gastrointestinal tract of patients with chronic renal failure","translated_title":"","metadata":{"abstract":"To evaluate the endoscopic and histological changes in upper gastrointestinal tract of patients with chronic renal failure 50 patients and 50 controls were studied. Upper gastrointestinal endoscopy was done and 2 biopsies each were taken from oesophagus, corpus and antrum of the stomach and duodenum. Sections were stained with haematoxylin \u0026 eosin, Alcian blue--Periodic acid Schiff\u0026#39;s (pH 2.5), and Loeffler\u0026#39;s methylene blue stains. Oesophagus was endoscopically normal in most of the patients. Predominant histological finding was chronic oesophagitis which was significantly higher in patients than controls (47.1% Vs 26%; p\u0026lt;0.05). Significantly higher (p\u0026lt;0.001) number of patients had gastritis, oedema and pale mucosa on endoscopic examination of stomach. Predominant histological changes were mucosal oedema (82.35%), gastritis (23.5%) and increase in number of bi- and multinucleated parietal cells with vacuolation and fragmentation of the cytoplasm (29%). Prevalence of H. ...","publication_date":{"day":null,"month":null,"year":2004,"errors":{}},"publication_name":"Indian journal of pathology \u0026 microbiology"},"translated_abstract":"To evaluate the endoscopic and histological changes in upper gastrointestinal tract of patients with chronic renal failure 50 patients and 50 controls were studied. Upper gastrointestinal endoscopy was done and 2 biopsies each were taken from oesophagus, corpus and antrum of the stomach and duodenum. Sections were stained with haematoxylin \u0026 eosin, Alcian blue--Periodic acid Schiff\u0026#39;s (pH 2.5), and Loeffler\u0026#39;s methylene blue stains. Oesophagus was endoscopically normal in most of the patients. Predominant histological finding was chronic oesophagitis which was significantly higher in patients than controls (47.1% Vs 26%; p\u0026lt;0.05). Significantly higher (p\u0026lt;0.001) number of patients had gastritis, oedema and pale mucosa on endoscopic examination of stomach. Predominant histological changes were mucosal oedema (82.35%), gastritis (23.5%) and increase in number of bi- and multinucleated parietal cells with vacuolation and fragmentation of the cytoplasm (29%). Prevalence of H. ...","internal_url":"https://www.academia.edu/109190297/Endoscopic_and_histological_changes_in_upper_gastrointestinal_tract_of_patients_with_chronic_renal_failure","translated_internal_url":"","created_at":"2023-11-15T01:49:33.407-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":48000299,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[],"downloadable_attachments":[],"slug":"Endoscopic_and_histological_changes_in_upper_gastrointestinal_tract_of_patients_with_chronic_renal_failure","translated_slug":"","page_count":null,"language":"en","content_type":"Work","owner":{"id":48000299,"first_name":"Vatsala","middle_initials":null,"last_name":"Misra","page_name":"VatsalaMisra","domain_name":"independent","created_at":"2016-04-30T07:36:38.954-07:00","display_name":"Vatsala Misra","url":"https://independent.academia.edu/VatsalaMisra"},"attachments":[],"research_interests":[{"id":3274,"name":"Gastroenterology","url":"https://www.academia.edu/Documents/in/Gastroenterology"},{"id":6947,"name":"Medical Microbiology","url":"https://www.academia.edu/Documents/in/Medical_Microbiology"},{"id":21460,"name":"Helicobacter pylori","url":"https://www.academia.edu/Documents/in/Helicobacter_pylori"},{"id":26327,"name":"Medicine","url":"https://www.academia.edu/Documents/in/Medicine"},{"id":65390,"name":"Internal Medicine","url":"https://www.academia.edu/Documents/in/Internal_Medicine"},{"id":98939,"name":"Pubmed","url":"https://www.academia.edu/Documents/in/Pubmed"},{"id":131233,"name":"Indian","url":"https://www.academia.edu/Documents/in/Indian"},{"id":405739,"name":"Esophagus","url":"https://www.academia.edu/Documents/in/Esophagus"},{"id":537759,"name":"Gastrointestinal Tract","url":"https://www.academia.edu/Documents/in/Gastrointestinal_Tract"},{"id":1030661,"name":"Stomach","url":"https://www.academia.edu/Documents/in/Stomach"},{"id":1106287,"name":"Chronic Kidney Failure","url":"https://www.academia.edu/Documents/in/Chronic_Kidney_Failure"},{"id":1819399,"name":"Case Control Studies","url":"https://www.academia.edu/Documents/in/Case_Control_Studies"},{"id":2090674,"name":"Duodenum","url":"https://www.academia.edu/Documents/in/Duodenum"}],"urls":[]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="109190296"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" rel="nofollow" href="https://www.academia.edu/109190296/Duodenal_metastases_from_squamous_cell_carcinoma_of_the_lung_endoscopic_management_of_bleeding_and_biliary_and_duodenal_obstruction"><img alt="Research paper thumbnail of Duodenal metastases from squamous cell carcinoma of the lung: endoscopic management of bleeding and biliary and duodenal obstruction" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" rel="nofollow" href="https://www.academia.edu/109190296/Duodenal_metastases_from_squamous_cell_carcinoma_of_the_lung_endoscopic_management_of_bleeding_and_biliary_and_duodenal_obstruction">Duodenal metastases from squamous cell carcinoma of the lung: endoscopic management of bleeding and biliary and duodenal obstruction</a></div><div class="wp-workCard_item"><span>Indian journal of gastroenterology : official journal of the Indian Society of Gastroenterology</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">Intestinal metastases from cancer of the lung are rare. We report a 65-year-old man with duodenal...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">Intestinal metastases from cancer of the lung are rare. We report a 65-year-old man with duodenal metastases from squamous cell cancer causing GI bleeding and biliary and duodenal obstruction; these were managed endoscopically.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109190296"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109190296"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109190296; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=109190296]").text(description); $(".js-view-count[data-work-id=109190296]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 109190296; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='109190296']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 109190296, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=109190296]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":109190296,"title":"Duodenal metastases from squamous cell carcinoma of the lung: endoscopic management of bleeding and biliary and duodenal obstruction","translated_title":"","metadata":{"abstract":"Intestinal metastases from cancer of the lung are rare. 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Sections were stained with hematoxylene and eosin, AB/PAS (Ph 2.5) and Loeffler&#39;s methylene blue stains. Infection by H. pylori, inflammation and atrophy were found to be significantly more in leprosy patients as compared to controls (p &lt; 0.01, &lt; 0.005 and &lt; 0.02 respectively). On comparing the histopathological changes in various subgroups of leprosy, H. pylori, inflammation and activity showed a statistically decreasing trend from tuberculoid to lepromatous subgroups (p &lt; 0.05, &lt; 0.001, &lt; 0.01 respectively). Atrophy showed a significant increasing trend from tuberculoid to lepromatous group (&lt; 0.001), it is concluded that despite a low prevalence of H. pylori and associated gastritis in patients with lepromatous leprosy, gastric epithelial damage is more marked due to altered immune response.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109190295"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109190295"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109190295; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=109190295]").text(description); $(".js-view-count[data-work-id=109190295]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 109190295; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='109190295']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 109190295, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=109190295]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":109190295,"title":"Helicobacter pylori and associated histopathological changes in gastric biopsies of patients with leprosy","translated_title":"","metadata":{"abstract":"Two antral biopsies each from 104 patients of leprosy and 100 controls were studied to find out the prevalence of H. pylori and associated histopathological changes. Sections were stained with hematoxylene and eosin, AB/PAS (Ph 2.5) and Loeffler\u0026#39;s methylene blue stains. Infection by H. pylori, inflammation and atrophy were found to be significantly more in leprosy patients as compared to controls (p \u0026lt; 0.01, \u0026lt; 0.005 and \u0026lt; 0.02 respectively). On comparing the histopathological changes in various subgroups of leprosy, H. pylori, inflammation and activity showed a statistically decreasing trend from tuberculoid to lepromatous subgroups (p \u0026lt; 0.05, \u0026lt; 0.001, \u0026lt; 0.01 respectively). Atrophy showed a significant increasing trend from tuberculoid to lepromatous group (\u0026lt; 0.001), it is concluded that despite a low prevalence of H. pylori and associated gastritis in patients with lepromatous leprosy, gastric epithelial damage is more marked due to altered immune response.","publication_date":{"day":null,"month":null,"year":2001,"errors":{}},"publication_name":"Indian journal of pathology \u0026 microbiology"},"translated_abstract":"Two antral biopsies each from 104 patients of leprosy and 100 controls were studied to find out the prevalence of H. pylori and associated histopathological changes. Sections were stained with hematoxylene and eosin, AB/PAS (Ph 2.5) and Loeffler\u0026#39;s methylene blue stains. Infection by H. pylori, inflammation and atrophy were found to be significantly more in leprosy patients as compared to controls (p \u0026lt; 0.01, \u0026lt; 0.005 and \u0026lt; 0.02 respectively). On comparing the histopathological changes in various subgroups of leprosy, H. pylori, inflammation and activity showed a statistically decreasing trend from tuberculoid to lepromatous subgroups (p \u0026lt; 0.05, \u0026lt; 0.001, \u0026lt; 0.01 respectively). Atrophy showed a significant increasing trend from tuberculoid to lepromatous group (\u0026lt; 0.001), it is concluded that despite a low prevalence of H. pylori and associated gastritis in patients with lepromatous leprosy, gastric epithelial damage is more marked due to altered immune response.","internal_url":"https://www.academia.edu/109190295/Helicobacter_pylori_and_associated_histopathological_changes_in_gastric_biopsies_of_patients_with_leprosy","translated_internal_url":"","created_at":"2023-11-15T01:49:32.696-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":48000299,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[],"downloadable_attachments":[],"slug":"Helicobacter_pylori_and_associated_histopathological_changes_in_gastric_biopsies_of_patients_with_leprosy","translated_slug":"","page_count":null,"language":"en","content_type":"Work","owner":{"id":48000299,"first_name":"Vatsala","middle_initials":null,"last_name":"Misra","page_name":"VatsalaMisra","domain_name":"independent","created_at":"2016-04-30T07:36:38.954-07:00","display_name":"Vatsala Misra","url":"https://independent.academia.edu/VatsalaMisra"},"attachments":[],"research_interests":[{"id":3274,"name":"Gastroenterology","url":"https://www.academia.edu/Documents/in/Gastroenterology"},{"id":6947,"name":"Medical Microbiology","url":"https://www.academia.edu/Documents/in/Medical_Microbiology"},{"id":21460,"name":"Helicobacter pylori","url":"https://www.academia.edu/Documents/in/Helicobacter_pylori"},{"id":22506,"name":"Adolescent","url":"https://www.academia.edu/Documents/in/Adolescent"},{"id":26327,"name":"Medicine","url":"https://www.academia.edu/Documents/in/Medicine"},{"id":73489,"name":"Leprosy","url":"https://www.academia.edu/Documents/in/Leprosy"},{"id":98939,"name":"Pubmed","url":"https://www.academia.edu/Documents/in/Pubmed"},{"id":131233,"name":"Indian","url":"https://www.academia.edu/Documents/in/Indian"},{"id":289271,"name":"Aged","url":"https://www.academia.edu/Documents/in/Aged"},{"id":357850,"name":"Atrophy","url":"https://www.academia.edu/Documents/in/Atrophy"},{"id":925054,"name":"Gastritis","url":"https://www.academia.edu/Documents/in/Gastritis"},{"id":1819399,"name":"Case Control Studies","url":"https://www.academia.edu/Documents/in/Case_Control_Studies"},{"id":2547982,"name":"Gastric mucosa","url":"https://www.academia.edu/Documents/in/Gastric_mucosa"}],"urls":[]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="109190294"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/109190294/AgNOR_counts_are_not_altered_in_alcoholic_cirrhosis"><img alt="Research paper thumbnail of AgNOR counts are not altered in alcoholic cirrhosis" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/109190294/AgNOR_counts_are_not_altered_in_alcoholic_cirrhosis">AgNOR counts are not altered in alcoholic cirrhosis</a></div><div class="wp-workCard_item"><span>Indian journal of pathology & microbiology</span><span>, 2003</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">One hundred and two liver biopsy specimens were stained for Argyrophilic nucleolar organizer regi...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">One hundred and two liver biopsy specimens were stained for Argyrophilic nucleolar organizer regions and associated proteins to assess its utility in differentiating normal, cirrhotic and neoplastic liver tissue. A statistically significant (p &lt; 0.001) difference was observed between mean AgNOR counts of normal (1.53 +/- 0.4), post-hepatitic cirrhosis (3.65 +/- 0.53) and hepatocellular carcinoma (7.94 +/- 1.18). In contrast the mean AgNOR count of biopsies with alcoholic cirrhosis (1.57 +/- 0.06) was significantly less (p &lt; 0.001) than post-hepatitic cirrhosis and was similar to that of normal liver tissue. It is concluded that AgNORs can act as a good adjuvant to histology in diagnosing liver diseases especially in differentiating post-hepatitic and alcoholic cirrhosis.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109190294"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109190294"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109190294; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=109190294]").text(description); $(".js-view-count[data-work-id=109190294]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 109190294; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='109190294']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 109190294, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=109190294]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":109190294,"title":"AgNOR counts are not altered in alcoholic cirrhosis","translated_title":"","metadata":{"abstract":"One hundred and two liver biopsy specimens were stained for Argyrophilic nucleolar organizer regions and associated proteins to assess its utility in differentiating normal, cirrhotic and neoplastic liver tissue. A statistically significant (p \u0026lt; 0.001) difference was observed between mean AgNOR counts of normal (1.53 +/- 0.4), post-hepatitic cirrhosis (3.65 +/- 0.53) and hepatocellular carcinoma (7.94 +/- 1.18). In contrast the mean AgNOR count of biopsies with alcoholic cirrhosis (1.57 +/- 0.06) was significantly less (p \u0026lt; 0.001) than post-hepatitic cirrhosis and was similar to that of normal liver tissue. It is concluded that AgNORs can act as a good adjuvant to histology in diagnosing liver diseases especially in differentiating post-hepatitic and alcoholic cirrhosis.","publication_date":{"day":null,"month":null,"year":2003,"errors":{}},"publication_name":"Indian journal of pathology \u0026 microbiology"},"translated_abstract":"One hundred and two liver biopsy specimens were stained for Argyrophilic nucleolar organizer regions and associated proteins to assess its utility in differentiating normal, cirrhotic and neoplastic liver tissue. A statistically significant (p \u0026lt; 0.001) difference was observed between mean AgNOR counts of normal (1.53 +/- 0.4), post-hepatitic cirrhosis (3.65 +/- 0.53) and hepatocellular carcinoma (7.94 +/- 1.18). In contrast the mean AgNOR count of biopsies with alcoholic cirrhosis (1.57 +/- 0.06) was significantly less (p \u0026lt; 0.001) than post-hepatitic cirrhosis and was similar to that of normal liver tissue. It is concluded that AgNORs can act as a good adjuvant to histology in diagnosing liver diseases especially in differentiating post-hepatitic and alcoholic cirrhosis.","internal_url":"https://www.academia.edu/109190294/AgNOR_counts_are_not_altered_in_alcoholic_cirrhosis","translated_internal_url":"","created_at":"2023-11-15T01:49:32.437-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":48000299,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[],"downloadable_attachments":[],"slug":"AgNOR_counts_are_not_altered_in_alcoholic_cirrhosis","translated_slug":"","page_count":null,"language":"en","content_type":"Work","owner":{"id":48000299,"first_name":"Vatsala","middle_initials":null,"last_name":"Misra","page_name":"VatsalaMisra","domain_name":"independent","created_at":"2016-04-30T07:36:38.954-07:00","display_name":"Vatsala Misra","url":"https://independent.academia.edu/VatsalaMisra"},"attachments":[],"research_interests":[{"id":165,"name":"Pathology","url":"https://www.academia.edu/Documents/in/Pathology"},{"id":6947,"name":"Medical Microbiology","url":"https://www.academia.edu/Documents/in/Medical_Microbiology"},{"id":12069,"name":"Histology","url":"https://www.academia.edu/Documents/in/Histology"},{"id":26327,"name":"Medicine","url":"https://www.academia.edu/Documents/in/Medicine"},{"id":37826,"name":"Biopsy","url":"https://www.academia.edu/Documents/in/Biopsy"},{"id":71343,"name":"Liver Cirrhosis","url":"https://www.academia.edu/Documents/in/Liver_Cirrhosis"},{"id":71437,"name":"Liver","url":"https://www.academia.edu/Documents/in/Liver"},{"id":80694,"name":"Silver","url":"https://www.academia.edu/Documents/in/Silver"},{"id":98939,"name":"Pubmed","url":"https://www.academia.edu/Documents/in/Pubmed"},{"id":111972,"name":"Hepatocellular Carcinoma","url":"https://www.academia.edu/Documents/in/Hepatocellular_Carcinoma"},{"id":144048,"name":"Hepatitis","url":"https://www.academia.edu/Documents/in/Hepatitis"},{"id":558694,"name":"Nucleolus Organizer Region","url":"https://www.academia.edu/Documents/in/Nucleolus_Organizer_Region"},{"id":600778,"name":"Cirrhosis","url":"https://www.academia.edu/Documents/in/Cirrhosis"},{"id":1819399,"name":"Case Control Studies","url":"https://www.academia.edu/Documents/in/Case_Control_Studies"},{"id":2815868,"name":"Alcoholic liver disease","url":"https://www.academia.edu/Documents/in/Alcoholic_liver_disease"},{"id":3562342,"name":"Liver neoplasms","url":"https://www.academia.edu/Documents/in/Liver_neoplasms"}],"urls":[]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="109190293"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/109190293/Diagnosing_ascites_Value_of_ascitic_fluid_total_protein_albumin_cholesterol_their_ratios_serum_ascites_albumin_and_cholesterol_gradient"><img alt="Research paper thumbnail of Diagnosing ascites: Value of ascitic fluid total protein, albumin, cholesterol, their ratios, serum-ascites albumin and cholesterol gradient" class="work-thumbnail" src="https://attachments.academia-assets.com/107388529/thumbnails/1.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/109190293/Diagnosing_ascites_Value_of_ascitic_fluid_total_protein_albumin_cholesterol_their_ratios_serum_ascites_albumin_and_cholesterol_gradient">Diagnosing ascites: Value of ascitic fluid total protein, albumin, cholesterol, their ratios, serum-ascites albumin and cholesterol gradient</a></div><div class="wp-workCard_item"><span>Journal of Gastroenterology and Hepatology</span><span>, 1995</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><a id="4200a97401e41ffdf22c3362a2482beb" class="wp-workCard--action" rel="nofollow" data-click-track="profile-work-strip-download" data-download="{"attachment_id":107388529,"asset_id":109190293,"asset_type":"Work","button_location":"profile"}" href="https://www.academia.edu/attachments/107388529/download_file?st=MTczMjc1MTU1OCw4LjIyMi4yMDguMTQ2&s=profile"><span><i class="fa fa-arrow-down"></i></span><span>Download</span></a><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109190293"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109190293"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109190293; 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The mean f s.d. ascitic fluid total protein, albumin, cholesterol, their respective ascitic fluid/serum ratios in cirrhotic ascites were lower than malignant and tuberculous groups (P \u003c 0.001 for each). The difference between malignant and tuberculous groups was sigtllficant for ascitic fluid/serum total protein (P \u003c 0.05) and ascitic fluidherum albumin (P \u003c 0.01) only. Mean serum-ascites albumin gradient in cirrhotics was higher than in the malignant and tuberculous groups (P \u003c 0.001 for each). The difference between malignant and tuberculous groups was sigtllficant (P \u003c 0.01). Mean f s.d. serum-ascites cholesterol gradient in cirrhotics was higher than that in malignant and tuberculous groups (P\u003cO.001 for each). The difference between malignant and tuberculous groups was also si@icant (P \u003c 0.01). Both serudascitic fluid total protein less than 0.5 and ascitic fluid cholesterol less than 55 mg/dL had 94% diagnostic accuracy for differentiating cirrhotic from malignant and tuberculous ascites. Serum ascitic fluid albumin gradient greater than 1.1 g/ dL, ascitic fluid/serum albumin less than 0.65 and ascitic fluid albumin less than 2 g/dL had diagnostic accuracy of 92, 92 and 91%, respectively. Ascitic fluid total protein had diagnostic accuracy of 88%. None of the tests was able to differentiate between malignant and tuberculous ascites. Measurement of ascitic fluid cholesterol concentration is a simple method of differentiating cirrhotic from non-cirrhotic ascites.","publication_date":{"day":null,"month":null,"year":1995,"errors":{}},"publication_name":"Journal of Gastroenterology and Hepatology","grobid_abstract_attachment_id":107388529},"translated_abstract":null,"internal_url":"https://www.academia.edu/109190293/Diagnosing_ascites_Value_of_ascitic_fluid_total_protein_albumin_cholesterol_their_ratios_serum_ascites_albumin_and_cholesterol_gradient","translated_internal_url":"","created_at":"2023-11-15T01:49:31.708-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":48000299,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[],"downloadable_attachments":[{"id":107388529,"title":"","file_type":"pdf","scribd_thumbnail_url":"https://attachments.academia-assets.com/107388529/thumbnails/1.jpg","file_name":"j.1440-1746.1995.tb01096.x20231115-1-yeiz6s.pdf","download_url":"https://www.academia.edu/attachments/107388529/download_file?st=MTczMjc1MTU1OCw4LjIyMi4yMDguMTQ2&","bulk_download_file_name":"Diagnosing_ascites_Value_of_ascitic_flui.pdf","bulk_download_url":"https://d1wqtxts1xzle7.cloudfront.net/107388529/j.1440-1746.1995.tb01096.x20231115-1-yeiz6s-libre.pdf?1700048619=\u0026response-content-disposition=attachment%3B+filename%3DDiagnosing_ascites_Value_of_ascitic_flui.pdf\u0026Expires=1732708236\u0026Signature=Wpb1AURv3o67ywEec2ZXbuoaRthkC-CjjocL58cw6m2QFOVY7nBwYpz5JoB~RipTvAVFjSpjjqpR~GHxT9700PETmCZjnpPRyU2WW9Uu1ZfYj45XDVSUVNVyKszggQ4knw~SVW5RVtvdVE0L3KfDqhcJYlvVN10evUxt9ygQgX1GdSUQaFPC6bDWlOTvM~IQ0ZAAY6zfXWF-X9ZasPTZBYOsc5H2AEdOytZ-RtiZZB-LTYx47OReaa9l82QTflEpBN0WVEAW8jvwmXjN4M5wuf3kLsxHso5gxtrvyC6Gw6uv~SJ7W6VEFEnJNcTSaMmf6mIPU2JD7XSdjW3RPykf9w__\u0026Key-Pair-Id=APKAJLOHF5GGSLRBV4ZA"}],"slug":"Diagnosing_ascites_Value_of_ascitic_fluid_total_protein_albumin_cholesterol_their_ratios_serum_ascites_albumin_and_cholesterol_gradient","translated_slug":"","page_count":5,"language":"en","content_type":"Work","owner":{"id":48000299,"first_name":"Vatsala","middle_initials":null,"last_name":"Misra","page_name":"VatsalaMisra","domain_name":"independent","created_at":"2016-04-30T07:36:38.954-07:00","display_name":"Vatsala Misra","url":"https://independent.academia.edu/VatsalaMisra"},"attachments":[{"id":107388529,"title":"","file_type":"pdf","scribd_thumbnail_url":"https://attachments.academia-assets.com/107388529/thumbnails/1.jpg","file_name":"j.1440-1746.1995.tb01096.x20231115-1-yeiz6s.pdf","download_url":"https://www.academia.edu/attachments/107388529/download_file?st=MTczMjc1MTU1OCw4LjIyMi4yMDguMTQ2&","bulk_download_file_name":"Diagnosing_ascites_Value_of_ascitic_flui.pdf","bulk_download_url":"https://d1wqtxts1xzle7.cloudfront.net/107388529/j.1440-1746.1995.tb01096.x20231115-1-yeiz6s-libre.pdf?1700048619=\u0026response-content-disposition=attachment%3B+filename%3DDiagnosing_ascites_Value_of_ascitic_flui.pdf\u0026Expires=1732708236\u0026Signature=Wpb1AURv3o67ywEec2ZXbuoaRthkC-CjjocL58cw6m2QFOVY7nBwYpz5JoB~RipTvAVFjSpjjqpR~GHxT9700PETmCZjnpPRyU2WW9Uu1ZfYj45XDVSUVNVyKszggQ4knw~SVW5RVtvdVE0L3KfDqhcJYlvVN10evUxt9ygQgX1GdSUQaFPC6bDWlOTvM~IQ0ZAAY6zfXWF-X9ZasPTZBYOsc5H2AEdOytZ-RtiZZB-LTYx47OReaa9l82QTflEpBN0WVEAW8jvwmXjN4M5wuf3kLsxHso5gxtrvyC6Gw6uv~SJ7W6VEFEnJNcTSaMmf6mIPU2JD7XSdjW3RPykf9w__\u0026Key-Pair-Id=APKAJLOHF5GGSLRBV4ZA"}],"research_interests":[{"id":26327,"name":"Medicine","url":"https://www.academia.edu/Documents/in/Medicine"},{"id":62112,"name":"Prospective studies","url":"https://www.academia.edu/Documents/in/Prospective_studies"},{"id":65390,"name":"Internal Medicine","url":"https://www.academia.edu/Documents/in/Internal_Medicine"},{"id":71343,"name":"Liver Cirrhosis","url":"https://www.academia.edu/Documents/in/Liver_Cirrhosis"},{"id":76408,"name":"Albumin","url":"https://www.academia.edu/Documents/in/Albumin"},{"id":90514,"name":"Cholesterol","url":"https://www.academia.edu/Documents/in/Cholesterol"},{"id":111972,"name":"Hepatocellular Carcinoma","url":"https://www.academia.edu/Documents/in/Hepatocellular_Carcinoma"},{"id":181569,"name":"Proteins","url":"https://www.academia.edu/Documents/in/Proteins"},{"id":244814,"name":"Clinical Sciences","url":"https://www.academia.edu/Documents/in/Clinical_Sciences"},{"id":260020,"name":"GASTROENTEROLOGY AND HEPATOLOGY.","url":"https://www.academia.edu/Documents/in/GASTROENTEROLOGY_AND_HEPATOLOGY"},{"id":790533,"name":"Liver Failure","url":"https://www.academia.edu/Documents/in/Liver_Failure"},{"id":901876,"name":"Sensitivity and Specificity","url":"https://www.academia.edu/Documents/in/Sensitivity_and_Specificity"},{"id":1281483,"name":"Ascites","url":"https://www.academia.edu/Documents/in/Ascites"},{"id":1593060,"name":"Blood Proteins","url":"https://www.academia.edu/Documents/in/Blood_Proteins"},{"id":2349258,"name":"Serum albumin","url":"https://www.academia.edu/Documents/in/Serum_albumin"},{"id":2836761,"name":"Total protein","url":"https://www.academia.edu/Documents/in/Total_protein"},{"id":3562342,"name":"Liver neoplasms","url":"https://www.academia.edu/Documents/in/Liver_neoplasms"},{"id":3960847,"name":"Gastroenterology and Hepatology","url":"https://www.academia.edu/Documents/in/Gastroenterology_and_Hepatology-1"},{"id":4074011,"name":" Ascitic Fluid","url":"https://www.academia.edu/Documents/in/Ascitic_Fluid"}],"urls":[{"id":35398403,"url":"http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1440-1746.1995.tb01096.x/fullpdf"}]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="109190292"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" rel="nofollow" href="https://www.academia.edu/109190292/Nodular_gastritis_in_adults_Clinical_features_endoscopic_appearance_histopathological_features_and_response_to_therapy"><img alt="Research paper thumbnail of Nodular gastritis in adults: Clinical features, endoscopic appearance, histopathological features, and response to therapy" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" rel="nofollow" href="https://www.academia.edu/109190292/Nodular_gastritis_in_adults_Clinical_features_endoscopic_appearance_histopathological_features_and_response_to_therapy">Nodular gastritis in adults: Clinical features, endoscopic appearance, histopathological features, and response to therapy</a></div><div class="wp-workCard_item"><span>Journal of Gastroenterology and Hepatology</span><span>, 2008</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">Background and Aims: The present study aims to determine the prevalence of nodular gastritis (NG...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">Background and Aims: The present study aims to determine the prevalence of nodular gastritis (NG) and ascertain its clinical presentation and histopathological features in adults. It also assesses its association with Helicobacter pylori and the normalization of endoscopic features, symptoms, and histology after anti H. pylori therapy.Methods: A total of 7140 patients undergoing upper gastrointestinal endoscopy were studied. Patients showing nodularity of the gastric mucosa at endoscopy and an age‐ and sex‐matched control group with normal gastric mucosa underwent biopsies from the gastric antrum and fundus. The biopsies were assessed for the presence of mucosal inflammation, activity, eosinophils, atrophy, lymphoid follicles, H. pylori, and the presence of intestinal metaplasia. Patients with NG were given triple therapy. Endoscopy and biopsy was repeated after 4 weeks of stopping therapy. The symptoms of the patients and histology were assessed pre‐ and post‐therapy.Results: Th...</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109190292"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109190292"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109190292; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=109190292]").text(description); $(".js-view-count[data-work-id=109190292]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 109190292; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='109190292']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 109190292, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=109190292]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":109190292,"title":"Nodular gastritis in adults: Clinical features, endoscopic appearance, histopathological features, and response to therapy","translated_title":"","metadata":{"abstract":"Background and Aims: The present study aims to determine the prevalence of nodular gastritis (NG) and ascertain its clinical presentation and histopathological features in adults. It also assesses its association with Helicobacter pylori and the normalization of endoscopic features, symptoms, and histology after anti H. pylori therapy.Methods: A total of 7140 patients undergoing upper gastrointestinal endoscopy were studied. Patients showing nodularity of the gastric mucosa at endoscopy and an age‐ and sex‐matched control group with normal gastric mucosa underwent biopsies from the gastric antrum and fundus. The biopsies were assessed for the presence of mucosal inflammation, activity, eosinophils, atrophy, lymphoid follicles, H. pylori, and the presence of intestinal metaplasia. Patients with NG were given triple therapy. Endoscopy and biopsy was repeated after 4 weeks of stopping therapy. The symptoms of the patients and histology were assessed pre‐ and post‐therapy.Results: Th...","publisher":"Wiley","publication_date":{"day":null,"month":null,"year":2008,"errors":{}},"publication_name":"Journal of Gastroenterology and Hepatology"},"translated_abstract":"Background and Aims: The present study aims to determine the prevalence of nodular gastritis (NG) and ascertain its clinical presentation and histopathological features in adults. It also assesses its association with Helicobacter pylori and the normalization of endoscopic features, symptoms, and histology after anti H. pylori therapy.Methods: A total of 7140 patients undergoing upper gastrointestinal endoscopy were studied. Patients showing nodularity of the gastric mucosa at endoscopy and an age‐ and sex‐matched control group with normal gastric mucosa underwent biopsies from the gastric antrum and fundus. The biopsies were assessed for the presence of mucosal inflammation, activity, eosinophils, atrophy, lymphoid follicles, H. pylori, and the presence of intestinal metaplasia. Patients with NG were given triple therapy. Endoscopy and biopsy was repeated after 4 weeks of stopping therapy. The symptoms of the patients and histology were assessed pre‐ and post‐therapy.Results: Th...","internal_url":"https://www.academia.edu/109190292/Nodular_gastritis_in_adults_Clinical_features_endoscopic_appearance_histopathological_features_and_response_to_therapy","translated_internal_url":"","created_at":"2023-11-15T01:49:31.346-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":48000299,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[],"downloadable_attachments":[],"slug":"Nodular_gastritis_in_adults_Clinical_features_endoscopic_appearance_histopathological_features_and_response_to_therapy","translated_slug":"","page_count":null,"language":"en","content_type":"Work","owner":{"id":48000299,"first_name":"Vatsala","middle_initials":null,"last_name":"Misra","page_name":"VatsalaMisra","domain_name":"independent","created_at":"2016-04-30T07:36:38.954-07:00","display_name":"Vatsala Misra","url":"https://independent.academia.edu/VatsalaMisra"},"attachments":[],"research_interests":[{"id":3274,"name":"Gastroenterology","url":"https://www.academia.edu/Documents/in/Gastroenterology"},{"id":21460,"name":"Helicobacter pylori","url":"https://www.academia.edu/Documents/in/Helicobacter_pylori"},{"id":26327,"name":"Medicine","url":"https://www.academia.edu/Documents/in/Medicine"},{"id":37826,"name":"Biopsy","url":"https://www.academia.edu/Documents/in/Biopsy"},{"id":43838,"name":"India","url":"https://www.academia.edu/Documents/in/India"},{"id":65390,"name":"Internal Medicine","url":"https://www.academia.edu/Documents/in/Internal_Medicine"},{"id":244814,"name":"Clinical Sciences","url":"https://www.academia.edu/Documents/in/Clinical_Sciences"},{"id":289271,"name":"Aged","url":"https://www.academia.edu/Documents/in/Aged"},{"id":289330,"name":"Prevalence","url":"https://www.academia.edu/Documents/in/Prevalence"},{"id":335984,"name":"Anti-Bacterial Agents","url":"https://www.academia.edu/Documents/in/Anti-Bacterial_Agents"},{"id":925054,"name":"Gastritis","url":"https://www.academia.edu/Documents/in/Gastritis"},{"id":1295203,"name":"Gastroscopy","url":"https://www.academia.edu/Documents/in/Gastroscopy"},{"id":1819399,"name":"Case Control Studies","url":"https://www.academia.edu/Documents/in/Case_Control_Studies"},{"id":2452785,"name":"Proton Pump Inhibitors","url":"https://www.academia.edu/Documents/in/Proton_Pump_Inhibitors"},{"id":2547982,"name":"Gastric mucosa","url":"https://www.academia.edu/Documents/in/Gastric_mucosa"}],"urls":[{"id":35398402,"url":"https://onlinelibrary.wiley.com/doi/pdf/10.1111/j.1440-1746.2007.05044.x"}]}, dispatcherData: dispatcherData }); 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$(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="109190290"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" rel="nofollow" href="https://www.academia.edu/109190290/Preoperative_sonographic_diagnosis_of_acute_appendicitis_caused_byAscaris_lumbricoides"><img alt="Research paper thumbnail of Preoperative sonographic diagnosis of acute appendicitis caused byAscaris lumbricoides" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" rel="nofollow" href="https://www.academia.edu/109190290/Preoperative_sonographic_diagnosis_of_acute_appendicitis_caused_byAscaris_lumbricoides">Preoperative sonographic diagnosis of acute appendicitis caused byAscaris lumbricoides</a></div><div class="wp-workCard_item"><span>Journal of Clinical Ultrasound</span><span>, 1999</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">Acute appendicitis caused by Ascaris lumbricoides is an uncommon variant of a common disease. We ...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">Acute appendicitis caused by Ascaris lumbricoides is an uncommon variant of a common disease. We describe a case in which sonography was used for preoperative diagnosis of ascaris appendicitis.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109190290"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109190290"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109190290; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=109190290]").text(description); $(".js-view-count[data-work-id=109190290]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 109190290; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='109190290']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 109190290, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=109190290]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":109190290,"title":"Preoperative sonographic diagnosis of acute appendicitis caused byAscaris lumbricoides","translated_title":"","metadata":{"abstract":"Acute appendicitis caused by Ascaris lumbricoides is an uncommon variant of a common disease. 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The factors influencing colonic involvement also are unknown. Methods: Seventy-one patients with amebic liver abscess were studied. Colonoscopy was performed in all patients. Results: Abdominal pain and fever were the most common presenting manifestations. Ten patients (14%) had diarrhea at admission. Ten other patients had a history of diarrhea during the preceding 2 months. Thirty-nine (55%) patients had colonic ulcers. Colonic ulcers were present in 18 of the 20 (90%) patients with ongoing diarrhea or a history of recent diarrhea, and in 21 of 51 (41%) patients without diarrhea (p \u003c 0.001). Thirty (42%) patients had small, discrete ulcers in the cecum, the ascending colon, or the region of the hepatic flexure. Nine patients had large, multiple ulcers with surrounding inflammation. In these patients, the ulcers were present either in the left colon (n = 7) or throughout the colon (n = 2). The mean (standard deviation) age of patients with 5 or more ulcers was significantly greater than that of patients with fewer than 5 ulcers (49.8 [14.6] years vs. 37 [11.7] years; p \u003c 0.05). Multiple ulcers were noted in 7 of the 10 patients (70%) with diarrhea at admission and in two of the 61 patients (3%) in whom diarrhea was not a presenting symptom (p \u003c 0.001). No association was noted between the location of the abscess in the liver and the colonic lesions. Conclusions: Colonic ulcers are a common finding, occurring in more than half of patients with amebic liver abscess. They are more likely to be present if the patient has diarrhea as a presenting symptom or has had diarrhea in the recent past. Multiple, large, and left-sided ulcers are more common in elderly patients and in those in whom diarrhea is the presenting symptom. However, there is no association between the location of the abscess in the liver and colonic lesions.","publication_date":{"day":null,"month":null,"year":2004,"errors":{}},"publication_name":"Gastrointestinal 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$(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="109190288"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/109190288/Esophageal_stricture_as_a_late_sequel_of_Stevens_Johnson_syndrome_in_adults_incidental_detection_because_of_foreign_body_impaction"><img alt="Research paper thumbnail of Esophageal stricture as a late sequel of Stevens-Johnson syndrome in adults: incidental detection because of foreign body impaction" class="work-thumbnail" src="https://attachments.academia-assets.com/107388527/thumbnails/1.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/109190288/Esophageal_stricture_as_a_late_sequel_of_Stevens_Johnson_syndrome_in_adults_incidental_detection_because_of_foreign_body_impaction">Esophageal stricture as a late sequel of Stevens-Johnson syndrome in adults: incidental detection because of foreign body impaction</a></div><div class="wp-workCard_item"><span>Gastrointestinal Endoscopy</span><span>, 2004</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><a id="ba06929e4c752a949764eb9563cea16c" class="wp-workCard--action" rel="nofollow" data-click-track="profile-work-strip-download" data-download="{"attachment_id":107388527,"asset_id":109190288,"asset_type":"Work","button_location":"profile"}" href="https://www.academia.edu/attachments/107388527/download_file?st=MTczMjc1MTU1OCw4LjIyMi4yMDguMTQ2&st=MTczMjc1MTU1Nyw4LjIyMi4yMDguMTQ2&s=profile"><span><i class="fa fa-arrow-down"></i></span><span>Download</span></a><span 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Endoscopy","url":"https://www.academia.edu/Documents/in/Gastrointestinal_Endoscopy"},{"id":364282,"name":"Impaction","url":"https://www.academia.edu/Documents/in/Impaction"},{"id":405739,"name":"Esophagus","url":"https://www.academia.edu/Documents/in/Esophagus"},{"id":620049,"name":"Risk Factors","url":"https://www.academia.edu/Documents/in/Risk_Factors-1"},{"id":1295297,"name":"Foreign Bodies","url":"https://www.academia.edu/Documents/in/Foreign_Bodies"},{"id":1971419,"name":"Esophagoscopy","url":"https://www.academia.edu/Documents/in/Esophagoscopy"},{"id":2460250,"name":"Esophageal Stenosis","url":"https://www.academia.edu/Documents/in/Esophageal_Stenosis"},{"id":2560984,"name":"foreign body","url":"https://www.academia.edu/Documents/in/foreign_body"}],"urls":[{"id":35398400,"url":"https://api.elsevier.com/content/article/PII:S001651070302710X?httpAccept=text/xml"}]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="109190287"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/109190287/Endoscopic_Biopsies_from_Normal_Appearing_Terminal_Ileum_and_Cecum_in_Patients_with_Suspected_Colonic_Tuberculosis"><img alt="Research paper thumbnail of Endoscopic Biopsies from Normal-Appearing Terminal Ileum and Cecum in Patients with Suspected Colonic Tuberculosis" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/109190287/Endoscopic_Biopsies_from_Normal_Appearing_Terminal_Ileum_and_Cecum_in_Patients_with_Suspected_Colonic_Tuberculosis">Endoscopic Biopsies from Normal-Appearing Terminal Ileum and Cecum in Patients with Suspected Colonic Tuberculosis</a></div><div class="wp-workCard_item"><span>Endoscopy</span><span>, 2004</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">Colonic tuberculosis is generally diagnosed by colonoscopy and targeted biopsy of lesions. Howeve...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">Colonic tuberculosis is generally diagnosed by colonoscopy and targeted biopsy of lesions. However, the diagnostic yield of colonic biopsies is not very good. So far as we are aware, there have been no studies investigating the role of biopsies from endoscopically normal-appearing cecum and terminal ileum in diagnosing colonic or ileal tuberculosis, or both. Patients with a clinical suspicion of colonic tuberculosis, in whom no endoscopic abnormalities were found on colonoscopy or ileoscopy, were included in the study. Multiple biopsies were obtained from the cecum and ileum. Fifty patients were studied. Intubation of the terminal ileum was possible in 43 patients (86 %). Histological examination of biopsies obtained from the cecum and terminal ileum showed noncaseating granuloma in two patients. Both of these biopsies were from the terminal ileum. In two other patients, collections of loosely arranged epithelioid cells were observed. This established the diagnosis in these four patients (8 %). In the remaining 46 patients, histology showed nonspecific inflammation in 18 patients (in the cecum in 15 and in the terminal ileum in seven). The other biopsies did not show any abnormalities (33 from the cecum, 34 from the terminal ileum). Histological examination of biopsies from the normal-appearing cecum and terminal ileum is useful in a small but significant number of patients with colonic tuberculosis.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109190287"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109190287"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109190287; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=109190287]").text(description); $(".js-view-count[data-work-id=109190287]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 109190287; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='109190287']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 109190287, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=109190287]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":109190287,"title":"Endoscopic Biopsies from Normal-Appearing Terminal Ileum and Cecum in Patients with Suspected Colonic Tuberculosis","translated_title":"","metadata":{"abstract":"Colonic tuberculosis is generally diagnosed by colonoscopy and targeted biopsy of lesions. However, the diagnostic yield of colonic biopsies is not very good. So far as we are aware, there have been no studies investigating the role of biopsies from endoscopically normal-appearing cecum and terminal ileum in diagnosing colonic or ileal tuberculosis, or both. Patients with a clinical suspicion of colonic tuberculosis, in whom no endoscopic abnormalities were found on colonoscopy or ileoscopy, were included in the study. Multiple biopsies were obtained from the cecum and ileum. Fifty patients were studied. Intubation of the terminal ileum was possible in 43 patients (86 %). Histological examination of biopsies obtained from the cecum and terminal ileum showed noncaseating granuloma in two patients. Both of these biopsies were from the terminal ileum. In two other patients, collections of loosely arranged epithelioid cells were observed. This established the diagnosis in these four patients (8 %). In the remaining 46 patients, histology showed nonspecific inflammation in 18 patients (in the cecum in 15 and in the terminal ileum in seven). The other biopsies did not show any abnormalities (33 from the cecum, 34 from the terminal ileum). Histological examination of biopsies from the normal-appearing cecum and terminal ileum is useful in a small but significant number of patients with colonic tuberculosis.","publisher":"Georg Thieme Verlag KG","publication_date":{"day":null,"month":null,"year":2004,"errors":{}},"publication_name":"Endoscopy"},"translated_abstract":"Colonic tuberculosis is generally diagnosed by colonoscopy and targeted biopsy of lesions. However, the diagnostic yield of colonic biopsies is not very good. So far as we are aware, there have been no studies investigating the role of biopsies from endoscopically normal-appearing cecum and terminal ileum in diagnosing colonic or ileal tuberculosis, or both. Patients with a clinical suspicion of colonic tuberculosis, in whom no endoscopic abnormalities were found on colonoscopy or ileoscopy, were included in the study. Multiple biopsies were obtained from the cecum and ileum. Fifty patients were studied. Intubation of the terminal ileum was possible in 43 patients (86 %). Histological examination of biopsies obtained from the cecum and terminal ileum showed noncaseating granuloma in two patients. Both of these biopsies were from the terminal ileum. In two other patients, collections of loosely arranged epithelioid cells were observed. This established the diagnosis in these four patients (8 %). In the remaining 46 patients, histology showed nonspecific inflammation in 18 patients (in the cecum in 15 and in the terminal ileum in seven). The other biopsies did not show any abnormalities (33 from the cecum, 34 from the terminal ileum). Histological examination of biopsies from the normal-appearing cecum and terminal ileum is useful in a small but significant number of patients with colonic tuberculosis.","internal_url":"https://www.academia.edu/109190287/Endoscopic_Biopsies_from_Normal_Appearing_Terminal_Ileum_and_Cecum_in_Patients_with_Suspected_Colonic_Tuberculosis","translated_internal_url":"","created_at":"2023-11-15T01:49:30.307-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":48000299,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[],"downloadable_attachments":[],"slug":"Endoscopic_Biopsies_from_Normal_Appearing_Terminal_Ileum_and_Cecum_in_Patients_with_Suspected_Colonic_Tuberculosis","translated_slug":"","page_count":null,"language":"en","content_type":"Work","owner":{"id":48000299,"first_name":"Vatsala","middle_initials":null,"last_name":"Misra","page_name":"VatsalaMisra","domain_name":"independent","created_at":"2016-04-30T07:36:38.954-07:00","display_name":"Vatsala Misra","url":"https://independent.academia.edu/VatsalaMisra"},"attachments":[],"research_interests":[{"id":3274,"name":"Gastroenterology","url":"https://www.academia.edu/Documents/in/Gastroenterology"},{"id":26327,"name":"Medicine","url":"https://www.academia.edu/Documents/in/Medicine"},{"id":37805,"name":"Endoscopy","url":"https://www.academia.edu/Documents/in/Endoscopy"},{"id":37826,"name":"Biopsy","url":"https://www.academia.edu/Documents/in/Biopsy"},{"id":62235,"name":"Tuberculosis","url":"https://www.academia.edu/Documents/in/Tuberculosis"},{"id":65390,"name":"Internal Medicine","url":"https://www.academia.edu/Documents/in/Internal_Medicine"},{"id":162159,"name":"Differential Diagnosis","url":"https://www.academia.edu/Documents/in/Differential_Diagnosis"},{"id":227324,"name":"Colonoscopy","url":"https://www.academia.edu/Documents/in/Colonoscopy"},{"id":244814,"name":"Clinical Sciences","url":"https://www.academia.edu/Documents/in/Clinical_Sciences"},{"id":829416,"name":"Colon","url":"https://www.academia.edu/Documents/in/Colon"},{"id":1901112,"name":"Ileum","url":"https://www.academia.edu/Documents/in/Ileum"}],"urls":[{"id":35398399,"url":"http://www.thieme-connect.de/products/ejournals/pdf/10.1055/s-2004-814518.pdf"}]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="109190286"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/109190286/AgNORs_in_benign_borderline_and_transitional_cell_neoplasms_of_the_urinary_bladder"><img alt="Research paper thumbnail of AgNORs in benign, borderline and transitional cell neoplasms of the urinary bladder" class="work-thumbnail" src="https://attachments.academia-assets.com/107388525/thumbnails/1.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/109190286/AgNORs_in_benign_borderline_and_transitional_cell_neoplasms_of_the_urinary_bladder">AgNORs in benign, borderline and transitional cell neoplasms of the urinary bladder</a></div><div class="wp-workCard_item"><span>APMIS</span><span>, 1998</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">Argyrophilic nucleolar organizer regions (AgNORs) were studied in 106 tissue samples from the uri...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">Argyrophilic nucleolar organizer regions (AgNORs) were studied in 106 tissue samples from the urinary bladder (6 normal transitional epithelium, 5 cystitis, 12 hyperplastic, 14 dysplastic lesions, 12 carcinoma in situ, 4 transitional cell carcinoma grade 0, 12 grade 1, 15 grade II and 12 grade III) to evaluate their role in differentiating benign, borderline and malignant lesions. The NOR counts presented a rising scale from normal (2.21), inflammatory (3.93 for both cystitis and hyperplasia), dysplastic (4.16), carcinoma in situ (5.08) to malignant lesions (5.28 grade I, 6.59 grade II and 8.37 grade III). It was concluded that AgNORs do not have a diagnostic role in these lesions, but that they can act as a reliable adjunct to existing parameters in the early detection of tumour recurrence and grading of malignant neoplasms.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><a id="304b2443c7acf6d249cf0b0a4e1a6ae6" class="wp-workCard--action" rel="nofollow" data-click-track="profile-work-strip-download" data-download="{"attachment_id":107388525,"asset_id":109190286,"asset_type":"Work","button_location":"profile"}" href="https://www.academia.edu/attachments/107388525/download_file?st=MTczMjc1MTU1OCw4LjIyMi4yMDguMTQ2&st=MTczMjc1MTU1OCw4LjIyMi4yMDguMTQ2&s=profile"><span><i class="fa fa-arrow-down"></i></span><span>Download</span></a><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109190286"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109190286"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109190286; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=109190286]").text(description); $(".js-view-count[data-work-id=109190286]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 109190286; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='109190286']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 109190286, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (true){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "304b2443c7acf6d249cf0b0a4e1a6ae6" } } $('.js-work-strip[data-work-id=109190286]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":109190286,"title":"AgNORs in benign, borderline and transitional cell neoplasms of the urinary bladder","translated_title":"","metadata":{"abstract":"Argyrophilic nucleolar organizer regions (AgNORs) were studied in 106 tissue samples from the urinary bladder (6 normal transitional epithelium, 5 cystitis, 12 hyperplastic, 14 dysplastic lesions, 12 carcinoma in situ, 4 transitional cell carcinoma grade 0, 12 grade 1, 15 grade II and 12 grade III) to evaluate their role in differentiating benign, borderline and malignant lesions. The NOR counts presented a rising scale from normal (2.21), inflammatory (3.93 for both cystitis and hyperplasia), dysplastic (4.16), carcinoma in situ (5.08) to malignant lesions (5.28 grade I, 6.59 grade II and 8.37 grade III). It was concluded that AgNORs do not have a diagnostic role in these lesions, but that they can act as a reliable adjunct to existing parameters in the early detection of tumour recurrence and grading of malignant neoplasms.","publisher":"Wiley","publication_date":{"day":null,"month":null,"year":1998,"errors":{}},"publication_name":"APMIS"},"translated_abstract":"Argyrophilic nucleolar organizer regions (AgNORs) were studied in 106 tissue samples from the urinary bladder (6 normal transitional epithelium, 5 cystitis, 12 hyperplastic, 14 dysplastic lesions, 12 carcinoma in situ, 4 transitional cell carcinoma grade 0, 12 grade 1, 15 grade II and 12 grade III) to evaluate their role in differentiating benign, borderline and malignant lesions. The NOR counts presented a rising scale from normal (2.21), inflammatory (3.93 for both cystitis and hyperplasia), dysplastic (4.16), carcinoma in situ (5.08) to malignant lesions (5.28 grade I, 6.59 grade II and 8.37 grade III). It was concluded that AgNORs do not have a diagnostic role in these lesions, but that they can act as a reliable adjunct to existing parameters in the early detection of tumour recurrence and grading of malignant neoplasms.","internal_url":"https://www.academia.edu/109190286/AgNORs_in_benign_borderline_and_transitional_cell_neoplasms_of_the_urinary_bladder","translated_internal_url":"","created_at":"2023-11-15T01:49:30.090-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":48000299,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[],"downloadable_attachments":[{"id":107388525,"title":"","file_type":"pdf","scribd_thumbnail_url":"https://attachments.academia-assets.com/107388525/thumbnails/1.jpg","file_name":"j.1699-0463.1998.tb00250.x20231115-1-xkw8vv.pdf","download_url":"https://www.academia.edu/attachments/107388525/download_file?st=MTczMjc1MTU1OCw4LjIyMi4yMDguMTQ2&st=MTczMjc1MTU1OCw4LjIyMi4yMDguMTQ2&","bulk_download_file_name":"AgNORs_in_benign_borderline_and_transiti.pdf","bulk_download_url":"https://d1wqtxts1xzle7.cloudfront.net/107388525/j.1699-0463.1998.tb00250.x20231115-1-xkw8vv-libre.pdf?1700048619=\u0026response-content-disposition=attachment%3B+filename%3DAgNORs_in_benign_borderline_and_transiti.pdf\u0026Expires=1732755158\u0026Signature=EMJ0HspAZcp33YrlNbpU9Z8Z0ZEcKaCiZWhCQ3P-RS4g~ODq6QZzKqoIASWEPIiCSi5oLZq~sYxz7q1lJpliJbfDN-DS9aUaocHafwLT3Jna4M4hJKVXth6AnabyzVaWRbtzPUSYSyqAMMGef3MXMXJttx2WpzcUZyHxBecl1dU2i3gxibGYpQoMnWPmBhxYBA~OsKqoWwj-ZZuu3tKvunOV28DJd4UOI1eJfvhviPIDz4-irPuGzpHTqPyDZvSWo5T332h~88yLl-CqI5wwEfYAUeUNVvsRfoq4lnzWEPcksohvJRDgZ3UqPU5dXQLt8mV8VNxdmBsQObrJN0HatA__\u0026Key-Pair-Id=APKAJLOHF5GGSLRBV4ZA"}],"slug":"AgNORs_in_benign_borderline_and_transitional_cell_neoplasms_of_the_urinary_bladder","translated_slug":"","page_count":6,"language":"en","content_type":"Work","owner":{"id":48000299,"first_name":"Vatsala","middle_initials":null,"last_name":"Misra","page_name":"VatsalaMisra","domain_name":"independent","created_at":"2016-04-30T07:36:38.954-07:00","display_name":"Vatsala Misra","url":"https://independent.academia.edu/VatsalaMisra"},"attachments":[{"id":107388525,"title":"","file_type":"pdf","scribd_thumbnail_url":"https://attachments.academia-assets.com/107388525/thumbnails/1.jpg","file_name":"j.1699-0463.1998.tb00250.x20231115-1-xkw8vv.pdf","download_url":"https://www.academia.edu/attachments/107388525/download_file?st=MTczMjc1MTU1OCw4LjIyMi4yMDguMTQ2&st=MTczMjc1MTU1OCw4LjIyMi4yMDguMTQ2&","bulk_download_file_name":"AgNORs_in_benign_borderline_and_transiti.pdf","bulk_download_url":"https://d1wqtxts1xzle7.cloudfront.net/107388525/j.1699-0463.1998.tb00250.x20231115-1-xkw8vv-libre.pdf?1700048619=\u0026response-content-disposition=attachment%3B+filename%3DAgNORs_in_benign_borderline_and_transiti.pdf\u0026Expires=1732755158\u0026Signature=EMJ0HspAZcp33YrlNbpU9Z8Z0ZEcKaCiZWhCQ3P-RS4g~ODq6QZzKqoIASWEPIiCSi5oLZq~sYxz7q1lJpliJbfDN-DS9aUaocHafwLT3Jna4M4hJKVXth6AnabyzVaWRbtzPUSYSyqAMMGef3MXMXJttx2WpzcUZyHxBecl1dU2i3gxibGYpQoMnWPmBhxYBA~OsKqoWwj-ZZuu3tKvunOV28DJd4UOI1eJfvhviPIDz4-irPuGzpHTqPyDZvSWo5T332h~88yLl-CqI5wwEfYAUeUNVvsRfoq4lnzWEPcksohvJRDgZ3UqPU5dXQLt8mV8VNxdmBsQObrJN0HatA__\u0026Key-Pair-Id=APKAJLOHF5GGSLRBV4ZA"}],"research_interests":[{"id":165,"name":"Pathology","url":"https://www.academia.edu/Documents/in/Pathology"},{"id":1290,"name":"Immunology","url":"https://www.academia.edu/Documents/in/Immunology"},{"id":6947,"name":"Medical Microbiology","url":"https://www.academia.edu/Documents/in/Medical_Microbiology"},{"id":26327,"name":"Medicine","url":"https://www.academia.edu/Documents/in/Medicine"},{"id":176968,"name":"Hyperplasia","url":"https://www.academia.edu/Documents/in/Hyperplasia"},{"id":244814,"name":"Clinical Sciences","url":"https://www.academia.edu/Documents/in/Clinical_Sciences"},{"id":558694,"name":"Nucleolus Organizer Region","url":"https://www.academia.edu/Documents/in/Nucleolus_Organizer_Region"},{"id":1433630,"name":"Transitional Cell Carcinoma","url":"https://www.academia.edu/Documents/in/Transitional_Cell_Carcinoma"},{"id":1902592,"name":"Urinary Bladder","url":"https://www.academia.edu/Documents/in/Urinary_Bladder"},{"id":3287751,"name":"cystitis","url":"https://www.academia.edu/Documents/in/cystitis"},{"id":3364752,"name":"Carcinoma in situ","url":"https://www.academia.edu/Documents/in/Carcinoma_in_situ"},{"id":4102912,"name":"Urinary bladder neoplasms","url":"https://www.academia.edu/Documents/in/Urinary_bladder_neoplasms"}],"urls":[{"id":35398398,"url":"https://onlinelibrary.wiley.com/doi/pdf/10.1111/j.1699-0463.1998.tb00250.x"}]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="109190235"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" rel="nofollow" href="https://www.academia.edu/109190235/Helicobacter_pylori_and_gastric_cancer"><img alt="Research paper thumbnail of Helicobacter pylori and gastric cancer" class="work-thumbnail" src="https://a.academia-assets.com/images/blank-paper.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" rel="nofollow" href="https://www.academia.edu/109190235/Helicobacter_pylori_and_gastric_cancer">Helicobacter pylori and gastric cancer</a></div><div class="wp-workCard_item"><span>The American Journal of Medicine</span><span>, 1999</span></div><div class="wp-workCard_item"><span class="js-work-more-abstract-truncated">This review focuses on the similarities between the epidemiology of gastric cancer and the epidem...</span><a class="js-work-more-abstract" data-broccoli-component="work_strip.more_abstract" data-click-track="profile-work-strip-more-abstract" href="javascript:;"><span> more </span><span><i class="fa fa-caret-down"></i></span></a><span class="js-work-more-abstract-untruncated hidden">This review focuses on the similarities between the epidemiology of gastric cancer and the epidemiology of Helicobacter pylori. Their demographic patterns and the results of studies regarding familial and environmental risk factors are described. The association of gastric cancer and H. pylori infection with both gastric ulcer and chronic atrophic gastritis is also characterized and the possibility that a H. pylori infection could lead to gastric cancer is discussed.</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="109190235"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="109190235"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 109190235; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=109190235]").text(description); $(".js-view-count[data-work-id=109190235]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 109190235; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='109190235']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 109190235, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (false){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "-1" } } $('.js-work-strip[data-work-id=109190235]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":109190235,"title":"Helicobacter pylori and gastric cancer","translated_title":"","metadata":{"abstract":"This review focuses on the similarities between the epidemiology of gastric cancer and the epidemiology of Helicobacter pylori. Their demographic patterns and the results of studies regarding familial and environmental risk factors are described. The association of gastric cancer and H. pylori infection with both gastric ulcer and chronic atrophic gastritis is also characterized and the possibility that a H. pylori infection could lead to gastric cancer is discussed.","publisher":"Elsevier BV","publication_date":{"day":null,"month":null,"year":1999,"errors":{}},"publication_name":"The American Journal of Medicine"},"translated_abstract":"This review focuses on the similarities between the epidemiology of gastric cancer and the epidemiology of Helicobacter pylori. Their demographic patterns and the results of studies regarding familial and environmental risk factors are described. The association of gastric cancer and H. pylori infection with both gastric ulcer and chronic atrophic gastritis is also characterized and the possibility that a H. pylori infection could lead to gastric cancer is discussed.","internal_url":"https://www.academia.edu/109190235/Helicobacter_pylori_and_gastric_cancer","translated_internal_url":"","created_at":"2023-11-15T01:48:28.923-08:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":48000299,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[],"downloadable_attachments":[],"slug":"Helicobacter_pylori_and_gastric_cancer","translated_slug":"","page_count":null,"language":"en","content_type":"Work","owner":{"id":48000299,"first_name":"Vatsala","middle_initials":null,"last_name":"Misra","page_name":"VatsalaMisra","domain_name":"independent","created_at":"2016-04-30T07:36:38.954-07:00","display_name":"Vatsala Misra","url":"https://independent.academia.edu/VatsalaMisra"},"attachments":[],"research_interests":[{"id":6021,"name":"Cancer","url":"https://www.academia.edu/Documents/in/Cancer"},{"id":9478,"name":"Diet","url":"https://www.academia.edu/Documents/in/Diet"},{"id":21460,"name":"Helicobacter pylori","url":"https://www.academia.edu/Documents/in/Helicobacter_pylori"},{"id":26327,"name":"Medicine","url":"https://www.academia.edu/Documents/in/Medicine"},{"id":52873,"name":"Virulence","url":"https://www.academia.edu/Documents/in/Virulence"},{"id":55269,"name":"Lymphoma","url":"https://www.academia.edu/Documents/in/Lymphoma"},{"id":192721,"name":"Risk factors","url":"https://www.academia.edu/Documents/in/Risk_factors"},{"id":244814,"name":"Clinical Sciences","url":"https://www.academia.edu/Documents/in/Clinical_Sciences"},{"id":620049,"name":"Risk Factors","url":"https://www.academia.edu/Documents/in/Risk_Factors-1"},{"id":925054,"name":"Gastritis","url":"https://www.academia.edu/Documents/in/Gastritis"},{"id":1030661,"name":"Stomach","url":"https://www.academia.edu/Documents/in/Stomach"},{"id":1745478,"name":"Adenocarcinoma","url":"https://www.academia.edu/Documents/in/Adenocarcinoma"},{"id":2345130,"name":"Socioeconomic Factors","url":"https://www.academia.edu/Documents/in/Socioeconomic_Factors"},{"id":2547982,"name":"Gastric mucosa","url":"https://www.academia.edu/Documents/in/Gastric_mucosa"},{"id":3461373,"name":"Stomach neoplasms","url":"https://www.academia.edu/Documents/in/Stomach_neoplasms"},{"id":3763225,"name":"Medical and Health Sciences","url":"https://www.academia.edu/Documents/in/Medical_and_Health_Sciences"},{"id":3960847,"name":"Gastroenterology and Hepatology","url":"https://www.academia.edu/Documents/in/Gastroenterology_and_Hepatology-1"},{"id":3992323,"name":"Metaplasia","url":"https://www.academia.edu/Documents/in/Metaplasia"}],"urls":[]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="104662124"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/104662124/Morphometric_Assessment_of_Chronic_Inflammatory_Cells_in_Colonic_Biopsies_of_Patients_with_Irritable_Bowel_Syndrome"><img alt="Research paper thumbnail of Morphometric Assessment of Chronic Inflammatory Cells in Colonic Biopsies of Patients with Irritable Bowel Syndrome" class="work-thumbnail" src="https://attachments.academia-assets.com/104330772/thumbnails/1.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/104662124/Morphometric_Assessment_of_Chronic_Inflammatory_Cells_in_Colonic_Biopsies_of_Patients_with_Irritable_Bowel_Syndrome">Morphometric Assessment of Chronic Inflammatory Cells in Colonic Biopsies of Patients with Irritable Bowel Syndrome</a></div><div class="wp-workCard_item"><span>JOURNAL OF CLINICAL AND DIAGNOSTIC RESEARCH</span><span>, 2019</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><a id="6a33a05de4b8adb2700230cf817b1129" class="wp-workCard--action" rel="nofollow" data-click-track="profile-work-strip-download" data-download="{"attachment_id":104330772,"asset_id":104662124,"asset_type":"Work","button_location":"profile"}" href="https://www.academia.edu/attachments/104330772/download_file?st=MTczMjc1MTU1OCw4LjIyMi4yMDguMTQ2&st=MTczMjc1MTU1OCw4LjIyMi4yMDguMTQ2&s=profile"><span><i class="fa fa-arrow-down"></i></span><span>Download</span></a><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="104662124"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="104662124"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 104662124; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=104662124]").text(description); $(".js-view-count[data-work-id=104662124]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 104662124; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='104662124']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 104662124, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (true){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); 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It is a functional bowel disorder characterised by abdominal pain, cramps, changes in bowel habit, gassiness, bloating, and nausea, but without any specific diagnostic markers. Thus, the diagnosis is based on clinical presentation for which several symptoms based diagnostic criteria have been suggested time to time over the last two decades [1]. These include Manning, Rome I, Rome II , Rome III and most recently Rome IV criteria. The Rome criteria is the most widely accepted worldwide because of its reasonable sensitivity and specificity [2]. IBS presents clinically as one of these three predominant subtypes-(1) IBS with constipation (IBS-C); (2) IBS with diarrhoea (IBS-D) and (3) Mixed IBS (IBS-M). According to onset of symptoms, IBS patients are further divided into Post Infectious IBS (PI-IBS) and Non Postinfectious IBS (NPI-IBS). PI-IBS is defined as Rome criteria positive IBS of acute onset developing after an infectious aetiology [3]. Lack of disease defining biomarkers makes the aetiology of this disease complicated with various factors like altered gastrointestinal motility, visceral hypersensitivity, post infectious reactivity, brain-gut interactions, alteration in faecal micro flora, bacterial overgrowth, barrier dysfunctions, food sensitivity, carbohydrate malabsorption, and intestinal inflammation being implicated in its pathogenesis. A genetic contribution also seems likely based upon the high degree of concordance seen among monozygotic twins as well as familial aggregations observed [4].","publication_date":{"day":null,"month":null,"year":2019,"errors":{}},"publication_name":"JOURNAL OF CLINICAL AND DIAGNOSTIC RESEARCH","grobid_abstract_attachment_id":104330772},"translated_abstract":null,"internal_url":"https://www.academia.edu/104662124/Morphometric_Assessment_of_Chronic_Inflammatory_Cells_in_Colonic_Biopsies_of_Patients_with_Irritable_Bowel_Syndrome","translated_internal_url":"","created_at":"2023-07-17T09:44:59.810-07:00","preview_url":null,"current_user_can_edit":null,"current_user_is_owner":null,"owner_id":48000299,"coauthors_can_edit":true,"document_type":"paper","co_author_tags":[],"downloadable_attachments":[{"id":104330772,"title":"","file_type":"pdf","scribd_thumbnail_url":"https://attachments.academia-assets.com/104330772/thumbnails/1.jpg","file_name":"40569_PDKM_V-2PoG_KM_CE_5BRa1_5D_PF1AG_OM_PFASL_PBNC_AG_PF2TR_SL_PNSL.pdf","download_url":"https://www.academia.edu/attachments/104330772/download_file?st=MTczMjc1MTU1OCw4LjIyMi4yMDguMTQ2&st=MTczMjc1MTU1OCw4LjIyMi4yMDguMTQ2&","bulk_download_file_name":"Morphometric_Assessment_of_Chronic_Infla.pdf","bulk_download_url":"https://d1wqtxts1xzle7.cloudfront.net/104330772/40569_PDKM_V-2PoG_KM_CE_5BRa1_5D_PF1AG_OM_PFASL_PBNC_AG_PF2TR_SL_PNSL-libre.pdf?1689612376=\u0026response-content-disposition=attachment%3B+filename%3DMorphometric_Assessment_of_Chronic_Infla.pdf\u0026Expires=1732755158\u0026Signature=Dk0RLf2HgFjC2rSxnG58jlw7aZB4B2WfhwECyDVxgnu59sRS5MbQRslraZTEEWP0iaodpNB99BC-xsq-uVsuch-nxf6ckSlenEt5stxK5OaJtMLKYQOfb4Gckx8w6eD49QbTMzDNVUcsKKt8Sodl9Km8FDrR1k9jrHukYJAdIJrbxp24wOxjcMEqb~I3bqNdTYF5AF4ArFwGUSjMLtjnus8NMD-OGyB~amcXypR3SL2ANvkJwA58JjG~PKFSJCrtykqzqzHlYP0hm0Xjaqxvdh5T8472qYHFTqcF0TgO~pWdYX4vN1WP1S9xROZl-IpTHFOZgZIRJKU8OLkzxtjung__\u0026Key-Pair-Id=APKAJLOHF5GGSLRBV4ZA"}],"slug":"Morphometric_Assessment_of_Chronic_Inflammatory_Cells_in_Colonic_Biopsies_of_Patients_with_Irritable_Bowel_Syndrome","translated_slug":"","page_count":4,"language":"en","content_type":"Work","owner":{"id":48000299,"first_name":"Vatsala","middle_initials":null,"last_name":"Misra","page_name":"VatsalaMisra","domain_name":"independent","created_at":"2016-04-30T07:36:38.954-07:00","display_name":"Vatsala Misra","url":"https://independent.academia.edu/VatsalaMisra"},"attachments":[{"id":104330772,"title":"","file_type":"pdf","scribd_thumbnail_url":"https://attachments.academia-assets.com/104330772/thumbnails/1.jpg","file_name":"40569_PDKM_V-2PoG_KM_CE_5BRa1_5D_PF1AG_OM_PFASL_PBNC_AG_PF2TR_SL_PNSL.pdf","download_url":"https://www.academia.edu/attachments/104330772/download_file?st=MTczMjc1MTU1OCw4LjIyMi4yMDguMTQ2&st=MTczMjc1MTU1OCw4LjIyMi4yMDguMTQ2&","bulk_download_file_name":"Morphometric_Assessment_of_Chronic_Infla.pdf","bulk_download_url":"https://d1wqtxts1xzle7.cloudfront.net/104330772/40569_PDKM_V-2PoG_KM_CE_5BRa1_5D_PF1AG_OM_PFASL_PBNC_AG_PF2TR_SL_PNSL-libre.pdf?1689612376=\u0026response-content-disposition=attachment%3B+filename%3DMorphometric_Assessment_of_Chronic_Infla.pdf\u0026Expires=1732755158\u0026Signature=Dk0RLf2HgFjC2rSxnG58jlw7aZB4B2WfhwECyDVxgnu59sRS5MbQRslraZTEEWP0iaodpNB99BC-xsq-uVsuch-nxf6ckSlenEt5stxK5OaJtMLKYQOfb4Gckx8w6eD49QbTMzDNVUcsKKt8Sodl9Km8FDrR1k9jrHukYJAdIJrbxp24wOxjcMEqb~I3bqNdTYF5AF4ArFwGUSjMLtjnus8NMD-OGyB~amcXypR3SL2ANvkJwA58JjG~PKFSJCrtykqzqzHlYP0hm0Xjaqxvdh5T8472qYHFTqcF0TgO~pWdYX4vN1WP1S9xROZl-IpTHFOZgZIRJKU8OLkzxtjung__\u0026Key-Pair-Id=APKAJLOHF5GGSLRBV4ZA"}],"research_interests":[{"id":3274,"name":"Gastroenterology","url":"https://www.academia.edu/Documents/in/Gastroenterology"},{"id":26327,"name":"Medicine","url":"https://www.academia.edu/Documents/in/Medicine"},{"id":65390,"name":"Internal Medicine","url":"https://www.academia.edu/Documents/in/Internal_Medicine"},{"id":214598,"name":"Irritable Bowel Syndrome","url":"https://www.academia.edu/Documents/in/Irritable_Bowel_Syndrome"}],"urls":[{"id":32938474,"url":"https://jcdr.net/article_fulltext.asp?issn=0973-709x\u0026year=2019\u0026volume=13\u0026issue=5\u0026page=EC06\u0026issn=0973-709x\u0026id=12852"}]}, dispatcherData: dispatcherData }); $(this).data('initialized', true); } }); $a.trackClickSource(".js-work-strip-work-link", "profile_work_strip") }); </script> <div class="js-work-strip profile--work_container" data-work-id="104662123"><div class="profile--work_thumbnail hidden-xs"><a class="js-work-strip-work-link" data-click-track="profile-work-strip-thumbnail" href="https://www.academia.edu/104662123/Ileoscopy_in_patients_with_ileocolonic_tuberculosis"><img alt="Research paper thumbnail of Ileoscopy in patients with ileocolonic tuberculosis" class="work-thumbnail" src="https://attachments.academia-assets.com/104330773/thumbnails/1.jpg" /></a></div><div class="wp-workCard wp-workCard_itemContainer"><div class="wp-workCard_item wp-workCard--title"><a class="js-work-strip-work-link text-gray-darker" data-click-track="profile-work-strip-title" href="https://www.academia.edu/104662123/Ileoscopy_in_patients_with_ileocolonic_tuberculosis">Ileoscopy in patients with ileocolonic tuberculosis</a></div><div class="wp-workCard_item"><span>World Journal of Gastroenterology</span><span>, 2007</span></div><div class="wp-workCard_item wp-workCard--actions"><span class="work-strip-bookmark-button-container"></span><a id="80e54b12649c6ee5bee95ba446abad9a" class="wp-workCard--action" rel="nofollow" data-click-track="profile-work-strip-download" data-download="{"attachment_id":104330773,"asset_id":104662123,"asset_type":"Work","button_location":"profile"}" href="https://www.academia.edu/attachments/104330773/download_file?st=MTczMjc1MTU1OCw4LjIyMi4yMDguMTQ2&st=MTczMjc1MTU1OCw4LjIyMi4yMDguMTQ2&s=profile"><span><i class="fa fa-arrow-down"></i></span><span>Download</span></a><span class="wp-workCard--action visible-if-viewed-by-owner inline-block" style="display: none;"><span class="js-profile-work-strip-edit-button-wrapper profile-work-strip-edit-button-wrapper" data-work-id="104662123"><a class="js-profile-work-strip-edit-button" tabindex="0"><span><i class="fa fa-pencil"></i></span><span>Edit</span></a></span></span><span id="work-strip-rankings-button-container"></span></div><div class="wp-workCard_item wp-workCard--stats"><span><span><span class="js-view-count view-count u-mr2x" data-work-id="104662123"><i class="fa fa-spinner fa-spin"></i></span><script>$(function () { var workId = 104662123; window.Academia.workViewCountsFetcher.queue(workId, function (count) { var description = window.$h.commaizeInt(count) + " " + window.$h.pluralize(count, 'View'); $(".js-view-count[data-work-id=104662123]").text(description); $(".js-view-count[data-work-id=104662123]").attr('title', description).tooltip(); }); });</script></span></span><span><span class="percentile-widget hidden"><span class="u-mr2x work-percentile"></span></span><script>$(function () { var workId = 104662123; window.Academia.workPercentilesFetcher.queue(workId, function (percentileText) { var container = $(".js-work-strip[data-work-id='104662123']"); container.find('.work-percentile').text(percentileText.charAt(0).toUpperCase() + percentileText.slice(1)); container.find('.percentile-widget').show(); container.find('.percentile-widget').removeClass('hidden'); }); });</script></span><span><script>$(function() { new Works.PaperRankView({ workId: 104662123, container: "", }); });</script></span></div><div id="work-strip-premium-row-container"></div></div></div><script> require.config({ waitSeconds: 90 })(["https://a.academia-assets.com/assets/wow_profile-f77ea15d77ce96025a6048a514272ad8becbad23c641fc2b3bd6e24ca6ff1932.js","https://a.academia-assets.com/assets/work_edit-ad038b8c047c1a8d4fa01b402d530ff93c45fee2137a149a4a5398bc8ad67560.js"], function() { // from javascript_helper.rb var dispatcherData = {} if (true){ window.WowProfile.dispatcher = window.WowProfile.dispatcher || _.clone(Backbone.Events); dispatcherData = { dispatcher: window.WowProfile.dispatcher, downloadLinkId: "80e54b12649c6ee5bee95ba446abad9a" } } $('.js-work-strip[data-work-id=104662123]').each(function() { if (!$(this).data('initialized')) { new WowProfile.WorkStripView({ el: this, workJSON: {"id":104662123,"title":"Ileoscopy in patients with ileocolonic tuberculosis","translated_title":"","metadata":{"publisher":"Baishideng Publishing Group Inc.","grobid_abstract":"AIM: To study the role of retrograde terminal ileoscopy in patients suspected to have ileocolonic tuberculosis. METHODS: A retrospective analysis was performed for patients undergoing colonoscopy for suspected ileocolonic tuberculosis between January 2000 and June 2004, in whom retrograde ileoscopy had been performed. Only patients diagnosed with tuberculosis on the basis of histological findings of either a caseating granuloma or those having non-caseating granuloma or a collection of epithelioid cells at a minimum of one endoscopic lesion (either in the colon or the terminal ileum) on histology with good responses to conventional anti-tuberculous drugs were enrolled for the study. RESULTS: Fifty-three patients were included. The terminal ileum was involved in only 11 patients. Eight of these patients had involvement of the cecum too. Two patients had ileal lesions without cecal involvement; however, lesions were noted in the ascending colon. In one patient the whole colon was normal and only the terminal ileum showed nodularity and ulceration. Histological examination of the ileal biopsies obtained from the ileal lesions showed noncaseating granulomas in five, collection of epithelioid cells in four and nonspecific histology in two patients. Mucosal biopsies obtained from the lesion in the terminal ileum, in the patient in whom mucosal abnormality was observed to be confined to the ileum, showed noncaseating granulomas. In two other patients the ileal biopsies alone showed histological evidence of tuberculosis with biopsies from the colonic lesions showing non-specific inflammatory changes only. CONCLUSION: Re t ro g ra d e i l e o s c o py s h o u l d b e performed in all patients undergoing colonoscopy for suspected ileocolonic tuberculosis. In some patients only the terminal ileum may be involved and histological examination may reveal evidence of tuberculosis only from the lesions in the terminal ileum. 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The appearance, behavior and management of the gastric mass was more like a bezoar than that of a foreign body, and therefore we strongly feel that it should be called a \"plastobezoar\". CASE REPORT An 11-year-old boy was admitted with complaints of vomiting and abdominal pain of six months duration and swelling over the face and feet for the last one month. On clinical examination the child was pale with puffiness over the face. Pedal edema was present. Abdominal examination found a fi rm lump palpable in the epigastrium, which moved with respiration. No crepitus was felt over the lump. There was mild tenderness present over the mass. The hemoglobin was 5.6 g/dL, total and differential leukocyte counts were normal. The serum urea was 36.4 mmol/L, creatinine 314 μmol/L, and serum albumin was 2 g/dL. Serum globulin, fasting and post-parandial blood glucose, serum amylase, bilirubin and transaminases were normal. Urine examination did not reveal any abnormality. Serum sodium was 135 mmol/L, potassium 2.8 mmol/L and calcium 1.8 mmol/L. Just after admission, the patient developed typical grandmal epileptic attack, which was managed by intravenous diazepam injection. Intravenous and oral supplements of potassium and calcium were given and the child did not have any further episodes of epilepsy. The patient was considered to have pre-renal failure due to severe vomiting and dehydration and was managed conservatively with intravenous fl uid. Two packs of whole blood were transfused and 100 mL of 20% albumin was given intravenously. Gradually, the serum creatinine and urea became normal. Clinically the puffiness over the face and pedal edema disappeared and the patient started feeling better. Dialysis was not required. A contrast enhanced CT scan revealed an irregular, large, non-attached mass of varying attenuation with pockets of air, occupying the stomach (Figure 1). A portion of the mass was also encroaching the duodenum. A few polypoidal masses were seen in the antrum of the stomach near the pylorus. There was no post-contrast enhancement of the mass lesion. UGI endoscopy revealed a large bezoar occupying nearly the whole stomach (Figure 2). 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